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Journal Article
Nature neuroscience, ISSN 1097-6256, 2014, Volume 17, Issue 7, pp. 908 - 910
Journal Article
Endocrinology, ISSN 0013-7227, 02/2016, Volume 157, Issue 2, pp. 737 - 751
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 03/2012, Volume 287, Issue 11, pp. 8598 - 8612
Perturbations in the adipocytokine profile, especially higher levels of leptin, are a major cause of breast tumor progression and metastasis; the underlying... 
HISTONE ACETYLTRANSFERASES | STEM-CELLS | MAMMARY-TUMOR GROWTH | CARCINOMA CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | IN-VIVO | FEMALE MICE | RECEPTOR | SIGNALING PROMOTES | E-CADHERIN | METASTASIS-ASSOCIATED PROTEIN-1 | Wnt1 Protein - genetics | Cadherins - metabolism | Vimentin - metabolism | Humans | Leptin - metabolism | Glycogen Synthase Kinase 3 beta | Wnt1 Protein - metabolism | Proto-Oncogene Proteins c-akt - genetics | Breast Neoplasms - metabolism | Phosphorylation - genetics | Cell Nucleus - metabolism | Cell Nucleus - pathology | Vimentin - genetics | Epithelial-Mesenchymal Transition | Female | Pyrimidinones - pharmacology | Leptin - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Cadherins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Gene Expression Regulation, Neoplastic - genetics | Repressor Proteins - metabolism | Wnt Signaling Pathway | Histone Deacetylases - genetics | Repressor Proteins - genetics | Histone Deacetylases - metabolism | Leptin - genetics | Glycogen Synthase Kinase 3 - metabolism | beta Catenin - metabolism | beta Catenin - genetics | Bridged Bicyclo Compounds, Heterocyclic - pharmacology | Breast Neoplasms - genetics | Cell Nucleus - genetics | Glycogen Synthase Kinase 3 - genetics | Breast Neoplasms - pathology | beta Catenin - antagonists & inhibitors | Cell Line, Tumor | Index Medicus | MTA1 | Signal Transduction | Adipokines | Breast Cancer | Leptin
Journal Article
Journal Article
Journal of Lipid Research, ISSN 0022-2275, 02/2014, Volume 55, Issue 2, pp. 276 - 288
Leptin is essential for energy homeostasis and regulation of food intake. Patients with congenital generalized lipodystrophy (CGL) due to mutations in... 
Acyltransferase | Diabetes mellitus | Phospholipids | BIOCHEMISTRY & MOLECULAR BIOLOGY | diabetes mellitus | ELEMENT-BINDING PROTEIN | phospholipids | NUCLEAR SREBP-1C | DIABETES-MELLITUS | RECOMBINANT LEPTIN | FATTY-ACID OXIDATION | OBESE GENE | MOUSE MODEL | OB/OB MICE | CONGENITAL GENERALIZED LIPODYSTROPHY | acyltransferase | ADIPOSE-TISSUE | 1-Acylglycerol-3-Phosphate O-Acyltransferase - genetics | Transcription, Genetic - drug effects | Liver - pathology | Receptors, Leptin - genetics | Fatty Liver - complications | Thyroxine - blood | Male | Hepatocytes - metabolism | Glycogen - metabolism | Gene Deletion | Oxidation-Reduction - drug effects | 1-Acylglycerol-3-Phosphate O-Acyltransferase - deficiency | Leptin - pharmacology | Corticosterone - blood | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Fatty Acids - metabolism | Hepatocytes - drug effects | Fatty Liver - metabolism | Fatty Liver - blood | Liver - metabolism | Insulin Resistance | Nuclear Proteins - metabolism | Lipodystrophy - complications | Triglycerides - metabolism | Gene Expression Regulation - drug effects | Transcription Factors - metabolism | Receptors, Leptin - deficiency | Animals | Lipogenesis - drug effects | Glucose - metabolism | Mice | Receptors, Leptin - metabolism | Fatty Acids - biosynthesis | Index Medicus
Journal Article