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Journal of the American College of Cardiology, ISSN 0735-1097, 10/2017, Volume 70, Issue 18, pp. 2278 - 2289
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2011, Volume 55, Issue 5, pp. 1086 - 1094
Journal Article
European Journal of Cancer, ISSN 0959-8049, 2013, Volume 49, Issue 12, pp. 2760 - 2770
Abstract Helicobacter pylori infection induced interleukin-1β (IL-1β) production and is associated with aberrant DNA methylation and gastric diseases... 
Hematology, Oncology and Palliative Medicine | Helicobacter pylori | Interleukin-1β | Nitric oxide | DNA methylation | Inflammation | E-cadherin | Gastric cancer | ANTAGONIST | CANCER | Interleukin-1 beta | NITRIC-OXIDE PRODUCTION | POLYMORPHISMS | EPITHELIAL-CELLS | GENE | ONCOLOGY | DISEASE | INFECTION | ERADICATION | CADHERIN PROMOTER HYPERMETHYLATION | Helicobacter pylori - immunology | DNA Methylation - immunology | Cadherins - metabolism | Receptors, Interleukin-1 Type I - immunology | Gastritis - genetics | Helicobacter pylori - drug effects | Male | Interleukin-1beta - genetics | Gastritis - prevention & control | Host-Pathogen Interactions - immunology | Receptors, Interleukin-1 Type I - metabolism | Cadherins - immunology | Nitric Oxide Synthase Type II - immunology | Interleukin-1beta - metabolism | Interleukin 1 Receptor Antagonist Protein - pharmacology | Cadherins - genetics | Nitric Oxide - immunology | Gene Expression | Mice, Inbred C57BL | Interleukin-1beta - immunology | Helicobacter Infections - immunology | Helicobacter Infections - genetics | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Animals | Nitric Oxide Synthase Type II - genetics | Gastritis - immunology | Receptors, Interleukin-1 Type I - genetics | Anti-Bacterial Agents - pharmacology | Helicobacter pylori - physiology | Mice | Nitric Oxide - metabolism | Helicobacter Infections - microbiology | Nitric Oxide Synthase Type II - metabolism
Journal Article
Journal Article
Journal Article
Journal Article
Immunity, ISSN 1074-7613, 02/2011, Volume 34, Issue 2, pp. 213 - 223
.... Here we showed that type I IFN inhibited interleukin-1 (IL-1) production through two distinct mechanisms... 
FAMILIAL MEDITERRANEAN FEVER | MULTIPLE-SCLEROSIS | CASPASE-1 | HOST-DEFENSE | INNATE | CRYSTALS | RECOMBINANT INTERLEUKIN-1 | MICE | BACTERIAL-INFECTION | IMMUNOLOGY | RECEPTOR ANTAGONIST | Interleukin-1 - genetics | Inflammasomes - metabolism | STAT1 Transcription Factor - deficiency | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Monocytes - metabolism | Monocytes - immunology | Interleukin-10 - physiology | Interleukin-1 - biosynthesis | Interferon-beta - therapeutic use | Interferon Type I - biosynthesis | STAT3 Transcription Factor - physiology | Candida albicans - physiology | Interferon Type I - physiology | Caspase 1 - physiology | Candidiasis - immunology | Carrier Proteins - physiology | Peritonitis - etiology | STAT1 Transcription Factor - physiology | Poly I-C - pharmacology | Disease Susceptibility | Mice, Inbred C57BL | Peritonitis - immunology | STAT1 Transcription Factor - genetics | Gene Expression Regulation - drug effects | Animals | Candidiasis - etiology | Caspase 1 - genetics | Caspase 1 - deficiency | Interferon Type I - genetics | Multiple Sclerosis - immunology | Multiple Sclerosis - pathology | Apoptosis Regulatory Proteins - physiology | Cells, Cultured - metabolism | Mice | Interferon Inducers - pharmacology | Multiple Sclerosis - drug therapy | Interferon | Infections | Cytokines | Viral infections | Molecular weight | Immune system
Journal Article