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Journal of immunology (Baltimore, Md, ISSN 0022-1767, 2013, Volume 191, Issue 1, pp. 353 - 362
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2014, Volume 193, Issue 4, pp. 1954 - 1965
Canonical neutrophil antimicrobial effector mechanisms, such as degranulation, production of reactive oxygen species, and release of neutrophil extracellular... 
MAMMALIAN TARGET | NADPH OXIDASE | NETTING NEUTROPHILS | SIGNALING PATHWAYS | RESPIRATORY BURST | SYNOVIAL-FLUID | SUPEROXIDE-PRODUCTION | IN-VIVO | IMMUNOLOGY | RECEPTOR-III | CELL-DEATH | Reactive Oxygen Species - metabolism | Nitriles - pharmacology | Humans | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Intracellular Signaling Peptides and Proteins - immunology | Macrophage-1 Antigen - immunology | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Receptors, IgG - antagonists & inhibitors | Protein-Tyrosine Kinases - immunology | Lymphocyte Function-Associated Antigen-1 - immunology | Macrophage-1 Antigen - metabolism | Syk Kinase | CD11a Antigen - metabolism | Butadienes - pharmacology | Neutrophil Activation - immunology | GPI-Linked Proteins - immunology | Cell Degranulation | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | src-Family Kinases - antagonists & inhibitors | Autoimmune Diseases - immunology | Cells, Cultured | Neutrophils - immunology | Imidazoles - pharmacology | Inflammation - immunology | Antioxidants - pharmacology | Onium Compounds - pharmacology | Pyrimidines - pharmacology | Receptors, IgG - immunology | src-Family Kinases - immunology | CD18 Antigens - metabolism | Mesalamine - pharmacology | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Aminopyrine - pharmacology | Ascorbic Acid - pharmacology | Pyridines - pharmacology | Antigen-Antibody Complex - immunology | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Protein-Tyrosine Kinases - antagonists & inhibitors | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Journal Article
The Journal of Immunology, ISSN 0022-1767, 01/2004, Volume 172, Issue 1, pp. 567 - 576
The signaling mechanism by which the anti-inflammatory cytokine IL-10 mediates suppression of proinflammatory cytokine synthesis remains largely unknown.... 
RHEUMATOID-ARTHRITIS | TUMOR-NECROSIS-FACTOR | DNA-BINDING | HUMAN NEUTROPHILS | TYROSINE PHOSPHORYLATION | INTERLEUKIN-10 RECEPTOR | GENE-EXPRESSION | KAPPA-B-ALPHA | IMMUNOLOGY | HUMAN MONOCYTES | MONONUCLEAR PHAGOCYTES | Protein Binding - genetics | Protein Biosynthesis | Interleukin-6 - antagonists & inhibitors | Humans | Tumor Necrosis Factor-alpha - genetics | Immunoglobulins - genetics | Lipopolysaccharides - antagonists & inhibitors | RNA, Messenger - metabolism | Suppressor of Cytokine Signaling Proteins | Repressor Proteins - antagonists & inhibitors | Antigens, CD - metabolism | Trans-Activators - physiology | Protein Tyrosine Phosphatases - antagonists & inhibitors | RNA, Messenger - biosynthesis | Protein Tyrosine Phosphatases - genetics | Inflammation Mediators - physiology | Glycoproteins - genetics | DNA-Binding Proteins - physiology | Protein Tyrosine Phosphatases - biosynthesis | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction - genetics | DNA - metabolism | Down-Regulation - genetics | Macrophages - metabolism | Protein Tyrosine Phosphatase, Non-Receptor Type 2 | Repressor Proteins - biosynthesis | Up-Regulation - immunology | Interleukin-10 - antagonists & inhibitors | Lipopolysaccharides - pharmacology | Adenoviruses, Human - genetics | Interleukin-10 - immunology | Tumor Necrosis Factor-alpha - biosynthesis | Phosphorylation | Tissue Inhibitor of Metalloproteinase-1 - biosynthesis | Antigens, CD - biosynthesis | Receptors, Cell Surface | Receptors, IgG - biosynthesis | Receptors, IgG - antagonists & inhibitors | Interleukin-10 - physiology | Signal Transduction - immunology | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Signaling Lymphocytic Activation Molecule Family Member 1 | Receptors, Tumor Necrosis Factor - antagonists & inhibitors | RNA, Messenger - antagonists & inhibitors | Receptors, Tumor Necrosis Factor, Type II | Trans-Activators - genetics | Inflammation Mediators - antagonists & inhibitors | Trans-Activators - biosynthesis | Immunoglobulins - biosynthesis | Macrophages - immunology | Inflammation Mediators - immunology | Receptors, Tumor Necrosis Factor - metabolism | Immune Sera - pharmacology | Proteins - physiology | Cells, Cultured | Glycoproteins - antagonists & inhibitors | Histocompatibility Antigens Class II - biosynthesis | Tissue Inhibitor of Metalloproteinase-1 - antagonists & inhibitors | Transcription Factors - antagonists & inhibitors | Transcription Factors - biosynthesis | Up-Regulation - genetics | DNA-Binding Proteins - genetics | DNA - antagonists & inhibitors | Glycoproteins - biosynthesis | Suppressor of Cytokine Signaling 3 Protein | Down-Regulation - immunology | Interleukin-6 - biosynthesis | Receptors, Tumor Necrosis Factor - biosynthesis | STAT3 Transcription Factor | Trans-Activators - antagonists & inhibitors | Genetic Vectors | DNA-Binding Proteins - biosynthesis | Tumor Necrosis Factor-alpha - antagonists & inhibitors | SOCS-3 protein | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2014, Volume 135, Issue 3, pp. 729 - 736.e5
Background Clinical observations suggest that anaphylaxis is more common in adult women compared with adult men, although the mechanistic basis for this sex... 
Allergy and Immunology | estrogen | Anaphylaxis | nitric oxide synthase | vascular permeability | mast cells | ACTIVATION | IDIOPATHIC ANAPHYLAXIS | SEX-HORMONES | CARDIOVASCULAR-SYSTEM | SENSITIVITY | BODY-TEMPERATURE | RELEASE | IMMUNOLOGY | SHOCK | ALLERGY | NO SYNTHASE | UP-REGULATION | Protein Aggregates | Capillary Permeability | Humans | Receptors, IgG - chemistry | Male | omega-N-Methylarginine - pharmacology | Histamine - immunology | Receptors, IgG - antagonists & inhibitors | Lung - enzymology | Receptors, Estrogen - immunology | Receptors, IgE - chemistry | Estrogen Receptor Antagonists - pharmacology | Anaphylaxis - physiopathology | Anaphylaxis - immunology | Female | Receptors, IgE - immunology | Estradiol - pharmacology | Disease Models, Animal | Estradiol - metabolism | Mast Cells - immunology | Nitric Oxide - biosynthesis | Nitric Oxide Synthase Type III - immunology | Gene Expression | Histamine - pharmacology | Ovariectomy | Receptors, Estrogen - antagonists & inhibitors | Receptors, IgE - antagonists & inhibitors | Lung - physiopathology | Receptors, IgG - immunology | Body Temperature | Anaphylaxis - enzymology | Mast Cells - drug effects | Nitric Oxide Synthase Type III - genetics | Mice, Knockout | Nitric Oxide Synthase Type III - antagonists & inhibitors | Anaphylaxis - genetics | Animals | Receptors, Estrogen - chemistry | Lung - drug effects | Mast Cells - pathology | Mice | Lung - immunology | Sex discrimination | Nitric oxide | Estrogen | Endothelium | Gender differences | Rodents | Allergies | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 06/2006, Volume 79, Issue 6, pp. 1242 - 1251
Fibrocytes are fibroblast‐like cells, which appear to participate in wound healing and are present in pathological lesions associated with asthma, pulmonary... 
monocytes | serum amyloid P | cellular differentiation | inflammation | FcγR | Monocytes | Inflammation | Cellular differentiation | Serum amyloid P | C-REACTIVE PROTEIN | RHEUMATOID-ARTHRITIS | AMYLOID-P COMPONENT | PERIPHERAL-BLOOD FIBROCYTES | CIRCULATING FIBROCYTES | IMMUNOLOGY | FC-GAMMA-RIIA | MEDIATED PHAGOCYTOSIS | CELL BIOLOGY | TYROSINE KINASE SYK | STRUCTURAL REQUIREMENTS | Fc gamma R | HEMATOLOGY | T-CELLS | Phosphorylation | Monocytes - cytology | Cells, Cultured - cytology | Humans | Serum Amyloid P-Component - physiology | Receptors, IgG - antagonists & inhibitors | Immunoglobulin M - immunology | Protein-Tyrosine Kinases - physiology | Immunoglobulin E - immunology | Immunoglobulin Fc Fragments - immunology | Immunoglobulin G - immunology | Immunoglobulin G - pharmacology | src-Family Kinases - physiology | Syk Kinase | Cells, Cultured - drug effects | Receptors, IgG - physiology | Antibodies, Monoclonal - pharmacology | Immunoglobulin E - pharmacology | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | src-Family Kinases - antagonists & inhibitors | Immunoglobulin A - pharmacology | Receptors, IgG - immunology | Monocytes - drug effects | Cell Differentiation - drug effects | Biopolymers - pharmacology | Fibroblasts - drug effects | Immunoglobulin A - immunology | Fibroblasts - cytology | Protein Processing, Post-Translational | Intracellular Signaling Peptides and Proteins - physiology | Immunoglobulin M - pharmacology | Protein-Tyrosine Kinases - antagonists & inhibitors | Index Medicus
Journal Article
Nature Communications, ISSN 2041-1723, 2015, Volume 6, Issue 1, pp. 6211 - 6211
Most macrophages remain uninfected in HIV-1-infected patients. Nevertheless, the phagocytic capacity of phagocytes from these patients is impaired, favouring... 
RECEPTOR-MEDIATED PHAGOCYTOSIS | MYCOBACTERIUM-AVIUM COMPLEX | MANNOSE RECEPTOR | PHOSPHATIDYLINOSITOL 4,5-BISPHOSPHATE | MULTIDISCIPLINARY SCIENCES | ACTIVE ANTIRETROVIRAL THERAPY | IN-VIVO | MONOCYTE-DERIVED MACROPHAGES | RHO-GTPASES | PLASMA-MEMBRANE | T-CELLS | Mannose-Binding Lectins - metabolism | Pseudopodia - ultrastructure | Humans | Neutrophils - ultrastructure | Monocytes - metabolism | cdc42 GTP-Binding Protein - metabolism | Protein Transport - drug effects | Bystander Effect | Recombinant Proteins - biosynthesis | Lectins, C-Type - genetics | Receptors, Cell Surface - antagonists & inhibitors | Receptors, IgG - antagonists & inhibitors | Receptors, IgG - metabolism | Pseudopodia - drug effects | Lectins, C-Type - metabolism | Phosphatidylinositol 4,5-Diphosphate - metabolism | Receptors, IgG - genetics | HIV-1 - physiology | Toxoplasma - growth & development | Neutrophils - metabolism | Monocytes - ultrastructure | Macrophages - ultrastructure | Phagocytosis - drug effects | Signal Transduction | Neutrophils - drug effects | Gene Expression Regulation | Receptors, Cell Surface - metabolism | tat Gene Products, Human Immunodeficiency Virus - biosynthesis | Recombinant Proteins - pharmacology | Mannose-Binding Lectins - antagonists & inhibitors | Phosphatidylinositol 4,5-Diphosphate - antagonists & inhibitors | Host-Pathogen Interactions | Monocytes - drug effects | Macrophages - metabolism | tat Gene Products, Human Immunodeficiency Virus - pharmacology | Mannose-Binding Lectins - genetics | Pseudopodia - metabolism | cdc42 GTP-Binding Protein - genetics | Macrophages - drug effects | Primary Cell Culture | Mycobacterium avium - growth & development | Lectins, C-Type - antagonists & inhibitors | Receptors, Cell Surface - genetics | Index Medicus | Life Sciences | Microbiology and Parasitology | Virology
Journal Article
Developmental Cell, ISSN 1534-5807, 02/2013, Volume 24, Issue 4, pp. 372 - 383
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2013, Volume 132, Issue 6, pp. 1375 - 1387
Journal Article