Journal of Internal Medicine, ISSN 0954-6820, 06/2009, Volume 265, Issue 6, pp. 663 - 679
. Cytokine production is necessary to protect against pathogens and promote tissue repair, but excessive cytokine release can lead to systemic inflammation,...
innate immunity | alpha7 | inflammation | cholinergic | Alpha7 | Innate immunity | Inflammation | Cholinergic | VAGUS NERVE | ANTIINFLAMMATORY PATHWAY | ISCHEMIA/REPERFUSION INJURY | NONNEURONAL ACETYLCHOLINE | IMPROVES SURVIVAL | MEDICINE, GENERAL & INTERNAL | TUMOR-NECROSIS-FACTOR | IMMUNE-SYSTEM | SYMPATHETIC INNERVATION | NICOTINIC ACETYLCHOLINE-RECEPTOR | NF-KAPPA-B | Tumor Necrosis Factor-alpha - metabolism | Receptors, Nicotinic - metabolism | Sepsis - immunology | Receptors, Cholinergic - metabolism | Cholinergic Agents - immunology | Inflammation - immunology | Acetylcholine - immunology | Mice, Knockout | Brain - metabolism | Vagus Nerve - physiology | Animals | Vagus Nerve - immunology | Neural Pathways - metabolism | Receptors, Cholinergic - immunology | Mice | Corticosteroids
innate immunity | alpha7 | inflammation | cholinergic | Alpha7 | Innate immunity | Inflammation | Cholinergic | VAGUS NERVE | ANTIINFLAMMATORY PATHWAY | ISCHEMIA/REPERFUSION INJURY | NONNEURONAL ACETYLCHOLINE | IMPROVES SURVIVAL | MEDICINE, GENERAL & INTERNAL | TUMOR-NECROSIS-FACTOR | IMMUNE-SYSTEM | SYMPATHETIC INNERVATION | NICOTINIC ACETYLCHOLINE-RECEPTOR | NF-KAPPA-B | Tumor Necrosis Factor-alpha - metabolism | Receptors, Nicotinic - metabolism | Sepsis - immunology | Receptors, Cholinergic - metabolism | Cholinergic Agents - immunology | Inflammation - immunology | Acetylcholine - immunology | Mice, Knockout | Brain - metabolism | Vagus Nerve - physiology | Animals | Vagus Nerve - immunology | Neural Pathways - metabolism | Receptors, Cholinergic - immunology | Mice | Corticosteroids
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2013, Volume 110, Issue 9, pp. 3573 - 3578
Symptoms of depression can be induced in humans through blockade of acetylcholinesterase (AChE) whereas antidepressant-like effects can be produced in animal...
Cholinergics | Pain | Genetic variation | Antidepressants | Mice | Anxiety | Cholinergic receptors | Behavioral neuroscience | Hippocampus | Depressive disorders | Affective disorders | Psychosocial stress | SYSTEM | psychosocial stress | ACETYLCHOLINE | MULTIDISCIPLINARY SCIENCES | SCOPOLAMINE | NUCLEUS-ACCUMBENS | ADRENERGIC HYPOTHESIS | affective disorders | ANIMAL-MODEL | MICE | RECEPTORS | ANTIDEPRESSANT-LIKE PROPERTIES | BRAIN | Humans | Male | Anxiety - drug therapy | Hippocampus - drug effects | Cholinergic Antagonists - pharmacology | Gene Knockdown Techniques | Depression - drug therapy | Depression - metabolism | Time Factors | Stress, Psychological - metabolism | Anxiety - complications | Behavior, Animal - drug effects | Depression - complications | Antidepressive Agents - pharmacology | Resilience, Psychological | Fluoxetine - therapeutic use | Anxiety - metabolism | Cholinergic Antagonists - therapeutic use | Fluoxetine - pharmacology | Dependovirus - metabolism | Receptors, Cholinergic - metabolism | Cholinergic Neurons - metabolism | Stress, Psychological - drug therapy | Mice, Inbred C57BL | Anxiety - psychology | Hippocampus - pathology | Physostigmine | Cholinergic Neurons - drug effects | Hindlimb Suspension | Antidepressive Agents - therapeutic use | Hippocampus - metabolism | Phenotype | Animals | Signal Transduction - drug effects | Depression - psychology | Cholinergic Neurons - pathology | Acetylcholinesterase - metabolism | Stress, Psychological - complications | RNA, Small Interfering - metabolism | Physiological aspects | Hippocampus (Brain) | Stress (Psychology) | Depression, Mental | Biological Sciences
Cholinergics | Pain | Genetic variation | Antidepressants | Mice | Anxiety | Cholinergic receptors | Behavioral neuroscience | Hippocampus | Depressive disorders | Affective disorders | Psychosocial stress | SYSTEM | psychosocial stress | ACETYLCHOLINE | MULTIDISCIPLINARY SCIENCES | SCOPOLAMINE | NUCLEUS-ACCUMBENS | ADRENERGIC HYPOTHESIS | affective disorders | ANIMAL-MODEL | MICE | RECEPTORS | ANTIDEPRESSANT-LIKE PROPERTIES | BRAIN | Humans | Male | Anxiety - drug therapy | Hippocampus - drug effects | Cholinergic Antagonists - pharmacology | Gene Knockdown Techniques | Depression - drug therapy | Depression - metabolism | Time Factors | Stress, Psychological - metabolism | Anxiety - complications | Behavior, Animal - drug effects | Depression - complications | Antidepressive Agents - pharmacology | Resilience, Psychological | Fluoxetine - therapeutic use | Anxiety - metabolism | Cholinergic Antagonists - therapeutic use | Fluoxetine - pharmacology | Dependovirus - metabolism | Receptors, Cholinergic - metabolism | Cholinergic Neurons - metabolism | Stress, Psychological - drug therapy | Mice, Inbred C57BL | Anxiety - psychology | Hippocampus - pathology | Physostigmine | Cholinergic Neurons - drug effects | Hindlimb Suspension | Antidepressive Agents - therapeutic use | Hippocampus - metabolism | Phenotype | Animals | Signal Transduction - drug effects | Depression - psychology | Cholinergic Neurons - pathology | Acetylcholinesterase - metabolism | Stress, Psychological - complications | RNA, Small Interfering - metabolism | Physiological aspects | Hippocampus (Brain) | Stress (Psychology) | Depression, Mental | Biological Sciences
Journal Article
Current Neuropharmacology, ISSN 1570-159X, 01/2016, Volume 14, Issue 1, pp. 101 - 115
Acetylcholine (ACh) has a crucial role in the peripheral and central nervous systems. The enzyme choline acetyltransferase (ChAT) is responsible for...
ChAT | AChE | Acetylcholine | Alzheimer’s disease | CHT1 | VAChT | M1 MUSCARINIC AGONISTS | NUCLEUS BASALIS | VESICULAR ACETYLCHOLINE TRANSPORTER | AMYLOID PRECURSOR PROTEIN | CHOLINESTERASE-INHIBITORS | RIVASTIGMINE TRANSDERMAL PATCH | NEUROSCIENCES | SPATIAL WORKING-MEMORY | PHARMACOLOGY & PHARMACY | CENTRAL-NERVOUS-SYSTEM | VOLTAGE-CLAMP ANALYSIS | Alzheimer's disease | BASAL FOREBRAIN LESIONS | Acetylcholine - genetics | Cholinergic Agents - administration & dosage | Receptors, Cholinergic - metabolism | Cholinergic Neurons - metabolism | Humans | Alzheimer Disease - drug therapy | Acetylcholine - metabolism | Cholinergic Neurons - drug effects | Animals | Cholinesterase Inhibitors - administration & dosage | Alzheimer Disease - metabolism | Drug Delivery Systems - trends | Acetylcholine - antagonists & inhibitors | Alzheimer Disease - genetics | Drug Delivery Systems - methods | Receptors, Cholinergic - genetics
ChAT | AChE | Acetylcholine | Alzheimer’s disease | CHT1 | VAChT | M1 MUSCARINIC AGONISTS | NUCLEUS BASALIS | VESICULAR ACETYLCHOLINE TRANSPORTER | AMYLOID PRECURSOR PROTEIN | CHOLINESTERASE-INHIBITORS | RIVASTIGMINE TRANSDERMAL PATCH | NEUROSCIENCES | SPATIAL WORKING-MEMORY | PHARMACOLOGY & PHARMACY | CENTRAL-NERVOUS-SYSTEM | VOLTAGE-CLAMP ANALYSIS | Alzheimer's disease | BASAL FOREBRAIN LESIONS | Acetylcholine - genetics | Cholinergic Agents - administration & dosage | Receptors, Cholinergic - metabolism | Cholinergic Neurons - metabolism | Humans | Alzheimer Disease - drug therapy | Acetylcholine - metabolism | Cholinergic Neurons - drug effects | Animals | Cholinesterase Inhibitors - administration & dosage | Alzheimer Disease - metabolism | Drug Delivery Systems - trends | Acetylcholine - antagonists & inhibitors | Alzheimer Disease - genetics | Drug Delivery Systems - methods | Receptors, Cholinergic - genetics
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2012, Volume 109, Issue 28, pp. 11360 - 11365
A large number of studies have demonstrated that the nucleus accumbens (NAC) is a critical site in the neuronal circuits controlling reward responses,...
Cholinergics | Phenotypes | Interneurons | Neurons | Antidepressants | Viruses | Mice | Physiological regulation | Cells | Depressive disorders | Antidepressant | Acetylcholine | S100a10 | Neurotransmission | neurotransmission | MULTIDISCIPLINARY SCIENCES | s100a10 | antidepressant | MAJOR DEPRESSION | REWARD | CNS CELL-TYPES | P11 | TRANSLATIONAL PROFILING APPROACH | HYPOTHESIS | acetylcholine | DEEP BRAIN-STIMULATION | RECEPTORS | STRIATUM | Depression - physiopathology | Molecular Chaperones - metabolism | Receptors, Cholinergic - metabolism | Mice, Inbred C57BL | Nucleus Accumbens - metabolism | Neurotransmitter Agents - metabolism | Male | S100 Proteins - metabolism | Acetylcholine - metabolism | Mice, Knockout | Behavior, Animal | Immunohistochemistry - methods | Depression - metabolism | Phenotype | Animals | Interneurons - metabolism | Models, Biological | Antidepressive Agents - pharmacology | Neurons - metabolism | Annexin A2 - metabolism | Physiological aspects | Cholinergic mechanisms | Research | Depression, Mental | Biological Sciences
Cholinergics | Phenotypes | Interneurons | Neurons | Antidepressants | Viruses | Mice | Physiological regulation | Cells | Depressive disorders | Antidepressant | Acetylcholine | S100a10 | Neurotransmission | neurotransmission | MULTIDISCIPLINARY SCIENCES | s100a10 | antidepressant | MAJOR DEPRESSION | REWARD | CNS CELL-TYPES | P11 | TRANSLATIONAL PROFILING APPROACH | HYPOTHESIS | acetylcholine | DEEP BRAIN-STIMULATION | RECEPTORS | STRIATUM | Depression - physiopathology | Molecular Chaperones - metabolism | Receptors, Cholinergic - metabolism | Mice, Inbred C57BL | Nucleus Accumbens - metabolism | Neurotransmitter Agents - metabolism | Male | S100 Proteins - metabolism | Acetylcholine - metabolism | Mice, Knockout | Behavior, Animal | Immunohistochemistry - methods | Depression - metabolism | Phenotype | Animals | Interneurons - metabolism | Models, Biological | Antidepressive Agents - pharmacology | Neurons - metabolism | Annexin A2 - metabolism | Physiological aspects | Cholinergic mechanisms | Research | Depression, Mental | Biological Sciences
Journal Article
Journal of Pharmacological Sciences, ISSN 1347-8613, 05/2017, Volume 134, Issue 1, pp. 1 - 21
T and B cells, macrophages and dendritic cells (DCs) all express most of the components necessary for a functional cholinergic system. This includes choline...
Dendritic cell | Choline acetyltransferase | Lymphocyte | Acetylcholine receptor | Macrophage | LEUKEMIC T-CELLS | NONNEURONAL ACETYLCHOLINE | FOS GENE-EXPRESSION | CIRCULATING MONONUCLEAR LEUKOCYTES | POLYSPECIFIC CATION TRANSPORTERS | INFLAMMATORY-BOWEL-DISEASE | ISOLATED HUMAN PLACENTA | NICOTINIC ACETYLCHOLINE-RECEPTOR | ACETYLTRANSFERASE MESSENGER-RNA | PHARMACOLOGY & PHARMACY | ANTIGEN-SPECIFIC IGG | Lymphocytes - metabolism | Receptors, Nicotinic - metabolism | Receptors, Cholinergic - metabolism | Lymphocyte Activation | Dendritic Cells - immunology | Humans | Acetylcholine - metabolism | Inflammation - immunology | Choline O-Acetyltransferase - metabolism | Inflammation - metabolism | Macrophages - metabolism | Animals | B-Lymphocytes - immunology | Lymphocytes - immunology | T-Lymphocytes - metabolism | Antibodies - immunology | Antibody Formation | Antigens - immunology | T-Lymphocytes - immunology | B-Lymphocytes - metabolism | Cytokines - biosynthesis | Dendritic Cells - metabolism | Macrophages - immunology | Physiological aspects | Cholinergic mechanisms | Research | B cells | Dendritic cells | T cells | Cell receptors | Genetic aspects | Immune response | Health aspects
Dendritic cell | Choline acetyltransferase | Lymphocyte | Acetylcholine receptor | Macrophage | LEUKEMIC T-CELLS | NONNEURONAL ACETYLCHOLINE | FOS GENE-EXPRESSION | CIRCULATING MONONUCLEAR LEUKOCYTES | POLYSPECIFIC CATION TRANSPORTERS | INFLAMMATORY-BOWEL-DISEASE | ISOLATED HUMAN PLACENTA | NICOTINIC ACETYLCHOLINE-RECEPTOR | ACETYLTRANSFERASE MESSENGER-RNA | PHARMACOLOGY & PHARMACY | ANTIGEN-SPECIFIC IGG | Lymphocytes - metabolism | Receptors, Nicotinic - metabolism | Receptors, Cholinergic - metabolism | Lymphocyte Activation | Dendritic Cells - immunology | Humans | Acetylcholine - metabolism | Inflammation - immunology | Choline O-Acetyltransferase - metabolism | Inflammation - metabolism | Macrophages - metabolism | Animals | B-Lymphocytes - immunology | Lymphocytes - immunology | T-Lymphocytes - metabolism | Antibodies - immunology | Antibody Formation | Antigens - immunology | T-Lymphocytes - immunology | B-Lymphocytes - metabolism | Cytokines - biosynthesis | Dendritic Cells - metabolism | Macrophages - immunology | Physiological aspects | Cholinergic mechanisms | Research | B cells | Dendritic cells | T cells | Cell receptors | Genetic aspects | Immune response | Health aspects
Journal Article
Nature, ISSN 0028-0836, 2016, Volume 538, Issue 7624, pp. 253 - 256
Atypical food intake is a primary cause of obesity and other eating and metabolic disorders. Insight into the neural control of feeding has previously focused...
LEPTIN | POMC | BODY-WEIGHT | ACETYLCHOLINE | FOOD-INTAKE | MULTIDISCIPLINARY SCIENCES | NEURONS | CENTRAL-NERVOUS-SYSTEM | SMOKING | MICE | BALANCE | Eating - psychology | Hyperphagia - enzymology | Homeostasis | Male | Acetylcholine - metabolism | Obesity - genetics | Eating - physiology | Satiety Response - physiology | Cell Death | Female | Cholinergic Agonists | Receptors, Cholinergic - metabolism | Cholinergic Neurons - metabolism | Hypothalamus - physiology | Feeding Behavior - physiology | Mice, Knockout | Obesity - pathology | Hyperphagia - genetics | Hyperphagia - pathology | Basal Forebrain - physiology | Animals | Nicotine - metabolism | Hypothalamus - cytology | Basal Forebrain - cytology | Choline O-Acetyltransferase - deficiency | Feeding Behavior - psychology | Mice | Obesity - enzymology | Appetite Regulation - physiology | Models, Neurological | Body Weight - physiology | Cholinergic Neurons - pathology | Appetite | Physiological aspects | Control | Neural circuitry | Prosencephalon | Brain | Obesity | Signal transduction | Neurons | Rodents | Food
LEPTIN | POMC | BODY-WEIGHT | ACETYLCHOLINE | FOOD-INTAKE | MULTIDISCIPLINARY SCIENCES | NEURONS | CENTRAL-NERVOUS-SYSTEM | SMOKING | MICE | BALANCE | Eating - psychology | Hyperphagia - enzymology | Homeostasis | Male | Acetylcholine - metabolism | Obesity - genetics | Eating - physiology | Satiety Response - physiology | Cell Death | Female | Cholinergic Agonists | Receptors, Cholinergic - metabolism | Cholinergic Neurons - metabolism | Hypothalamus - physiology | Feeding Behavior - physiology | Mice, Knockout | Obesity - pathology | Hyperphagia - genetics | Hyperphagia - pathology | Basal Forebrain - physiology | Animals | Nicotine - metabolism | Hypothalamus - cytology | Basal Forebrain - cytology | Choline O-Acetyltransferase - deficiency | Feeding Behavior - psychology | Mice | Obesity - enzymology | Appetite Regulation - physiology | Models, Neurological | Body Weight - physiology | Cholinergic Neurons - pathology | Appetite | Physiological aspects | Control | Neural circuitry | Prosencephalon | Brain | Obesity | Signal transduction | Neurons | Rodents | Food
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 4, p. e19155
The neurotransmitter glutamate is released by excitatory projection neurons throughout the brain. However, non-glutamatergic cells, including cholinergic and...
DOPAMINE NEURONS | TRANSPORTER-3 | TONICALLY ACTIVE NEURONS | SEROTONINERGIC NEURONS | ACETYLCHOLINE | VESICULAR GLUTAMATE | BIOLOGY | RAT-BRAIN | RECEPTORS | MODULATION | NUCLEUS-ACCUMBENS | Central Nervous System Stimulants - pharmacology | Receptors, Cholinergic - metabolism | Amino Acid Transport Systems, Acidic - metabolism | Interneurons - drug effects | Receptors, N-Methyl-D-Aspartate - metabolism | Channelrhodopsins | Male | Corpus Striatum - metabolism | Evoked Potentials - drug effects | Scopolamine Hydrobromide - pharmacology | Animals | Interneurons - metabolism | Mecamylamine - pharmacology | Corpus Striatum - drug effects | Picrotoxin - pharmacology | Female | Mice | Hostages | Amino acids | Glutamate | Neurons | Choline | Brain | Neurosciences | Interneurons | Synaptogenesis | Glutamic acid receptors (ionotropic) | Neurobiology | Homeostasis | Glutamic acid receptors | Choline O-acetyltransferase | Recombinase | Medical schools | Receptors | Transmitters | Rodents | Neostriatum | Physiology | Acetyltransferase | Localization | Adenosine | Departments | Dopamine | Cortex | N-Methyl-D-aspartic acid receptors | Artificial chromosomes | Metabolism | Spiny neurons | Medicine | Neurology | Acetylcholine receptors | Cocaine | Glutamic acid transporter | Glutamatergic transmission | Transporter | Endocrinology | Synapses | Questioning
DOPAMINE NEURONS | TRANSPORTER-3 | TONICALLY ACTIVE NEURONS | SEROTONINERGIC NEURONS | ACETYLCHOLINE | VESICULAR GLUTAMATE | BIOLOGY | RAT-BRAIN | RECEPTORS | MODULATION | NUCLEUS-ACCUMBENS | Central Nervous System Stimulants - pharmacology | Receptors, Cholinergic - metabolism | Amino Acid Transport Systems, Acidic - metabolism | Interneurons - drug effects | Receptors, N-Methyl-D-Aspartate - metabolism | Channelrhodopsins | Male | Corpus Striatum - metabolism | Evoked Potentials - drug effects | Scopolamine Hydrobromide - pharmacology | Animals | Interneurons - metabolism | Mecamylamine - pharmacology | Corpus Striatum - drug effects | Picrotoxin - pharmacology | Female | Mice | Hostages | Amino acids | Glutamate | Neurons | Choline | Brain | Neurosciences | Interneurons | Synaptogenesis | Glutamic acid receptors (ionotropic) | Neurobiology | Homeostasis | Glutamic acid receptors | Choline O-acetyltransferase | Recombinase | Medical schools | Receptors | Transmitters | Rodents | Neostriatum | Physiology | Acetyltransferase | Localization | Adenosine | Departments | Dopamine | Cortex | N-Methyl-D-aspartic acid receptors | Artificial chromosomes | Metabolism | Spiny neurons | Medicine | Neurology | Acetylcholine receptors | Cocaine | Glutamic acid transporter | Glutamatergic transmission | Transporter | Endocrinology | Synapses | Questioning
Journal Article
8.
Full Text
C. elegans Punctin specifies cholinergic versus GABAergic identity of postsynaptic domains
Nature, ISSN 0028-0836, 2014, Volume 511, Issue 7510, pp. 466 - 470
Because most neurons receive thousands of synaptic inputs, the neuronal membrane is a mosaic of specialized microdomains where neurotransmitter receptors...
RECEPTOR ALPHA-SUBUNIT | COMPLEX | CAENORHABDITIS-ELEGANS | GABA RECEPTOR | PROTEIN | MULTIDISCIPLINARY SCIENCES | EXTRACELLULAR-MATRIX | NEUROMUSCULAR-JUNCTION | GENE ENCODES | ACETYLCHOLINE-RECEPTOR | Caenorhabditis elegans - metabolism | Receptors, Cholinergic - metabolism | Cholinergic Neurons - metabolism | Caenorhabditis elegans Proteins - chemistry | Nerve Tissue Proteins - deficiency | Caenorhabditis elegans Proteins - metabolism | Acetylcholine - metabolism | Motor Neurons - metabolism | Nerve Tissue Proteins - metabolism | ADAM Proteins - metabolism | Nerve Tissue Proteins - chemistry | Animals | Protein Isoforms - metabolism | Protein Isoforms - chemistry | Neuromuscular Junction | Protein Isoforms - deficiency | Receptors, GABA-A - metabolism | GABAergic Neurons - metabolism | Post-Synaptic Density - metabolism | Extracellular Matrix Proteins - metabolism | Caenorhabditis elegans | Analysis | Physiological aspects | GABA | Genetic aspects | Research | Health aspects | Proteins | Medical research | Neurology | Neurotransmitters | Neurons
RECEPTOR ALPHA-SUBUNIT | COMPLEX | CAENORHABDITIS-ELEGANS | GABA RECEPTOR | PROTEIN | MULTIDISCIPLINARY SCIENCES | EXTRACELLULAR-MATRIX | NEUROMUSCULAR-JUNCTION | GENE ENCODES | ACETYLCHOLINE-RECEPTOR | Caenorhabditis elegans - metabolism | Receptors, Cholinergic - metabolism | Cholinergic Neurons - metabolism | Caenorhabditis elegans Proteins - chemistry | Nerve Tissue Proteins - deficiency | Caenorhabditis elegans Proteins - metabolism | Acetylcholine - metabolism | Motor Neurons - metabolism | Nerve Tissue Proteins - metabolism | ADAM Proteins - metabolism | Nerve Tissue Proteins - chemistry | Animals | Protein Isoforms - metabolism | Protein Isoforms - chemistry | Neuromuscular Junction | Protein Isoforms - deficiency | Receptors, GABA-A - metabolism | GABAergic Neurons - metabolism | Post-Synaptic Density - metabolism | Extracellular Matrix Proteins - metabolism | Caenorhabditis elegans | Analysis | Physiological aspects | GABA | Genetic aspects | Research | Health aspects | Proteins | Medical research | Neurology | Neurotransmitters | Neurons
Journal Article
The Neuroscientist, ISSN 1073-8584, 6/2016, Volume 22, Issue 3, pp. 238 - 251
Functional overviews of cholinergic mechanisms in the cerebral cortex have traditionally focused on the release of acetylcholine with modulator and transmitter...
volume transmission | acetylcholine | amyloid β | p75 neurotrophin receptor | Alzheimer's disease | tau protein | ALZHEIMERS-DISEASE | AMYLOID PRECURSOR PROTEIN | EXACERBATES TAU PATHOLOGY | NICOTINIC ACETYLCHOLINE-RECEPTORS | NEUROSCIENCES | amyloid beta | CLINICAL NEUROLOGY | BASAL FOREBRAIN NEURONS | IN-VIVO | BETA-SECRETASE BACE1 | CENTRAL-NERVOUS-SYSTEM | EXTRACELLULAR TAU | LONG-TERM POTENTIATION | Receptors, Cholinergic - metabolism | Cholinergic Neurons - metabolism | Humans | Basal Ganglia - metabolism | tau Proteins - metabolism | Acetylcholine - metabolism | Cerebral Cortex - metabolism | Synaptic Transmission | Animals | Amyloid beta-Peptides - metabolism | Amyloid beta-Protein Precursor - metabolism | Neural Pathways - metabolism | Neurons - metabolism | Care and treatment | Analysis | Acetylcholine | Glycoproteins | Research | Neurotrophic functions
volume transmission | acetylcholine | amyloid β | p75 neurotrophin receptor | Alzheimer's disease | tau protein | ALZHEIMERS-DISEASE | AMYLOID PRECURSOR PROTEIN | EXACERBATES TAU PATHOLOGY | NICOTINIC ACETYLCHOLINE-RECEPTORS | NEUROSCIENCES | amyloid beta | CLINICAL NEUROLOGY | BASAL FOREBRAIN NEURONS | IN-VIVO | BETA-SECRETASE BACE1 | CENTRAL-NERVOUS-SYSTEM | EXTRACELLULAR TAU | LONG-TERM POTENTIATION | Receptors, Cholinergic - metabolism | Cholinergic Neurons - metabolism | Humans | Basal Ganglia - metabolism | tau Proteins - metabolism | Acetylcholine - metabolism | Cerebral Cortex - metabolism | Synaptic Transmission | Animals | Amyloid beta-Peptides - metabolism | Amyloid beta-Protein Precursor - metabolism | Neural Pathways - metabolism | Neurons - metabolism | Care and treatment | Analysis | Acetylcholine | Glycoproteins | Research | Neurotrophic functions
Journal Article