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Cancer Cell, ISSN 1535-6108, 06/2012, Volume 21, Issue 6, pp. 822 - 835
Cancer-associated inflammation is thought to be a barrier to immune surveillance, particularly in pancreatic ductal adenocarcinoma (PDA). Gr-1 CD11b cells are... 
OVEREXPRESSION | MICROENVIRONMENT | ONCOLOGY | DUCTAL ADENOCARCINOMA | INTERLEUKIN-1-BETA | MICE | SUPPRESSOR-CELLS | INDUCTION | DIFFERENTIATION | MAMMARY CARCINOMAS | MOUSE MODELS | CELL BIOLOGY | Immunohistochemistry | Adenocarcinoma - pathology | CD8-Positive T-Lymphocytes - pathology | Pancreatic Neoplasms - metabolism | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Humans | Carcinoma, Pancreatic Ductal - metabolism | Inflammation - metabolism | Adenocarcinoma - metabolism | RNA Interference | T-Lymphocytes - metabolism | Myeloid Cells - immunology | CD8-Positive T-Lymphocytes - metabolism | T-Lymphocytes - pathology | Carcinoma, Pancreatic Ductal - immunology | CD11b Antigen - immunology | Adenocarcinoma - immunology | Pancreatic Neoplasms - pathology | Receptors, Chemokine - metabolism | Mice, Transgenic | Inflammation - immunology | Receptors, Chemokine - immunology | Carcinoma, Pancreatic Ductal - pathology | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Mice, Knockout | Tumor Microenvironment - immunology | Models, Immunological | Animals | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Pancreatic Neoplasms - immunology | Cell Line, Tumor | Myeloid Cells - metabolism | T-Lymphocytes - immunology | Mice | CD11b Antigen - metabolism | Myeloid Cells - pathology | CD8-Positive T-Lymphocytes - immunology | Medical colleges | Analysis | Pancreatic cancer | Oncology, Experimental | Genetically modified organisms | Inflammation | Macrophage colony stimulating factor | Research | T cells | Tumors | Cancer | granulocyte-macrophage colony stimulating factor | myeloid cells | T lymphocyte | carcinoma | pancreas
Journal Article
Blood, ISSN 0006-4971, 2013, Volume 122, Issue 24, pp. 3918 - 3928
Journal Article
Developmental and Comparative Immunology, ISSN 0145-305X, 2004, Volume 28, Issue 5, pp. 509 - 554
The colony-stimulating factors (CSFs) are a group of cytokines central to the hematopoiesis of blood cells, the modulation of their functional responses, as... 
Signal transduction | Function | GM-CSF | Multi-CSF/IL-3 | Colony-stimulating factors | Proliferation | Differentiation | M-CSF/CSF-1 | G-CSF | Hemopoiesis/hematopoiesis | Y-PSEUDOAUTOSOMAL REGION | FMS PROTO-ONCOGENE | differentiation | hemopoiesis/hematopoiesis | proliferation | signal transduction | HUMAN-ENDOTHELIAL CELLS | SEVERE CONGENITAL NEUTROPENIA | BONE-MARROW-CELLS | COMMON BETA-SUBUNIT | IMMUNOLOGY | HEMATOPOIETIC GROWTH-FACTORS | TUMOR-NECROSIS-FACTOR | FISHERIES | ZOOLOGY | function | MONOCYTE-DERIVED MACROPHAGES | GM-CSF RECEPTOR | multi-CSF/IL-3 | colony-stimulating factors | Granulocyte Colony-Stimulating Factor - physiology | Myelopoiesis - physiology | Signal Transduction | Humans | Receptors, Interleukin-3 - genetics | Receptor, Macrophage Colony-Stimulating Factor - genetics | Receptors, Granulocyte Colony-Stimulating Factor - genetics | Granulocyte-Macrophage Colony-Stimulating Factor - physiology | Colony-Stimulating Factors - genetics | Interleukin-3 - genetics | Receptors, Granulocyte Colony-Stimulating Factor - physiology | Granulocyte Colony-Stimulating Factor - genetics | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Animals | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - physiology | Receptor, Macrophage Colony-Stimulating Factor - physiology | Colony-Stimulating Factors - physiology | Macrophage Colony-Stimulating Factor - genetics | Macrophage Colony-Stimulating Factor - physiology | Interleukin-3 - physiology | Receptors, Interleukin-3 - physiology
Journal Article
Journal Article
Blood, ISSN 0006-4971, 02/2017, Volume 129, Issue 6, pp. 667 - 679
The genetic landscape of classicalmyelopro-liferative neoplasm (MPN) is in large part elucidated. The MPN-restricted driver mutations, including those in JAK2,... 
POLYCYTHEMIA-VERA | ACTIVATING MUTATION | CLONAL HEMATOPOIESIS | ACQUIRED UNIPARENTAL DISOMY | JAK2 V617F MUTATION | ACUTE MYELOID-LEUKEMIA | ESSENTIAL THROMBOCYTHEMIA | HEMATOLOGY | CHRONIC MYELOMONOCYTIC LEUKEMIA | THROMBOPOIETIN RECEPTOR | DISEASE PROGRESSION | Thrombocythemia, Essential - physiopathology | Epigenesis, Genetic | Humans | Gene Expression Regulation, Neoplastic | STAT Transcription Factors - metabolism | Polycythemia Vera - genetics | Receptors, Granulocyte Colony-Stimulating Factor - genetics | Thrombocythemia, Essential - genetics | Calreticulin - genetics | Receptors, Erythropoietin - genetics | DNA (Cytosine-5-)-Methyltransferases - metabolism | DNA-Binding Proteins - metabolism | Polycythemia Vera - metabolism | Janus Kinase 2 - metabolism | Calreticulin - metabolism | Receptors, Granulocyte Colony-Stimulating Factor - metabolism | Polycythemia Vera - physiopathology | Primary Myelofibrosis - metabolism | Repressor Proteins - metabolism | Proto-Oncogene Proteins - metabolism | Enhancer of Zeste Homolog 2 Protein - genetics | Enhancer of Zeste Homolog 2 Protein - metabolism | Primary Myelofibrosis - physiopathology | Janus Kinase 2 - genetics | Repressor Proteins - genetics | Proto-Oncogene Proteins - genetics | DNA-Binding Proteins - genetics | Receptors, Thrombopoietin - metabolism | Disease Progression | DNA (Cytosine-5-)-Methyltransferases - genetics | Receptors, Thrombopoietin - genetics | Primary Myelofibrosis - genetics | STAT Transcription Factors - genetics | Mutation | Receptors, Erythropoietin - metabolism | Thrombocythemia, Essential - metabolism
Journal Article
Hepatology, ISSN 0270-9139, 01/2012, Volume 55, Issue 1, pp. 209 - 221
Journal Article