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1. Full Text Control of Immunity by the TNFR-related molecule OX40 (CD134)
by Croft, Michael
Annual Review of Immunology, ISSN 0732-0582, 04/2010, Volume 28, Issue 1, pp. 57 - 78
TNFR/TNF superfamily members can control diverse aspects of immune function. Research over the past 10 years has shown that one of the most important and... 
Autoimmunity | Infectious disease | T cells | Costimulation | OX40L | Vaccination | OX40-OX40 LIGAND INTERACTION | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | PLASMACYTOID-DENDRITIC-CELLS | HUMAN NK CELLS | IMMUNOLOGY | GROWTH-FACTOR RECEPTOR | TUMOR-NECROSIS-FACTOR | T-CELL RESPONSES | IL-1 RECEPTOR ANTAGONIST | costimulation | autoimmunity | infectious disease | IN-VIVO | VASCULAR ENDOTHELIAL-CELLS | vaccination | Receptors, OX40 - metabolism | Animals | OX40 Ligand - immunology | Signal Transduction | T-Lymphocytes - metabolism | OX40 Ligand - metabolism | Humans | Cell Communication | Killer Cells, Natural - immunology | T-Lymphocytes - immunology | Killer Cells, Natural - metabolism | Receptors, OX40 - immunology | Care and treatment | Physiological aspects | Antigen presenting cells | Research | Properties | Identification and classification | Immune system | Cancer | Immunological research
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2. Full Text OX40 costimulation turns off Foxp3 + Tregs
by Vu, Minh Diem and Xiao, Xiang and Gao, Wenda and Degauque, Nicolas and Chen, Ming and Kroemer, Alexander and Killeen, Nigel and Ishii, Naoto and Li, Xian Chang
Blood, ISSN 0006-4971, 10/2007, Volume 110, Issue 7, pp. 2501 - 2510
OX40 is a recently identified T-cell costimulatory molecule that belongs to the TNF/TNFR superfamily. OX40 can be expressed by both activated T effector cells... 
MURINE MODEL | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | TRANSPLANTATION TOLERANCE | CD4(+)CD25(+) | TRANSCRIPTION FACTOR FOXP3 | IN-VIVO | REGULATORY T-CELLS | IMMUNOLOGICAL SELF-TOLERANCE | TNFR FAMILY | HEMATOLOGY | VERSUS-HOST-DISEASE | T-Lymphocytes, Regulatory - metabolism | Lymphocyte Activation - immunology | Receptors, OX40 - metabolism | Animals | Forkhead Transcription Factors - metabolism | Mice | Forkhead Transcription Factors - genetics | Interferon-gamma - biosynthesis | Receptors, OX40 - immunology | Mice, Knockout | T-Lymphocytes, Regulatory - immunology | Immunobiology
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3. Full Text Ox40+ regulatory T cells in cutaneous squamous cell carcinoma suppress effector T-cell responses and associate with metastatic potential
by Lai, Chester and August, Suzannah and Albibas, Amel and Behar, Ramnik and Cho, Shin-Young and Polak, Marta E and Theaker, Jeffrey and Macleod, Amanda S and French, Ruth R and Glennie, Martin J and Al-Shamkhani, Aymen and Healy, Eugene
Clinical Cancer Research, ISSN 1078-0432, 08/2016, Volume 22, Issue 16, pp. 4236 - 4248
Purpose: Cutaneous squamous cell carcinoma (cSCC) is the most common In-flan cancer with metastatic potential. Despite T cells accumulating around cSCCs, these... 
TUMOR-INFILTRATING LYMPHOCYTES | INDIVIDUALS | RECRUITMENT | CANCER-PATIENTS | MELANOMA | ONCOLOGY | LYMPH-NODE | SKIN | EXPRESSION | CD134 | PROGRESSION | Immunohistochemistry | Skin Neoplasms - pathology | T-Lymphocyte Subsets - immunology | T-Lymphocytes, Regulatory - metabolism | Receptors, OX40 - agonists | Skin Neoplasms - immunology | Immunomodulation | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Immunophenotyping | Carcinoma, Squamous Cell - immunology | Skin Neoplasms - metabolism | T-Lymphocytes, Regulatory - immunology | Lymphocyte Activation - immunology | Neoplasm Metastasis | Phenotype | Receptors, OX40 - metabolism | T-Lymphocyte Subsets - metabolism | Biomarkers | Lymphocyte Count | Immunologic Memory | Lymphocytes, Tumor-Infiltrating - metabolism | Lymphocytes, Tumor-Infiltrating - immunology
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4. Full Text OX40 and IL‐7 play synergistic roles in the homeostatic proliferation of effector memory CD4+ T cells
by Yamaki, Satoshi and Ine, Shouji and Kawabe, Takeshi and Okuyama, Yuko and Suzuki, Nobu and Soroosh, Pejman and Mousavi, Seyed Fazlollah and Nagashima, Hiroyuki and Sun, Shu‐lan and So, Takanori and Sasaki, Takeshi and Harigae, Hideo and Sugamura, Kazuo and Kudo, Hironori and Wada, Motoshi and Nio, Masaki and Ishii, Naoto
European Journal of Immunology, ISSN 0014-2980, 10/2014, Volume 44, Issue 10, pp. 3015 - 3025
T‐cell homeostasis preserves the numbers, the diversity and functional competence of different T‐cell subsets that are required for adaptive immunity. Naïve... 
OX40 | Memory CD4+ T cells | Th17 | Homeostatic proliferation | T cells | Memory CD4 | OX40-OX40 LIGAND INTERACTION | IMMUNITY | Memory CD4(+) T cells | MOLECULE OX40 | PHENOTYPE CELLS | IMMUNOLOGY | NAIVE | CUTTING EDGE | IN-VIVO | EXPANSION | MICE | EXPRESSION | T-Lymphocyte Subsets - immunology | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Cell Proliferation - physiology | Homeostasis - immunology | Mice, Transgenic | Adoptive Transfer | Receptors, OX40 - immunology | CD4-Positive T-Lymphocytes - immunology | Receptors, OX40 - metabolism | Animals | Flow Cytometry | Interleukin-7 - immunology | T-Lymphocyte Subsets - metabolism | Mice | Immunologic Memory - immunology | Interleukin-7 - metabolism | T cells
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5. Full Text OX40 Ligand Contributes to Human Lupus Pathogenesis by Promoting T Follicular Helper Response
by Jacquemin, Clément and Schmitt, Nathalie and Contin-Bordes, Cécile and Liu, Yang and Narayanan, Priya and Seneschal, Julien and Maurouard, Typhanie and Dougall, David and Davizon, Emily Spence and Dumortier, Hélène and Douchet, Isabelle and Raffray, Loïc and Richez, Christophe and Lazaro, Estibaliz and Duffau, Pierre and Truchetet, Marie-Elise and Khoryati, Liliane and Mercié, Patrick and Couzi, Lionel and Merville, Pierre and Schaeverbeke, Thierry and Viallard, Jean-François and Pellegrin, Jean-Luc and Moreau, Jean-François and Muller, Sylviane and Zurawski, Sandy and Coffman, Robert L and Pascual, Virginia and Ueno, Hideki and Blanco, Patrick
Immunity, ISSN 1074-7613, 06/2015, Volume 42, Issue 6, pp. 1159 - 1170
Increased activity of T follicular helper (Tfh) cells plays a major pathogenic role in systemic lupus erythematosus (SLE). However, the mechanisms that cause... 
MESSENGER-RNAS | B-CELL | DENDRITIC CELLS | RECEPTOR | INDUCTION | IMMUNOLOGY | CELL-DIFFERENTIATION | ICOS | EXPRESSION | IL-21 | ERYTHEMATOSUS | OX40 Ligand - metabolism | Humans | Middle Aged | Male | Molecular Targeted Therapy | Young Adult | Receptors, OX40 - metabolism | Antigen Presentation | Myeloid Cells - immunology | T-Lymphocytes, Helper-Inducer - immunology | Lupus Erythematosus, Systemic - immunology | Adult | Female | Cell Differentiation | Toll-Like Receptor 7 - metabolism | Cytokines - metabolism | RNA - immunology | Signal Transduction | Lymphocyte Activation | Cells, Cultured | Disease Progression | B-Lymphocytes - immunology | Adolescent | Immunologic Memory | Aged | Inducible T-Cell Co-Stimulator Protein - metabolism | Lupus | Systemic lupus erythematosus | B cells | RNA | T cells | Studies | Antigens | Pediatrics | Genotype & phenotype | Statistical analysis | Pathogenesis | Rodents | Ligands | Autoimmune diseases | Experiments
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6. Full Text Timing of PD-1 blockade is critical to effective combination immunotherapy with anti-OX40
by Messenheimer, David J and Jensen, Shawn M and Afentoulis, Michael E and Wegmann, Keith W and Feng, Zipei and Friedman, David J and Gough, Michael J and Urba, Walter J and Fox, Bernard A
Clinical Cancer Research, ISSN 1078-0432, 10/2017, Volume 23, Issue 20, pp. 6165 - 6177
Purpose: Antibodies specific for inhibitory checkpoints PD-1 and CTLA-4 have shown impressive results against solid tumors. This has fueled interest in novel... 
ONCOLOGY | THERAPEUTIC-EFFICACY | IMMUNE-RESPONSES | POOR-PROGNOSIS | CD4(+) T-CELLS | COSTIMULATORY MOLECULE | ESTABLISHED MELANOMA | OVARIAN-CANCER | HUMAN BREAST-CANCER | LYMPHOPENIC HOSTS | ANTITUMOR EFFICACY | T-Lymphocyte Subsets - immunology | Immunotherapy - methods | Humans | Programmed Cell Death 1 Receptor - antagonists & inhibitors | Receptors, OX40 - metabolism | Time Factors | T-Lymphocyte Subsets - drug effects | Female | Lymphocytes, Tumor-Infiltrating - metabolism | Molecular Targeted Therapy - methods | Cytokines - blood | Disease Models, Animal | Cytokines - metabolism | Receptors, Antigen, T-Cell - metabolism | Programmed Cell Death 1 Receptor - metabolism | Immunophenotyping | Mice, Transgenic | Survival Rate | Treatment Outcome | Lymphocytes, Tumor-Infiltrating - drug effects | Antineoplastic Agents, Immunological - pharmacology | Xenograft Model Antitumor Assays | Animals | Antineoplastic Agents, Immunological - therapeutic use | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | T-Lymphocyte Subsets - metabolism | Lymphocyte Count | Mice | Lymphocytes, Tumor-Infiltrating - immunology | Receptors, OX40 - antagonists & inhibitors | Cell proliferation | Animal models | PD-1 protein | CD8 antigen | Clinical trials | Antibodies | Lymphocytes T | Contraction | Anticancer properties | Receptors | CTLA-4 protein | Cell activation | Cell growth | Acquired immune deficiency syndrome--AIDS | Lymphocytes | Immunotherapy | Medical research | Cytokines | T cell receptors | CD4 antigen | Immune checkpoint | Experimental design | Tumor necrosis factor | Exhaustion | Antitumor activity | Solid tumors | Tumors | Cancer | Combination immunotherapy | T cells | Costimulation | Checkpoint blockade
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7. Full Text OX40 signaling favors the induction of T(H)9 cells and airway inflammation
by Xiao, X and Balasubramanian, S and Liu, WT and Chu, XF and Wang, HB and Taparowsky, EJ and Fu, YX and Choi, Y and Walsh, MC and Li, XC
NATURE IMMUNOLOGY, ISSN 1529-2908, 10/2012, Volume 13, Issue 10, pp. 981 - 990
The mechanisms that regulate the T(H)9 subset of helper T cells and diseases mediated by T(H)9 cells remain poorly defined. Here we found that the... 
TGF-BETA | NUCLEAR-FACTOR | FAMILY-MEMBERS | MOLECULE OX40 | MAST-CELLS | REGULATORY T-CELLS | LIGAND | IMMUNOLOGY | NF-KAPPA-B | EXPRESSION | IL-9 | TNF Receptor-Associated Factor 6 - biosynthesis | OX40 Ligand - metabolism | Humans | NF-kappa B - metabolism | Receptors, OX40 - immunology | Interleukin-9 - biosynthesis | Signal Transduction - immunology | Inflammation - metabolism | Receptors, OX40 - metabolism | T-Lymphocytes, Helper-Inducer - immunology | Proto-Oncogene Proteins - immunology | Interleukin-9 - metabolism | Protein-Serine-Threonine Kinases - metabolism | Respiratory System - immunology | Trans-Activators - immunology | Proto-Oncogene Proteins - metabolism | Transforming Growth Factor beta - immunology | T-Lymphocytes, Helper-Inducer - metabolism | Inflammation - immunology | Protein-Serine-Threonine Kinases - biosynthesis | Animals | OX40 Ligand - immunology | TNF Receptor-Associated Factor 6 - metabolism | Trans-Activators - metabolism | Mice | CD4 Antigens - biosynthesis | Transforming Growth Factor beta - metabolism
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8. Full Text Small‐molecule modulators of the OX40–OX40 ligand co‐stimulatory protein–protein interaction
by Song, Yun and Margolles‐Clark, Emilio and Bayer, Allison and Buchwald, Peter
British Journal of Pharmacology, ISSN 0007-1188, 11/2014, Volume 171, Issue 21, pp. 4955 - 4969
Background and Purpose The OX40–OX40L protein–protein interaction (PPI) is an important cell‐surface signalling co‐stimulatory regulator within the TNFR... 
IMMUNITY | NOD MICE | INHIBITION | FOXP3(+) TREGS | OX40 CD134 | PHARMACOLOGY | REGULATORY T-CELLS | PREVENTION | PHARMACOLOGY & PHARMACY | TNF | CUTTING EDGE | Receptors, OX40 - agonists | Receptors, OX40 - genetics | OX40 Ligand - genetics | OX40 Ligand - metabolism | Humans | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Mice, Transgenic | Forkhead Transcription Factors - genetics | Receptors, OX40 - metabolism | Animals | HEK293 Cells | Cell Line, Tumor | Receptors, OX40 - antagonists & inhibitors | Sensors | T cells | Transforming growth factors | Protein-protein interactions | Proteins | Research Papers
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9. Full Text Inherited human OX40 deficiency underlying classic kaposi sarcoma of childhood
by Byun, Minji and Ma Cindy S, C.S and Akçay, Arzu and Pedergnana, Vincent and Palendira, Umaimainthan and Myoung, Jinjong and Avery, Danielle T and Liu, Yifang and Abhyankar, Avinash and Lorenzo, Lazaro and Schmidt, Monika and Lim, Hye Kyung and Cassar, Olivier and Migaud, Melanie and Rozenberg, Flore and Canpolat, Nur and Aydogan, Gönül and Fleckenstein, Bernhard and Bustamante, Jacinta and Picard, Capucine and Gessain, Antoine and Jouanguy, Emmanuelle and Cesarman, Ethel and Olivier, Martin and Gros, Philippe and Abel, Laurent and Croft, Michael and Tangye, Stuart G and Casanova, Jean-Laurent
Journal of Experimental Medicine, ISSN 0022-1007, 2013, Volume 210, Issue 9, pp. 1743 - 1759
Kaposi sarcoma (KS), a human herpes virus 8 (HHV-8; also called KSHV)-induced endothelial tumor, develops only in a small fraction of individuals infected with... 
MEDICINE, RESEARCH & EXPERIMENTAL | HERPESVIRUS | B-CELL | OX40/GP34 SYSTEM | KALA-AZAR | VASCULAR ENDOTHELIAL-CELLS | IMMUNOLOGY | LEISHMANIA-DONOVANI | EPIDEMIOLOGY | PRIMARY HUMAN-HERPESVIRUS-8 INFECTION | CD8 T-CELLS | CHILDREN | Receptors, OX40 - genetics | Sarcoma, Kaposi - blood | Humans | Molecular Sequence Data | Cysteine - genetics | Young Adult | Receptors, OX40 - metabolism | Base Sequence | HEK293 Cells | Inheritance Patterns - genetics | Adult | Female | Cell Membrane - metabolism | Sarcoma, Kaposi - immunology | Child | Immunologic Memory - immunology | Amino Acid Sequence | Antibody Formation - immunology | Receptors, OX40 - deficiency | Mutant Proteins - metabolism | Mutation - genetics | Sarcoma, Kaposi - pathology | Receptors, OX40 - chemistry | Homozygote | T-Lymphocyte Subsets - metabolism | Intracellular Space - metabolism | Mutant Proteins - chemistry | Age of Onset | Lymphocyte Count | Amino Acid Substitution - genetics | Sarcoma, Kaposi - genetics | Life Sciences | Genetics | Human genetics | Cancer
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10. Full Text The Ox40/Ox40 ligand pathway promotes pathogenic Th cell responses, plasmablast accumulation, and lupus nephritis in NZB/W F1 mice
by Sitrin, Jonathan and Suto, Eric and Wuster, Arthur and Eastham-Anderson, Jeffrey and Kim, Jeong M and Austin, Cary D and Lee, Wyne P and Behrens, Timothy W
Journal of Immunology, ISSN 0022-1767, 08/2017, Volume 199, Issue 4, pp. 1238 - 1249
Ox40 ligand (Ox40L) locus genetic variants are associated with the risk for systemic lupus erythematosus (SLE); however, it is unclear how Ox40L contributes to... 
NOD MICE | FOLLICULAR-HELPER-CELLS | B-CELL | OX40 LIGAND | CLONAL EXPANSION | IN-VIVO | CHEMOKINE RECEPTOR | MURINE LUPUS | IL-21 RECEPTOR | IMMUNOLOGY | SYSTEMIC AUTOIMMUNITY | Autoimmunity | Kidney - pathology | Membrane Glycoproteins - metabolism | Antibodies, Antinuclear - immunology | Humans | Kidney - immunology | CD4-Positive T-Lymphocytes - immunology | Receptors, OX40 - metabolism | T-Lymphocytes, Helper-Inducer - immunology | Female | Antibodies, Monoclonal - immunology | Disease Models, Animal | T-Lymphocytes, Helper-Inducer - metabolism | Proteinuria - immunology | Signal Transduction | CD4-Positive T-Lymphocytes - metabolism | Tumor Necrosis Factors - metabolism | Lupus Nephritis - immunology | Kidney Glomerulus - pathology | Mice, Inbred NZB | Animals | Lupus Nephritis - physiopathology | Mice | Kidney Glomerulus - immunology | Plasma Cells - immunology | Immunoglobulin M | Nephritis | Syngeneic grafts | Pathogenesis | Medical services | Activation | Lymphocytes T | Lupus nephritis | Cell activation | Systemic lupus erythematosus | Lymphocytes | Fusion protein | Spleen | Lupus | Medical treatment | Inflammation | Gene expression | CD4 antigen | Genetic variance | Fc receptors | Chronic conditions | Ox40L protein | Tumor necrosis factor | Ligands | Interferon | Autoimmune diseases | Proteinuria
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11. Full Text OX40 is required for regulatory T cell-mediated control of colitis
by Griseri, Thibault and Asquith, Mark and Thompson, Claire and Powrie, Fiona
Journal of Experimental Medicine, ISSN 0022-1007, 04/2010, Volume 207, Issue 4, pp. 699 - 709
The immune response in the gastrointestinal tract is a tightly controlled balance between effector and regulatory cell responses. Here, we have investigated... 
MEDICINE, RESEARCH & EXPERIMENTAL | RESPONSES | GITR | INFLAMMATORY-BOWEL-DISEASE | DENDRITIC CELLS | SCID MICE | FAMILY-MEMBERS | IN-VIVO | EXPANSION | ANTIGEN-PRESENTING CELLS | IMMUNOLOGY | EXPRESSION | T-Lymphocyte Subsets - immunology | Tumor Necrosis Factors - genetics | T-Lymphocytes, Regulatory - metabolism | Receptors, OX40 - genetics | Cell Proliferation | T-Lymphocyte Subsets - cytology | Gene Expression - genetics | Cell Count | Colitis - pathology | Colon - immunology | Interferon-gamma - metabolism | Integrins - metabolism | Lymphoid Tissue - immunology | T-Lymphocytes, Regulatory - immunology | CD4-Positive T-Lymphocytes - immunology | Cell Movement - immunology | Intestinal Mucosa - cytology | Lymphocyte Activation - immunology | Receptors, OX40 - metabolism | Forkhead Transcription Factors - metabolism | Intestinal Mucosa - immunology | Gene Expression - immunology | Colitis - immunology | Disease Models, Animal | Colon - pathology | CD4-Positive T-Lymphocytes - cytology | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Mice, Transgenic | Forkhead Transcription Factors - genetics | T-Lymphocyte Subsets - transplantation | Colon - metabolism | Homeodomain Proteins - genetics | Membrane Glycoproteins - genetics | Mice, Knockout | Interleukin-17 - metabolism | Lymphoid Tissue - cytology | Animals | Apoptosis - immunology | CD4-Positive T-Lymphocytes - transplantation | T-Lymphocyte Subsets - metabolism | Colitis - metabolism | Leukocyte Common Antigens - genetics | Mice | T-Lymphocytes, Regulatory - transplantation | Brief Definitive Report
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12. Full Text OX40 regulates pressure overload-induced cardiac hypertrophy and remodelling via CD4+ T-cells
by Wu, Qing-Qing and Yuan, Yuan and Jiang, Xiao-Han and Xiao, Yang and Yang, Zheng and Ma, Zhen-Guo and Liao, Hai-Han and Liu, Yuan and Chang, Wei and Bian, Zhou-Yan and Tang, Qi-Zhu
Clinical Science, ISSN 0143-5221, 2016, Volume 130, Issue 22, pp. 2061 - 2071
OX40, which belongs to the tumour necrosis factor (TNF)-receptor family, is a costimulatory receptor that can potentiate T-cell receptor signalling on the... 
OX40 | Pressure overload | CD4 | T-lymphocytes | Cardiac remodelling | Ang II | MEDICINE, RESEARCH & EXPERIMENTAL | cardiac remodelling | ACTIVATION | MYOCARDIAL-INFARCTION | CD4(+) T-lymphocytes | MICE | LIGAND | pressure overload | EXPRESSION | LYMPHOCYTES | Receptors, OX40 - genetics | Cell Proliferation | Humans | CD4-Positive T-Lymphocytes - metabolism | Cardiomegaly - physiopathology | Rats | Cardiomegaly - pathology | Mice, Knockout | Receptors, OX40 - metabolism | Animals | Myocytes, Cardiac - metabolism | Mice | Cardiomegaly - genetics | Cardiomegaly - metabolism
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13. Full Text Roquin Paralogs 1 and 2 Redundantly Repress the Icos and Ox40 Costimulator mRNAs and Control Follicular Helper T Cell Differentiation
by Vogel, Katharina U and Edelmann, Stephanie L and Jeltsch, Katharina M and Bertossi, Arianna and Heger, Klaus and Heinz, Gitta A and Zöller, Jessica and Warth, Sebastian C and Hoefig, Kai P and Lohs, Claudia and Neff, Frauke and Kremmer, Elisabeth and Schick, Joel and Repsilber, Dirk and Geerlof, Arie and Blum, Helmut and Wurst, Wolfgang and Heikenwälder, Mathias and Schmidt-Supprian, Marc and Heissmeyer, Vigo
Immunity, ISSN 1074-7613, 04/2013, Volume 38, Issue 4, pp. 655 - 668
The Roquin-1 protein binds to messenger RNAs (mRNAs) and regulates gene expression posttranscriptionally. A single point mutation in Roquin-1, but not gene... 
B-CELLS | PROTEIN | TRANSCRIPTION FACTOR IRF4 | BCL6 | LEADS | GENERATION | KAPPA-B | IMMUNOLOGY | PHOSPHOINOSITIDE 3-KINASE | EXPRESSION | SYSTEMIC AUTOIMMUNITY | Receptors, OX40 - genetics | Humans | Mice, Inbred C57BL | Ubiquitin-Protein Ligases - metabolism | Repressor Proteins - genetics | RNA, Messenger - metabolism | Mice, Knockout | Cell Differentiation - genetics | Lymphocyte Activation - genetics | Receptors, OX40 - metabolism | Animals | Mice, Mutant Strains | T-Lymphocytes, Helper-Inducer - immunology | HEK293 Cells | Inducible T-Cell Co-Stimulator Protein - genetics | Protein Binding | Mice | Inducible T-Cell Co-Stimulator Protein - metabolism | Ubiquitin-Protein Ligases - genetics | Repressor Proteins - metabolism | CD4 Antigens - metabolism | T cells | Messenger RNA | Proteins | Transcription factors | Rodents | Ligands | RNA polymerase | Gene expression | Chromatography | Deoxyribonucleic acid--DNA | Cell adhesion & migration | Biological Sciences | Naturvetenskap | Natural Sciences | Bioinformatik och systembiologi | Bioinformatics and Systems Biology | Biologiska vetenskaper
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