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1. Full Text Somatostatin receptor 2 signaling promotes growth and tumor survival in small‐cell lung cancer
by Lehman, Jonathan M and Hoeksema, Megan D and Staub, Jeremy and Qian, Jun and Harris, Bradford and Callison, J. Clay and Miao, Jennifer and Shi, Chanjuan and Eisenberg, Rosana and Chen, Heidi and Chen, Sheau‐Chiann and Massion, Pierre P
International Journal of Cancer, ISSN 0020-7136, 03/2019, Volume 144, Issue 5, pp. 1104 - 1114
Somatostatin receptor 2 (SSTR2) is overexpressed in a majority of neuroendocrine neoplasms, including small‐cell lung carcinomas (SCLCs). SSTR2 was previously... 
cancer progression | somatostatin | survival | SUBTYPE | OCTREOTIDE | APOPTOSIS | ANALOGS | PANCREATIC-CANCER | ANTINEOPLASTIC AGENTS | COMBINATION | THERAPY | ONCOLOGY | EXPRESSION | CARCINOMA | Lung Neoplasms - genetics | Cell Proliferation - genetics | Basic Helix-Loop-Helix Transcription Factors - genetics | Humans | RNA, Messenger - genetics | Apoptosis - genetics | Lung Neoplasms - pathology | Signal Transduction - genetics | Small Cell Lung Carcinoma - genetics | Nerve Tissue Proteins - genetics | Disease Progression | Down-Regulation - genetics | Receptors, Somatostatin - genetics | Small Cell Lung Carcinoma - pathology | Animals | Mice, Nude | Cell Line, Tumor | Biomarkers, Tumor - genetics | Mice | AMP-Activated Protein Kinases - genetics | Medical research | Phosphorylation | Cell survival | Therapeutic applications | Oxidative metabolism | Lung cancer | Clinical trials | Lung carcinoma | Metabolism | Neoplasms | Beta2 protein | Signaling | Correlation analysis | Cell lines | Biomarkers | Bioindicators | Somatostatin | Cancer | Tumors | Apoptosis | Index Medicus
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2. Full Text A subpopulation of itch‐sensing neurons marked by Ret and somatostatin expression
by Stantcheva, Kalina K and Iovino, Loredana and Dhandapani, Rahul and Martinez, Concepcion and Castaldi, Laura and Nocchi, Linda and Perlas, Emerald and Portulano, Carla and Pesaresi, Martina and Shirlekar, Kalyanee S and Castro Reis, Fernanda and Paparountas, Triantafillos and Bilbao, Daniel and Heppenstall, Paul A
EMBO reports, ISSN 1469-221X, 04/2016, Volume 17, Issue 4, pp. 585 - 600
Itch, the unpleasant sensation that elicits a desire to scratch, is mediated by specific subtypes of cutaneous sensory neuron. Here, we identify a... 
DRG | 5HT | sensory neurons | IL‐31 | IL-31 | Heparin-binding EGF-like Growth Factor - genetics | Somatostatin - genetics | Receptor, Nerve Growth Factor - genetics | Neuropeptides - metabolism | Gene Expression Profiling | Pruritus - genetics | Lectins - metabolism | Animals | In Situ Hybridization | Mice | Sensory Receptor Cells - metabolism | Neurons, Afferent - metabolism | Microfilament Proteins - genetics | Ganglia, Spinal - metabolism | Proto-Oncogene Proteins c-ret - genetics | Kinases | Flow cytometry | Neurons | Index Medicus | Neuroscience | 5HT1F
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3. Full Text Aberrant methylation inactivates somatostatin and somatostatin receptor type 1 in head and neck squamous cell carcinoma
by Misawa, Kiyoshi and Misawa, Yuki and Kondo, Haruki and Mochizuki, Daiki and Imai, Atsushi and Fukushima, Hirofumi and Uehara, Takayuki and Kanazawa, Takeharu and Mineta, Hiroyuki
PLoS ONE, ISSN 1932-6203, 03/2015, Volume 10, Issue 3, pp. e0118588 - e0118588
Purpose The aim of this study was to define somatostatin (SST) and somatostatin receptor type 1 (SSTR1) methylation profiles for head and neck squamous cell... 
PATHOGENESIS | POTENTIAL BIOMARKER | HYPERMETHYLATION | PROMOTER METHYLATION | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | GALANIN | TUMOR-SUPPRESSOR ACTIVITY | TACHYKININ-1 | EXPRESSION | ASSOCIATION | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Oropharyngeal Neoplasms - mortality | Male | Oropharyngeal Neoplasms - pathology | Galanin - metabolism | Carcinoma, Squamous Cell - diagnosis | Receptors, Somatostatin - genetics | DNA Methylation | Mouth Neoplasms - diagnosis | Squamous Cell Carcinoma of Head and Neck | Carcinoma, Squamous Cell - mortality | Tachykinins - genetics | Aged, 80 and over | Biomarkers, Tumor - metabolism | Receptor, Galanin, Type 2 - genetics | Adult | Female | Transcription, Genetic | Receptors, Somatostatin - metabolism | Retrospective Studies | Odds Ratio | Mouth Neoplasms - genetics | Somatostatin - metabolism | Oropharyngeal Neoplasms - diagnosis | Promoter Regions, Genetic | Oropharyngeal Neoplasms - genetics | Somatostatin - genetics | Tachykinins - metabolism | Risk Factors | Mouth Neoplasms - mortality | Head and Neck Neoplasms - pathology | Survival Analysis | CpG Islands | Mouth Neoplasms - pathology | Head and Neck Neoplasms - diagnosis | Head and Neck Neoplasms - genetics | Receptor, Galanin, Type 2 - metabolism | Aged | Biomarkers, Tumor - genetics | Head and Neck Neoplasms - mortality | Galanin - genetics | Neoplasm Staging | Neuropeptides | Squamous cell carcinoma | Methylation | Analysis | Tachykinin receptors | Correlation | Galanin | Mucosa | Otolaryngology | Inactivation | Tissues | Epidermal growth factor | DNA methylation | Measurement methods | Tumorigenesis | Bioindicators | Neck | Somatostatin | CpG islands | Head | Deactivation | Oropharynx | Regression analysis | Gene expression | Survival | Promoters | Polymerase chain reaction | Oral cavity | Epigenetics | Biomarkers | Head and neck cancer | Aberration | Tachykinin | Tumors | Cancer | Index Medicus
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4. Full Text Deletion of Men1 and somatostatin induces hypergastrinemia and gastric carcinoids
by Sundaresan, Sinju and Kang, Anthony J and Hayes, Michael M and Choi, Eun-Young K and Merchant, Juanita L
Gut, ISSN 0017-5749, 06/2017, Volume 66, Issue 6, pp. 1012 - 1021
BackgroundGastric carcinoids are slow growing neuroendocrine tumours arising from enterochromaffin-like (ECL) cells in the corpus of stomach. Although most of... 
ACID SECRETION | GASTRIN | CANCER GENETICS | GASTRIN RECEPTORS | ENDOCRINE TUMOURS | MASTOMYS-NATALENSIS | INHIBITOR P27(KIP1) | AFRICAN RODENT | CANCER CELLS | RAT STOMACH | GASTROENTEROPANCREATIC NEUROENDOCRINE TUMORS | ENDOCRINE TUMORS | ENTEROCHROMAFFIN-LIKE CELLS | ZOLLINGER-ELLISON-SYNDROME | GASTROENTEROLOGY & HEPATOLOGY | TRANSGENIC MICE | Proton Pump Inhibitors - pharmacology | Humans | Receptor, Cholecystokinin B - metabolism | Stomach Neoplasms - metabolism | Male | RNA, Messenger - metabolism | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Adenocarcinoma - metabolism | Gene Deletion | Adult | Female | Omeprazole - pharmacology | Gastrins - genetics | Phosphorylation - drug effects | Gastrins - pharmacology | Gastrins - blood | Stomach Neoplasms - genetics | Hormones - pharmacology | Somatostatin - genetics | Benzodiazepines - pharmacology | Signal Transduction | Carcinogenesis - genetics | Mice, Transgenic | Proto-Oncogene Proteins - genetics | Gastrins - metabolism | Receptor, Cholecystokinin B - genetics | Hormone Antagonists - pharmacology | Carcinogenesis - drug effects | Animals | Carcinoid Tumor - genetics | Cell Line, Tumor | Mice | Causes of | Genetic aspects | Carcinoid | Somatostatin | Research | Stomach diseases | Index Medicus | Abridged Index Medicus | gastrin receptor | cancer genetics | endocrine tumors | Acid secretion | gastrin
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5. Full Text Transcriptional and Functional Characterization of the G Protein-Coupled Receptor Repertoire of Gastric Somatostatin Cells
by Egerod, Kristoffer L and Engelstoft, Maja S and Lund, Mari L and Grunddal, Kaare V and Zhao, Mirabella and Barir-Jensen, Dominique and Nygaard, Eva B and Petersen, Natalia and Holst, Jens J and Schwartz, Thue W
Endocrinology, ISSN 0013-7227, 11/2015, Volume 156, Issue 11, pp. 3909 - 3923
In the stomach, somatostatin (SST) acts as a general paracrine negative regulator of exocrine secretion of gastric acid and pepsinogen and endocrine secretion... 
PERFUSED RAT STOMACH | CALCIUM-SENSING RECEPTOR | GLUCAGON-LIKE PEPTIDE-1 | GROWTH-HORMONE | GHRELIN SECRETION | ENTEROENDOCRINE CELLS | ACID-SECRETION | ENDOCRINOLOGY & METABOLISM | ISLET AMYLOID POLYPEPTIDE | RELEASE-INHIBITING HORMONE | ENDOCRINE-CELLS | Peptide YY - metabolism | Receptors, G-Protein-Coupled - metabolism | Humans | Transcriptome | Male | Receptors, Muscarinic - metabolism | Receptors, Neuropeptide - metabolism | Islet Amyloid Polypeptide - genetics | Stomach - metabolism | Gastric Mucosa - metabolism | Receptors, Somatostatin - genetics | Glucagon-Like Peptide-1 Receptor - genetics | Glucagon-Like Peptide-1 Receptor - metabolism | In Situ Hybridization | Receptors, Adrenergic - genetics | Receptors, Somatostatin - metabolism | Peptide YY - genetics | Somatostatin - metabolism | Receptors, Muscarinic - genetics | Somatostatin - secretion | Somatostatin - genetics | Receptors, Adrenergic - metabolism | Cells, Cultured | Islet Amyloid Polypeptide - metabolism | Receptors, Neuropeptide - genetics | Mice, Transgenic | Gastric Mucosa - cytology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Microscopy, Confocal | Animals | Stomach - cytology | Luminescent Proteins - genetics | Receptors, G-Protein-Coupled - genetics | Somatostatin-Secreting Cells - metabolism | Luminescent Proteins - metabolism | Index Medicus | Abridged Index Medicus
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6. Full Text Dopamine receptor type 2 (DRD2) and somatostatin receptor type 2 (SSTR2) agonists are effective in inhibiting proliferation of progenitor/stem‐like cells isolated from nonfunctioning pituitary tumors
by Peverelli, E and Giardino, E and Treppiedi, D and Meregalli, M and Belicchi, M and Vaira, V and Corbetta, S and Verdelli, C and Verrua, E and Serban, A. L and Locatelli, M and Carrabba, G and Gaudenzi, G and Malchiodi, E and Cassinelli, L and Lania, A. G and Ferrero, S and Bosari, S and Vitale, G and Torrente, Y and Spada, A and Mantovani, G
International Journal of Cancer, ISSN 0020-7136, 04/2017, Volume 140, Issue 8, pp. 1870 - 1880
The role of progenitor/stem cells in pituitary tumorigenesis, resistance to pharmacological treatments and tumor recurrence is still unclear. This study... 
drug resistance | dopamine | tumor stem cells | pituitary adenomas | somatostatin | NEUROENDOCRINE TUMORS | ANALOG | BROMOCRIPTINE | SCINTIGRAPHY | IN-VITRO | THERAPY | ONCOLOGY | CABERGOLINE | STEM-CELL | EXPRESSION | ADENOMAS | Neoplastic Stem Cells - drug effects | Humans | Middle Aged | Spheroids, Cellular - pathology | Neoplasm Recurrence, Local - drug therapy | Male | Neoplasm Recurrence, Local - pathology | Receptors, Somatostatin - genetics | Receptors, Somatostatin - agonists | Adult | DAX-1 Orphan Nuclear Receptor - biosynthesis | Female | Dopamine Agents - administration & dosage | Spheroids, Cellular - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Gonadotropins - biosynthesis | Pituitary Neoplasms - genetics | Receptors, Dopamine D2 - agonists | Carcinogenesis - genetics | Pituitary Neoplasms - pathology | ERG1 Potassium Channel - biosynthesis | Receptors, Dopamine D2 - genetics | Carcinogenesis - drug effects | Drug Resistance, Neoplasm - genetics | Pituitary Neoplasms - drug therapy | Cyclin D3 - biosynthesis | Cyclin-Dependent Kinase Inhibitor p27 - biosynthesis | RNA Splicing Factors - biosynthesis | Neoplasm Recurrence, Local - genetics | Cell Proliferation - drug effects | Dopamine | Pituitary gland tumors | Analysis | Stem cells | Phenols | Gonadotropin | Drug resistance | Dopamine receptors | Index Medicus
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7. Full Text Preoperative Normalization of Cortisol Levels in Cushing's Disease After Medical Treatment: Consequences for Somatostatin and Dopamine Receptor Subtype Expression and In Vitro Response to Somatostatin Analogs and Dopamine Agonists
by van der Pas, R and Feelders, R. A and Gatto, F and de Bruin, C and Pereira, A. M and van Koetsveld, P. M and Sprij-Mooij, D. M and Waaijers, A. M and Dogan, F and Schulz, S and Kros, J. M and Lamberts, S. W. J and Hofland, L. J
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 12/2013, Volume 98, Issue 12, pp. E1880 - E1890
Context: Corticotroph pituitary adenomas often highly express the dopamine 2 receptor (D2R) and somatostatin receptor subtype 5 (sst5). The sst2 expression is... 
POTENT INHIBITOR | OCTREOTIDE | ACTH-SECRETION | INTERNALIZATION | GLUCOCORTICOIDS | CORTICOTROPH ADENOMAS | ENDOCRINOLOGY & METABOLISM | ADRENOCORTICOTROPIN SECRETION | NELSONS SYNDROME | TUMOR-CELLS | PITUITARY-ADENOMAS | ACTH-Secreting Pituitary Adenoma - drug therapy | Pituitary Gland - metabolism | Humans | Middle Aged | Hydrocortisone - secretion | Hydrocortisone - urine | Male | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - metabolism | Receptors, Dopamine D2 - metabolism | ACTH-Secreting Pituitary Adenoma - physiopathology | Somatostatin - pharmacology | Receptors, Somatostatin - genetics | Young Adult | Receptors, Somatostatin - agonists | Protein Isoforms - metabolism | Adult | Female | Receptors, Somatostatin - antagonists & inhibitors | Adrenal Glands - secretion | Antineoplastic Agents - pharmacology | Receptors, Somatostatin - metabolism | Tumor Cells, Cultured | Adrenal Glands - drug effects | Neoplasm Proteins - genetics | Pituitary ACTH Hypersecretion - prevention & control | Dopamine Agonists - pharmacology | Somatostatin - analogs & derivatives | Pituitary ACTH Hypersecretion - etiology | ACTH-Secreting Pituitary Adenoma - metabolism | Pituitary Gland - pathology | Receptors, Dopamine D2 - genetics | Gene Expression Regulation - drug effects | Neoplasm Proteins - agonists | Adolescent | Aged | Dopamine D2 Receptor Antagonists | ACTH-Secreting Pituitary Adenoma - pathology | Pituitary Gland - drug effects | Protein Isoforms - genetics | Index Medicus | Abridged Index Medicus
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8. Full Text Modification of ghrelin receptor signaling by somatostatin receptor-5 regulates insulin release
by Seongjoon Park and Hong Jiang and Hongjie Zhang and Roy G. Smith
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2012, Volume 109, Issue 46, pp. 19003 - 19008
Both ghrelin and somatostatin (SST) inhibit glucose-stimulated insulin secretion (GSIS) from pancreatic β-cells, but how these independent actions are... 
Receptors | HEK293 cells | Secretion | Agonists | Growth hormones | Islets of Langerhans | Physiological regulation | Insulin | Somatostatin receptors | Insulin secretion | Glucose homeostasis | GPCR oligomers | ALPHA-CELLS | MULTIDISCIPLINARY SCIENCES | BETA-CELLS | IDENTIFICATION | HETERODIMERS | PEPTIDE | GLUCAGON-SECRETION | ACYLTRANSFERASE | glucose homeostasis | GROWTH-HORMONE | MICE | HORMONE SECRETAGOGUE RECEPTOR | GTP-Binding Protein alpha Subunits, Gi-Go - genetics | Humans | Receptors, Ghrelin - genetics | Receptors, Somatostatin - genetics | Insulin-Secreting Cells - metabolism | Protein Multimerization - physiology | HEK293 Cells | Receptors, Ghrelin - metabolism | Insulin-Secreting Cells - cytology | Receptors, Somatostatin - metabolism | GTP-Binding Protein alpha Subunits, Gq-G11 - metabolism | Insulin - genetics | Insulin Secretion | Somatostatin - metabolism | GTP-Binding Protein alpha Subunits, Gq-G11 - genetics | Somatostatin - genetics | Ghrelin - genetics | Rats | Ghrelin - metabolism | Insulin - metabolism | Animals | Cell Line, Tumor | Signal Transduction - physiology | Mice | GTP-Binding Protein alpha Subunits, Gi-Go - metabolism | Physiological aspects | Pancreatic beta cells | Somatostatin | Ghrelin | Health aspects | Proteins | Signal transduction | Biochemistry | Bioenergetics | Statins | Index Medicus | Biological Sciences
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9. Full Text A Transcriptome-Led Exploration of Molecular Mechanisms Regulating Somatostatin-Producing D-Cells in the Gastric Epithelium
by Adriaenssens, Alice and Lam, Brian Yee Hong and Billing, Lawrence and Skeffington, Katie and Sewing, Sabine and Reimann, Frank and Gribble, Fiona
Endocrinology, ISSN 0013-7227, 11/2015, Volume 156, Issue 11, pp. 3924 - 3936
The stomach epithelium contains a myriad of enteroendocrine cells that modulate a range of physiological functions, including postprandial secretion of... 
CHOLECYSTOKININ | IMMUNOREACTIVE SOMATOSTATIN | GLUCAGON-LIKE PEPTIDE-1 | CALCIUM-SENSING RECEPTOR | BOMBESIN | RAT STOMACH | ACID-SECRETION | ENDOCRINOLOGY & METABOLISM | PROTEIN-COUPLED RECEPTORS | H-3 RECEPTORS | RELEASE | Epithelial Cells - metabolism | Receptors, G-Protein-Coupled - metabolism | Calcium - metabolism | Transcriptome | Male | Receptors, G-Protein-Coupled - classification | Stomach - metabolism | Gastric Mucosa - metabolism | Hormones - genetics | Single-Cell Analysis - methods | Female | Somatostatin - metabolism | Somatostatin - secretion | Somatostatin - genetics | Cells, Cultured | Receptors, Cell Surface - metabolism | Mice, Transgenic | Gastric Mucosa - cytology | Reverse Transcriptase Polymerase Chain Reaction | Receptors, Cell Surface - classification | Sequence Analysis, RNA - methods | Animals | Hormones - metabolism | Stomach - cytology | Luminescent Proteins - genetics | Mice, Inbred NOD | Receptors, G-Protein-Coupled - genetics | Somatostatin-Secreting Cells - metabolism | Microscopy, Fluorescence | Luminescent Proteins - metabolism | Receptors, Cell Surface - genetics | Index Medicus | Abridged Index Medicus | Original Research
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10. Full Text Differential somatostatin receptor (SSTR) 1–5 expression and downstream effectors in histologic subtypes of growth hormone pituitary tumors
by Kiseljak-Vassiliades, Katja and Xu, Mei and Mills, Taylor S and Smith, Elizabeth E and Silveira, Lori J and Lillehei, Kevin O and Kerr, Janice M and Kleinschmidt-DeMasters, B.K and Wierman, Margaret E
Molecular and Cellular Endocrinology, ISSN 0303-7207, 12/2015, Volume 417, Issue C, pp. 73 - 83
The aim of this study was to examine whether differential expression of somatostatin receptors (SSTR) 1–5 and downstream effectors are different in densely... 
Acromegaly | Dense GH tumors | Somatostatin receptor | Sparse GH tumors | OCTREOTIDE | GRANULATION PATTERN | ANALOGS | DOWN-REGULATION | CELL-PROLIFERATION | E-CADHERIN | MICE LACKING | CELL BIOLOGY | THERAPY | ENDOCRINOLOGY & METABOLISM | SOMATOTROPH ADENOMAS | CLINICOPATHOLOGICAL FEATURES | Cadherins - metabolism | Signal Transduction | Pituitary Neoplasms - genetics | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Pituitary Neoplasms - pathology | Pituitary Neoplasms - metabolism | Receptors, Somatostatin - genetics | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Biomarkers, Tumor - metabolism | Cell Line, Tumor | Adult | Female | Biomarkers, Tumor - genetics | Receptors, Somatostatin - metabolism | Retrospective Studies | Cadherins - genetics | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Physiological aspects | Somatotropin | Tumors | Index Medicus
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11. Full Text Somatostatin Secreted by Islet δ-Cells Fulfills Multiple Roles as a Paracrine Regulator of Islet Function
by Astrid C. Hauge-Evans and Aileen J. King and Danielle Carmignac and Carolyn C. Richardson and Iain C.A.F. Robinson and Malcolm J. Low and Michael R. Christie and Shanta J. Persaud and Peter M. Jones
Diabetes, ISSN 0012-1797, 02/2009, Volume 58, Issue 2, pp. 403 - 411
Somatostatin Secreted by Islet δ-Cells Fulfills Multiple Roles as a Paracrine Regulator of Islet Function Astrid C. Hauge-Evans 1 , Aileen J. King 1 , Danielle... 
GLUCAGON-SECRETION | B-CELLS | RECEPTOR SUBTYPES | RAT PANCREAS | INSULIN-SECRETION | ENDOCRINOLOGY & METABOLISM | MIN6 PSEUDOISLETS | RELEASE | PHORBOL ESTER | PANCREATIC ALPHA-CELLS | LANGERHANS | Immunohistochemistry | Somatostatin - genetics | Islets of Langerhans - physiology | Male | Somatostatin - physiology | Mice, Knockout | Insulin - metabolism | Animals | Islets of Langerhans - metabolism | Islets of Langerhans - cytology | Radioimmunoassay | Somatostatin - deficiency | Polymerase Chain Reaction | Female | Glucagon - metabolism | Mice | Insulin Secretion | Somatostatin-Secreting Cells - metabolism | Index Medicus | Abridged Index Medicus | Islet Studies
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