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Journal of Clinical Investigation, ISSN 0021-9738, 11/2012, Volume 122, Issue 11, pp. 4094 - 4104
TNF, an inflammatory cytokine that is enriched in the tumor microenvironment, promotes tumor growth and subverts innate immune responses to cancer cells. We... 
MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-NECROSIS-FACTOR | INHIBITION | INFLAMMATION | MACROPHAGES | DEATH | EXPRESSION | CANCER | ANTITUMOR | T-CELLS | FACTOR-ALPHA | Neoplasms - metabolism | Tumor Necrosis Factor-alpha - metabolism | Neoplasm Transplantation | Tumor Escape | Receptors, Tumor Necrosis Factor, Type I - immunology | Tumor Necrosis Factor-alpha - genetics | Caspase 8 - metabolism | Graft Rejection - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Receptors, Tumor Necrosis Factor, Type II - immunology | Caspase 8 - genetics | Signal Transduction - immunology | Neoplasms - genetics | T-Lymphocytes - metabolism | Myeloid Cells - immunology | Receptors, Tumor Necrosis Factor, Type II - metabolism | Tumor Necrosis Factor-alpha - immunology | T-Lymphocytes - pathology | Graft Rejection - pathology | CASP8 and FADD-Like Apoptosis Regulating Protein - genetics | Signal Transduction - genetics | Receptors, Tumor Necrosis Factor, Type I - genetics | CASP8 and FADD-Like Apoptosis Regulating Protein - biosynthesis | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Caspase 8 - immunology | Animals | Neoplasms - immunology | Cell Line, Tumor | Myeloid Cells - metabolism | T-Lymphocytes - immunology | Mice | Mice, Inbred BALB C | CASP8 and FADD-Like Apoptosis Regulating Protein - immunology | Graft Rejection - immunology | Myeloid Cells - pathology | Graft Rejection - genetics | Neoplasms - pathology | Tumor necrosis factor | Genetic aspects | Cellular signal transduction | Research | Properties | Gene expression | Apoptosis
Journal Article
Journal of Bone and Mineral Metabolism, ISSN 0914-8779, 11/2011, Volume 29, Issue 6, pp. 671 - 681
Journal Article
Immunological Reviews, ISSN 0105-2896, 11/2011, Volume 244, Issue 1, pp. 9 - 28
Tumor necrosis factor (TNF) is crucial for innate immunity, but deregulated TNF signaling also plays an eminent role in the pathogenesis of many chronic... 
signaling proteins | cytokines | signal transduction | apoptosis/autophagy | cytotoxicity | Signal transduction | Cytotoxicity | Apoptosis/autophagy | Cytokines | Signaling proteins | DEUBIQUITINATING ENZYME | autophagy | CRYSTAL-STRUCTURE | AFFINITY PURIFICATION | apoptosis | FACTOR RECEPTOR-1 | IMMUNOLOGY | ASSEMBLY COMPLEX | FACTOR-ALPHA | SIGNALING COMPLEX | TUMOR-NECROSIS-FACTOR | UNANCHORED POLYUBIQUITIN CHAINS | NF-KAPPA-B | Neoplasms - metabolism | Tumor Necrosis Factor-alpha - metabolism | Protein Binding - genetics | Receptors, Tumor Necrosis Factor, Type I - immunology | Humans | Tumor Necrosis Factor-alpha - genetics | Ubiquitin - metabolism | Apoptosis - genetics | Receptors, Tumor Necrosis Factor, Type I - metabolism | Receptors, Tumor Necrosis Factor, Type II - immunology | Transcriptional Activation - immunology | Autophagy - immunology | Signal Transduction - immunology | Inflammation - metabolism | Neoplasms - genetics | Receptors, Tumor Necrosis Factor, Type II - metabolism | Tumor Necrosis Factor-alpha - immunology | Autophagy - genetics | Inflammation - immunology | Signal Transduction - genetics | Ubiquitin - genetics | Ubiquitination - immunology | Immunity, Innate | Receptors, Tumor Necrosis Factor, Type I - genetics | Receptors, Tumor Necrosis Factor, Type II - genetics | Apoptosis - immunology | Neoplasms - immunology | Inflammation - genetics | Ubiquitin - immunology | Ubiquitination - genetics | Ubiquitin | Tumor necrosis factor | Cell death | Genes | Physiological aspects | Genetic research | Biochemistry | Inflammation | Genetic aspects | Cancer
Journal Article
Nature, ISSN 0028-0836, 08/2012, Volume 488, Issue 7412, pp. 508 - 511
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2011, Volume 141, Issue 6, pp. 2026 - 2038
Journal Article
Arthritis & Rheumatology, ISSN 2326-5191, 08/2014, Volume 66, Issue 8, pp. 2281 - 2289
Journal Article
Hepatology, ISSN 0270-9139, 12/2012, Volume 56, Issue 6, pp. 2353 - 2362
Interleukin (IL)‐33, a member of the IL‐1 cytokine family, positively correlates with acute hepatitis and chronic liver failure in mice and humans. IL‐33 is... 
CONCANAVALIN-A | TUMOR-NECROSIS-FACTOR | DEPENDENT LIVER-INJURY | A-INDUCED HEPATITIS | IFN-GAMMA | IN-VIVO | MICE | INTERLEUKIN-33 | LIGAND | GASTROENTEROLOGY & HEPATOLOGY | CELL-DEATH | Tumor Necrosis Factor-alpha - metabolism | Fas Ligand Protein - metabolism | Perforin - genetics | Tumor Necrosis Factor-alpha - genetics | Hepatocytes - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Interleukins - metabolism | Hepatitis, Animal - metabolism | Interleukins - genetics | Receptors, Tumor Necrosis Factor, Type II - metabolism | Acute Lung Injury - metabolism | Hepatitis, Animal - chemically induced | Natural Killer T-Cells | Galactosamine | Gene Expression | Interleukin-33 | Hepatitis, Animal - immunology | Antibodies, Monoclonal | Concanavalin A | Receptors, Tumor Necrosis Factor, Type I - genetics | TNF-Related Apoptosis-Inducing Ligand - metabolism | Receptors, Tumor Necrosis Factor, Type II - genetics | Animals | Acute Lung Injury - chemically induced | Mice | Perforin - metabolism | Primary Cell Culture | Fas Ligand Protein - genetics | RNA, Messenger | TNF-Related Apoptosis-Inducing Ligand - genetics | Hépatology and Gastroenterology | Receptors, Tumor Necrosis Factor, Type I | Tumor Necrosis Factor-alpha | Fas Ligand Protein | Life Sciences | Hepatitis, Animal | Human health and pathology | TNF-Related Apoptosis-Inducing Ligand | Hepatocytes | Interleukins | Receptors, Tumor Necrosis Factor, Type II | Perforin | Acute Lung Injury
Journal Article