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American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 05/2009, Volume 296, Issue 5, pp. 980 - 991
Journal Article
Scientific reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 1480 - 11
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Retina - drug effects | Glial Fibrillary Acidic Protein - genetics | Retina - metabolism | Nitriles - pharmacology | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Ependymoglial Cells - metabolism | Gliosis - chemically induced | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Ocular Hypertension - metabolism | Glial Fibrillary Acidic Protein - metabolism | Ocular Hypertension - drug therapy | Gliosis - pathology | Mitogen-Activated Protein Kinase 1 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | JNK Mitogen-Activated Protein Kinases - genetics | Ependymoglial Cells - pathology | Disease Models, Animal | Barium Compounds - administration & dosage | Butadienes - pharmacology | Gliosis - genetics | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Ocular Hypertension - pathology | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Gene Expression Regulation | Proto-Oncogene Proteins c-fos - metabolism | Rats | p38 Mitogen-Activated Protein Kinases - genetics | Potassium Channels, Inwardly Rectifying - genetics | Ependymoglial Cells - drug effects | Rats, Sprague-Dawley | Ocular Hypertension - genetics | Cyclic AMP Response Element-Binding Protein - genetics | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Chlorides - administration & dosage | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cyclic AMP Response Element-Binding Protein - metabolism | Proto-Oncogene Proteins c-fos - genetics | Gliosis - drug therapy | Protein Kinase Inhibitors - pharmacology | Chronic Disease | Potassium Channels, Inwardly Rectifying - metabolism | Retina - pathology | Mitogen-Activated Protein Kinase 1 - metabolism | Transcription factors | Potassium channels (inwardly-rectifying) | P elements | Glial fibrillary acidic protein | c-Jun protein | Extracellular signal-regulated kinase | MAP kinase | Retina | Injection | Cyclic AMP response element-binding protein | Kinases | Proteins | Signal transduction | Gliosis | Protein kinase | Rodents | c-Fos protein | Glutamic acid receptors (metabotropic) | Index Medicus
Journal Article
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 2016, Volume 311, Issue 4, pp. H871 - H880
Heart failure | Brain | Sympathetic activity | Hypothalamic paraventricular nucleus | Mitogen-activated protein kinase | Subfornical organ | Endoplasmic reticulum stress | Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cholagogues and Choleretics - pharmacology | Tumor Necrosis Factor-alpha - genetics | Heart Failure - physiopathology | Male | NF-KappaB Inhibitor alpha - genetics | Peptidyl-Dipeptidase A - drug effects | Interleukin-1beta - genetics | Sympathetic Nervous System - physiopathology | RNA, Messenger - metabolism | Activating Transcription Factor 6 - genetics | Subfornical Organ - drug effects | Brain - metabolism | Heat-Shock Proteins - genetics | Inflammation - metabolism | Receptor, Angiotensin, Type 1 - genetics | Cyclooxygenase 2 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Real-Time Polymerase Chain Reaction | Echocardiography | Signal Transduction | Rats | Cyclooxygenase 2 - drug effects | Heart Failure - metabolism | Rats, Sprague-Dawley | Blotting, Western | Brain - drug effects | Tumor Necrosis Factor-alpha - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Endoplasmic Reticulum Stress | Paraventricular Hypothalamic Nucleus - metabolism | Infusions, Intraventricular | Mitogen-Activated Protein Kinases - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism | Interleukin-1beta - drug effects | Sympathetic Nervous System - drug effects | Mitogen-Activated Protein Kinase 1 - drug effects | X-Box Binding Protein 1 - drug effects | Sympathetic Nervous System - metabolism | Transcription Factor RelA - genetics | Activating Transcription Factor 6 - drug effects | Mitogen-Activated Protein Kinase 3 - drug effects | Taurochenodeoxycholic Acid - pharmacology | Peptidyl-Dipeptidase A - genetics | Renin-Angiotensin System | Receptor, Angiotensin, Type 1 - drug effects | RNA, Messenger - drug effects | Subfornical Organ - metabolism | Heat-Shock Proteins - drug effects | Activating Transcription Factor 4 - genetics | Activating Transcription Factor 4 - drug effects | NF-KappaB Inhibitor alpha - drug effects | Paraventricular Hypothalamic Nucleus - drug effects | p38 Mitogen-Activated Protein Kinases - drug effects | Animals | Transcription Factor RelA - drug effects | X-Box Binding Protein 1 - genetics | Mitogen-Activated Protein Kinases - metabolism | Physiological aspects | Cellular signal transduction | Endoplasmic reticulum | Health aspects | Mitogen-activated protein kinases | Signal transduction | Inflammation | Kinases | Index Medicus | mitogen-activated protein kinase | heart failure | Cardiovascular Neurohormonal Regulation | subfornical organ | sympathetic activity | brain | endoplasmic reticulum stress | hypothalamic paraventricular nucleus
Journal Article
Journal of clinical immunology, ISSN 0271-9142, 6/2011, Volume 31, Issue 3, pp. 472 - 478
Journal Article
Journal of molecular and cellular cardiology, ISSN 0022-2828, 2015, Volume 90, pp. 59 - 69
Cardiovascular | NF-κB | Cardiac fibroblasts | Collagen type I | Angiotensin II | p38 MAPK | Discoidin domain receptor 2 | P38 MAPK | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cell Biology | Protein Kinase C - genetics | RNA, Small Interfering - genetics | Angiotensin II - genetics | Reactive Oxygen Species - metabolism | NADPH Oxidases - metabolism | Type C Phospholipases - metabolism | Male | NF-kappa B - metabolism | Discoidin Domain Receptors | Collagen Type I - genetics | Protein Kinase C - metabolism | Mitogen-Activated Protein Kinase 1 - genetics | Myocardium - metabolism | NADPH Oxidases - genetics | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Transcription, Genetic | p38 Mitogen-Activated Protein Kinases - metabolism | Fibroblasts - metabolism | Angiotensin II - pharmacology | Collagen Type I - metabolism | Angiotensin II - metabolism | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Receptors, Mitogen - antagonists & inhibitors | Collagen Type I - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Gene Expression Regulation | Rats | p38 Mitogen-Activated Protein Kinases - genetics | Receptors, Mitogen - metabolism | Receptor Protein-Tyrosine Kinases - metabolism | Rats, Sprague-Dawley | Myocardium - cytology | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | NF-kappa B - genetics | Receptor Protein-Tyrosine Kinases - genetics | Fibroblasts - drug effects | Type C Phospholipases - genetics | Fibroblasts - cytology | Primary Cell Culture | Mitogen-Activated Protein Kinase 1 - metabolism | RNA, Small Interfering - metabolism | Receptors, Mitogen - genetics | Oxidases | Memory (Computers) | Analysis | Collagen | Genes | Angiotensin | Genetic research | Gene expression | Index Medicus
Journal Article