X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (2672) 2672
Book Review (531) 531
Publication (337) 337
Book Chapter (14) 14
Conference Proceeding (6) 6
Dissertation (5) 5
Data Set (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
index medicus (2516) 2516
humans (1664) 1664
animals (1498) 1498
mice (964) 964
immunology (915) 915
male (818) 818
female (737) 737
inflammation (693) 693
cytokines (622) 622
interleukin-18 - metabolism (581) 581
mice, inbred c57bl (452) 452
expression (436) 436
cells, cultured (425) 425
cell biology (410) 410
activation (377) 377
signal transduction (351) 351
middle aged (337) 337
adult (328) 328
interleukin-18 (327) 327
research (320) 320
abridged index medicus (318) 318
proteins (312) 312
mice, knockout (307) 307
biochemistry & molecular biology (305) 305
research article (300) 300
nf-kappa-b (295) 295
gene expression (291) 291
t-cells (285) 285
receptor (282) 282
il-18 (279) 279
interleukin-18 - genetics (268) 268
apoptosis (259) 259
rodents (257) 257
health aspects (244) 244
cytokines - metabolism (240) 240
toll-like receptors (239) 239
dendritic cells (237) 237
ifn-gamma (235) 235
interleukin-18 - immunology (233) 233
cytokine (231) 231
interleukins (228) 228
cells (227) 227
medicine (225) 225
interferon-gamma (224) 224
analysis (223) 223
physiological aspects (221) 221
article (215) 215
multidisciplinary sciences (213) 213
rats (212) 212
aged (204) 204
interleukin 18 (201) 201
disease models, animal (194) 194
interleukin-18 - pharmacology (187) 187
caspase 1 - metabolism (186) 186
medicine, research & experimental (185) 185
neurosciences (184) 184
immune system (181) 181
innate immunity (181) 181
necrosis-factor-alpha (181) 181
gene-expression (179) 179
macrophages (178) 178
science (178) 178
tumor-necrosis-factor (173) 173
cell line (172) 172
enzyme-linked immunosorbent assay (167) 167
flow cytometry (166) 166
disease (165) 165
interleukin-1beta - metabolism (165) 165
receptors (165) 165
review (165) 165
inflammasomes - metabolism (162) 162
interferon-gamma - biosynthesis (162) 162
rna, messenger - metabolism (157) 157
lymphocytes (156) 156
biology (155) 155
genetic aspects (154) 154
killer cells, natural - immunology (147) 147
immune response (146) 146
pathogenesis (143) 143
interferon-gamma - metabolism (140) 140
interleukin-18 - blood (140) 140
risk factors (140) 140
gamma-inducing factor (139) 139
physiology (138) 138
endocrinology & metabolism (137) 137
inflammation - immunology (137) 137
interleukin-18 - biosynthesis (136) 136
receptors, interleukin-18 (135) 135
gene expression regulation (134) 134
in-vivo (134) 134
ligands (134) 134
pathology (133) 133
rheumatology (132) 132
inflammation - metabolism (131) 131
cancer (129) 129
interleukin-18 - physiology (129) 129
interleukin-18 receptor alpha subunit (129) 129
nf-kappa b - metabolism (129) 129
immunity (128) 128
infection (128) 128
more...
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (2646) 2646
Chinese (12) 12
Polish (8) 8
Japanese (5) 5
Russian (3) 3
Romanian (1) 1
Swedish (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


American Journal of Physiology : Heart and Circulatory Physiology, ISSN 0363-6135, 2014, Volume 306, Issue 7, pp. H1025 - 31
Patients with heart failure (HF) have enhanced systemic IL-1 activity, and, in the experimental mouse model, IL-1 induces left ventricular (LV) systolic... 
Heart failure | Inflammation | Interleukins | Systolic dysfunction | heart failure | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | CARDIOVASCULAR EVENTS | INCREASED CIRCULATING INTERLEUKIN-18 | MONONUCLEAR-CELLS | BINDING-PROTEIN | systolic dysfunction | NECROSIS-FACTOR-ALPHA | RECEPTOR ANTAGONIST | interleukins | inflammation | ACUTE MYOCARDIAL-INFARCTION | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | BLOCKING INTERLEUKIN-1 | CONGESTIVE-HEART-FAILURE | Humans | Heart Failure - physiopathology | Male | Systole | Heart Failure - blood | Ventricular Dysfunction, Left - chemically induced | Time Factors | Ventricular Dysfunction, Left - blood | Ventricular Dysfunction, Left - genetics | Interleukin-6 - blood | Interleukin 1 Receptor Antagonist Protein - pharmacology | Disease Models, Animal | Signal Transduction | Mice, Inbred C57BL | Ventricular Function, Left - drug effects | Interleukin-18 - deficiency | Heart Failure - genetics | Heart Failure - metabolism | Interleukin-1beta | Ventricular Dysfunction, Left - physiopathology | Mice, Knockout | Antibodies - pharmacology | Ventricular Dysfunction, Left - metabolism | Animals | Intercellular Signaling Peptides and Proteins - pharmacology | Mice | Interleukin-18 - genetics | Heart Failure - chemically induced | Interleukin-18 - metabolism | Cardiac patients | Physiological aspects | Research | Cardiovascular research | Health aspects | Protein-protein interactions | Plasma | Cytokines | Rodents | Index Medicus | Signaling and Stress Response
Journal Article
Blood, ISSN 0006-4971, 12/2012, Volume 120, Issue 24, pp. 4751 - 4760
Natural killer (NK) cells are lymphocytes that play an important role in the immune response to infection and malignancy. Recent studies in mice have shown... 
INNATE | NATURAL-KILLER-CELLS | INFECTION | HEMATOLOGY | EXPANSION | Receptors, Interleukin-12 - immunology | Antigens, CD - immunology | Interleukin-18 - immunology | Humans | Lectins, C-Type - immunology | Interferon-gamma - metabolism | Interleukin-15 - immunology | Antigens, CD - metabolism | NK Cell Lectin-Like Receptor Subfamily D - immunology | Lectins, C-Type - metabolism | NK Cell Lectin-Like Receptor Subfamily C - immunology | Lymphocyte Activation - immunology | Flow Cytometry | Time Factors | Killer Cells, Natural - immunology | Receptors, Interleukin-12 - genetics | Interferon-gamma - genetics | CD56 Antigen - immunology | Immunologic Memory - immunology | Cytokines - immunology | Interleukin-12 - pharmacology | Receptors, Interleukin-18 - metabolism | CD56 Antigen - metabolism | NK Cell Lectin-Like Receptor Subfamily C - metabolism | Antigens, Differentiation, T-Lymphocyte - immunology | Antigens, Differentiation, T-Lymphocyte - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Interleukin-15 - pharmacology | Receptors, Interleukin-12 - metabolism | Lymphocyte Activation - drug effects | Interferon-gamma - immunology | Interleukin-12 - immunology | K562 Cells | Receptors, Interleukin-18 - genetics | Receptors, Interleukin-18 - immunology | Cell Proliferation - drug effects | Interleukin-18 - pharmacology | Killer Cells, Natural - drug effects | Killer Cells, Natural - metabolism | NK Cell Lectin-Like Receptor Subfamily D - metabolism | Cytokines - pharmacology | Index Medicus | Abridged Index Medicus | Immunobiology
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 07/2002, Volume 22, Issue 14, pp. 5910 - 5919
Journal Article
Diabetes, ISSN 0012-1797, 09/2013, Volume 62, Issue 9, pp. 3064 - 3074
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2012, Volume 53, Issue 1, pp. 113 - 124
Abstract Chronic elevation of angiotensin (Ang)-II can lead to myocardial inflammation, hypertrophy and cardiac failure. The adaptor molecule CIKS (connection... 
Cardiovascular | Act1 | Cardiac hypertrophy | TRAF3IP2 | NADPH oxidase | RAAS | Fibrosis | SIGNALING PATHWAYS | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | NOX2-CONTAINING NADPH OXIDASE | PROTEIN-KINASE | HEART-FAILURE | CELL BIOLOGY | IN-VIVO | GENE-EXPRESSION | INDUCED CARDIAC-HYPERTROPHY | NF-KAPPA-B | REQUIREMENT | Reactive Oxygen Species - metabolism | Gene Expression - drug effects | Membrane Glycoproteins - metabolism | NADPH Oxidases - metabolism | Male | NF-kappa B - metabolism | Transcription Factor AP-1 - metabolism | Matrix Metalloproteinase 9 - genetics | Endomyocardial Fibrosis - genetics | Angiotensin II - toxicity | Angiotensin II - pharmacology | Cells, Cultured | NADPH Oxidase 2 | Collagen - metabolism | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Receptor, Angiotensin, Type 1 - metabolism | Adaptor Proteins, Signal Transducing - genetics | Cardiomegaly - chemically induced | Myocytes, Cardiac - metabolism | Mice | Adaptor Proteins, Signal Transducing - metabolism | Cardiomegaly - genetics | Interleukin-18 - genetics | Interleukin-18 - metabolism | rac1 GTP-Binding Protein - metabolism | Cardiomegaly - metabolism | Oxidases | Phosphates | Niacinamide | RNA | Superoxide | Purines | Interleukins | Collagen | Angiotensin | Fibroblast growth factors | Natriuretic peptides | Protein binding | Hypertrophy | Index Medicus | fibrosis | cardiac hypertrophy
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2013, Volume 123, Issue 1, pp. 236 - 246
Nephrocalcinosis, acute calcium oxalate (CaOx) nephropathy, and renal stone disease can lead to inflammation and subsequent renal failure, but the underlying... 
KIDNEY-DISEASE | NEPHROLITHIASIS | MEDICINE, RESEARCH & EXPERIMENTAL | DANGER | ACTIVATION | DENDRITIC CELLS | NLRP3 INFLAMMASOME | STONE FORMATION | FAILURE | NEPHROPATHY | NALP3 INFLAMMASOME | Interleukin-18 - immunology | Cytoskeletal Proteins - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Dendritic Cells - immunology | Nephrocalcinosis - immunology | Receptors, Interleukin-1 - genetics | Interleukin-1beta - genetics | Inflammation - metabolism | Nephrocalcinosis - metabolism | Myeloid Differentiation Factor 88 - immunology | Cytoskeletal Proteins - metabolism | Kidney Tubules - pathology | Immunologic Deficiency Syndromes - immunology | Kidney Tubules - metabolism | Carrier Proteins - immunology | Dendritic Cells - metabolism | Interleukin-1beta - secretion | Kidney Tubules - immunology | Calcium Oxalate - immunology | Receptors, Interleukin-1 - immunology | Myeloid Differentiation Factor 88 - genetics | Interleukin-1beta - immunology | Inflammation - immunology | Calcium Oxalate - metabolism | Mice, Knockout | Receptors, Interleukin-1 - metabolism | Carrier Proteins - genetics | Nephrocalcinosis - pathology | Animals | Apoptosis Regulatory Proteins | Carrier Proteins - metabolism | Cytoskeletal Proteins - immunology | Nephrocalcinosis - genetics | CARD Signaling Adaptor Proteins | Immunologic Deficiency Syndromes - genetics | Immunologic Deficiency Syndromes - metabolism | Inflammation - genetics | Mice | Interleukin-18 - genetics | Myeloid Differentiation Factor 88 - metabolism | Interleukin-18 - metabolism | Phagocytosis | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Journal Article
Circulation Journal, ISSN 1346-9843, 2012, Volume 76, Issue 7, pp. 1780 - 1791
Journal Article
Immunity, ISSN 1074-7613, 01/2014, Volume 40, Issue 1, pp. 128 - 139
Commensal gut microflora and dietary fiber protect against colonic inflammation and colon cancer through unknown targets. Butyrate, a bacterial product from... 
INTESTINAL INFLAMMATION | NICOTINIC-ACID | DENDRITIC CELLS | CHAIN FATTY-ACIDS | INTERLEUKIN-10 RECEPTOR | RETINOIC-ACID | REGULATORY T-CELLS | APC(MIN/+) MICE | GUT MICROBIOTA | IMMUNOLOGY | BOWEL-DISEASE | T-Lymphocyte Subsets - immunology | Receptors, G-Protein-Coupled - metabolism | Colonic Neoplasms - prevention & control | Dendritic Cells - immunology | Epithelial Cells - drug effects | Colitis - complications | Colon - immunology | Receptors, G-Protein-Coupled - immunology | T-Lymphocytes, Regulatory - immunology | Niacin - administration & dosage | Microbiota | Butyrates - immunology | Interleukin-10 - metabolism | Colitis - drug therapy | Colitis - immunology | Colonic Neoplasms - etiology | Macrophages - immunology | Receptors, Nicotinic - metabolism | Disease Susceptibility | Colon - pathology | Carcinogenesis - immunology | Mice, Inbred C57BL | Cells, Cultured | Mice, Knockout | Receptors, Nicotinic - immunology | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Lymphocyte Activation - drug effects | Epithelial Cells - immunology | Colon - microbiology | Mice | Receptors, G-Protein-Coupled - genetics | Interleukin-18 - genetics | Receptors, Nicotinic - genetics | Interleukin-18 - metabolism | Colon cancer | Metabolites | Dendritic cells | Esters | Inflammation | T cells | Niacin | Inflammatory bowel disease | Colon | Lymphocytes | Rodents | Vitamin B | Colorectal cancer | Index Medicus
Journal Article