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Journal of Clinical Investigation, ISSN 0021-9738, 10/2012, Volume 122, Issue 10, pp. 3647 - 3651
Although long considered a promising treatment option for type 1 diabetes, pancreatic islet cell transformation has been hindered by immune system rejection of... 
MEDICINE, RESEARCH & EXPERIMENTAL | RECEPTOR | TISSUE FACTOR | T-CELLS | Receptors, Interleukin-8A - antagonists & inhibitors | Diabetes Mellitus, Experimental - surgery | Diabetes Mellitus, Type 1 - surgery | Humans | Middle Aged | Diabetes Mellitus, Experimental - genetics | Interleukin-8 - physiology | Male | Chemokine CXCL1 - genetics | Receptors, Interleukin-8B - antagonists & inhibitors | Receptors, Interleukin-8B - deficiency | Diabetes Mellitus, Experimental - blood | Islets of Langerhans Transplantation - immunology | Islets of Langerhans - metabolism | Chemokine CXCL1 - physiology | Adult | Female | Diabetes Mellitus, Type 1 - immunology | Drug Evaluation, Preclinical | Receptors, Interleukin-8B - physiology | Cell Survival - drug effects | Blood Glucose - analysis | Islets of Langerhans - pathology | Graft Rejection - prevention & control | Mice, Inbred C57BL | Neutrophils - immunology | Islets of Langerhans - immunology | Chemokine CXCL1 - biosynthesis | Treatment Outcome | Sulfonamides - pharmacology | Pilot Projects | Animals | Sulfonamides - therapeutic use | Receptors, Interleukin-8B - genetics | Mice | Mice, Inbred BALB C | Graft Survival - drug effects | Natural Killer T-Cells - immunology | Receptors, Interleukin-8A - physiology | Islet cell transplantation | Care and treatment | Type 1 diabetes | Allosteric proteins | Physiological aspects | Research | Health aspects | Chemokine receptors | Brief Report
Journal Article
Science Translational Medicine, ISSN 1946-6234, 05/2014, Volume 6, Issue 237, pp. 237ra67 - 237ra67
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2016, Volume 11, Issue 4, p. e0153829
Phagocytes restrict the germination of Aspergillus fumigatus conidia and prevent the establishment of invasive pulmonary aspergillosis in immunecompetent mice.... 
DEFENSE | CXC CHEMOKINE | MULTIDISCIPLINARY SCIENCES | CONIDIA | ALVEOLAR MACROPHAGES | RECEPTOR | NEUTROPHIL RECRUITMENT | Lung - microbiology | Bone Marrow - immunology | Lectins, C-Type - immunology | Disease Resistance - immunology | Host-Pathogen Interactions - immunology | Lectins, C-Type - metabolism | Flow Cytometry | Bone Marrow - metabolism | Aspergillus fumigatus - physiology | Lung - metabolism | Chemokines - immunology | Cytokines - immunology | Spores, Fungal - immunology | Cytokines - metabolism | Mice, Inbred C57BL | Receptors, Interleukin-8B - immunology | Phagocytes - metabolism | Phagocytes - immunology | Mice, Knockout | Spores, Fungal - physiology | Animals | Phagocytes - microbiology | Aspergillus fumigatus - immunology | Receptors, Interleukin-8B - metabolism | Aspergillosis - microbiology | Chemokines - metabolism | Mice, Inbred BALB C | Bone Marrow - microbiology | Lung - immunology | Aspergillosis - immunology | Aspergillus | Phagocytes | Aspergillosis | Physiological aspects | Development and progression | Genetic aspects | Research | Bioluminescence | Cytokines | CXCR2 protein | Germination | Neutrophils | Priming | Inflammation | Infections | Fungal infections | Lethal dose | Lymphocytes B | Lungs | Bone marrow | Mice | Life sciences | Chemokines | Phagocytosis | Viral infections | Immune system | Conidia | Disease Resistance | Lung | Lectins, C-Type | Host-Pathogen Interactions | Life Sciences | Microbiology and Parasitology | Spores, Fungal | Receptors, Interleukin-8B | Aspergillus fumigatus | Bone Marrow
Journal Article
Hepatology, ISSN 0270-9139, 11/2012, Volume 56, Issue 5, pp. 1971 - 1982
Acetaminophen (APAP) is a safe analgesic and antipyretic drug. However, APAP overdose leads to massive hepatocyte death. Cell death during APAP toxicity occurs... 
ACETAMINOPHEN-INDUCED HEPATOTOXICITY | CELL INJURY | HEPATOCYTES | STERILE INFLAMMATION | DEPLETION PROTECTS | INFLAMMATORY RESPONSES | FORMYL PEPTIDE RECEPTOR | AUTOIMMUNE HEPATITIS | MICE | CORTICOSTEROIDS | GASTROENTEROLOGY & HEPATOLOGY | Chemokines - blood | Liver - pathology | Acute-Phase Reaction - immunology | Coculture Techniques | Humans | Middle Aged | Toll-Like Receptor 9 - genetics | Male | Receptors, Formyl Peptide - immunology | Acute-Phase Reaction - metabolism | Acute Lung Injury - blood | Necrosis - immunology | Toll-Like Receptor 9 - immunology | Young Adult | Receptors, Interleukin-8B - blood | Systemic Inflammatory Response Syndrome - blood | Mitochondrial Proteins - metabolism | Receptors, Formyl Peptide - metabolism | Systemic Inflammatory Response Syndrome - immunology | Adult | Female | Interleukin-8 - blood | Child | Chemokines - immunology | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Neutrophils - immunology | Liver Failure, Acute - chemically induced | Receptors, Interleukin-8B - immunology | DNA, Mitochondrial - blood | Hep G2 Cells | Mice, Knockout | Animals | Analysis of Variance | Acetaminophen | Liver Failure, Acute - immunology | Receptors, Interleukin-8B - metabolism | Adolescent | Acute Lung Injury - immunology | Chemokines - metabolism | Mice | Mitochondrial Proteins - immunology | Cell Movement | Mitochondrial DNA | Liver | Rodents | Hepatology | Chemokines | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 09/2012, Volume 122, Issue 9, pp. 3127 - 3144
The chemokine receptor CXCR2 is a key mediator of neutrophil migration that also plays a role in tumor development. However, CXCR2 influences tumors through... 
ATHEROSCLEROTIC LESIONS | COLON-CANCER | MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-NECROSIS-FACTOR | IMMUNE CELLS | INTESTINAL NEOPLASIA | PROSTATE-CANCER | SKIN CARCINOGENESIS | RECEPTOR D6 | MYELOID CELLS | BEARING MICE | Dextran Sulfate | Tetradecanoylphorbol Acetate | Adenocarcinoma - pathology | Colitis - pathology | Colonic Neoplasms - chemically induced | Mice, 129 Strain | Receptors, Interleukin-8B - antagonists & inhibitors | Receptors, Interleukin-8B - deficiency | Colonic Neoplasms - metabolism | Adenoma - metabolism | Adenocarcinoma - metabolism | Azoxymethane | Colitis - chemically induced | Statistics, Nonparametric | Papilloma - pathology | Precancerous Conditions - pathology | Neutrophils - metabolism | Papilloma - chemically induced | Papilloma - metabolism | Skin Neoplasms - pathology | Dermatitis, Contact - pathology | Gene Expression | Neutrophils - enzymology | Mice, Inbred C57BL | Skin Neoplasms - chemically induced | Tumor Burden | Chemokines, CXC - genetics | Chemokines, CXC - secretion | Mice, Knockout | Skin Neoplasms - metabolism | Animals | Cell Transformation, Neoplastic | Adenoma - secretion | Chemokines, CXC - metabolism | Receptors, Interleukin-8B - genetics | Animals, Inbred Strains | 9,10-Dimethyl-1,2-benzanthracene | Mice | Mice, Inbred BALB C | Precancerous Conditions - chemically induced | Colonic Neoplasms - secretion | Adenoma - chemically induced | Peroxidase - metabolism | Care and treatment | Cancer cells | Neutrophils | Papilloma | Genetic aspects | Diagnosis | Adenoma | Research | Gene expression
Journal Article
The Journal of Pathology, ISSN 0022-3417, 09/2015, Volume 237, Issue 1, pp. 85 - 97
Pancreatitis is a significant clinical problem and the lack of effective therapeutic options means that treatment is often palliative rather than curative. A... 
pancreatitis | chemokines | inflammation | CXCR2 | ATHEROSCLEROTIC LESIONS | ACTIVATION | NEUTROPHIL | TRYPSINOGEN | KAPPA-B | PATHOLOGY | PEPTIDE | ONCOLOGY | ACINAR-CELLS | MICE | CHEMOKINE RECEPTORS | EXPRESSION | Pancreatitis, Chronic - chemically induced | Pancreatitis - prevention & control | Receptors, Interleukin-8B - antagonists & inhibitors | Receptors, Interleukin-8B - deficiency | Pancreatitis - genetics | Time Factors | Pancreatitis - immunology | Neutrophils - metabolism | Neutrophil Infiltration - drug effects | Cytoprotection | Disease Models, Animal | Pancreatitis, Chronic - immunology | Acute Disease | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Neutrophils - drug effects | Neutrophils - immunology | Pancreas - drug effects | Pancreas - pathology | Receptors, Interleukin-8B - immunology | Pancreas - metabolism | Pancreas - immunology | Pancreatitis - chemically induced | Pancreatitis, Chronic - genetics | Pancreatitis, Chronic - prevention & control | Mice, Knockout | Peptides - pharmacology | Pancreatitis, Chronic - pathology | Animals | Signal Transduction - drug effects | Receptors, Interleukin-8B - genetics | Pancreatitis - pathology | Mice, Inbred BALB C | Pancreatitis - metabolism | Ceruletide | Pancreatitis, Chronic - metabolism | Analysis | Inflammation | Pancreatitis | Original Paper | Original Papers
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 1685 - 12
Journal Article
Journal Article