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Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 11/2009, Volume 54, Issue 19, pp. 1747 - 1762
Heart failure is a syndrome characterized initially by left ventricular dysfunction that triggers countermeasures aimed to restore cardiac output. These... 
adrenergic receptor | beta-blockers | heart failure | sympathomimetic inotropes | sympatholytics | sympathetic nervous system | CARDIAC-INSUFFICIENCY BISOPROLOL | OBSTRUCTIVE SLEEP-APNEA | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | AUTONOMIC FUNCTION | RANDOMIZED CONTROLLED-TRIAL | DILATED CARDIOMYOPATHY | LEFT-VENTRICULAR DYSFUNCTION | BETA-BLOCKADE | POSITIVE AIRWAY PRESSURE | BETA-ADRENERGIC RECEPTOR | Receptors, Adrenergic, alpha-1 - metabolism | Sympathetic Nervous System - drug effects | Humans | Heart Failure - physiopathology | Adrenergic alpha-Antagonists - therapeutic use | Receptors, Adrenergic, beta-2 - drug effects | Sympathetic Nervous System - physiopathology | Sympatholytics - therapeutic use | Cardiotonic Agents - therapeutic use | Receptors, Adrenergic - genetics | Parasympathetic Nervous System - physiopathology | Digoxin - therapeutic use | Adrenergic beta-Antagonists - therapeutic use | Sympathomimetics - therapeutic use | Receptors, Adrenergic, alpha-1 - drug effects | Heart Failure - genetics | Heart Failure - metabolism | Receptors, Adrenergic, beta-2 - metabolism | Heart Failure - drug therapy | Polymorphism, Genetic | Randomized Controlled Trials as Topic | Parasympathetic Nervous System - drug effects | Exercise Tolerance | Renin-Angiotensin System - drug effects | Hemodynamics - drug effects | Proteins | Confidence intervals | Heart rate | Heart failure | Heart attacks | Rodents | Mortality | Hospitalization | Kinases | Beta blockers
Journal Article
Europace, ISSN 1099-5129, 07/2017, Volume 19, Issue 7, pp. 1211 - 1219
Postural tachycardia syndrome (POTS), a common and debilitating cardiovascular disorder, is characterized by an exaggerated heart rate increase during... 
Autoimmunity | Adrenergic receptors | Postural tachycardia syndrome | Allosteric activation | Vasovagal syncope | DIAGNOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | MANAGEMENT | SYNCOPE | INAPPROPRIATE SINUS TACHYCARDIA | ORTHOSTATIC INTOLERANCE | NITROGLYCERIN | HYPOTENSION | CONSENSUS STATEMENT | Receptors, Adrenergic, alpha-1 - metabolism | Cricetulus | Immunoglobulin G - blood | Autoantibodies - blood | Humans | Male | Receptors, Adrenergic, beta-2 - immunology | Receptors, Adrenergic, beta-2 - drug effects | Adrenergic beta-1 Receptor Agonists - pharmacology | Case-Control Studies | Adrenergic alpha-1 Receptor Agonists - pharmacology | Adrenergic beta-2 Receptor Agonists - pharmacology | Receptors, Adrenergic, alpha-1 - immunology | Dose-Response Relationship, Drug | Young Adult | Transfection | Receptors, Adrenergic, beta-1 - immunology | Adult | Female | Receptors, Adrenergic, beta-1 - drug effects | CHO Cells | Postural Orthostatic Tachycardia Syndrome - immunology | Receptors, Adrenergic, alpha-1 - drug effects | Receptors, Adrenergic, beta-2 - genetics | Rats | Receptors, Adrenergic, beta-1 - metabolism | Abdominal Muscles - blood supply | Arterioles - drug effects | Vasoconstriction - drug effects | Postural Orthostatic Tachycardia Syndrome - blood | Receptors, Adrenergic, beta-2 - metabolism | Animals | Arterioles - metabolism | Postural Orthostatic Tachycardia Syndrome - diagnosis | Receptors, Adrenergic, alpha-1 - genetics | Adolescent | Receptors, Adrenergic, beta-1 - genetics | In Vitro Techniques | Clinical Research | Editor's Choice | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Immunologi inom det medicinska området | Immunology in the medical area
Journal Article
Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 2007, Volume 320, Issue 1, pp. 1 - 13
Journal Article
Circulation Research, ISSN 0009-7330, 02/2013, Volume 112, Issue 3, pp. 476 - 486
RATIONALE:Short-term β-adrenergic stimulation promotes contractility in response to stress but is ultimately detrimental in the failing heart because of... 
heart failure | cardiac progenitor cells | adrenergic regulation | adrenergic receptors | adult stem cells | PROTEIN-KINASE-A | CHRONIC HEART-FAILURE | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | DILATED CARDIOMYOPATHY | LEFT-VENTRICULAR DYSFUNCTION | FAILING HEART | GENE-EXPRESSION | MEDIATED DNA-SYNTHESIS | PERIPHERAL VASCULAR DISEASE | CYCLE PROGRESSION | BETA-ADRENERGIC RECEPTORS | HEMATOLOGY | Cyclin D1 - metabolism | Phosphorylation | Coculture Techniques | Humans | Male | G-Protein-Coupled Receptor Kinase 2 - metabolism | Receptors, Adrenergic, beta-2 - drug effects | Proto-Oncogene Proteins c-kit - metabolism | Stem Cells - metabolism | Adrenergic beta-2 Receptor Agonists - pharmacology | Dose-Response Relationship, Drug | Stem Cell Transplantation | Transfection | RNA Interference | Time Factors | Myocardial Infarction - pathology | Cell Death | Receptors, Adrenergic, beta-1 - drug effects | Nitric Oxide Synthase Type III - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Adrenergic beta-2 Receptor Antagonists - pharmacology | Disease Models, Animal | Myocardial Infarction - surgery | Cell Survival | Receptors, Adrenergic, beta-2 - genetics | Cells, Cultured | Receptors, Adrenergic, beta-1 - metabolism | Myocardial Infarction - metabolism | Receptors, Adrenergic, beta-2 - metabolism | Myocytes, Cardiac - pathology | Myocytes, Cardiac - transplantation | Animals | Myocytes, Cardiac - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Myocytes, Cardiac - metabolism | Stem Cells - drug effects | Stem Cells - pathology | Receptors, Adrenergic, beta-1 - genetics | Cell Proliferation - drug effects | Mice | Mitogen-Activated Protein Kinase 1 - metabolism
Journal Article
Cardiovascular Research, ISSN 0008-6363, 06/2015, Volume 106, Issue 3, pp. 408 - 420
Journal Article
Kidney International, ISSN 0085-2538, 07/2017, Volume 92, Issue 1, pp. 101 - 113
Macrophage activation is increased in diabetes and correlated with the onset and progression of vascular complications. To identify drugs that could inhibit... 
macrophages | fibrosis | inflammation | diabetes | PROTEIN | PERIPHERAL-BLOOD | NEPHROPATHY | MONOCYTIC CELLS | BETA | ALPHA-TOCOPHEROL SUPPLEMENTATION | CYTOKINE PRODUCTION | INSULIN-RESISTANCE | UROLOGY & NEPHROLOGY | PLASMA-GLUCOSE | Tumor Necrosis Factor-alpha - metabolism | Diabetes Mellitus, Experimental - drug therapy | Phosphorylation | Diabetic Cardiomyopathies - metabolism | Kidney - pathology | Streptozocin | Humans | Diabetes Mellitus, Experimental - genetics | Male | NF-kappa B - metabolism | beta-Arrestin 2 - metabolism | Receptors, Adrenergic, beta-2 - drug effects | Adrenergic beta-2 Receptor Agonists - pharmacology | Kidney - metabolism | Transfection | RNA Interference | Time Factors | Protein Kinase C - metabolism | Myocardium - metabolism | Diabetic Cardiomyopathies - prevention & control | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Diabetic Nephropathies - prevention & control | Adrenergic beta-2 Receptor Antagonists - pharmacology | Kidney - drug effects | Anti-Inflammatory Agents - pharmacology | Receptors, Adrenergic, beta-2 - genetics | Diabetic Nephropathies - metabolism | Diabetic Nephropathies - chemically induced | THP-1 Cells | beta-Arrestin 2 - genetics | Diabetic Nephropathies - genetics | Myocardium - pathology | Rats, Sprague-Dawley | Receptors, Adrenergic, beta-2 - metabolism | Diabetic Cardiomyopathies - genetics | Rats, Zucker | Macrophages - metabolism | Animals | NF-KappaB Inhibitor alpha - metabolism | Fibrosis | Macrophages - drug effects | Diabetic Cardiomyopathies - chemically induced | Macrophage Activation - drug effects
Journal Article
Journal Article
Cardiovascular Research, ISSN 0008-6363, 05/2017, Volume 113, Issue 6, pp. 656 - 670
Journal Article
Brain, Behavior, and Immunity, ISSN 0889-1591, 2014, Volume 41, Issue 1, pp. 200 - 209
Abstract Infectious complications are the leading cause of death in the post-acute phase of stroke. Post-stroke immunodeficiency is believed to result from... 
Psychiatry | Allergy and Immunology | Catecholamines | Stroke-induced immunosuppression | Glucocorticoids | Lymphocytes | BETA-2-ADRENERGIC RECEPTOR | ACTIVATION | LYMPHOCYTE | PSYCHIATRY | DEATH | IMMUNOLOGY | NEUROSCIENCES | ACUTE ISCHEMIC-STROKE | IMMUNODEPRESSION | INFECTION | BETA-ADRENERGIC RECEPTORS | T-CELLS | Receptors, Adrenergic, beta-2 - analysis | Infarction, Middle Cerebral Artery - physiopathology | Receptors, Glucocorticoid - antagonists & inhibitors | Apoptosis - drug effects | Male | Neuroimmunomodulation - physiology | Infarction, Middle Cerebral Artery - immunology | Receptors, Adrenergic, beta-2 - drug effects | Sympathetic Nervous System - physiopathology | Receptors, Glucocorticoid - analysis | Lymphopenia - etiology | Metanephrine - blood | Corticosterone - blood | Hypothalamo-Hypophyseal System - physiopathology | Leukocytes - cytology | Pituitary-Adrenal System - physiopathology | Mice, Inbred C57BL | Cells, Cultured | Cortisone - blood | Prednisolone - pharmacology | Infarction, Middle Cerebral Artery - blood | Receptors, Adrenergic, beta-2 - biosynthesis | Feedback, Physiological | Animals | Leukocytes - drug effects | Mice | Mifepristone - pharmacology | Propanolamines - pharmacology | Epinephrine - pharmacology | Interferon-gamma - biosynthesis | Neuroimmunomodulation - drug effects | Stroke (Disease) | Epinephrine | Brain | Corticosterone | Analysis | Mortality | ACTH | Biological response modifiers | Prednisolone | Cortisone
Journal Article
Oncogene, ISSN 0950-9232, 05/2007, Volume 26, Issue 26, pp. 3777 - 3788
Journal Article