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Neuron (Cambridge, Mass.), ISSN 0896-6273, 2010, Volume 65, Issue 5, pp. 597 - 611
To investigate the role of microRNAs in regulating oligodendrocyte (OL) differentiation and myelination, we utilized transgenic mice in which microRNA... 
DEVBIO | MOLNEURO | TRANSCRIPTION FACTORS | NERVOUS-SYSTEM | GLIAL PROGENITOR-CELL | PROTEIN | CYCLE EXIT | NEURONS | GROWTH-FACTOR | IDENTIFICATION | MICRORNA EXPRESSION | LINEAGE | NEUROSCIENCES | Central Nervous System - metabolism | MicroRNAs - metabolism | Green Fluorescent Proteins - genetics | S100 Proteins - genetics | Oligodendroglia - drug effects | Basic Helix-Loop-Helix Transcription Factors - metabolism | Animals, Newborn | Basic Helix-Loop-Helix Transcription Factors - genetics | Receptor, Platelet-Derived Growth Factor alpha - metabolism | S100 Calcium Binding Protein beta Subunit | Oligonucleotide Array Sequence Analysis - methods | Rats | Mice, Transgenic | Oligodendrocyte Transcription Factor 2 | Myelin Proteins - genetics | Rats, Sprague-Dawley | Nerve Growth Factors - genetics | Mice | MicroRNAs - genetics | Myelin Proteins - metabolism | SOXD Transcription Factors - genetics | Optic Nerve - metabolism | Age Factors | SOXD Transcription Factors - metabolism | Gene Expression Regulation, Developmental - genetics | Sciatic Nerve - growth & development | Myelin Sheath - metabolism | Central Nervous System - growth & development | DNA-Binding Proteins - metabolism | Sciatic Nerve - metabolism | Oligodendroglia - physiology | Transfection | DEAD-box RNA Helicases - metabolism | Cell Differentiation - physiology | Optic Nerve - growth & development | Brain - cytology | 2',3'-Cyclic-Nucleotide Phosphodiesterases - genetics | Ribonuclease III - genetics | Ribonuclease III - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Profiling - methods | Transcription Factors - genetics | 2',3'-Cyclic-Nucleotide Phosphodiesterases - metabolism | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | DEAD-box RNA Helicases - genetics | Animals | Cell Differentiation - drug effects | Receptor, Platelet-Derived Growth Factor alpha - genetics | Stem Cells - drug effects | Stem Cells - physiology | Proteins | Genotype & phenotype | Gene expression | Experiments | Rodents | Cell cycle
Journal Article
Nature (London), ISSN 1476-4687, 2010, Volume 466, Issue 7302, pp. 133 - 137
...Cancers derive by clonal progression to appear as abnormal growths. At diagnosis, they can be at a stage ranging from low risk of metastasis and probable cure... 
PROGENITORS | PROSPECTIVE IDENTIFICATION | MULTIDISCIPLINARY SCIENCES | CANCER STEM-CELLS | Bone and Bones - pathology | Neoplasm Transplantation | Neoplastic Stem Cells - cytology | Humans | Nerve Tissue Proteins - deficiency | Neural Crest - pathology | Receptors, Nerve Growth Factor - metabolism | Neoplasm Proteins - metabolism | DNA-Binding Proteins - deficiency | Neural Crest - metabolism | Neoplasm Metastasis | Lung Neoplasms - secondary | Neoplastic Stem Cells - metabolism | Antigens, Neoplasm - metabolism | Neoplastic Stem Cells - pathology | Skin Transplantation | Antigens, Neoplasm - analysis | Skin - pathology | Melanoma - metabolism | Melanoma-Specific Antigens | Neural Crest - cytology | Melanoma - pathology | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Neoplastic Stem Cells - transplantation | Receptors, Nerve Growth Factor - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Bone Transplantation | Neoplasm Proteins - analysis | Transplantation, Heterologous - pathology | Mice | Receptors, Nerve Growth Factor - deficiency | Care and treatment | Melanoma | Stem cells | Physiological aspects | Development and progression | Nerve growth factor | Genetic aspects | Growth factor receptors | Research | Risk factors | Transplants & implants | Skin & tissue grafts | Cancer | Tumors | Human | Enrichment | Antigens | Tumours | Bones | Transplantation
Journal Article
Circulation research, ISSN 0009-7330, 04/2010, Volume 106, Issue 7, pp. 1275 - 1284
RATIONALE:Nerve growth factor (NGF) promotes angiogenesis and cardiomyocyte survival, which are both desirable for postinfarction myocardial healing... 
Myocardial infarction | Angiogenesis | Gene therapy | Apoptosis | SURVIVAL | MIGRATION | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTEIN | angiogenesis | myocardial infarction | BONE-MARROW | gene therapy | apoptosis | FOXO TRANSCRIPTION FACTORS | STEM-CELL | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | Genetic Therapy | Myocardial Infarction - genetics | Nerve Growth Factor - biosynthesis | Rats, Wistar | Ventricular Function, Left | Humans | Middle Aged | Male | Recombinant Proteins - biosynthesis | Proto-Oncogene Proteins c-kit - metabolism | Stem Cells - metabolism | Case-Control Studies | Myocardial Infarction - therapy | RNA, Messenger - biosynthesis | Transfection | Time Factors | Autopsy | Forkhead Transcription Factors - metabolism | Regeneration - genetics | Myocardial Infarction - pathology | Myocardium - metabolism | Female | Myocardial Infarction - physiopathology | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Coronary Circulation | Stem Cell Factor - metabolism | Signal Transduction | Endothelial Cells - metabolism | Cells, Cultured | Rats | Myocardium - pathology | Nerve Growth Factor - genetics | Myocardial Infarction - metabolism | Animals | Receptor, trkA - metabolism | Stem Cells - pathology | Aged | Mice | Endothelial Cells - pathology | Forkhead Box Protein O3 | Neovascularization, Physiologic
Journal Article
Journal Article
Clinical cancer research, ISSN 1078-0432, 04/2011, Volume 17, Issue 7, pp. 1741 - 1752
Purpose: Given that nerve growth factor has previously been shown to be involved in breast cancer progression, we have tested here the hypothesis that the other neurotrophins (NT... 
SIGNALING PATHWAYS | IN-VITRO | ONCOLOGY | PROSTATE-CANCER | FACTOR RECEPTORS | TRK RECEPTORS | PROLIFERATION | NEUROBLASTOMA-CELLS | CARCINOMA | NERVE GROWTH-FACTOR | P75 | Neoplasm Transplantation | RNA, Small Interfering - genetics | Carcinoma, Lobular - pathology | Receptor, Nerve Growth Factor - genetics | Humans | Carcinoma, Intraductal, Noninfiltrating - metabolism | Gene Expression Regulation, Neoplastic | Carcinoma, Medullary - metabolism | Neurotrophin 3 - genetics | Transplantation, Heterologous | Brain-Derived Neurotrophic Factor - immunology | Nerve Growth Factors - metabolism | Receptor, trkB - genetics | Breast Neoplasms - metabolism | Nerve Growth Factors - pharmacology | Brain-Derived Neurotrophic Factor - pharmacology | Receptor, trkC - genetics | Carcinoma, Ductal, Breast - pathology | Female | Transcription, Genetic | Antineoplastic Agents - pharmacology | Brain-Derived Neurotrophic Factor - metabolism | Carcinoma, Ductal, Breast - metabolism | Receptor, Nerve Growth Factor - metabolism | Receptor, Nerve Growth Factor - immunology | Cell Survival | Carcinoma, Medullary - pathology | Lymphatic Metastasis | Nerve Growth Factors - immunology | Mice, SCID | Antibodies - pharmacology | Animals | Carcinoma, Intraductal, Noninfiltrating - pathology | Neurotrophin 3 - metabolism | Breast Neoplasms - pathology | Cell Line, Tumor | Carcinoma, Lobular - metabolism | Mice | Receptor, trkB - metabolism | Immunologic Factors - pharmacology | Receptor, trkC - metabolism | Apoptosis
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2014, Volume 111, Issue 48, pp. 17272 - 17277
.... However, its actions on the peripheral nervous system have been less characterized. We examined whether VEGF-B mediates peripheral nerve repair... 
Sensation | Debridement | Cornea | Neurites | Neurons | Nerves | Peripheral nerves | Mice | Epithelium | Nerve regeneration | Nerve injury | Neuronal growth | VEGF-B | neuronal growth | cornea | nerve injury | ANGIOGENESIS | PERMEABILITY | ENDOTHELIAL-GROWTH-FACTOR | MULTIDISCIPLINARY SCIENCES | ISCHEMIA | DEFICIENT MICE | KNOCKOUT MICE | RECOVERY | DISPLAY | PATHWAY | DISEASE | Neurites - physiology | Receptors, Notch - metabolism | Nerve Regeneration - physiology | Cell Survival - genetics | Phosphatidylinositol 3-Kinases - metabolism | Peripheral Nerves - drug effects | Cornea - drug effects | Peripheral Nerve Injuries - physiopathology | Neurons - physiology | Neurons - metabolism | Neurons - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Neurites - drug effects | Nerve Regeneration - genetics | Cell Survival - drug effects | Trigeminal Ganglion - cytology | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Peripheral Nerves - pathology | Blotting, Western | Mice, Knockout | Peripheral Nerves - physiopathology | Vascular Endothelial Growth Factor B - pharmacology | Animals | Signal Transduction - drug effects | Vascular Endothelial Growth Factor B - metabolism | Nerve Regeneration - drug effects | Cornea - innervation | Vascular Endothelial Growth Factor B - genetics | Cell Proliferation - drug effects | Cornea - physiopathology | Peripheral Nerve Injuries - genetics | Microscopy, Fluorescence | Sensory receptors | Vascular endothelial growth factor | Health aspects | Biological Sciences
Journal Article
Osteoarthritis and cartilage, ISSN 1063-4584, 2015, Volume 23, Issue 3, pp. 478 - 486
Journal Article
International journal of molecular sciences, ISSN 1422-0067, 2017, Volume 18, Issue 5, p. 1028
.... A key mediator in cross-talk between the two systems is nerve growth factor (NGF), which can influence both neuronal cell function and immune cell activity... 
Innate immunity | Neuropeptides | Neurotransmitters | Sensory sympathetic and parasympathetic neurons | Inflammatory diseases | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | SYSTEMIC-LUPUS-ERYTHEMATOSUS | MESSENGER-RNA EXPRESSION | PRIMARY AFFERENT NEURONS | BIOCHEMISTRY & MOLECULAR BIOLOGY | neuropeptides | inflammatory diseases | FACTOR ENHANCES SURVIVAL | sensory sympathetic and parasympathetic neurons | NECROSIS-FACTOR-ALPHA | ALLERGIC AIRWAY INFLAMMATION | HUMAN B-LYMPHOCYTES | CHEMISTRY, MULTIDISCIPLINARY | NERVE GROWTH-FACTOR | innate immunity | neurotransmitters | PRIMARY SENSORY NEURONS | Adaptive Immunity - physiology | Nerve Growth Factor - metabolism | Cytokines - metabolism | Humans | Rats | Neuropeptides - metabolism | Inflammation - immunology | Receptors, Nerve Growth Factor - metabolism | Nerve Tissue Proteins - metabolism | Animals | Receptor, trkA - metabolism | Immunity, Innate - physiology | Arthritis - immunology | Cell Differentiation | Mice | Neurons - metabolism | Innervation | Homeostasis | Inflammatory response | Nerve growth factor | Nervous system | Activation | Arthritis | Dynamical systems | Receptors | Control | Functional anatomy | Pathways | TrkA protein | TrkA receptors | Damage | Immune system | Immune response | Neurons | Tropomyosin | Inflammation | Maintenance | Patients | Diseases | Nerve growth factor receptors | Differentiation
Journal Article
Journal Article
Nature (London), ISSN 1476-4687, 2004, Volume 427, Issue 6977, pp. 843 - 848
Journal Article