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Cancer Science, ISSN 1347-9032, 09/2014, Volume 105, Issue 9, pp. 1142 - 1151
We previously established that COX‐2 overexpression promotes breast cancer progression and metastasis. As long‐term use of COX‐2 inhibitors (COX‐2i) can... 
RQ‐00015986 | macrophages | cyclooxygenase 2 | prostaglandin E2 receptor EP4 subtype | Cancer stem cells | Prostaglandin E2 receptor EP4 subtype | Macrophages | RQ-00015986 | Cyclooxygenase 2 | FACTOR VEGF | ANTAGONIST | HUMAN BREAST-CANCER | MURINE | PROSTANOID RECEPTORS | ONCOLOGY | GROWTH | TUMOR-ASSOCIATED MACROPHAGES | CYCLOOXYGENASE-2 EXPRESSION | UP-REGULATION | PROGRESSION | Lung Neoplasms - drug therapy | Neoplasm Transplantation | Pyrazoles - therapeutic use | Cell Proliferation | Antineoplastic Agents - therapeutic use | Vascular Endothelial Growth Factor A - metabolism | Molecular Targeted Therapy | Benzamides - therapeutic use | Lung Neoplasms - secondary | Receptors, Prostaglandin E, EP4 Subtype - antagonists & inhibitors | Mammary Neoplasms, Experimental - pathology | Female | Mammary Neoplasms, Experimental - blood supply | Antineoplastic Agents - pharmacology | Benzamides - pharmacology | Lymphangiogenesis | Lung Neoplasms - blood supply | Mammary Neoplasms, Experimental - drug therapy | Pyrazoles - pharmacology | Receptors, Prostaglandin E, EP4 Subtype - metabolism | Lymphatic Metastasis | Neoplastic Stem Cells | Celecoxib | Tumor Burden | Sulfonamides - pharmacology | Adenocarcinoma - drug therapy | Adenocarcinoma - secondary | Mice, Inbred C3H | Adenocarcinoma - blood supply | Macrophages - metabolism | Animals | Sulfonamides - therapeutic use | Neovascularization, Pathologic - drug therapy | Cyclooxygenase 2 - metabolism | Cell Line, Tumor | Mice | Apoptosis | Drug Screening Assays, Antitumor | Original
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2015, Volume 112, Issue 31, pp. 9716 - 9721
Journal Article
Pharmacology and Therapeutics, ISSN 0163-7258, 06/2013, Volume 138, Issue 3, pp. 485 - 502
The large variety of biological functions governed by prostaglandin (PG) E is mediated by signaling through four distinct E-type prostanoid (EP) receptors. The... 
Vascular disease | Osteoporosis | Prostaglandins | Inflammation | Renal function | Cancerogenesis | HUMAN DENDRITIC CELLS | DIETARY SALT INTAKE | DUODENAL BICARBONATE SECRETION | MICROVASCULAR ENDOTHELIAL-CELLS | PROSTAGLANDIN-E-RECEPTOR | AGONIST-INDUCED INTERNALIZATION | BREAST-CANCER CELLS | INFLAMMATORY-BOWEL-DISEASE | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | PHARMACOLOGY & PHARMACY | NF-KAPPA-B | Neoplasms - metabolism | Receptors, Prostaglandin E, EP4 Subtype - agonists | Lung Diseases - metabolism | Cardiovascular Diseases - metabolism | Humans | Bone Diseases - metabolism | Dinoprostone - metabolism | Animals | Gastrointestinal Diseases - metabolism | Receptors, Prostaglandin E, EP4 Subtype - antagonists & inhibitors | Immunologic Factors - metabolism | Kidney Diseases - metabolism | Receptors, Prostaglandin E, EP4 Subtype - metabolism | CREB, cAMP-response element-binding protein | Associate editor | TX, thromboxane receptor | EPRAP, EP4 receptor-associated protein | PK, protein kinase | COX, cyclooxygenase | AMPK, AMP-activated protein kinase | ICAM-1, intercellular adhesion molecule-1 | PI3K, phosphatidyl insositol 3-kinase | CFTR, cystic fibrosis transmembrane conductance regulator | IP, I-type prostanoid receptor | Epac, exchange protein activated by cAMP | NF-κB, nuclear factor kappa-light-chain-enhancer of activated B cells | NSAID, non-steroidal anti-inflammatory drug | IFN, interferon | MEK, MAP kinase kinase | PG, prostaglandin | DSS, dextran sodium sulfate | eNOS, endothelial nitric oxide synthase | Ig, immunoglobulin | P. Holzer | ICER, inducible cAMP early repressor | EGFR, epidermal growth factor receptor | FEM1a, feminization 1 homolog a | ERK, extracellular signal-regulated kinase | MCP, monocyte chemoattractant protein | 5-HETE, 5-hydroxyeicosatetraenoic acid | IL, interleukin | GRK, G protein-coupled receptor kinase | MAP, mitogen-activated protein kinase | LPS, lipopolysaccharide | ClC, chloride channel | DP, D-type prostanoid receptor | FP, F-type prostanoid receptor | TP, T-type prostanoid receptor | cAMP, cyclic adenylyl monophosphate | EP, E-type prostanoid receptor
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 03/2016, Volume 173, Issue 6, pp. 992 - 1004
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2012, Volume 181, Issue 1, pp. 313 - 321
Journal Article
Science, ISSN 0036-8075, 03/2016, Volume 351, Issue 6279, pp. 1333 - 1338
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 04/2018, Volume 19, Issue 4, p. 1019
Journal Article