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Journal Article
Hepatology, ISSN 0270-9139, 10/2009, Volume 50, Issue 4, pp. 1140 - 1151
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 03/2015, Volume 74, Issue 3, pp. e20 - e20
Journal Article
Molecular Endocrinology, ISSN 0888-8809, 03/2004, Volume 18, Issue 3, pp. 653 - 665
Growth differentiation factor-9 (GDF-9) is an oocyte-derived growth factor and a member of the TGF-β superfamily that includes TGF-β, activin, and bone... 
ALK7 SIGNALS | BONE MORPHOGENETIC PROTEIN | MAD-RELATED PROTEIN | ENDOCRINOLOGY & METABOLISM | OSTEOGENIC PROTEIN-1 | FOLLICULAR-GROWTH | TGF-BETA RECEPTOR | OVARIAN GRANULOSA-CELLS | IDENTIFICATION | HOMOLOG GDF-9B | LUTEAL CELLS | Phosphorylation | Receptors, Transforming Growth Factor beta - genetics | Trans-Activators - drug effects | Protein-Serine-Threonine Kinases | Activin Receptors - drug effects | Activins - metabolism | COS Cells - drug effects | Activin Receptors - genetics | Granulosa Cells - drug effects | Granulosa Cells - metabolism | COS Cells - metabolism | DNA-Binding Proteins - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Bone Morphogenetic Proteins - metabolism | Growth Differentiation Factor 9 | Trans-Activators - genetics | Female | Smad7 Protein | Promoter Regions, Genetic | Signal Transduction | Smad6 Protein | Cells, Cultured | DNA-Binding Proteins - drug effects | Rats | Activin Receptors, Type I - metabolism | DNA-Binding Proteins - genetics | Activin Receptors, Type I - genetics | Rats, Sprague-Dawley | Activin Receptors, Type I - drug effects | Transforming Growth Factor beta - pharmacology | Bone Morphogenetic Protein 15 | Smad3 Protein | Activin Receptors - metabolism | Animals | Activins - pharmacology | Receptors, Transforming Growth Factor beta - drug effects | Receptors, Transforming Growth Factor beta - metabolism | Intercellular Signaling Peptides and Proteins - pharmacology | Smad Proteins | Trans-Activators - metabolism | RNA, Small Interfering | Transforming Growth Factor beta - metabolism | Smad2 Protein
Journal Article
Journal Article
Osteoarthritis and Cartilage, ISSN 1063-4584, 2014, Volume 23, Issue 3, pp. 478 - 486
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 10/2015, Volume 764, pp. 413 - 423
Transforming growth factor-β (TGF-β) has both tumor suppressive and oncogenic activities. Autocrine TGF-β signaling supports tumor survival and growth in... 
TGF-β | SB431542 | p21 | Specificity protein 1 (Sp1) | Chemical compounds studied in this article SB431542 (PubChem CID: 4521392) | SP600125 (PubChem CID: 8515) | Cycloheximide (PubChem CID: 6197) | MG132 (PubChem CID: 462382) | PROTEIN | MICROSATELLITE INSTABILITY | TRANSCRIPTION | C-MYC | CELL-CYCLE ARREST | DEPENDENT KINASE INHIBITOR | BREAST-CANCER | TGF-beta | NASOPHARYNGEAL CARCINOMA | SP1 GENE | II GENE MUTATION | PHARMACOLOGY & PHARMACY | Receptors, Transforming Growth Factor beta - genetics | Humans | Gene Expression Regulation, Neoplastic | Cellular Senescence - drug effects | Cercopithecus aethiops | Male | Sp1 Transcription Factor - metabolism | Autocrine Communication - drug effects | Breast Neoplasms - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Transfection | MCF-7 Cells | RNA Interference | Time Factors | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Dioxoles - pharmacology | Female | Benzamides - pharmacology | Protein-Serine-Threonine Kinases - metabolism | Caco-2 Cells | HCT116 Cells | Protein-Serine-Threonine Kinases - genetics | Breast Neoplasms - drug therapy | Transforming Growth Factor beta - pharmacology | Animals | Breast Neoplasms - genetics | Receptors, Transforming Growth Factor beta - drug effects | Receptors, Transforming Growth Factor beta - metabolism | Signal Transduction - drug effects | Sp1 Transcription Factor - genetics | Breast Neoplasms - pathology | Protein-Serine-Threonine Kinases - drug effects | Cell Proliferation - drug effects | COS Cells
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 07/2015, Volume 74, Issue 7, pp. 1408 - 1416
ObjectivesWe have previously described the antifibrotic role of the soluble guanylate cyclase (sGC). The mode of action, however, remained elusive. In the... 
NO | GROWTH-FACTOR-BETA | TRANS-DIFFERENTIATION | FIBROTIC DISORDERS | DISEASE | KINASE | EXPERIMENTAL DERMAL FIBROSIS | RHEUMATOLOGY | SYSTEMIC-SCLEROSIS | HYPERTENSION | INDUCED FIBROBLAST ACTIVATION | MAP Kinase Signaling System - physiology | Receptors, Cytoplasmic and Nuclear - deficiency | Skin - metabolism | Humans | Scleroderma, Systemic - pathology | Fibrosis - metabolism | Case-Control Studies | Scleroderma, Systemic - physiopathology | Transforming Growth Factor beta - antagonists & inhibitors | Fibrosis - prevention & control | Skin - pathology | Disease Models, Animal | Fibroblasts - metabolism | Pyrazoles - pharmacology | Scleroderma, Systemic - metabolism | Cells, Cultured | Guanylate Cyclase - metabolism | Soluble Guanylyl Cyclase | Receptors, Cytoplasmic and Nuclear - agonists | Fibroblasts - pathology | Mice, Knockout | Collagen - metabolism | Guanylate Cyclase - deficiency | Animals | MAP Kinase Signaling System - drug effects | Cyclic GMP - metabolism | Receptors, Transforming Growth Factor beta - drug effects | Receptors, Transforming Growth Factor beta - metabolism | Signal Transduction - drug effects | Signal Transduction - physiology | Mice | Pyridines - pharmacology | In Vitro Techniques | Smad Proteins - metabolism | Transforming Growth Factor beta - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism
Journal Article