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Journal of Biological Chemistry, ISSN 0021-9258, 09/2016, Volume 291, Issue 36, pp. 18947 - 18958
The thromboxane A2 receptor (TP) has been implicated in restenosis after vascular injury, which induces vascular smooth muscle cell (VSMC) migration and... 
TISSUE-GROWTH | F-ACTIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | BALLOON INJURY | HIPPO PATHWAY | NEOINTIMAL HYPERPLASIA | HYPERTENSIVE RATS | SIZE-CONTROL | RECEPTOR ANTAGONIST | CORONARY ANGIOPLASTY | Muscle, Smooth, Vascular - metabolism | Humans | Phosphoproteins - metabolism | Cell Movement - physiology | GTP-Binding Protein alpha Subunits, G12-G13 - metabolism | Gene Knockdown Techniques | Receptors, Thromboxane A2, Prostaglandin H2 - agonists | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Thromboxane A2 - genetics | Receptors, Thromboxane A2, Prostaglandin H2 - metabolism | Cell Proliferation - physiology | GTP-Binding Protein alpha Subunits, G12-G13 - genetics | Fatty Acids, Unsaturated - pharmacology | Phosphoproteins - genetics | Transcription Factors - genetics | Muscle, Smooth, Vascular - cytology | Receptors, Thromboxane A2, Prostaglandin H2 - genetics | Thromboxane A2 - metabolism | Transcription Factors - metabolism | Bridged Bicyclo Compounds, Heterocyclic - pharmacology | Adaptor Proteins, Signal Transducing - genetics | Signal Transduction - physiology | HeLa Cells | Adaptor Proteins, Signal Transducing - metabolism | Hippo pathway | Signal Transduction | yes-associated protein (YAP) | cell migration | G protein-coupled receptor (GPCR) | TAZ | vascular smooth muscle cells | thromboxane A2
Journal Article
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2014, Volume 211, Issue 13, pp. 2507 - 2517
Journal Article
Thrombosis and Haemostasis, ISSN 0340-6245, 09/2007, Volume 98, Issue 3, pp. 662 - 669
Journal Article
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 02/2014, Volume 171, Issue 3, pp. 676 - 687
Background and Purpose Dysregulation of the thromboxane A2 (TP) receptor, resulting in agonist hypersensitivity and hyper‐responsiveness, contributes to... 
thromboxane | hypoxia | pulmonary hypertension | PKA | PROTEIN-KINASE-C | HOMOLOGOUS DESENSITIZATION | HUMAN PLATELETS | BETA ISOFORMS | ARRIVE GUIDELINES | A RECEPTOR | ARACHIDONIC-ACID METABOLITES | PHARMACOLOGY & PHARMACY | MEDIATED DESENSITIZATION | BETA-ADRENERGIC RECEPTOR | MAJOR SITE | Enzyme Activators - pharmacology | Humans | Cyclic AMP-Dependent Protein Kinases - chemistry | Myocytes, Smooth Muscle - pathology | Cell Hypoxia | Pulmonary Artery - metabolism | Protein Processing, Post-Translational - drug effects | Receptors, Thromboxane A2, Prostaglandin H2 - agonists | HEK293 Cells | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Phosphorylation - drug effects | Myocytes, Smooth Muscle - drug effects | Mutant Proteins - antagonists & inhibitors | Myocytes, Smooth Muscle - metabolism | Thromboxane A2 - pharmacology | Disease Models, Animal | Animals, Newborn | Cyclic AMP-Dependent Protein Kinases - metabolism | Recombinant Proteins - metabolism | Thromboxane A2 - analogs & derivatives | Mutagenesis, Site-Directed | Specific Pathogen-Free Organisms | Receptors, Thromboxane A2, Prostaglandin H2 - metabolism | Cells, Cultured | Mutant Proteins - genetics | Recombinant Proteins - chemistry | Mutant Proteins - metabolism | Persistent Fetal Circulation Syndrome - metabolism | Pulmonary Artery - drug effects | Receptors, Thromboxane A2, Prostaglandin H2 - genetics | Serine - metabolism | Thromboxane A2 - metabolism | Animals | Calcium Signaling - drug effects | Protein Kinase Inhibitors - pharmacology | Receptors, Thromboxane A2, Prostaglandin H2 - antagonists & inhibitors | Persistent Fetal Circulation Syndrome - pathology | Sus scrofa | Pulmonary Artery - pathology | Amino Acid Substitution | Mutant Proteins - agonists | Persistent Fetal Circulation Syndrome - enzymology | Swine | Serine | Pulmonary hypertension | Hypoxia | Phosphorylation | Kinases | Pulmonary arteries | Hypertension | Protein kinase A | Neonates | Alanine | Calcium mobilization | Thromboxane A2 | Hypersensitivity | Vasoconstriction | Pulmonary artery | Agonists | Affinity | Research Papers
Journal Article
Journal of Vascular Research, ISSN 1018-1172, 03/2016, Volume 52, Issue 4, pp. 279 - 288
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2016, Volume 11, Issue 6, p. e0158438
Chronic rhinosinusitis without nasal polyps (CRSsNP) is a common chronic disease and the etiology remains unclear. Thromboxane A2 (TXA2) participates in... 
Interleukin-8 - genetics | Sinusitis - metabolism | Receptors, Thromboxane A2, Prostaglandin H2 - metabolism | Cells, Cultured | Fatty Acids, Unsaturated - pharmacology | Chemokine CXCL1 - genetics | Phosphatidylinositol 3-Kinases - metabolism | Case-Control Studies | Thromboxane A2 - metabolism | Bridged Bicyclo Compounds, Heterocyclic - pharmacology | Receptors, Thromboxane A2, Prostaglandin H2 - agonists | Rhinitis - metabolism | Protein Kinase C - metabolism | Fibroblasts - drug effects | 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid - pharmacology | Cyclic AMP Response Element-Binding Protein - metabolism | Nasal Mucosa - cytology | Sinusitis - pathology | Nasal Mucosa - metabolism | Interleukin-8 - metabolism | Rhinitis - pathology | Chemokine CXCL1 - metabolism | Second Messenger Systems | Fibroblasts - metabolism | Protein kinase A | Immunohistochemistry | Phosphorylation | Protein kinase C | Disease | Transcription | Rhinosinusitis | Mucosa | Otolaryngology | Kinases | Western blotting | Proteins | Receptors | Pathways | Metabolites | Etiology | Surgery | Nose | Fibroblasts | Enzyme-linked immunosorbent assay | Cytokines | Thromboxane A2 | Cyclic AMP | Pharmacology | Inflammation | Platelet aggregation | siRNA | Cyclic AMP response element-binding protein | Gene expression | Patients | Allergies | Polymerase chain reaction | Medicine | Hospitals | Polyps | Chemokines | Binding sites
Journal Article
Molecular Carcinogenesis, ISSN 0899-1987, 08/2014, Volume 53, Issue 8, pp. 659 - 666
Journal Article
CIRCULATION RESEARCH, ISSN 0009-7330, 10/2008, Volume 103, Issue 9, pp. 1037 - U302
Isoprostanes are endogenously formed end products of lipid peroxidation. Furthermore, they are markers of oxidative stress and independent risk markers of... 
thromboxane A receptor | CARDIAC & CARDIOVASCULAR SYSTEMS | SERIES | Rho kinase | angiogenesis | QUANTIFICATION | RISK | 8-ISO-PROSTAGLANDIN F2-ALPHA | isoprostanes | 8-EPI-PROSTAGLANDIN F-2 ALPHA | ADHESION | F-2-ISOPROSTANES | DISEASE | migration | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | Neovascularization, Physiologic - drug effects | Phosphorylation | Oxidative Stress | Humans | Actins - metabolism | Vascular Endothelial Growth Factor A - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Dinoprost - metabolism | Coronary Vessels - metabolism | Isoprostanes - metabolism | RNA Interference | Sulfonylurea Compounds - pharmacology | Receptors, Thromboxane A2, Prostaglandin H2 - drug effects | Dinoprost - analogs & derivatives | Dioxanes - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Focal Adhesion Kinase 1 - metabolism | Chorioallantoic Membrane - blood supply | Coronary Vessels - drug effects | Hydrazines - pharmacology | Endothelial Cells - metabolism | Receptors, Thromboxane A2, Prostaglandin H2 - metabolism | Tissue Culture Techniques | Cells, Cultured | Angiogenesis Inhibitors - pharmacology | Receptors, Thromboxane A2, Prostaglandin H2 - genetics | Chick Embryo | Dinoprostone - metabolism | Cell Movement - drug effects | Stress Fibers - metabolism | Angiogenesis Inhibitors - metabolism | Animals | 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid - pharmacology | Dinoprostone - analogs & derivatives | Mice | Prostaglandins A - metabolism | Endothelial Cells - pathology | Endothelial Cells - enzymology | Apoptosis | Endothelial Cells - drug effects | RNA, Small Interfering - metabolism
Journal Article
Journal of Agricultural and Food Chemistry, ISSN 0021-8561, 02/2009, Volume 57, Issue 4, pp. 1589 - 1594
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2011, Volume 31, Issue 1, pp. 125 - 132
OBJECTIVE—The thromboxane receptor (TPr) and multiple TPr ligands, including thromboxane A2 (TxA2) and prostaglandin H2, are elevated during vascular and... 
peroxynitrite | nitric oxide synthase | prostacyclin | signal transduction | thromboxanes | Phosphorylation | Apoptosis - drug effects | NADPH Oxidases - metabolism | Cytochrome P-450 Enzyme System - metabolism | Phosphoproteins - metabolism | Biopterin - analogs & derivatives | Tyrosine - analogs & derivatives | Biopterin - metabolism | RNA Interference | Receptors, Thromboxane A2, Prostaglandin H2 - agonists | Time Factors | Cattle | Protein Kinase C - metabolism | Superoxides - metabolism | NADPH Oxidases - genetics | Nitric Oxide Synthase Type III - metabolism | Intramolecular Oxidoreductases - metabolism | Cell Survival - drug effects | Receptors, Thromboxane A2, Prostaglandin H2 - metabolism | Mice, Inbred C57BL | NADPH Oxidases - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Receptors, Immunologic - deficiency | Fatty Acids, Unsaturated - pharmacology | Protein Kinase C - antagonists & inhibitors | Mice, Knockout | Protein Transport | Bridged Bicyclo Compounds, Heterocyclic - pharmacology | Tyrosine - metabolism | Animals | Cyclic GMP - metabolism | Signal Transduction - drug effects | 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid - pharmacology | Peroxynitrous Acid - metabolism | Mice | Protein Processing, Post-Translational | Enzyme Activation | Oxidative Stress - drug effects | Endothelial Cells - pathology | Nitric Oxide - metabolism | Receptors, Immunologic - genetics | Endothelial Cells - enzymology | Endothelial Cells - drug effects | Thromboxane receptor | oxidant stress | prostacyclin synthase | eNOS uncoupling
Journal Article