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PloS one, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, pp. e65860 - e65860
Dopamine (DA), a neurotransmitter in the nervous system, has been shown to modulate immune function... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Enzyme Activators - pharmacology | Phosphorylation | Receptors, Dopamine D3 - metabolism | Receptors, Dopamine D4 - genetics | Receptors, Dopamine D3 - genetics | Isoquinolines - pharmacology | Killer Cells, Natural - immunology | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Cyclic AMP - metabolism | Second Messenger Systems | Cyclic AMP-Dependent Protein Kinases - metabolism | Dopamine Agonists - pharmacology | Gene Expression | Colforsin - pharmacology | Adenylyl Cyclases - metabolism | Benzazepines - pharmacology | Sulfonamides - pharmacology | Mice, Inbred ICR | Animals | Dopamine Antagonists - pharmacology | Cyclic AMP Response Element-Binding Protein - metabolism | 2,3,4,5-Tetrahydro-7,8-dihydroxy-1-phenyl-1H-3-benzazepine - pharmacology | Cell Line, Tumor | Receptors, Dopamine D5 - metabolism | Receptors, Dopamine D4 - metabolism | Protein Processing, Post-Translational | Quinpirole - pharmacology | Receptors, Dopamine D5 - genetics | Killer Cells, Natural - metabolism | Cytotoxicity, Immunologic | Phenols | Cyclic adenylic acid | Dopamine | Lymphocytes | Dopamine receptors | Drugs | Flow cytometry | Toxicity | Cytotoxicity | Nervous system | Lymphocytes T | mRNA | Haloperidol | Kinases | Dopamine D4 receptors | Immunosuppressive agents | Proteins | Signal transduction | Receptors | Cell activation | Modulation | Clonal selection | Physiology | Natural killer cells | Dopamine D2 receptors | Spleen | Dopamine D5 receptors | Adenosine | Immune response | Cytokines | Dopamine D3 receptors | T cell receptors | Cyclic AMP response element-binding protein | Gene expression | Lymphoma | Medicine | Cytometry | Signaling | Dopamine D1 receptors | Forskolin | Receptor mechanisms | Laboratory animals | Index Medicus
Journal Article
Science (American Association for the Advancement of Science), ISSN 1095-9203, 09/2017, Volume 357, Issue 6357, pp. 1255 - 1261
... enzymatic activity, lysosomal dysfunction, and a-synuclein accumulation. This toxic cascade was observed in human, but not in mouse, PD neurons at least in part because of species-specific differences in dopamine metabolism... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Mitochondria - enzymology | Mesencephalon - metabolism | Humans | Protein Deglycase DJ-1 - genetics | Substantia Nigra - metabolism | Melanins - metabolism | Glucosylceramidase - deficiency | Lysosomes - metabolism | Dopaminergic Neurons - metabolism | Tacrolimus - pharmacology | Parkinson Disease - metabolism | Dopamine - metabolism | Disease Models, Animal | Substantia Nigra - enzymology | Cell Line | Calcineurin Inhibitors - pharmacology | Oxidation-Reduction | Mesencephalon - enzymology | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - drug effects | Parkinson Disease - genetics | Mice, Knockout | Animals | Parkinson Disease - enzymology | Mice | Oxidative Stress - drug effects | alpha-Synuclein - metabolism | Oxidation-reduction reaction | Development and progression | Mitochondria | Dopamine | Parkinson's disease | Health aspects | Brain | Energy metabolism | Animal models | Target recognition | Mesencephalon | Pathogenesis | Substantia nigra | Parkinsons disease | Lysosomes | Synuclein | Accumulation | Pathways | Enzymatic activity | Neurodegeneration | Rodents | Oxidation | Degeneration | Species | Movement disorders | Dopamine receptors | Neurodegenerative diseases | Neurons | Medical treatment | Metabolism | Patients | Glucosylceramidase | Index Medicus
Journal Article
10.