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Journal of Cellular Physiology, ISSN 0021-9541, 04/2006, Volume 207, Issue 1, pp. 261 - 270
Recent evidence indicates that cyclooxygenase‐2 (COX‐2) and epidermal growth factor receptor (EGFR) are involved in hepatocarcinogenesis. This study was... 
Proto-Oncogene Proteins c-met - metabolism | RNA, Small Interfering - genetics | src-Family Kinases | Gene Expression - genetics | Protein-Tyrosine Kinases - metabolism | Caproates - pharmacology | Humans | Receptors, Prostaglandin - metabolism | Receptors, Prostaglandin E - genetics | Alprostadil - analogs & derivatives | Cell Movement - physiology | Membrane Proteins - analysis | Receptor, Epidermal Growth Factor - metabolism | Receptors, Prostaglandin - genetics | Transfection | RNA Interference | Cyclooxygenase 2 - genetics | Carcinoma, Hepatocellular - genetics | Protein Binding - drug effects | Liver Neoplasms - pathology | Membrane Proteins - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins - metabolism | Dinoprostone - pharmacology | Bridged Bicyclo Compounds - pharmacology | Liver Neoplasms - genetics | Membrane Proteins - genetics | Neoplasm Invasiveness | Alprostadil - pharmacology | Cyclooxygenase 2 - analysis | Receptors, Prostaglandin E - physiology | Cell Movement - drug effects | Carcinoma, Hepatocellular - pathology | Cyclooxygenase 2 - metabolism | Liver Neoplasms - metabolism | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Protein Kinase Inhibitors - pharmacology | Receptors, Prostaglandin E, EP1 Subtype | Enzyme Activation | Carcinoma, Hepatocellular - metabolism | Index Medicus
Journal Article
Journal Article
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 2015, Volume 309, Issue 10, pp. 639 - 649
Obesity, a known risk factor for pancreatic cancer, is associated with inflammation and insulin resistance. Proinflammatory prostaglandin E2 (PGE(2)) and... 
Prostaglandin E2 | Obesity | Mammalian target of rapamycin complex 1 | Pancreatic cancer | PHYSIOLOGY | HIGH-FAT DIET | COUPLED RECEPTOR | COLON CARCINOGENESIS | KRAS(G12D) MOUSE MODEL | CELL BIOLOGY | CANCER GROWTH | BREAST-CANCER | INSULIN | pancreatic cancer | CYCLIC-AMP | prostaglandin E2 | PROTEIN S6 PHOSPHORYLATION | mammalian target of rapamycin complex 1 | obesity | TUMOR MICROENVIRONMENT | Cyclic AMP-Dependent Protein Kinases - metabolism | Receptors, Prostaglandin E, EP4 Subtype - genetics | Dinoprostone - pharmacology | Receptors, Prostaglandin E, EP2 Subtype - metabolism | TOR Serine-Threonine Kinases - metabolism | Calcium - metabolism | Humans | Receptors, Prostaglandin E, EP2 Subtype - genetics | Gene Expression Regulation - physiology | Multiprotein Complexes - genetics | Receptors, Prostaglandin E, EP1 Subtype - genetics | Receptors, Prostaglandin E, EP1 Subtype - metabolism | Mechanistic Target of Rapamycin Complex 1 | Dinoprostone - metabolism | Interleukin-23 Subunit p19 - metabolism | Multiprotein Complexes - metabolism | Cyclic AMP-Dependent Protein Kinases - genetics | TOR Serine-Threonine Kinases - genetics | Interleukin-23 Subunit p19 - genetics | Cell Line, Tumor | Cyclic AMP - genetics | Cyclic AMP - metabolism | Receptors, Prostaglandin E, EP4 Subtype - metabolism | Index Medicus | Call for Papers
Journal Article
Journal of Cellular Biochemistry, ISSN 0730-2312, 12/2017, Volume 118, Issue 12, pp. 4383 - 4393
Mice lacking the PGE2 receptor subtype 1 (EP1) exhibit accelerated fracture repair and maintain higher bone mass upon aging and ovariectomy. Here, we examined... 
MESENCHYMAL STROMAL CELL | PGE2 | OXIDATIVE PHOSPHORYLATION | EP1 | OSTEOBLAST DIFFERENTIATION | MITOCHONDRIAL BIOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | BONE-MARROW | INCREASES | NICHE | PGE | CELL BIOLOGY | HEMATOPOIETIC STEM-CELLS | IN-VIVO | MICE | PROSTAGLANDIN E-2 | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Oxygen Consumption | Mesenchymal Stromal Cells - metabolism | Receptors, Prostaglandin E, EP1 Subtype - genetics | Receptors, Prostaglandin E, EP1 Subtype - metabolism | Mice, Knockout | Animals | Energy Metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Cell Differentiation | Mice | Osteoblasts - metabolism | Osteogenesis | Physiological aspects | Cell differentiation | Prostaglandins E | Stem cells | Energy metabolism | Phosphorylation | Hypoxia-inducible factor 1 | Mesenchyme | Differentiation (biology) | Prostaglandin E2 | Stem cell transplantation | Bone (trabecular) | Inactivation | Osteoblasts | Prostaglandin endoperoxide synthase | Signal transduction | Receptors | Bone growth | Mitochondria | Biomedical materials | Allografts | Clonal deletion | Bioenergetics | Deletion | Bones | Biocompatibility | Bone (cortical) | Repair | Deactivation | Oxygen consumption | Bone healing | Metabolism | Fracture toughness | Osteoblastogenesis | Oxidative phosphorylation | Stromal cells | Bone | Cyclooxygenase-2 | Index Medicus | Mesenchymal Stromal Cell | Oxidative Phosphorylation
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2015, Volume 5, Issue 1, pp. 17956 - 17956
Cyclooxygenase-2 (COX-2) is activated in response to ischemia and significantly contributes to the neuroinflammatory process. Accumulation of COX-2-derived... 
HEMORRHAGIC TRANSFORMATION | MATRIX METALLOPROTEINASES | NEUTROPHIL INFILTRATION | TYROSINE PHOSPHORYLATION | CYCLOOXYGENASE INHIBITION | MULTIDISCIPLINARY SCIENCES | FOCAL CEREBRAL-ISCHEMIA | REPERFUSION INJURY | NECROSIS-FACTOR-ALPHA | NF-KAPPA-B | MATRIX-METALLOPROTEINASE-9 GENE-EXPRESSION | Brain Infarction - metabolism | Brain Infarction - pathology | Male | Neutrophil Infiltration | Stroke - genetics | Stroke - pathology | Proteolysis - drug effects | Matrix Metalloproteinase 9 - metabolism | Neurons - metabolism | Tight Junction Proteins - metabolism | Disease Models, Animal | Gene Expression | Hydrazines - pharmacology | Permeability - drug effects | Endothelial Cells - metabolism | Rats | Matrix Metalloproteinase 3 - metabolism | Receptors, Prostaglandin E, EP1 Subtype - genetics | Gene Knockout Techniques | Blood-Brain Barrier - drug effects | Receptors, Prostaglandin E, EP1 Subtype - metabolism | Oxazepines - pharmacology | Blood-Brain Barrier - metabolism | Mice, Knockout | Blood-Brain Barrier - pathology | Stroke - metabolism | Animals | Mice | Receptors, Prostaglandin E, EP1 Subtype - antagonists & inhibitors | Stroke | Transformation | Preservation | Prostaglandin E2 | Inflammation | Hemorrhage | Zonula occludens-1 protein | Gelatinase B | Prostaglandin endoperoxide synthase | Signal transduction | Blood-brain barrier | Clonal deletion | Ischemia | Cyclooxygenase-2 | Brain injury | Index Medicus
Journal Article
Journal Article
Journal Article
Journal Article