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Neuron (Cambridge, Mass.), ISSN 0896-6273, 2010, Volume 65, Issue 5, pp. 597 - 611
To investigate the role of microRNAs in regulating oligodendrocyte (OL) differentiation and myelination, we utilized transgenic mice in which microRNA... 
DEVBIO | MOLNEURO | TRANSCRIPTION FACTORS | NERVOUS-SYSTEM | GLIAL PROGENITOR-CELL | PROTEIN | CYCLE EXIT | NEURONS | GROWTH-FACTOR | IDENTIFICATION | MICRORNA EXPRESSION | LINEAGE | NEUROSCIENCES | Central Nervous System - metabolism | MicroRNAs - metabolism | Green Fluorescent Proteins - genetics | S100 Proteins - genetics | Oligodendroglia - drug effects | Basic Helix-Loop-Helix Transcription Factors - metabolism | Animals, Newborn | Basic Helix-Loop-Helix Transcription Factors - genetics | Receptor, Platelet-Derived Growth Factor alpha - metabolism | S100 Calcium Binding Protein beta Subunit | Oligonucleotide Array Sequence Analysis - methods | Rats | Mice, Transgenic | Oligodendrocyte Transcription Factor 2 | Myelin Proteins - genetics | Rats, Sprague-Dawley | Nerve Growth Factors - genetics | Mice | MicroRNAs - genetics | Myelin Proteins - metabolism | SOXD Transcription Factors - genetics | Optic Nerve - metabolism | Age Factors | SOXD Transcription Factors - metabolism | Gene Expression Regulation, Developmental - genetics | Sciatic Nerve - growth & development | Myelin Sheath - metabolism | Central Nervous System - growth & development | DNA-Binding Proteins - metabolism | Sciatic Nerve - metabolism | Oligodendroglia - physiology | Transfection | DEAD-box RNA Helicases - metabolism | Cell Differentiation - physiology | Optic Nerve - growth & development | Brain - cytology | 2',3'-Cyclic-Nucleotide Phosphodiesterases - genetics | Ribonuclease III - genetics | Ribonuclease III - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Profiling - methods | Transcription Factors - genetics | 2',3'-Cyclic-Nucleotide Phosphodiesterases - metabolism | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | DEAD-box RNA Helicases - genetics | Animals | Cell Differentiation - drug effects | Receptor, Platelet-Derived Growth Factor alpha - genetics | Stem Cells - drug effects | Stem Cells - physiology | Proteins | Genotype & phenotype | Gene expression | Experiments | Rodents | Cell cycle
Journal Article
Neurobiology of disease, ISSN 0969-9961, 09/2018, Volume 117, pp. 125 - 136
...) formation during the progression of Alzheimer's disease (AD). nbM neurons require the neurotrophin nerve growth factor (NGF... 
Nucleus basalis of Meynert | Neurotrophin | Mild cognitive impairment | Dopamine receptor | Cholinergic basal forebrain | Glutamate receptor | Adrenoceptor | Trk receptor | Acetylcholine receptor | Neurotransmission | NEUROFIBRILLARY TANGLES | PROTEIN-KINASE-B | PHOSPHATIDYLINOSITOL 3-KINASE | DEPENDENT AXONAL-TRANSPORT | NEUROSCIENCES | NERVE GROWTH-FACTOR | CA1 PYRAMIDAL NEURONS | NEUROPATHOLOGIC ASSESSMENT | FOREBRAIN NEURONS | MESSENGER-RNA | Neurofibrillary Tangles - genetics | Receptor, Nerve Growth Factor - genetics | Humans | Male | Nerve Growth Factors - metabolism | Receptors, Nerve Growth Factor - metabolism | Receptors, Neurotransmitter - genetics | Basal Nucleus of Meynert - metabolism | Alzheimer Disease - pathology | Neurofibrillary Tangles - metabolism | Aged, 80 and over | Female | Receptor, Nerve Growth Factor - metabolism | Neurofibrillary Tangles - pathology | Nerve Growth Factor - metabolism | Cholinergic Neurons - metabolism | Nerve Growth Factor - genetics | Nerve Tissue Proteins - genetics | Disease Progression | Receptors, Nerve Growth Factor - genetics | Nerve Tissue Proteins - metabolism | Basal Nucleus of Meynert - pathology | Nerve Growth Factors - genetics | Alzheimer Disease - metabolism | Aged | Receptors, Neurotransmitter - metabolism | Alzheimer Disease - genetics | Cholinergic Neurons - pathology | Tyrosine | Neurosciences | Dopamine | Neurons | Genes | Development and progression | Nerve growth factor | Neuropeptides | Glutamate | DNA microarrays | Analysis | Physiological aspects | GABA | Genetic research | Alzheimer's disease | Dopamine receptors
Journal Article
Molecular therapy, ISSN 1525-0016, 03/2015, Volume 23, Issue 3, pp. 423 - 433
Journal Article
PloS one, ISSN 1932-6203, 05/2014, Volume 9, Issue 5, p. e92596
...: Here we confirmed that the nerve growth factor receptor (CD271) is a crucial determinant of tumorigenicity, stem-like properties, heterogeneity and plasticity in melanoma cells... 
CANCER-CELLS | METASTATIC MELANOMA | FACTOR-BETA | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | EPIGENETIC REGULATION | NEURAL CREST | SOX10 | PROMOTES | TUMOR MICROENVIRONMENT | CD133 | Meta-Analysis as Topic | Antigens, CD - biosynthesis | Humans | Peptides - genetics | Male | Antigens, CD - genetics | Gene Knockdown Techniques | Neoplastic Stem Cells - metabolism | Melanoma - genetics | Nerve Tissue Proteins - biosynthesis | Neoplastic Stem Cells - pathology | Female | Neoplasm Proteins - genetics | SOXE Transcription Factors - biosynthesis | Glycoproteins - genetics | Melanoma - metabolism | Receptors, Nerve Growth Factor - biosynthesis | Neoplasm Proteins - biosynthesis | Melanoma - pathology | AC133 Antigen | Nerve Tissue Proteins - genetics | Receptors, Nerve Growth Factor - genetics | Glycoproteins - biosynthesis | Animals | Cell Line, Tumor | Mice, Inbred NOD | Biomarkers, Tumor - genetics | Mice | Biomarkers, Tumor - biosynthesis | SOXE Transcription Factors - genetics | Nerve growth factor | Drug resistance | Analysis | Genomics | Stem cells | Transcription factors | Laboratories | Genes | Genomes | Metastasis | Cell surface | Metastases | Genotype & phenotype | Heterogeneity | Rodents | Fibroblasts | Growth factors | Antigens | Colonies | Markers | Melanoma | Cell division | Tumorigenicity | Gene expression | Neural crest | Sox10 protein | Pathology | Properties (attributes) | Epigenetics | Neural stem cells | Surface markers | Cancer | Tumors
Journal Article
Nature (London), ISSN 1476-4687, 2010, Volume 466, Issue 7302, pp. 133 - 137
...Cancers derive by clonal progression to appear as abnormal growths. At diagnosis, they can be at a stage ranging from low risk of metastasis and probable cure... 
PROGENITORS | PROSPECTIVE IDENTIFICATION | MULTIDISCIPLINARY SCIENCES | CANCER STEM-CELLS | Bone and Bones - pathology | Neoplasm Transplantation | Neoplastic Stem Cells - cytology | Humans | Nerve Tissue Proteins - deficiency | Neural Crest - pathology | Receptors, Nerve Growth Factor - metabolism | Neoplasm Proteins - metabolism | DNA-Binding Proteins - deficiency | Neural Crest - metabolism | Neoplasm Metastasis | Lung Neoplasms - secondary | Neoplastic Stem Cells - metabolism | Antigens, Neoplasm - metabolism | Neoplastic Stem Cells - pathology | Skin Transplantation | Antigens, Neoplasm - analysis | Skin - pathology | Melanoma - metabolism | Melanoma-Specific Antigens | Neural Crest - cytology | Melanoma - pathology | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Neoplastic Stem Cells - transplantation | Receptors, Nerve Growth Factor - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Bone Transplantation | Neoplasm Proteins - analysis | Transplantation, Heterologous - pathology | Mice | Receptors, Nerve Growth Factor - deficiency | Care and treatment | Melanoma | Stem cells | Physiological aspects | Development and progression | Nerve growth factor | Genetic aspects | Growth factor receptors | Research | Risk factors | Transplants & implants | Skin & tissue grafts | Cancer | Tumors | Human | Enrichment | Antigens | Tumours | Bones | Transplantation
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 10/2016, Volume 11, Issue 10, p. e0165586
.... Signaling via the nerve growth factor (NGF) pathway between pancreatic cancer cells and the surrounding nerves has been implicated in PNI, and increased levels of these proteins have been correlated to poor prognosis... 
PAIN | PERINEURAL INVASION | MULTIDISCIPLINARY SCIENCES | NEUROTROPHINS | EXPRESSION | PARACRINE | PCR | Humans | Nerve Tissue Proteins - deficiency | Receptor, trkA - antagonists & inhibitors | Cell Survival - genetics | Cell Movement - genetics | Nervous System - pathology | Indoles - pharmacology | Neurites - drug effects | Cell Survival - drug effects | Cell Proliferation - genetics | Nerve Growth Factor - metabolism | Neoplasm Invasiveness | Pancreatic Neoplasms - pathology | Nerve Growth Factor - genetics | Neurites - metabolism | Signal Transduction - genetics | Gene Knockout Techniques | Nerve Tissue Proteins - genetics | Receptor, trkA - deficiency | Receptors, Nerve Growth Factor - genetics | Cell Movement - drug effects | Nervous System - drug effects | Signal Transduction - drug effects | Ganglia, Spinal - pathology | Cell Line, Tumor | Nerve Growth Factor - deficiency | Receptor, trkA - genetics | Cell Proliferation - drug effects | Receptors, Nerve Growth Factor - deficiency | Proteins | Nerve growth factor | Cancer cells | Cell proliferation | Cell culture | Genomics | Clinical trials | Antibodies | Paracrine signalling | Kinases | Signal transduction | Receptors | Pain | Dorsal root ganglia | Surgery | Penicillin | Nerves | TrkA protein | Inhibition | Conditioning | Growth factors | Medical research | Gene expression | Ganglia | Pancreatic cancer | Medical prognosis | Cell migration | Cancer | Media (culture)
Journal Article
Nature genetics, ISSN 1546-1718, 2001, Volume 28, Issue 2, pp. 131 - 138
Hypoxia stimulates angiogenesis through the binding of hypoxia-inducible factors to the hypoxia-response element in the vascular endothelial growth factor (Vegf) promotor... 
ANGIOGENESIS | VEGF | GENETICS & HEREDITY | FACTOR MESSENGER-RNA | SPINAL-CORD | FACTOR EXPRESSION | MECHANISMS | AMYOTROPHIC-LATERAL-SCLEROSIS | MODEL | INDUCIBLE FACTOR-1 | BINDING | Receptors, Vascular Endothelial Growth Factor | Sequence Deletion | Vascular Endothelial Growth Factor A | Endothelial Growth Factors - metabolism | Vascular Endothelial Growth Factors | Humans | Nerve Degeneration - physiopathology | Electrophysiology | Nerve Degeneration - genetics | Axons - physiology | Motor Neurons - pathology | Neuropilin-1 | Receptors, Growth Factor - genetics | Lymphokines - genetics | Lymphokines - metabolism | Response Elements - genetics | Binding Sites | Promoter Regions, Genetic | Motor Neurons - physiology | Amyotrophic Lateral Sclerosis - genetics | Muscular Atrophy - pathology | Muscular Atrophy - genetics | Receptor Protein-Tyrosine Kinases - metabolism | Nerve Tissue Proteins - genetics | Peripheral Nerves - pathology | Nerve Degeneration - pathology | Mice, Knockout | Nerve Tissue Proteins - metabolism | Amyotrophic Lateral Sclerosis - pathology | Animals | Muscle Contraction | Receptor Protein-Tyrosine Kinases - genetics | Endothelial Growth Factors - genetics | Spinal Cord - physiology | Muscle Fibers, Skeletal - pathology | Mice | Cell Hypoxia - genetics | Receptors, Growth Factor - metabolism | Gene mutations | Physiological aspects | Hypoxia | Genetic aspects | Research | Neovascularization | Health aspects | Vascular endothelial growth factor
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 7, p. e0177962
Adult neural crest stem-derived cells (NCSC) are of extraordinary high plasticity and promising candidates for use in regenerative medicine. Several locations... 
PROGENITOR CELLS | MESENCHYMAL STROMAL CELLS | STEM-CELLS | TRUNK | MULTIDISCIPLINARY SCIENCES | PERIODONTAL-LIGAMENT | ONTOGENY | GENERATION | SKIN-DERIVED PRECURSORS | DIFFERENTIATION | MELANOCYTES | RNA-Binding Proteins - genetics | Humans | Adipose Tissue - cytology | Melanocytes - metabolism | Neurons - cytology | Receptors, Nerve Growth Factor - metabolism | Neural Stem Cells - cytology | Schwann Cells - cytology | Neural Crest - metabolism | Adipose Tissue - metabolism | Mesenchymal Stromal Cells - cytology | Snail Family Transcription Factors - genetics | Melanocytes - cytology | Nestin - genetics | Adult | Female | Cell Differentiation | Neurons - metabolism | Dermis - cytology | Nuclear Proteins - genetics | Biomarkers - metabolism | SOX9 Transcription Factor - metabolism | Gene Expression | Neural Crest - cytology | Dermis - metabolism | Snail Family Transcription Factors - metabolism | Microinjections | Bone Marrow Cells - cytology | Neural Crest - growth & development | Mesenchymal Stromal Cells - metabolism | Nuclear Proteins - metabolism | Schwann Cells - metabolism | Transcription Factor Brn-3A - metabolism | Chick Embryo | Nerve Tissue Proteins - genetics | Receptors, Nerve Growth Factor - genetics | Nerve Tissue Proteins - metabolism | Nestin - metabolism | Animals | Transcription Factor Brn-3A - genetics | Twist-Related Protein 1 - genetics | Twist-Related Protein 1 - metabolism | Neural Stem Cells - metabolism | SOX9 Transcription Factor - genetics | Bone Marrow Cells - metabolism | Mesenchymal Stem Cell Transplantation | RNA-Binding Proteins - metabolism | Adipose tissues | Comparative analysis | Skin | Neurosciences | Nestin | Adipose tissue | Leukocyte migration | Laboratories | Sox9 protein | Dermis | Melanocytes | Nervous system | Human tissues | Regeneration (physiology) | Cell adhesion & migration | Msi1 protein | Plasticity (neural) | Ethics | Immunology | Pathways | Surgery | Bone marrow | Hair | Hematology | Neurons | Tissue engineering | Schwann cells | Brn-3 protein | Gene expression | Embryos | Neural crest | Medicine | Regeneration | Human performance | Stromal cells | Stem cells | Cell migration | Dental pulp
Journal Article