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Cancer Cell, ISSN 1535-6108, 2010, Volume 17, Issue 6, pp. 535 - 546
Castration-recurrent prostate cancer (CRPC) is suspected to depend on androgen receptor (AR). The AF-1 region in the amino-terminal domain (NTD) of AR contains... 
CELLCYCLE | CROSS-TALK | SIGNAL-TRANSDUCTION PATHWAYS | STEROID-BINDING | ONCOLOGY | INDEPENDENT TRANSCRIPTIONAL ACTIVATION | NUCLEAR RECEPTORS | CREB-BINDING-PROTEIN | FACTORS INDUCE | BISPHENOL-A | PROLIFERATION | EXPRESSION | CELL BIOLOGY | Protein Binding - genetics | Apoptosis - drug effects | Humans | Protein Conformation - drug effects | Transcriptional Activation - drug effects | Male | Neoplasm Recurrence, Local - pathology | Antineoplastic Agents, Hormonal - adverse effects | CREB-Binding Protein - metabolism | Prostate - pathology | Protein Binding - drug effects | Serine Endopeptidases - genetics | Prostate - drug effects | Prostate-Specific Antigen - metabolism | Benzhydryl Compounds - therapeutic use | Prostatic Neoplasms - drug therapy | Chlorohydrins - therapeutic use | Prostate-Specific Antigen - genetics | Prostate - anatomy & histology | DNA - metabolism | Prostate-Specific Antigen - blood | Androgens - pharmacology | Cell Line, Tumor | Ligands | Mice, Inbred NOD | Mice | Protein Multimerization - drug effects | Androgen Receptor Antagonists | Gene Expression - drug effects | Receptors, Androgen - metabolism | Neoplasm Recurrence, Local - drug therapy | Benzhydryl Compounds - adverse effects | Castration | Antineoplastic Agents, Hormonal - therapeutic use | Molecular Structure | Response Elements - genetics | Chlorohydrins - pharmacology | Chlorohydrins - adverse effects | Prostatic Neoplasms - pathology | Antineoplastic Agents, Hormonal - pharmacology | Prostatic Neoplasms - surgery | Protein Interaction Domains and Motifs - drug effects | Receptors, Steroid - drug effects | Mice, SCID | DNA - genetics | Xenograft Model Antitumor Assays | Animals | Benzhydryl Compounds - pharmacology | Cell Proliferation - drug effects | Chemical oceanography | Prostate cancer | Index Medicus
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 10/2013, Volume 272, Issue 1, pp. 127 - 136
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 04/2013, Volume 19, Issue 4, pp. 418 - 420
Co-therapy with rifampicin (RIF) and isoniazid (INH) used to treat tuberculosis in humans frequently causes liver injury. Here, using a pregnane X receptor... 
MEDICINE, RESEARCH & EXPERIMENTAL | DRUG-INTERACTIONS | GENOTYPE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ERYTHROPOIETIC-PROTOPORPHYRIA | INJURY | LIVER TOXICITY | MODEL | PREGNANE-X-RECEPTOR | CELL BIOLOGY | DISEASE | GENE-EXPRESSION | DEFINES | Antitubercular Agents - adverse effects | Liver - pathology | Receptors, Steroid - metabolism | Humans | Isoniazid - adverse effects | Male | Alanine Transaminase - blood | Drug Therapy, Combination - adverse effects | Rifampin - adverse effects | Liver - drug effects | Heme - biosynthesis | Isoniazid - therapeutic use | Rifampin - therapeutic use | Rifampin - administration & dosage | Chemical and Drug Induced Liver Injury - pathology | Alkaline Phosphatase - blood | Chemical and Drug Induced Liver Injury - etiology | Antitubercular Agents - therapeutic use | Liver - metabolism | Protoporphyrins - biosynthesis | Heme - analysis | Mice, Transgenic | Receptors, Steroid - drug effects | Protoporphyrins - analysis | Liver - chemistry | Isoniazid - administration & dosage | Animals | Chemical and Drug Induced Liver Injury - metabolism | Receptors, Steroid - physiology | Antitubercular Agents - administration & dosage | Mice | Complications and side effects | Transcription factors | Usage | Liver | Analysis | Antitubercular agents | Research | Injuries | Side effects | Pharmacology | Tuberculosis | Drug therapy | Toxicity
Journal Article
Journal Article
Reproductive Toxicology, ISSN 0890-6238, 2011, Volume 31, Issue 4, pp. 409 - 417
Bisphenol A (BPA) is used in the manufacture of many products and is ubiquitous in the environment. Adverse effects of BPA on animal reproductive health have... 
Kisspeptin | Steroidogenesis | GPR-54 | Gonadotrophin | ENDOCRINE-DISRUPTING CHEMICALS | FETAL EXPOSURE | ENVIRONMENT | INCREASES | UDP-GLUCURONOSYLTRANSFERASE | REPRODUCTIVE BIOLOGY | ESTROGENIC CHEMICALS | HORMONE | XENOESTROGENS | TOXICOLOGY | PLACENTAL-TRANSFER | BINDING | Prenatal Exposure Delayed Effects | Testis - metabolism | Age Factors | Phenols - toxicity | Gonadal Steroid Hormones - biosynthesis | Male | RNA, Messenger - metabolism | Receptors, Peptide - drug effects | Hypothalamo-Hypophyseal System - growth & development | Ovary - growth & development | Receptors, Peptide - genetics | Testis - drug effects | Dose-Response Relationship, Drug | Aging | Female | Ovary - drug effects | Environmental Pollutants - toxicity | Receptors, G-Protein-Coupled - drug effects | Ovary - metabolism | Hypothalamo-Hypophyseal System - drug effects | Benzhydryl Compounds | Gonadotropins, Pituitary - metabolism | Gene Expression Regulation, Developmental - drug effects | Receptors, Steroid - drug effects | Testis - growth & development | Gestational Age | Hypothalamo-Hypophyseal System - metabolism | Endocrine Disruptors - toxicity | Pregnancy | Reproduction - drug effects | No-Observed-Adverse-Effect Level | Animals | Analysis of Variance | Receptors, Steroid - genetics | Mice | Receptors, G-Protein-Coupled - genetics | Enzymes | Testosterone | Messenger RNA | Bisphenol-A | Analysis | Estrogen | Phenols | Hormones | Animal models | Follicle-stimulating hormone | Perinatal exposure | Circuits | Estrogens | mRNA | Hypothalamus | Hypothalamic-pituitary-gonadal axis | Bisphenol A | Side effects | Feedback | Aromatase | Gonadotropin-releasing hormone | Kiss1 protein | Testes | Steroid hormones
Journal Article