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FASEB Journal, ISSN 0892-6638, 01/2015, Volume 29, Issue 1, pp. 286 - 299
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 07/2014, Volume 124, Issue 7, pp. 2935 - 2946
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 06/2012, Volume 27, Issue 6, pp. 1252 - 1262
As they age, mice deficient for the β2‐adrenergic receptor (Adrb2−/−) maintain greater trabecular bone microarchitecture, as a result of lower bone resorption... 
β‐ADRENERGIC | ISOPROTERENOL | MECHANICAL LOADING | PEAK BONE MASS ACQUISITION | β-ADRENERGIC | ss-ADRENERGIC | SYMPATHETIC-NERVOUS-SYSTEM | BETA-2-ADRENERGIC RECEPTORS | OSTEOBLAST-LIKE CELLS | FRACTURE RISK | NORADRENERGIC REGULATION | LEPTIN DEFICIENCY | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | PARATHYROID-HORMONE | ADAPTIVE RESPONSE | GROWTH-FACTOR-I | Bone and Bones - pathology | Femur - pathology | Stress, Mechanical | Male | Bone Remodeling - drug effects | Bone and Bones - drug effects | Body Composition - drug effects | Bone and Bones - metabolism | Gene Deletion | Biomechanical Phenomena - drug effects | Receptors, Adrenergic, beta-2 - deficiency | Receptors, Adrenergic, beta-1 - deficiency | Femur - metabolism | Receptors, Adrenergic, beta-2 - genetics | Osteoblasts - drug effects | Osteogenesis - drug effects | Femur - drug effects | Receptors, Adrenergic, beta-1 - metabolism | Femur - physiopathology | Receptors, Adrenergic, beta-2 - metabolism | Gene Expression Regulation - drug effects | Bone and Bones - physiopathology | Organ Size - drug effects | Phenotype | Animals | Isoproterenol - pharmacology | Receptors, Adrenergic, beta-1 - genetics | Mice | Osteoblasts - metabolism | Insulin-Like Growth Factor I - metabolism | Femur | Vertebrae | Insulin-like growth factor I | Spine | Insulin-like growth factors | isoproterenol | Bone (trabecular) | Bone resorption | Bone histomorphometry | Bone growth | NF- Kappa B protein | Bone mass | Loading | Tibia | Adrenergic receptors | Bone mineral density | Mechanical stimuli | Osteogenesis | Bone (cancellous)
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 10, p. e26581
Sustained beta-adrenergic receptors (beta AR) activation leads to cardiac hypertrophy and prevents left ventricular (LV) atrophy during LV unloading. The... 
GLYCOGEN-SYNTHASE KINASE-3-BETA | CARDIOMYOCYTE HYPERTROPHY | ACTIVATION | PROTEIN-KINASE-B | MUSCLE ATROPHY | MULTIDISCIPLINARY SCIENCES | CARDIAC-HYPERTROPHY | MYOCARDIAL-CONTRACTILITY | AKT | FOXO TRANSCRIPTION FACTORS | BETA-ADRENERGIC RECEPTORS | TOR Serine-Threonine Kinases - metabolism | SKP Cullin F-Box Protein Ligases - genetics | Glycogen Synthase Kinase 3 beta | Male | Phosphatidylinositol 3-Kinases - metabolism | RNA, Messenger - metabolism | Tripartite Motif Proteins | Forkhead Transcription Factors - metabolism | Myocardium - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Receptors, Adrenergic, beta-2 - deficiency | Receptors, Adrenergic, beta-1 - deficiency | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | Receptors, Adrenergic, beta-2 - genetics | RNA, Messenger - genetics | Adrenergic beta-Agonists - pharmacology | Receptors, Adrenergic, beta-1 - metabolism | Gene Knockout Techniques | Glycogen Synthase Kinase 3 - metabolism | Organ Specificity | Receptors, Adrenergic, beta-2 - metabolism | Myocardium - cytology | Muscle Proteins - genetics | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Receptors, Adrenergic, beta-1 - genetics | Mice | Ubiquitin-Protein Ligases - genetics | Mitogen-Activated Protein Kinase 1 - metabolism | TOR protein | Heart | Pediatrics | Transcription factors | Phosphorylation | Target recognition | Pathogenesis | Crosstalk | AKT protein | Stimulation | Activation | Kinases | Medical schools | Proteins | Atrophy | Signal transduction | Receptors | Pathways | FOXO1 protein | Rodents | Cascades | Cell cycle | Forkhead protein | Formoterol | Lesions | Growth factors | Heart diseases | Heart failure | Cardiac muscle | Glycogen | Extracellular signal-regulated kinase | Glycogen synthase kinase 3 | Unloading | Cardiomyocytes | Rapamycin | Ribosomal protein S6 | Insulin | 1-Phosphatidylinositol 3-kinase | Studies | Musculoskeletal system | Signaling | Lungs | Adrenergic receptors | Ventricle | Laboratory animals | Receptors (physiology) | Hypertrophy
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 09/2016, Volume 126, Issue 9, pp. 3467 - 3478
Journal Article
Journal Article
Journal Article
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 12/2015, Volume 289, Issue 2, pp. 313 - 322
The generation of an immune response against infectious and other foreign agents is substantially modified by allostatic load, which is increased with... 
Oxidative stress | Beta Adrenoceptor | Listeria | Neuroimmunology | Stress response | Autophagy | ACTIVATION | BIOMEDICAL-RESEARCH | TRAIL DEFICIENCY | RESTRAINT STRESS | BETA-1-ADRENERGIC RECEPTORS | ACUTE COLD/RESTRAINT STRESS | PHARMACOLOGY & PHARMACY | TOXICOLOGY | HEALTH | LISTERIA-MONOCYTOGENES INFECTION | T-CELLS | HOST-RESISTANCE | Oxidative Stress | Glutathione - metabolism | Macrophages, Peritoneal - pathology | Stress, Physiological | Endoplasmic Reticulum - metabolism | Endoplasmic Reticulum - microbiology | Stress, Psychological - immunology | Endoplasmic Reticulum - pathology | Time Factors | Stress, Psychological - metabolism | Listeriosis - immunology | Restraint, Physical | Macrophages, Peritoneal - drug effects | Disease Models, Animal | Receptors, Adrenergic, beta-2 - deficiency | Cold Temperature | Listeriosis - metabolism | Receptors, Adrenergic, beta-1 - deficiency | Signal Transduction | Receptors, Adrenergic, beta-2 - genetics | Heat-Shock Proteins - metabolism | Cells, Cultured | Gene Expression Regulation | Macrophages, Peritoneal - immunology | Unfolded Protein Response | Mice, Knockout | Host-Pathogen Interactions | Stress, Psychological - pathology | Animals | Macrophages, Peritoneal - microbiology | Endoplasmic Reticulum Stress | Endoplasmic Reticulum - immunology | Listeriosis - pathology | Listeriosis - microbiology | Receptors, Adrenergic, beta-1 - genetics | Mice, Inbred BALB C | Macrophages, Peritoneal - metabolism | Phagocytosis | Stress, Psychological - etiology | Proteins | Glucose | Nitric oxide | Dextrose | Index Medicus | OXIDATION | NITRIC OXIDE | NERVES | MACROPHAGES | CATECHOLAMINES | 60 APPLIED LIFE SCIENCES | BIOLOGICAL STRESS | THIOLS | ENDOPLASMIC RETICULUM | REDUCTION | GLUTATHIONE | GLUCOSE | IN VITRO | INFLAMMATION | LIVER | MICE | NORADRENALINE | LYMPHOCYTES | PATHOGENS | autophagy
Journal Article
BRITISH JOURNAL OF PHARMACOLOGY, ISSN 0007-1188, 02/2008, Volume 153, Issue 4, pp. 684 - 692
Background and purpose: The role of beta-adrenoceptors in heart disease remains controversial. Although beta-blockers ameliorate the progression of heart... 
NERVOUS-SYSTEM | catecholamines | hypertrophy | HEART-FAILURE | ADRENERGIC-RECEPTOR SUBTYPES | BETA-2-ADRENERGIC RECEPTORS | DILATED CARDIOMYOPATHY | beta-adrenoceptor | LEFT-VENTRICULAR HYPERTROPHY | MICE LACKING | SYMPATHETIC DRIVE | TISSUE GROWTH-FACTOR | PHARMACOLOGY & PHARMACY | fibrosis | METABOLIC ALTERATIONS | Adaptation, Physiological | Blood Pressure | Ventricular Function, Left - genetics | Male | Intercellular Signaling Peptides and Proteins - metabolism | Hypertrophy, Left Ventricular - chemically induced | Inflammation - metabolism | Hypertrophy, Left Ventricular - genetics | Ligation | Time Factors | Hypertrophy, Left Ventricular - pathology | Mice, Inbred DBA | Angiotensin II | Cytokines - genetics | Disease Models, Animal | Receptors, Adrenergic, beta-2 - deficiency | Receptors, Adrenergic, beta-1 - deficiency | Cytokines - metabolism | Receptors, Adrenergic, beta-2 - genetics | Hypertrophy, Left Ventricular - metabolism | Hypertrophy, Left Ventricular - prevention & control | Mice, Inbred C57BL | Gene Expression Regulation | Intercellular Signaling Peptides and Proteins - genetics | Genotype | Receptors, Adrenergic, beta-1 - metabolism | Signal Transduction - genetics | Receptors, Adrenergic, beta-2 - metabolism | Mice, Knockout | Phenotype | Animals | Aorta - surgery | Fibrosis | Receptors, Adrenergic, beta-1 - genetics | Inflammation - prevention & control | Mice | Hypertrophy, Left Ventricular - physiopathology | Inflammation - physiopathology | Index Medicus
Journal Article
Cell Stress & Chaperones, ISSN 1355-8145, 5/2013, Volume 18, Issue 3, pp. 279 - 291
Acute cold restraint stress (ACRS) has been reported to suppress host defenses against Listeria monocytogenes, and this suppression was mediated by... 
T lymphocytes | Oxidative stress | Granulocytes | Lymphocytes | Neutrophils | Bone marrow | Infections | Mice | Leukocytes | Blood | Biochemistry, general | Neurosciences | Biomedicine | Immunology | Nitric oxide | Cold restraint stress | Cancer Research | Norepinephrine | Sympathetic nervous system | Biomedicine general | Cell Biology | LISTERIA-MONOCYTOGENES | COLONY-STIMULATING FACTOR | CXCR4 EXPRESSION | BONE-MARROW | FACTOR-I | NEUTROPHIL HOMEOSTASIS | G-CSF | CELL BIOLOGY | BETA-1-ADRENERGIC RECEPTORS | ACUTE COLD/RESTRAINT STRESS | T-CELLS | Leukocytes - pathology | Glutathione - metabolism | Male | Lymphopenia - pathology | RNA, Messenger - metabolism | Chemotaxis, Leukocyte - drug effects | Bone Marrow - metabolism | Restraint, Physical | Stress, Physiological - drug effects | Neutrophils - metabolism | Bone Marrow - drug effects | Lymphocytes - metabolism | Nitric Oxide - biosynthesis | Cold Temperature | Receptors, Adrenergic, beta-1 - deficiency | Lymphopenia - blood | Neutrophils - drug effects | RNA, Messenger - genetics | Receptors, Adrenergic, beta-1 - metabolism | Receptors, Adrenergic, beta-2 - metabolism | Mice, Knockout | Gene Expression Regulation - drug effects | Chemotactic Factors - blood | Animals | Chemokine CXCL12 - pharmacology | Lymphocytes - drug effects | Leukocytes - drug effects | Receptors, Adrenergic, beta-1 - genetics | Lymphopenia - immunology | Mice, Inbred BALB C | Oxidative Stress - drug effects | Chemotaxis, Leukocyte - immunology | Leukocytes - metabolism | Stress, Physiological - immunology | Genetic engineering | T cells | Noradrenaline | Original Paper
Journal Article
Journal Article