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1. Full Text Type 2 cytokines: Mechanisms and therapeutic strategies
by Wynn, Thomas A
Nature Reviews Immunology, ISSN 1474-1733, 04/2015, Volume 15, Issue 5, pp. 271 - 282
Type 2 immune responses are defined by the cytokines interleukin-4 (IL-4), IL-5, IL-9 and IL-13, which can either be host protective or have pathogenic... 
ALTERNATIVELY ACTIVATED MACROPHAGES | IL-13 RECEPTOR ALPHA-2 | BRONCHIAL EPITHELIAL-CELLS | REGULATORY T-CELLS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | TUMOR-ASSOCIATED MACROPHAGES | ALLERGIC AIRWAY INFLAMMATION | INNATE LYMPHOID-CELLS | IMMUNOLOGY | HOUSE-DUST MITE | HELMINTH INFECTION PROTECTS | Mast Cells - immunology | Humans | Interleukin-17 - immunology | Interleukin-13 - immunology | Th2 Cells - immunology | Eosinophils - immunology | Interleukin-5 - immunology | Immunity, Innate - immunology | Interleukin-4 - immunology | Basophils - immunology | Adaptive Immunity - immunology | Interleukin-9 - immunology | Cytokines - immunology | Interleukin-4 | B cells | Cytokines | Health aspects | Analysis
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2. Full Text IL-2 family cytokines: new insights into the complex roles of IL-2 as a broad regulator of T helper cell differentiation
by Liao, Wei and Lin, Jian-Xin and Leonard, Warren J
Current Opinion in Immunology, ISSN 0952-7915, 2011, Volume 23, Issue 5, pp. 598 - 604
Highlights ► We examined the role of IL-2 in T helper cell differentiation. ► IL-2 induces its own receptor chains, IL-2Rα and IL-2Rβ. ► Additionally, IL-2... 
Allergy and Immunology | GAMMA | SIGNALING PATHWAYS | ACTIVATION | GENE | CYTOPLASMIC DOMAINS | INTERLEUKIN-2-RECEPTOR BETA-CHAIN | RECEPTOR | HETERODIMERIZATION | IMMUNOLOGY | EXPRESSION | LOCUS | Cell Proliferation | Humans | Cell Death - immunology | X-Linked Combined Immunodeficiency Diseases - genetics | Th2 Cells - immunology | X-Linked Combined Immunodeficiency Diseases - immunology | Th1 Cells - immunology | Immunity | Signal Transduction - immunology | Lymphocyte Activation - immunology | Interleukin-2 - immunology | Killer Cells, Natural - immunology | Receptors, Cytokine - metabolism | Transcription Factors - immunology | Interleukin-2 - metabolism | Gene Expression Regulation - immunology | Immunologic Factors - immunology | Mice, Transgenic | Th2 Cells - cytology | Signal Transduction - genetics | Transcription Factors - genetics | Cell Differentiation - immunology | Killer Cells, Natural - cytology | Animals | Receptors, Cytokine - immunology | Mice | Th1 Cells - cytology | Immunologic Factors - metabolism | Cytokines
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3. Full Text Cytokine activation induces human memory-like NK cells
by Romee, Rizwan and Schneider, Stephanie E and Leong, Jeffrey W and Chase, Julie M and Keppel, Catherine R and Sullivan, Ryan P and Cooper, Megan A and Fehniger, Todd A
Blood, ISSN 0006-4971, 12/2012, Volume 120, Issue 24, pp. 4751 - 4760
Natural killer (NK) cells are lymphocytes that play an important role in the immune response to infection and malignancy. Recent studies in mice have shown... 
INNATE | NATURAL-KILLER-CELLS | INFECTION | HEMATOLOGY | EXPANSION | Receptors, Interleukin-12 - immunology | Antigens, CD - immunology | Interleukin-18 - immunology | Humans | Lectins, C-Type - immunology | Interferon-gamma - metabolism | Interleukin-15 - immunology | Antigens, CD - metabolism | NK Cell Lectin-Like Receptor Subfamily D - immunology | Lectins, C-Type - metabolism | NK Cell Lectin-Like Receptor Subfamily C - immunology | Lymphocyte Activation - immunology | Flow Cytometry | Time Factors | Killer Cells, Natural - immunology | Receptors, Interleukin-12 - genetics | Interferon-gamma - genetics | CD56 Antigen - immunology | Immunologic Memory - immunology | Cytokines - immunology | Interleukin-12 - pharmacology | Receptors, Interleukin-18 - metabolism | CD56 Antigen - metabolism | NK Cell Lectin-Like Receptor Subfamily C - metabolism | Antigens, Differentiation, T-Lymphocyte - immunology | Antigens, Differentiation, T-Lymphocyte - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Interleukin-15 - pharmacology | Receptors, Interleukin-12 - metabolism | Lymphocyte Activation - drug effects | Interferon-gamma - immunology | Interleukin-12 - immunology | K562 Cells | Receptors, Interleukin-18 - genetics | Receptors, Interleukin-18 - immunology | Cell Proliferation - drug effects | Interleukin-18 - pharmacology | Killer Cells, Natural - drug effects | Killer Cells, Natural - metabolism | NK Cell Lectin-Like Receptor Subfamily D - metabolism | Cytokines - pharmacology | Immunobiology
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4. Full Text Tissue-Specific Distribution of iNKT Cells Impacts Their Cytokine Response
by Lee, You Jeong and Wang, Haiguang and Starrett, Gabriel J and Phuong, Vanessa and Jameson, Stephen C and Hogquist, Kristin A
Immunity, ISSN 1074-7613, 09/2015, Volume 43, Issue 3, pp. 566 - 578
Three subsets of invariant natural killer T (iNKT) cells have been identified, NKT1, NKT2, and NKT17, which produce distinct cytokines when stimulated, but... 
ACTIVATION | HOMEOSTASIS | ALPHA-GALACTOSYLCERAMIDE | INVARIANT NKT CELLS | KILLER T-CELLS | ORPHAN RECEPTOR | IMMUNOLOGY | IDENTIFICATION | EXPRESSION | PROGRAM | CUTTING EDGE | Organ Specificity - drug effects | T-Lymphocyte Subsets - immunology | Spleen - immunology | T-Lymphocyte Subsets - cytology | Thymocytes - metabolism | Interferon-gamma - metabolism | Galactosylceramides - pharmacology | Lung - cytology | Organ Specificity - immunology | Liver - immunology | Lymphocyte Activation - immunology | Flow Cytometry | Natural Killer T-Cells - metabolism | Lung - metabolism | Thymus Gland - metabolism | Cytokines - immunology | Cytokines - metabolism | Interleukin-4 - metabolism | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Thymus Gland - cytology | Lymph Nodes - metabolism | Mice, Transgenic | Spleen - cytology | Lymph Nodes - immunology | Lymph Nodes - cytology | Natural Killer T-Cells - classification | Mice, Knockout | Thymocytes - immunology | Interleukin-4 - immunology | Animals | Galactosylceramides - immunology | Spleen - metabolism | Lymphocyte Activation - drug effects | T-Lymphocyte Subsets - metabolism | Interferon-gamma - immunology | Fluorescent Antibody Technique | Mice, Inbred NOD | Thymus Gland - immunology | Liver - cytology | Mice, Inbred BALB C | Lung - immunology | Natural Killer T-Cells - immunology | Cytokines | Biological response modifiers | T cells | Studies | Flow cytometry | Transcription factors | Statistical analysis | Lymphocytes | Rodents | Lipids | T cell receptors | Software | Experiments
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5. Full Text Cholesterol crystals induce complement-dependent inflammasome activation and cytokine release
by Samstad, Eivind O and Niyonzima, Nathalie and Nymo, Stig and Aune, Marie H and Ryan, Liv and Bakke, Siril S and Lappegård, Knut T and Brekke, Ole-Lars and Lambris, John D and Damås, Jan K and Latz, Eicke and Mollnes, Tom E and Espevik, Terje
Journal of Immunology, ISSN 0022-1767, 03/2014, Volume 192, Issue 6, pp. 2837 - 2845
Inflammation is associated with development of atherosclerosis, and cholesterol crystals (CC) have long been recognized as a hallmark of atherosclerotic... 
ATHEROSCLEROTIC LESIONS | TUMOR-NECROSIS-FACTOR | C5A RECEPTOR | MEMBRANE ATTACK | STERILE INFLAMMATION | EPITHELIAL-CELLS | CARDIOVASCULAR EVENTS | INTERLEUKIN-1-BETA | OXIDATIVE BURST | ANAPHYLATOXINS | IMMUNOLOGY | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | Complement C1q - immunology | Humans | Complement Pathway, Classical - drug effects | Complement C5a - metabolism | Complement System Proteins - immunology | Caspase 1 - metabolism | Monocytes - metabolism | Caspase 1 - immunology | Macrophage-1 Antigen - immunology | Monocytes - immunology | Interleukin-1beta - genetics | Complement Activation - immunology | Complement C1q - metabolism | Complement System Proteins - metabolism | Inflammasomes - drug effects | Reactive Oxygen Species - immunology | Complement C5 - immunology | Tumor Necrosis Factor-alpha - immunology | Macrophage-1 Antigen - metabolism | Complement Activation - drug effects | Cytokines - immunology | Interleukin-1beta - secretion | Complement C5 - metabolism | Tumor Necrosis Factor-alpha - secretion | Cells, Cultured | Cytokines - secretion | Phagocytosis - immunology | Interleukin-1beta - immunology | Cholesterol - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Monocytes - drug effects | Complement Pathway, Classical - immunology | Microscopy, Confocal | Complement Pathway, Alternative - drug effects | Complement Pathway, Alternative - immunology | Inflammasomes - immunology | Cholesterol - pharmacology | Complement C5a - immunology
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6. Full Text Cytokines
by Hiscott, John and Ware, Carl
Current Opinion in Immunology, ISSN 0952-7915, 2011, Volume 23, Issue 5, pp. 561 - 563
Allergy and Immunology | Signal Transduction - immunology | Receptors, Cytokine - immunology | Immunity, Innate | Humans | Cytokines - immunology
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7. Full Text Epithelial-Derived Cytokines in Asthma
by Mitchell, Patrick D and O’Byrne, Paul M
Chest, ISSN 0012-3692, 06/2017, Volume 151, Issue 6, pp. 1338 - 1344
The interaction between the airway epithelium and the inhaled environment is crucial to understanding the pathobiology of asthma. Several studies have... 
airway inflammation | asthma | cytokines | EFFECTOR FUNCTION | INNATE LYMPHOID-CELLS | RECEPTOR ST2 | IL-33 | CD8(+) T-CELLS | THYMIC STROMAL LYMPHOPOIETIN | RESPIRATORY SYSTEM | ALLERGIC-ASTHMA | IN-VIVO | EXPRESSION | CRITICAL CARE MEDICINE | Immunity, Humoral - immunology | Humans | Interleukin-17 - immunology | Inflammation - immunology | Molecular Targeted Therapy | Respiratory Hypersensitivity - immunology | Interleukin-33 - immunology | Immunity, Innate - immunology | Animals | Allergens - immunology | Epithelial Cells - immunology | Respiratory Mucosa - immunology | Asthma - immunology | Alarmins - immunology | Cytokines - immunology | Care and treatment | Research | Cytokines | Asthma
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8. Full Text Peripheral CD4+ T‐cell differentiation regulated by networks of cytokines and transcription factors
by Zhu, Jinfang and Paul, William E
Immunological Reviews, ISSN 0105-2896, 11/2010, Volume 238, Issue 1, pp. 247 - 262
CD4+ T cells, also known as T‐helper (Th) cells, play an important role in orchestrating adaptive immune responses to various infectious agents. They are also... 
T‐helper cells | cytokines | transcription factors | T‐cell differentiation | Transcription factors | T-cell differentiation | T-helper cells | Cytokines | TGF-BETA | TH2 CELLS | ROR-GAMMA-T | CD4(+) T-CELLS | IMMUNOLOGY | HYPER-IGE SYNDROME | LOCUS-CONTROL REGION | GROWTH-FACTOR-BETA | FOLLICULAR-HELPER-CELLS | IN-VIVO | GENE-EXPRESSION | CD4-Positive T-Lymphocytes - immunology | Receptor Cross-Talk - immunology | T-Lymphocyte Subsets - immunology | Cell Lineage | Signal Transduction - immunology | Feedback, Physiological | Animals | Humans | Cell Differentiation | Gene Regulatory Networks - immunology | Transcription Factors - immunology | Cytokines - immunology | Cell activation | Helper cells | Allergic diseases | Lymphocytes T | Differentiation | double prime T-cell receptor | CD4 antigen | T helper cells | T cell differentiation
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9. Full Text Proinflammatory cytokine signaling required for the generation of natural killer cell memory
by Sun, Joseph C and Madera, Sharline and Bezman, Natalie A and Beilke, Joshua N and Kaplan, Mark H and Lanier, Lewis L
Journal of Experimental Medicine, ISSN 0022-1007, 05/2012, Volume 209, Issue 5, pp. 947 - 954
Although natural killer (NK) cells are classified as innate immune cells, recent studies demonstrate that NK cells can become long-lived memory cells and... 
ADAPTIVE IMMUNITY | MEDICINE, RESEARCH & EXPERIMENTAL | RESPONSES | IFN-ALPHA-BETA | INNATE | MICE | IMMUNOLOGY | IL-12 | Mice, Inbred C57BL | Adoptive Transfer | Killer Cells, Natural - physiology | Mice, Knockout | Muromegalovirus - immunology | Signal Transduction - immunology | Animals | Flow Cytometry | Interleukin-12 - immunology | Statistics, Nonparametric | Killer Cells, Natural - immunology | Receptors, Interleukin-12 - genetics | Mice | STAT4 Transcription Factor - immunology | Immunologic Memory - immunology | Cytokines - immunology | Herpesviridae Infections - immunology | Brief Definitive Report
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10. Full Text T-helper-1-cell cytokines drive cancer into senescence
by Braumüller, Heidi and Wieder, Thomas and Brenner, Ellen and Aßmann, Sonja and Hahn, Matthias and Alkhaled, Mohammed and Schilbach, Karin and Essmann, Frank and Kneilling, Manfred and Griessinger, Christoph and Ranta, Felicia and Ullrich, Susanne and Mocikat, Ralph and Braungart, Kilian and Mehra, Tarun and Fehrenbacher, Birgit and Berdel, Julia and Niessner, Heike and Meier, Friedegund and Van Den Broek, Maries and Häring, Hans-Ulrich and Handgretinger, Rupert and Quintanilla-Martinez, Leticia and Fend, Falko and Pesic, Marina and Bauer, Jürgen and Zender, Lars and Schaller, Martin and Schulze-Osthoff, Klaus and Röcken, Martin
Nature, ISSN 0028-0836, 02/2013, Volume 494, Issue 7437, pp. 361 - 365
Cancer control by adaptive immunity involves a number of defined death(1-8) and clearance(9-11) mechanisms. However, efficient inhibition of exponential cancer... 
REGRESSION | PATHWAY | MULTIDISCIPLINARY SCIENCES | TUMOR | SURVEILLANCE | CD4(+) T-CELLS | ONCOGENE-INDUCED SENESCENCE | P53 | Cell Proliferation | Humans | Male | Receptors, Tumor Necrosis Factor, Type I - metabolism | Th1 Cells - immunology | Cellular Senescence - immunology | STAT1 Transcription Factor - metabolism | Time Factors | Tumor Necrosis Factor-alpha - immunology | Female | Tumor Cells, Cultured | Retinoblastoma Protein - chemistry | Cytokines - immunology | Disease Models, Animal | Oncogenes - genetics | Cyclin-Dependent Kinase Inhibitor p16 - deficiency | Antigens, Polyomavirus Transforming - genetics | Antigens, Polyomavirus Transforming - metabolism | Retinoblastoma Protein - metabolism | Tumor Suppressor Protein p53 - metabolism | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Mice, Transgenic | Mice, SCID | Phosphoserine - metabolism | Disease Progression | Animals | Cell Cycle | Neoplasms - immunology | Interferon-gamma - immunology | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Mice, Inbred NOD | Mice | Neoplasms - pathology | Care and treatment | Cytokines | Cellular control mechanisms | Cancer cells | Research | Observations | Testing | Cancer | Proteins | Medical research | Incorporation | Cytotoxicity | Arrests | Chemokines | Tumors | Apoptosis
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