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Journal Article
American journal of human genetics, ISSN 0002-9297, 2015, Volume 96, Issue 4, pp. 597 - 611
Esophageal squamous cell carcinoma (ESCC) is one of the most common cancers worldwide and the fourth most lethal cancer in China... 
POPULATION | DNA | SUSCEPTIBILITY | GENETICS & HEREDITY | ARCHITECTURE | WIDE ASSOCIATION | DIFFERENTIATION | IDENTIFICATION | SOMATIC MUTATIONS | HUMAN CANCER | BREAST | Immunohistochemistry | Carcinoma, Squamous Cell - genetics | Humans | Molecular Sequence Data | Immunoblotting | Gene Knockdown Techniques | CREB-Binding Protein - genetics | Polycomb Repressive Complex 1 - genetics | Tetrazolium Salts | APOBEC-1 Deaminase | Ligases | Base Sequence | China | Tumor Suppressor Proteins - genetics | Thiazoles | Patched Receptors | Real-Time Polymerase Chain Reaction | Esophageal Squamous Cell Carcinoma | Genetic Predisposition to Disease - genetics | Cytidine Deaminase - genetics | In Situ Hybridization, Fluorescence | DNA Copy Number Variations - genetics | Signal Transduction - genetics | Transcription Factors - genetics | Mutation - genetics | Ubiquitin Thiolesterase - genetics | Sequence Analysis, DNA | Genome, Human - genetics | Phosphatidylinositol 3-Kinases - genetics | Class I Phosphatidylinositol 3-Kinases | Analysis of Variance | Esophageal Neoplasms - genetics | Polycomb-Group Proteins - genetics | Cell Line, Tumor | LIM Domain Proteins - genetics | Ubiquitin-Protein Ligases - genetics | Patched-1 Receptor | Receptors, Cell Surface - genetics | Squamous cell carcinoma | Genetic aspects | Gene mutations | Identification and classification | Esophageal cancer
Journal Article
PLoS genetics, ISSN 1553-7390, 06/2017, Volume 13, Issue 6, p. e1006832
.... Using a reverse-genetic approach, we identified the Arabidopsis thaliana leucine-rich repeat receptor kinase MIK2 as an important regulator of cell wall damage responses triggered upon cellulose biosynthesis inhibition... 
Journal Article | ORGANIZATION | CORTICAL MICROTUBULES | FERONIA | MUTANT | PROTEIN | BIOSYNTHESIS | GENES | GENETICS & HEREDITY | RESISTANCE | HELICAL GROWTH | CELLULOSE SYNTHASE COMPLEXES | Arabidopsis Proteins - genetics | Protein Kinases - genetics | Arabidopsis - drug effects | Stress, Physiological - genetics | Cell Wall - genetics | Plant Roots - genetics | Plant Diseases - microbiology | Sodium Chloride - toxicity | Plant Roots - drug effects | Arabidopsis - genetics | Cellulose - biosynthesis | Oxylipins - metabolism | Gene Expression Regulation, Plant - drug effects | Cell Wall - drug effects | Lignin - biosynthesis | Arabidopsis Proteins - biosynthesis | Cyclopentanes - metabolism | Plant Diseases - genetics | Stress, Physiological - drug effects | Protein Kinases - biosynthesis | Disease Resistance - genetics | Fusarium - pathogenicity | Receptors, Cell Surface - genetics | Arabidopsis thaliana | Physiological aspects | Genetic aspects | Gene expression | Observations | Phosphotransferases | Plant cell walls | Lignin | Research parks | Laboratories | Genomics | Cellulose | Biosynthesis | Leucine | Kinases | Salinity tolerance | Cell adhesion & migration | Fungi | Fusarium | Life sciences | Charitable foundations | Jasmonic acid | Plates | Stresses | Cues | Pathogens | Plants (botany) | Fusarium oxysporum | Cell walls | Abiotic stress | Grants | Acid production | College campuses | Immunological tolerance | Alleles | Molecular biology | Detection | Integrity | Life Sciences | Vegetal Biology | Engineering Sciences | Food engineering | Chemical and Process Engineering
Journal Article
Bioimpacts, ISSN 2228-5660, 12/2018, Volume 8, Issue Suppl 1, pp. S1 - S129
Journal Article
The Journal of experimental medicine, ISSN 1540-9538, 2013, Volume 210, Issue 1, pp. 31 - 40
Journal Article
The Journal of experimental medicine, ISSN 1540-9538, 2008, Volume 205, Issue 8, pp. 1903 - 1916
Journal Article
Nature cell biology, ISSN 1476-4679, 2009, Volume 11, Issue 10, pp. 1254 - 1260
.... Despite the identification of many components of this pathway, it remains unclear how the BR signal is transduced from the cell surface to the nucleus... 
TYROSINE PHOSPHORYLATION | GSK3 | PROTEIN-KINASE | BRI1 | BZR1 | MECHANISMS | ARABIDOPSIS | PLASMA-MEMBRANE | PLANT-GROWTH | FAMILY | CELL BIOLOGY | Genes, Plant | Recombinant Fusion Proteins - metabolism | Phytosterols - pharmacology | Cell Nucleus - metabolism | Plant Proteins - chemistry | Plants, Genetically Modified | Gene Expression Regulation, Plant | Plant Proteins - metabolism | Phosphorylation - drug effects | Nuclear Proteins - genetics | Steroids, Heterocyclic - metabolism | Steroids, Heterocyclic - pharmacology | Amino Acid Sequence | Glycogen Synthase Kinase 3 - chemistry | Glutathione Transferase - metabolism | Receptors, Cell Surface - metabolism | Brassinosteroids | Nuclear Proteins - metabolism | Signal Transduction - genetics | Binding Sites - genetics | Recombinant Fusion Proteins - chemistry | Glycogen Synthase Kinase 3 - metabolism | Nuclear Proteins - chemistry | Subcellular Fractions - metabolism | Cholestanols - pharmacology | Plant Proteins - genetics | Cell Nucleus - genetics | Glycogen Synthase Kinase 3 - genetics | Phytosterols - metabolism | Protein Binding | Signal Transduction - physiology | Mutation | Plant Growth Regulators - pharmacology | Receptors, Cell Surface - genetics | Development | Physiological aspects | Genetic aspects | Cellular signal transduction | Plants | Research | DNA binding proteins | Gene expression
Journal Article
Nature medicine, ISSN 1546-170X, 2007, Volume 13, Issue 3, pp. 332 - 339
Journal Article
Blood, ISSN 1528-0020, 2017, Volume 129, Issue 6, pp. 759 - 770
Contrasting patterns of basal phosphorylation levels and α-BCR–induced signaling between CLL and MCL tumors. Direct association between BCR-induced signaling... 
SURFACE IGM | BCR | CHRONIC-LYMPHOCYTIC-LEUKEMIA | BRUTONS TYROSINE KINASE | HUMAN FOLLICULAR LYMPHOMA | GENE-EXPRESSION | KINASE INHIBITOR IBRUTINIB | MUTATIONS | TARGETING BTK | HEMATOLOGY | NF-KAPPA-B | Lymphoma, Follicular - metabolism | Protein-Tyrosine Kinases - metabolism | Humans | Lymphoma, Mantle-Cell - pathology | Leukemia, Lymphocytic, Chronic, B-Cell - genetics | Receptors, Antigen, B-Cell - metabolism | STAT1 Transcription Factor - metabolism | Lymphoma, Follicular - genetics | Mitogen-Activated Protein Kinase 1 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Single-Cell Analysis | Immunoglobulin M - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | STAT1 Transcription Factor - genetics | Leukemia, Lymphocytic, Chronic, B-Cell - pathology | Lymphoma, Large B-Cell, Diffuse - diagnosis | Mitogen-Activated Protein Kinase 3 - metabolism | Leukemia, Lymphocytic, Chronic, B-Cell - metabolism | Receptors, Antigen, B-Cell - genetics | CD79 Antigens - genetics | src-Family Kinases - genetics | Mitogen-Activated Protein Kinase 1 - metabolism | Phosphorylation | Gene Expression Regulation, Neoplastic | Lymphoma, Follicular - diagnosis | Lymphoma, Large B-Cell, Diffuse - metabolism | Phosphoproteins - metabolism | Proto-Oncogene Proteins c-akt - genetics | Lymphoma, Mantle-Cell - diagnosis | STAT5 Transcription Factor - genetics | STAT5 Transcription Factor - metabolism | Protein-Tyrosine Kinases - genetics | Flow Cytometry | Syk Kinase - genetics | Phospholipase C gamma - genetics | src-Family Kinases - metabolism | Lymphoma, Mantle-Cell - genetics | Lymphoma, Mantle-Cell - metabolism | Diagnosis, Differential | Phospholipase C gamma - metabolism | Lymphoma, Large B-Cell, Diffuse - pathology | Lymphoma, Follicular - pathology | p38 Mitogen-Activated Protein Kinases - genetics | Phosphoproteins - genetics | CD79 Antigens - metabolism | Syk Kinase - metabolism | Lymphoma, Large B-Cell, Diffuse - genetics | Leukemia, Lymphocytic, Chronic, B-Cell - diagnosis | Life Sciences | Human health and pathology | Hematology | Cancer | Lymphoid Neoplasia
Journal Article
Journal of Virology, ISSN 0022-538X, 2015, Volume 89, Issue 17, pp. 8880 - 8896
.... We show that human dermal fibroblasts, epidermal keratinocytes, and immature dendritic cells are permissive to the most recent ZIKV isolate, responsible for the epidemic in French Polynesia... 
ACTIVATION | DENGUE-VIRUS | ENTRY | VIROLOGY | MOSQUITOS | WEST-NILE-VIRUS | MACHINERY | HEPARAN-SULFATE | AUTOPHAGY | RECEPTORS | I INTERFERON | Keratinocytes - virology | Membrane Glycoproteins - metabolism | Dendritic Cells - immunology | Humans | Flaviviridae - physiology | Virus Internalization | Interferon-Induced Helicase, IFIH1 | Lectins, C-Type - metabolism | Phagosomes - immunology | RNA Interference | Vero Cells | Myxovirus Resistance Proteins - biosynthesis | DEAD Box Protein 58 | Toll-Like Receptor 3 - immunology | Receptor Protein-Tyrosine Kinases - metabolism | Toll-Like Receptor 3 - metabolism | Cell Adhesion Molecules - metabolism | RNA, Small Interfering | Receptors, Cell Surface - genetics | Cell Adhesion Molecules - genetics | Ubiquitins - biosynthesis | Interferon-beta - immunology | Receptors, Virus - metabolism | Cercopithecus aethiops | Receptors, Virus - genetics | Lectins, C-Type - genetics | Insect Vectors - virology | Autophagy - immunology | Aedes - virology | Flaviviridae - immunology | Toll-Like Receptor 7 - immunology | HEK293 Cells | Toll-Like Receptor 3 - genetics | DEAD-box RNA Helicases - metabolism | Dendritic Cells - virology | Skin - virology | Flaviviridae Infections - immunology | Skin - immunology | Fibroblasts - virology | Proto-Oncogene Proteins - metabolism | Cells, Cultured | Interferon-beta - biosynthesis | Receptors, Cell Surface - metabolism | Proto-Oncogene Proteins - genetics | Hepatitis A Virus Cellular Receptor 1 | Flaviviridae Infections - virology | Membrane Glycoproteins - genetics | DEAD-box RNA Helicases - genetics | Animals | Receptor Protein-Tyrosine Kinases - genetics | Virus Replication | Cytokines - biosynthesis | Life Sciences | Microbiology and Parasitology | Cellular Biology | Biochemistry, Molecular Biology | Virology
Journal Article