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Oncogene, ISSN 1476-5594, 2014, Volume 34, Issue 29, pp. 3760 - 3769
.... We show that estrogen, acting via estrogen receptor alpha (ER alpha), induces rapid anticipatory activation of the UPR, resulting in increased production... 
REGULATED GENE-EXPRESSION | MICROARRAY | TUMOR-DEVELOPMENT | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | KINASE PERK | ENDOPLASMIC-RETICULUM STRESS | CHEMOTHERAPY | CELL BIOLOGY | B-CELLS | ONCOLOGY | GROWTH | GENETICS & HEREDITY | XBP-1 | Estrogens - pharmacology | Oligonucleotide Array Sequence Analysis | Humans | Transplantation, Heterologous | Gene Expression Profiling | Breast Neoplasms - metabolism | Heat-Shock Proteins - genetics | MCF-7 Cells | RNA Interference | Antineoplastic Agents, Hormonal - therapeutic use | Estrogen Receptor alpha - metabolism | Female | Gene Expression Regulation, Neoplastic - drug effects | Estradiol - pharmacology | Unfolded Protein Response - drug effects | Cell Proliferation - genetics | Ovariectomy | Unfolded Protein Response - genetics | Heat-Shock Proteins - metabolism | Kaplan-Meier Estimate | Reverse Transcriptase Polymerase Chain Reaction | Breast Neoplasms - drug therapy | Blotting, Western | Microscopy, Confocal | Animals | Breast Neoplasms - genetics | Estrogen Receptor alpha - genetics | Mice, Nude | Tamoxifen - therapeutic use | Cell Line, Tumor | Cell Proliferation - drug effects | Cell proliferation | Cell culture | Calcium channels | Cell survival | Estrogen | Estrogen receptors | 17β-Estradiol | Breast cancer | siRNA | Chaperones | Tamoxifen | Cytosol | Ovarian cancer | Phospholipase C | Proteins | Phospholipase | Cell activation | Cell growth | Protein folding | Xenografts | Hormone replacement therapy | Endoplasmic reticulum | Inositol 1,4,5-trisphosphate receptors | Apoptosis | Estrogen receptor α | breast cancer | ovarian cancer | unfolded protein response (UPR)
Journal Article
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 8/2005, Volume 102, Issue 33, pp. 11651 - 11656
Journal Article
Fertility and sterility, ISSN 0015-0282, 2012, Volume 98, Issue 5, pp. 1200 - 1208
Objective To analyze the expression of estrogen receptors α and β as well as their target genes implicated in proliferation, c-myc, cyclin D1, and GREB1, in the endometrium of women with or without endometriosis... 
Internal Medicine | Obstetrics and Gynecology | Endometriosis | estrogen-regulated genes | proliferation | estrogen receptors | TRANSCRIPTIONAL ACTIVATION | UTERINE ENDOMETRIUM | PROGESTERONE-RECEPTORS | EUTOPIC ENDOMETRIUM | OBSTETRICS & GYNECOLOGY | STEROIDOGENIC-FACTOR-I | REPRODUCTIVE BIOLOGY | OVARIAN ENDOMETRIOSIS | IMMUNOHISTOCHEMICAL ANALYSIS | ECTOPIC ENDOMETRIUM | PROTEIN EXPRESSION | RESPONSE ELEMENTS | Immunohistochemistry | Proto-Oncogene Proteins c-myc - analysis | Cell Proliferation | Endometriosis - genetics | Humans | Middle Aged | Estrogen Receptor beta - analysis | RNA, Messenger - analysis | Case-Control Studies | Young Adult | Cyclin D1 - analysis | Peritoneal Diseases - pathology | Estrogen Receptor beta - genetics | Choristoma - metabolism | Estrogen Receptor alpha - analysis | Adult | Female | Choristoma - genetics | Neoplasm Proteins - genetics | Real-Time Polymerase Chain Reaction | Choristoma - surgery | Endometrium | Endometriosis - surgery | Gene Expression Regulation | Peritoneal Diseases - surgery | Endometriosis - pathology | Reverse Transcriptase Polymerase Chain Reaction | Peritoneal Diseases - genetics | Switzerland | Estrogen Receptor alpha - genetics | Cyclin D1 - genetics | Biopsy | Endometriosis - metabolism | Neoplasm Proteins - analysis | Proto-Oncogene Proteins c-myc - genetics | Choristoma - pathology | Peritoneal Diseases - metabolism | Germany | Analysis | Genes | Estrogen | Medical genetics | Phenols | Universities and colleges
Journal Article
International journal of cancer, ISSN 0020-7136, 2019, Volume 144, Issue 4, pp. 730 - 740
Journal Article
Oncogene, ISSN 1476-5594, 2014, Volume 34, Issue 4, pp. 506 - 515
Progesterone and estrogen are important drivers of breast cancer proliferation. Herein, we probed estrogen receptor-a (ER... 
breast cancer | cathepsin-D | estrogen receptor | progesterone receptor | IGF1R | PELP1 | HORMONE-RECEPTORS | ACTIVATION | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | TAMOXIFEN RESISTANCE | KINASE | CELL BIOLOGY | ONCOLOGY | PATHWAY | GROWTH | GENETICS & HEREDITY | ADJUVANT TAMOXIFEN | EXPRESSION | C-SRC | Protein Structure, Tertiary | Insulin-Like Growth Factor I - pharmacology | Transcription Factors - physiology | Humans | Co-Repressor Proteins - physiology | DNA - metabolism | Breast Neoplasms - drug therapy | Receptor Cross-Talk - physiology | MCF-7 Cells | Receptors, Progesterone - physiology | Breast Neoplasms - pathology | Tamoxifen - therapeutic use | Cathepsin D - genetics | Receptor, IGF Type 1 - physiology | Co-Repressor Proteins - analysis | Phosphatidylinositol 3-Kinases - physiology | Female | Transcription, Genetic | Cell Proliferation - drug effects | Transcription Factors - analysis | Estrogen Receptor alpha - physiology | Estradiol - pharmacology | Receptors, Progesterone - chemistry | Receptors | Estrogen | Physiological aspects | Development and progression | Breast cancer | Genetic aspects | Research | Progesterone | Neurons | Cells | Tyrosine | Phosphorylation | Transcription | Insulin-like growth factor I | Estrogen receptors | Proline | Genomes | Tumor cell lines | Leucine | Tamoxifen | Kinases | Insulin | Estradiol | Lysates | Onapristone | DNA microarrays | Progestin | Hormone replacement therapy | Protein-tyrosine kinase | Progesterone Receptor | Cathepsin D | Breast Cancer | Estrogen Receptor
Journal Article
Nature (London), ISSN 1476-4687, 2009, Volume 462, Issue 7269, pp. 58 - 64
Journal Article