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Journal of Cellular Physiology, ISSN 0021-9541, 04/2006, Volume 207, Issue 1, pp. 261 - 270
Recent evidence indicates that cyclooxygenase‐2 (COX‐2) and epidermal growth factor receptor (EGFR) are involved in hepatocarcinogenesis. This study was... 
Proto-Oncogene Proteins c-met - metabolism | RNA, Small Interfering - genetics | src-Family Kinases | Gene Expression - genetics | Protein-Tyrosine Kinases - metabolism | Caproates - pharmacology | Humans | Receptors, Prostaglandin - metabolism | Receptors, Prostaglandin E - genetics | Alprostadil - analogs & derivatives | Cell Movement - physiology | Membrane Proteins - analysis | Receptor, Epidermal Growth Factor - metabolism | Receptors, Prostaglandin - genetics | Transfection | RNA Interference | Cyclooxygenase 2 - genetics | Carcinoma, Hepatocellular - genetics | Protein Binding - drug effects | Liver Neoplasms - pathology | Membrane Proteins - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins - metabolism | Dinoprostone - pharmacology | Bridged Bicyclo Compounds - pharmacology | Liver Neoplasms - genetics | Membrane Proteins - genetics | Neoplasm Invasiveness | Alprostadil - pharmacology | Cyclooxygenase 2 - analysis | Receptors, Prostaglandin E - physiology | Cell Movement - drug effects | Carcinoma, Hepatocellular - pathology | Cyclooxygenase 2 - metabolism | Liver Neoplasms - metabolism | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Protein Kinase Inhibitors - pharmacology | Receptors, Prostaglandin E, EP1 Subtype | Enzyme Activation | Carcinoma, Hepatocellular - metabolism | Index Medicus
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 03/2009, Volume 206, Issue 3, pp. 535 - 548
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2011, Volume 286, Issue 3, pp. 2331 - 2342
A major immunological response during neuroinflammation is the activation of microglia, which subsequently release proinflammatory mediators such as... 
RAT SPINAL-CORD | TUMOR-NECROSIS-FACTOR | ISCHEMIC TOLERANCE | LIGAND-BINDING | NEONATAL-RAT | E RECEPTOR SUBTYPES | MESSENGER-RNA | BIOCHEMISTRY & MOLECULAR BIOLOGY | PAIN HYPERSENSITIVITY | LIPOPOLYSACCHARIDE | INNATE IMMUNITY | Inflammation - chemically induced | Tumor Necrosis Factor-alpha - metabolism | Microglia - metabolism | Myelitis - chemically induced | Myelitis - enzymology | Tumor Necrosis Factor-alpha - genetics | Interleukin-1beta - genetics | Receptors, Prostaglandin E - genetics | Cyclooxygenase 2 - genetics | Interleukin-1beta - metabolism | Intramolecular Oxidoreductases - metabolism | Prostaglandins - genetics | Disease Models, Animal | Prostaglandin-Endoperoxide Synthases - genetics | Receptors, Epoprostenol - metabolism | Lipopolysaccharides - toxicity | Myelitis - genetics | Receptors, Thromboxane A2, Prostaglandin H2 - metabolism | Cells, Cultured | Rats | Receptors, Thromboxane A2, Prostaglandin H2 - genetics | Rats, Sprague-Dawley | Mice, Knockout | Up-Regulation - drug effects | Receptors, Epoprostenol - genetics | Animals | Intramolecular Oxidoreductases - genetics | Prostaglandin-Endoperoxide Synthases - metabolism | Prostaglandin-E Synthases | Cyclooxygenase 2 - metabolism | Inflammation - genetics | Mice | Inflammation - enzymology | Receptors, Prostaglandin E - metabolism | Prostaglandins - metabolism | Index Medicus | Prostaglandins | mPGES-1 | Immunology | Spinal Cord | Innate Immunity | Tumor Necrosis Factor (TNF) | Inflammation | Cyclooxygenase (COX) Pathway | Cyclic AMP (cAMP) | Microglia
Journal Article
Journal Article
PLoS Genetics, ISSN 1553-7390, 04/2007, Volume 3, Issue 4, pp. 0538 - 0543
Journal Article
Fertility and Sterility, ISSN 0015-0282, 2010, Volume 94, Issue 4, pp. 1271 - 1278
Objective To define the role of prostaglandins (PG) in the endometrium of patients with repeated failure of embryo implantation. Prostaglandins are produced... 
Internal Medicine | Obstetrics and Gynecology | in vitro fertilization | prostaglandins | endometrial receptivity | repeated implantation failure | Endometrium | window of implantation | IIA | ACID RELEASE | CYCLOOXYGENASE-2 | EMBRYO IMPLANTATION | UTERINE RECEPTIVITY | PHOSPHOLIPASE A | PROLIFERATION | E-2 BIOSYNTHESIS | OBSTETRICS & GYNECOLOGY | E SYNTHASE | REPRODUCTIVE BIOLOGY | INTEGRINS | Group IV Phospholipases A2 - genetics | Embryo Loss - diagnosis | Humans | Embryo Implantation - genetics | Male | Lipid Metabolism Disorders - diagnosis | Abortion, Habitual - metabolism | Phospholipases A2, Secretory - genetics | Receptors, Prostaglandin E - genetics | Case-Control Studies | Fertility - physiology | Embryo Loss - metabolism | Young Adult | Fertility - genetics | Group IV Phospholipases A2 - metabolism | Cyclooxygenase 2 - genetics | Abortion, Habitual - diagnosis | Lipid Metabolism Disorders - complications | Abortion, Habitual - genetics | Embryo Loss - etiology | Adult | Female | Abortion, Habitual - etiology | Embryo Loss - genetics | Endometrium - metabolism | Embryo Implantation - physiology | Lipid Metabolism Disorders - metabolism | Prostaglandins - biosynthesis | Fertilization in Vitro | Lipid Metabolism Disorders - genetics | Cyclooxygenase 2 - metabolism | Phospholipases A2, Secretory - metabolism | Endometrium - pathology | Receptors, Prostaglandin E - metabolism | Unsaturated fatty acids | Prostaglandins | Universities and colleges | Arachidonic acid | Pharmacy | Index Medicus
Journal Article
Molecular Human Reproduction, ISSN 1360-9947, 05/2017, Volume 23, Issue 5, pp. 271 - 281
STUDY QUESTION: Do intraluteal prostaglandins (PG) contribute to luteal regulation in women? SUMMARY ANSWER: Prostaglandin E (PGE), which is produced in human... 
Corpus luteum | Luteinized granulosa cell | Prostaglandin | HCG | Luteolysis | PROGESTERONE | prostaglandin | luteinized granulosa cell | DEVELOPMENTAL BIOLOGY | HUMAN CHORIONIC-GONADOTROPIN | hCG | luteolysis | OBSTETRICS & GYNECOLOGY | DEHYDROGENASE GENE | ACTIVIN-A | IN-VITRO | REPRODUCTIVE BIOLOGY | EARLY-PREGNANCY | corpus luteum | RECEPTORS | HUMAN CORPUS-LUTEUM | LUTEINIZED GRANULOSA-CELLS | EXPRESSION | Aldehyde Reductase - genetics | Progesterone Reductase - metabolism | Corpus Luteum - drug effects | Luteinization - physiology | Humans | Prostaglandins E - pharmacology | Multienzyme Complexes - metabolism | Phosphoproteins - metabolism | Granulosa Cells - drug effects | Granulosa Cells - metabolism | Chorionic Gonadotropin - pharmacology | Isoenzymes - metabolism | Luteal Phase - physiology | Prostaglandin-E Synthases - genetics | Aldehyde Reductase - metabolism | Female | Progesterone - biosynthesis | Steroid Isomerases - genetics | Inhibin-beta Subunits - genetics | Inhibin-beta Subunits - metabolism | Progesterone - secretion | 20-Hydroxysteroid Dehydrogenases - genetics | Prostaglandin-E Synthases - metabolism | Signal Transduction | Isoenzymes - genetics | Menstruation - physiology | Steroid Isomerases - metabolism | Gene Expression Regulation | Multienzyme Complexes - genetics | Phosphoproteins - genetics | Progesterone Reductase - genetics | 20-Hydroxysteroid Dehydrogenases - metabolism | Corpus Luteum - cytology | Animals | Corpus Luteum - metabolism | Luteolysis - genetics | Primary Cell Culture | Prostaglandins E - genetics | Placenta Growth Factor - pharmacology | Prostaglandins E - deficiency | Granulosa Cells - cytology | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2010, Volume 107, Issue 27, pp. 12233 - 12238
Experimental autoimmune encephalomyelitis (EAE) is an animal model of multiple sclerosis (MS). Although prostaglandin (PG) concentrations are increased in... 
Immunization | Receptors | Prostaglandins | Spinal cord | Multiple sclerosis | Demyelinating diseases | Experimental autoimmune encephalomyelitis | Agonists | Mice | Neutrophil infiltration | Prostaglandin receptor | Knockout mice | Prostaglandin | Disease model | Humans | Encephalomyelitis, Autoimmune, Experimental - immunology | Male | Interferon-gamma - metabolism | Th1 Cells - immunology | Dinoprostone - immunology | Receptors, Prostaglandin E - genetics | Dose-Response Relationship, Drug | Receptors, Prostaglandin E, EP4 Subtype | T-Lymphocytes, Helper-Inducer - drug effects | Th1 Cells - metabolism | Signal Transduction - immunology | T-Lymphocytes - metabolism | T-Lymphocytes - drug effects | T-Lymphocytes, Helper-Inducer - immunology | Phenylbutyrates - pharmacology | Female | Receptors, Prostaglandin E - immunology | Encephalomyelitis, Autoimmune, Experimental - genetics | Th1 Cells - drug effects | Blood-Brain Barrier - immunology | T-Lymphocytes, Helper-Inducer - metabolism | Mice, Inbred C57BL | Methyl Ethers - pharmacology | Signal Transduction - genetics | Mice, Inbred Strains | Blood-Brain Barrier - drug effects | Blood-Brain Barrier - metabolism | Mice, Knockout | Naphthalenes - pharmacology | Dinoprostone - metabolism | Interleukin-17 - metabolism | Animals | Signal Transduction - drug effects | Receptors, Prostaglandin E - antagonists & inhibitors | Receptors, Prostaglandin E, EP2 Subtype | Encephalomyelitis, Autoimmune, Experimental - prevention & control | T-Lymphocytes - immunology | Index Medicus | Biological Sciences | prostaglandin receptor | knockout mice | prostaglandin | multiple sclerosis | disease model
Journal Article