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Cell death and differentiation, ISSN 1476-5403, 06/2014, Volume 21, Issue 10, pp. 1511 - 1521
...) and RIPK3 by receptors such as tumor necrosis factor (TNF) receptor-1. Structural studies indicate that activation of RIPK3 by RIPK1 involves the formation of oligomers via interactions of the RIP homotypic interaction motif (RHIM... 
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Protein Kinases - metabolism | Protein Structure, Tertiary | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Tumor Necrosis Factor-alpha - metabolism | Cell Line | Phosphorylation | Protein Kinases - genetics | Cell Survival | Humans | Protein Multimerization | Receptors, Tumor Necrosis Factor, Type I | Caspase 8 - metabolism | Imidazoles - pharmacology | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Necrosis - physiopathology | RNA Interference | Indoles - pharmacology | RNA, Small Interfering | Apoptosis - physiology | Enzyme Activation | Receptor-Interacting Protein Serine-Threonine Kinases - antagonists & inhibitors | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Index Medicus | Receptor-Interacting Protein Serine-Threonine Kinases/genetics | Protein Kinases/genetics | Receptor-Interacting Protein Serine-Threonine Kinases/antagonists & inhibitors | Caspase 8/metabolism | Receptor-Interacting Protein Serine-Threonine Kinases/metabolism | Tumor Necrosis Factor-alpha/antagonists & inhibitors | Indoles/pharmacology | Life Sciences | Tumor Necrosis Factor-alpha/metabolism | Imidazoles/pharmacology | Immunology | Apoptosis/physiology | Protein Kinases/metabolism | Necrosis/physiopathology | Original Paper
Journal Article
PloS one, ISSN 1932-6203, 12/2018, Volume 13, Issue 12, pp. e0209583 - e0209583
Tumor necrosis factor-like weak inducer of apoptosis (TWEAK), along with its receptor fibroblast growth factor-inducible (Fn... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Inflammation - pathology | Interleukin-8 - genetics | TWEAK Receptor - genetics | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Apoptosis - genetics | Male | Interleukin-1beta - genetics | Inflammation - blood | Adult | Female | Cytokine TWEAK - blood | Fibroblasts - metabolism | Interleukin-6 - genetics | TWEAK Receptor - blood | Gene Expression Regulation - genetics | Chemokine CCL2 - genetics | Signal Transduction - genetics | Graves Ophthalmopathy - genetics | Fibroblasts - pathology | Receptors, Tumor Necrosis Factor, Type I - genetics | Receptors, Tumor Necrosis Factor, Type II - genetics | Graves Ophthalmopathy - blood | Inflammation - genetics | Cytokine TWEAK - genetics | Graves Ophthalmopathy - pathology | Care and treatment | Cytokines | Graves' disease | Fibroblast growth factors | Genetic aspects | Research | Gene expression | T cells | Risk factors | Tumor necrosis factor receptors | TWEAK protein | Fibroblast growth factor | Transcription | Disease | Pathogenesis | Interleukin | Arthritis | Thyroid gland | Kinases | Tissues | Interleukin 6 | Proteins | Angiogenesis | Hyaluronan | Cell growth | Surgery | Fibroblasts | Tumor necrosis factor-TNF | Bioindicators | Growth factors | Interleukin 8 | Thyroid | Inflammation | Tumor necrosis factor-α | Patients | Myogenesis | Medicine | Signaling | Government agencies | Monocytes | Hospitals | Monocyte chemoattractant protein | Biomarkers | Diagnostic systems | Hyaluronic acid | Monocyte chemoattractant protein 1 | Apoptosis | Tumors | Index Medicus
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 03/2017, Volume 152, Issue 4, pp. 867 - 879
Journal Article
Neuroscience, ISSN 0306-4522, 2008, Volume 157, Issue 2, pp. 414 - 423
Abstract Recent studies suggest that tumor necrosis factor-alpha (TNF) sensitizes primary afferent neurons, and thus facilitates neuropathic pain... 
Neurology | DRG | neuropathic pain | ectopic activity | cytokines | TNF receptor | TNF | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Hyperalgesia - chemically induced | Receptors, Tumor Necrosis Factor - classification | Receptors, Tumor Necrosis Factor, Type II - chemistry | Humans | Male | Ganglia, Spinal - cytology | Ligation - methods | Dose-Response Relationship, Drug | Nerve Fibers - physiology | Receptors, Tumor Necrosis Factor, Type I - chemistry | Time Factors | Reaction Time - drug effects | Receptors, Tumor Necrosis Factor, Type I - pharmacology | Pain Threshold - drug effects | Action Potentials - drug effects | Receptors, Tumor Necrosis Factor - genetics | Physical Stimulation | Receptors, Tumor Necrosis Factor - chemistry | Rats | Nerve Fibers - drug effects | Receptors, Tumor Necrosis Factor - administration & dosage | Mutation - genetics | Receptors, Tumor Necrosis Factor, Type I - genetics | Rats, Sprague-Dawley | Receptors, Tumor Necrosis Factor, Type II - genetics | Spinal Cord - surgery | Hyperalgesia - physiopathology | Animals | Receptors, Tumor Necrosis Factor, Type II - pharmacology | Sensory Receptor Cells - drug effects | Analysis of Variance | In Vitro Techniques | Pain Measurement | Serum albumin | Tumor necrosis factor | Neurons | Tumors | Necrosis | Index Medicus
Journal Article
The Journal of cell biology, ISSN 1540-8140, 08/2007, Volume 178, Issue 5, pp. 829 - 841
The tumor necrosis factor type 1 death receptor (TNFR1) contributes to apoptosis... 
Pathology | Neurons | Transgenic animals | Entorhinal cortex | Memory | Antibodies | Amyloids | Alzheimers disease | Hippocampus | Microglia | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Tumor Necrosis Factor-alpha - metabolism | Amyloid Precursor Protein Secretases - genetics | Memory Disorders - physiopathology | Microglia - metabolism | Memory Disorders - genetics | Humans | Memory Disorders - metabolism | NF-kappa B - metabolism | Neurons - cytology | Aspartic Acid Endopeptidases - genetics | Receptors, Tumor Necrosis Factor, Type I - metabolism | Alzheimer Disease - pathology | Amyloid beta-Peptides - genetics | Gene Deletion | Amyloid beta-Peptides - metabolism | Neprilysin - genetics | Neurons - metabolism | Cerebral Amyloid Angiopathy - genetics | Disease Models, Animal | Microglia - cytology | Insulysin - metabolism | Promoter Regions, Genetic | Alzheimer Disease - physiopathology | Cerebral Amyloid Angiopathy - physiopathology | Gene Expression Regulation | Behavior, Animal - physiology | Mice, Transgenic | Neprilysin - metabolism | Receptors, Tumor Necrosis Factor, Type I - genetics | Mice, Knockout | Learning - physiology | Amyloid Precursor Protein Secretases - metabolism | Cerebral Amyloid Angiopathy - pathology | Animals | Aspartic Acid Endopeptidases - metabolism | Alzheimer Disease - metabolism | Insulysin - genetics | Mice | Alzheimer Disease - genetics | Index Medicus
Journal Article
Gut, ISSN 0017-5749, 03/2006, Volume 55, Issue 3, pp. 415 - 424
Background: While tumour necrosis factor α (TNF-α) appears to be associated with the development of non-alcoholic steatohepatitis (NASH... 
Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Receptors, Tumor Necrosis Factor, Type II - physiology | Tissue Inhibitor of Metalloproteinase-1 - biosynthesis | Fatty Liver - pathology | Fatty Liver - complications | Liver Cirrhosis, Experimental - etiology | Choline Deficiency - complications | Male | Mitochondria, Liver - physiology | Receptors, Tumor Necrosis Factor, Type I - deficiency | Reverse Transcriptase Polymerase Chain Reaction - methods | Liver Cirrhosis, Experimental - pathology | Kupffer Cells - metabolism | Methionine - deficiency | Fatty Liver - metabolism | Signal Transduction | Cell Adhesion Molecules - biosynthesis | RNA, Messenger - genetics | Gene Expression Regulation | Receptors, Tumor Necrosis Factor, Type I - genetics | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Receptors, Tumor Necrosis Factor, Type II - deficiency | Tissue Inhibitor of Metalloproteinase-1 - genetics | Animals | Receptors, Tumor Necrosis Factor, Type I - physiology | Tumor Necrosis Factor-alpha - physiology | Mice | Mutation | Tumor Necrosis Factor-alpha - biosynthesis | Liver Cirrhosis, Experimental - metabolism | Apoptosis | Index Medicus | Abridged Index Medicus | Liver Fibrosis | non‐alcoholic steatohepatitis | tissue inhibitor of metalloproteinase 1 | kupffer cell | tumour necrosis factor‐α
Journal Article
Journal Article