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Journal of Steroid Biochemistry and Molecular Biology, ISSN 0960-0760, 2010, Volume 118, Issue 3, pp. 177 - 187
Aldo-keto reductase (AKR) 1C3 (type 5 17β-hydroxysteroid dehydrogenase and prostaglandin F synthase), may stimulate proliferation via steroid hormone and... 
Estrogen receptor | Peroxisome proliferator activated receptor γ | Prostaglandin F synthase | Prostaglandin D 2 | 17β-Hydroxysteroid dehydrogenase | Prostaglandin D | SIGNALING PATHWAYS | 17-BETA-HYDROXYSTEROID DEHYDROGENASES | BIOCHEMISTRY & MOLECULAR BIOLOGY | AKR1C3 | PROLIFERATOR-ACTIVATED RECEPTOR | RISK | Prostaglandin D-2 | CELECOXIB | ENDOCRINOLOGY & METABOLISM | PROGNOSTIC MARKER | F SYNTHASE | INHIBITORS | 17 beta-Hydroxysteroid dehydrogenase | AROMATASE | Peroxisome proliferator activated receptor | gamma | Up-Regulation | Prostaglandins - pharmacology | Humans | Gene Expression Regulation, Neoplastic | Dinoprost - metabolism | Hydroxyprostaglandin Dehydrogenases - metabolism | Androstenedione - metabolism | Breast Neoplasms - metabolism | Testosterone - metabolism | Breast Neoplasms - enzymology | Transfection | Prostaglandin D2 - metabolism | Prostaglandin D2 - analogs & derivatives | 3-Hydroxysteroid Dehydrogenases - metabolism | Female | 5-alpha-Dihydroprogesterone - metabolism | Dinoprost - pharmacology | Etiocholanolone - metabolism | Estradiol - pharmacology | Dihydrotestosterone - metabolism | Recombinant Proteins - metabolism | Estradiol - metabolism | Estrone - pharmacology | Gonadal Steroid Hormones - metabolism | Biocatalysis | Etiocholanolone - analogs & derivatives | Gonadal Steroid Hormones - pharmacology | Progesterone - analogs & derivatives | Recombinant Proteins - genetics | 20-alpha-Dihydroprogesterone - metabolism | Androsterone - metabolism | Hydroxyprostaglandin Dehydrogenases - genetics | Breast Neoplasms - genetics | Aldo-Keto Reductase Family 1 Member C3 | Estrone - metabolism | Progesterone - metabolism | Cell Line, Tumor | Cell Proliferation - drug effects | Prostaglandin D2 - pharmacology | Ketosteroids - metabolism | Kinetics | 3-Hydroxysteroid Dehydrogenases - genetics | Prostaglandins - metabolism | Physiological aspects | Testosterone | Breast cancer | Progesterone | Estradiol | Synthetic prostaglandins F | Index Medicus
Journal Article
Cancer Research, ISSN 0008-5472, 09/2005, Volume 65, Issue 17, pp. 7917 - 7925
Journal Article
Experimental Dermatology, ISSN 0906-6705, 09/2007, Volume 16, Issue 9, pp. 762 - 769
Journal Article
CANCER LETTERS, ISSN 0304-3835, 06/2014, Volume 348, Issue 1-2, pp. 1 - 11
The anticancer activity of n-3 fatty acids, especially those derived from fish, such as eicosapentaenoic acid (EPA) and docosahexaenoic acid) (DHA), has been... 
n-3 Fatty acids | ARACHIDONIC-ACID | Metabolism | Tumor tissues | PGE | FISH-OIL SUPPLEMENTATION | PROSTANOID RECEPTORS | DIETARY EICOSAPENTAENOIC ACID | ONCOLOGY | DOCOSAHEXAENOIC ACID | COLORECTAL-CANCER | Cancer cells | POLYUNSATURATED FATTY-ACIDS | MAJOR URINARY METABOLITE | OMEGA-3-FATTY-ACID SUPPLEMENTATION | ALPHA-LINOLENIC ACID
Journal Article
Science, ISSN 0036-8075, 11/2011, Volume 334, Issue 6057, pp. 809 - 813
Phospholipase A₂ (PLA₂) enzymes are considered the primary source of arachidonic acid for cyclooxygenase (COX)-mediated biosynthesis of prostaglandins. Here,... 
Datasets | Brain | Enzymes | Prostaglandins | Cytokines | Neurodegenerative diseases | REPORTS | Eicosanoids | Lipids | Endocannabinoids | Vehicles | SYSTEM | INHIBITION | CYCLOOXYGENASE-2 | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | INJURY | LIPOPOLYSACCHARIDE | PHOSPHOLIPASE A | MICE | PARKINSONS-DISEASE | Cannabinoid Receptor Modulators - metabolism | Inflammation - pathology | Metabolomics | Arachidonic Acid - metabolism | Monoacylglycerol Lipases - antagonists & inhibitors | Monoacylglycerol Lipases - genetics | Brain - metabolism | Parkinsonian Disorders - metabolism | Monoacylglycerol Lipases - metabolism | Arachidonic Acids - metabolism | Inflammation - metabolism | Neuroprotective Agents - pharmacology | Piperidines - pharmacology | Inflammation Mediators - pharmacology | Lung - metabolism | Cytokines - metabolism | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Phospholipases A2 - metabolism | Prostaglandins - biosynthesis | Glycerides - metabolism | Brain - drug effects | Hydrolysis | Animals | Parkinsonian Disorders - pathology | Eicosanoids - metabolism | Lipopolysaccharides - pharmacology | Brain - pathology | Mice | Cyclooxygenase 1 - metabolism | Benzodioxoles - pharmacology | Phospholipases A2 - genetics | Prostaglandins - metabolism | Physiological aspects | Inflammation | Research | Risk factors | Hydrology | Neurology | Inflammatory diseases
Journal Article
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 04/2006, Volume 207, Issue 1, pp. 261 - 270
Recent evidence indicates that cyclooxygenase‐2 (COX‐2) and epidermal growth factor receptor (EGFR) are involved in hepatocarcinogenesis. This study was... 
Proto-Oncogene Proteins c-met - metabolism | RNA, Small Interfering - genetics | src-Family Kinases | Gene Expression - genetics | Protein-Tyrosine Kinases - metabolism | Caproates - pharmacology | Humans | Receptors, Prostaglandin - metabolism | Receptors, Prostaglandin E - genetics | Alprostadil - analogs & derivatives | Cell Movement - physiology | Membrane Proteins - analysis | Receptor, Epidermal Growth Factor - metabolism | Receptors, Prostaglandin - genetics | Transfection | RNA Interference | Cyclooxygenase 2 - genetics | Carcinoma, Hepatocellular - genetics | Protein Binding - drug effects | Liver Neoplasms - pathology | Membrane Proteins - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins - metabolism | Dinoprostone - pharmacology | Bridged Bicyclo Compounds - pharmacology | Liver Neoplasms - genetics | Membrane Proteins - genetics | Neoplasm Invasiveness | Alprostadil - pharmacology | Cyclooxygenase 2 - analysis | Receptors, Prostaglandin E - physiology | Cell Movement - drug effects | Carcinoma, Hepatocellular - pathology | Cyclooxygenase 2 - metabolism | Liver Neoplasms - metabolism | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Protein Kinase Inhibitors - pharmacology | Receptors, Prostaglandin E, EP1 Subtype | Enzyme Activation | Carcinoma, Hepatocellular - metabolism
Journal Article