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Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 07/2014, Volume 34, Issue 7, pp. 1477 - 1485
OBJECTIVE—17β-Estradiol (E2) offers cardiovascular protection in young female animals and postmenopausal women. In contrast, randomized trials of menopausal... 
estrogen | vascular system injuries | aging | c-reactive protein | INJURED ARTERIES | CAROTID-ARTERY | NEOINTIMA FORMATION | POSTMENOPAUSAL HORMONE-THERAPY | RANDOMIZED CONTROLLED-TRIAL | CARDIOVASCULAR-DISEASE | REPLACEMENT THERAPY | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | TRANSGENIC MICE | Age Factors | Muscle, Smooth, Vascular - metabolism | Receptors, G-Protein-Coupled - metabolism | Humans | Muscle, Smooth, Vascular - immunology | RNA, Messenger - metabolism | Estrogen Receptor beta - metabolism | Inflammation - metabolism | C-Reactive Protein - metabolism | Estrogen Receptor alpha - agonists | Estrogen Receptor beta - agonists | Estrogen Receptor beta - genetics | Neointima | Inflammation Mediators - metabolism | Estrogen Receptor alpha - metabolism | Female | Myocytes, Smooth Muscle - drug effects | Receptors, G-Protein-Coupled - drug effects | Myocytes, Smooth Muscle - metabolism | Carotid Artery Injuries - metabolism | Estradiol - pharmacology | Macrophages - immunology | Carotid Artery Injuries - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Receptors, Estrogen | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Mice, Transgenic | C-Reactive Protein - genetics | Inflammation - immunology | Mice, Knockout | Macrophages - metabolism | Animals | Estrogen Receptor alpha - genetics | Myocytes, Smooth Muscle - immunology | Macrophages - drug effects | Carotid Artery Injuries - immunology | Inflammation - prevention & control | Mice | vascular injury | vascular inflammation | Estrogen
Journal Article
Cell (Cambridge), ISSN 0092-8674, 2007, Volume 130, Issue 5, pp. 811 - 823
Estrogen prevents osteoporotic bone loss by attenuating bone resorption; however, the molecular basis for this is... 
HUMDISEASE | DEVBIO | SIGNALING | ANDROGEN RECEPTOR | APOPTOSIS | RESORPTION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MECHANISMS | OSTEOPETROSIS | MICE LACKING | CELL BIOLOGY | Fas Ligand Protein - metabolism | Male | fas Receptor - metabolism | Bone Remodeling - drug effects | Selective Estrogen Receptor Modulators - therapeutic use | Estrogen Receptor alpha - metabolism | Female | Integrases - metabolism | Mice, Inbred DBA | Cell Differentiation | Estradiol - metabolism | Osteoclasts - pathology | Bone Density | Cathepsin K | Ovariectomy | Signal Transduction | Bone Diseases, Metabolic - prevention & control | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Osteoclasts - metabolism | Estrogen Receptor alpha - deficiency | Mice, Knockout | Bone Diseases, Metabolic - metabolism | Estrogen Receptor alpha - drug effects | Phenotype | Animals | Estrogen Receptor alpha - genetics | Cathepsins - genetics | Cathepsins - metabolism | Tamoxifen - therapeutic use | Tamoxifen - pharmacology | Mice | Bone Diseases, Metabolic - physiopathology | Fas Ligand Protein - genetics | Integrases - genetics | Selective Estrogen Receptor Modulators - pharmacology | Osteoclasts - drug effects | Apoptosis | Cathepsins | Antigens, CD95 | Estradiol | Life Sciences | Estrogen Receptor alpha | Biochemistry, Molecular Biology | Tamoxifen | Bone Diseases, Metabolic | Integrases | Fas Ligand Protein | Selective Estrogen Receptor Modulators | Osteoclasts | Molecular biology | Bone Remodeling
Journal Article
Journal Article
Environmental health perspectives, ISSN 1552-9924, 2016, Volume 124, Issue 7, pp. 1023 - 1033
Journal Article
European journal of cancer (1990), ISSN 0959-8049, 2014, Volume 50, Issue 2, pp. 457 - 468
Abstract Purpose Our publications demonstrate that physiological concentrations of oestrogen (E2... 
Hematology, Oncology and Palliative Medicine | Epithelial–mesenchymal transition | c-Src | Oestrogen | Breast cancer | Apoptosis | Epithelial-mesenchymal transition | TYROSINE KINASE | TAMOXIFEN | E-CADHERIN | ATHYMIC MICE | RESISTANT | ONCOLOGY | DEPRIVATION | ESTRADIOL | GENE-EXPRESSION | POSTMENOPAUSAL WOMEN | FACTOR-I RECEPTOR | Receptor, IGF Type 1 - metabolism | Estradiol - analogs & derivatives | Receptor, IGF Type 1 - antagonists & inhibitors | Receptors, Estrogen - metabolism | Estrogens - pharmacology | Cadherins - metabolism | Apoptosis - drug effects | Humans | Immunoblotting | Receptors, Progesterone - genetics | Breast Neoplasms - metabolism | Receptors, Progesterone - metabolism | MCF-7 Cells | src-Family Kinases - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Snail Family Transcription Factors | Estradiol - pharmacology | Receptors, Estrogen - genetics | src-Family Kinases - antagonists & inhibitors | Nuclear Proteins - metabolism | Tyrphostins - pharmacology | Pyrimidines - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Receptor, IGF Type 1 - genetics | Transcription Factors - metabolism | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Cell Proliferation - drug effects | Twist-Related Protein 1 - metabolism | Mitogen-Activated Protein Kinases - metabolism | Cancer cells | Estrogen | apoptosis | breast cancer | estrogen | epithelial-mesenchymal transition
Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 2006, Volume 52, Issue 2, pp. 271 - 280
The mechanisms through which estrogen regulates gonadotropin-releasing hormone (GnRH) neurons to control mammalian ovulation are unknown... 
MOLNEURO | SIGNALING | NERVOUS-SYSTEM | LHRH NEURONS | FEMALE RAT | PSEUDORABIES VIRUS | SURGE | DEPENDENT PROTEIN-KINASE | ALPHA | PREOPTIC AREA | RAT-BRAIN | NEUROSCIENCES | LUTEINIZING-HORMONE | Neural Pathways - drug effects | Estrous Cycle - physiology | Hypothalamus, Middle - secretion | Hypothalamus, Middle - drug effects | Luteinizing Hormone - metabolism | Hypothalamo-Hypophyseal System - secretion | Fertility - physiology | Estrogen Receptor beta - metabolism | Estrogen Receptor alpha - metabolism | Female | Neurons - secretion | Neurons - metabolism | Estrogens - metabolism | Neurons - drug effects | Luteinizing Hormone - secretion | Gonadotropin-Releasing Hormone - secretion | Hypothalamo-Hypophyseal System - drug effects | Neural Pathways - secretion | Estradiol Congeners - pharmacology | Herpesvirus 1, Suid - physiology | Mice, Transgenic | Hypothalamus, Middle - metabolism | Hypothalamo-Hypophyseal System - metabolism | Estrogens - agonists | Estrous Cycle - drug effects | Animals | Estrogen Receptor alpha - genetics | Gonadotropin-Releasing Hormone - metabolism | Neural Pathways - metabolism | Mice | Feedback, Physiological - physiology | Cell research | Neurons | Oncology, Experimental | Estrogen | Physiological aspects | Phenols | Hormones | Universities and colleges | Research | Cancer | Brain research | Fertility | Rodents | Ligands | Mammals | Labeling | Females
Journal Article
Experimental cell research, ISSN 0014-4827, 2009, Volume 315, Issue 7, pp. 1287 - 1301
Journal Article