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Cell death and differentiation, ISSN 1476-5403, 06/2014, Volume 21, Issue 10, pp. 1511 - 1521
...) and RIPK3 by receptors such as tumor necrosis factor (TNF) receptor-1. Structural studies indicate that activation of RIPK3 by RIPK1 involves the formation of oligomers via interactions of the RIP homotypic interaction motif (RHIM... 
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Protein Kinases - metabolism | Protein Structure, Tertiary | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Tumor Necrosis Factor-alpha - metabolism | Cell Line | Phosphorylation | Protein Kinases - genetics | Cell Survival | Humans | Protein Multimerization | Receptors, Tumor Necrosis Factor, Type I | Caspase 8 - metabolism | Imidazoles - pharmacology | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Necrosis - physiopathology | RNA Interference | Indoles - pharmacology | RNA, Small Interfering | Apoptosis - physiology | Enzyme Activation | Receptor-Interacting Protein Serine-Threonine Kinases - antagonists & inhibitors | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Index Medicus | Receptor-Interacting Protein Serine-Threonine Kinases/genetics | Protein Kinases/genetics | Receptor-Interacting Protein Serine-Threonine Kinases/antagonists & inhibitors | Caspase 8/metabolism | Receptor-Interacting Protein Serine-Threonine Kinases/metabolism | Tumor Necrosis Factor-alpha/antagonists & inhibitors | Indoles/pharmacology | Life Sciences | Tumor Necrosis Factor-alpha/metabolism | Imidazoles/pharmacology | Immunology | Apoptosis/physiology | Protein Kinases/metabolism | Necrosis/physiopathology | Original Paper
Journal Article
Journal Article
Cardiovascular research, ISSN 1755-3245, 11/2009, Volume 84, Issue 2, pp. 201 - 208
...) pathway, which involves the activation of the cytokine tumour necrosis factor alpha (TNFα) and signal transducer and activator of transcription-3... 
Ischaemia | Infarction | Reperfusion | Postconditioning | Preconditioning | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Tumor Necrosis Factor-alpha - metabolism | Phosphorylation | Tumor Necrosis Factor-alpha - genetics | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Glycogen Synthase Kinase 3 beta | Male | Phosphatidylinositol 3-Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Tumor Necrosis Factor-alpha - deficiency | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Myocardial Reperfusion Injury - pathology | Receptors, Tumor Necrosis Factor, Type II - metabolism | Myocardial Infarction - pathology | Myocardium - metabolism | STAT3 Transcription Factor - deficiency | Flavonoids - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Mice, Inbred C57BL | Myocardium - pathology | Tyrphostins - pharmacology | Myocardial Infarction - metabolism | Glycogen Synthase Kinase 3 - metabolism | Receptors, Tumor Necrosis Factor, Type I - genetics | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Receptors, Tumor Necrosis Factor, Type II - deficiency | Myocardial Reperfusion Injury - metabolism | Animals | Androstadienes - pharmacology | Signal Transduction - drug effects | Mice | Myocardial Infarction - prevention & control | Protein Kinase Inhibitors - pharmacology | STAT3 Transcription Factor - antagonists & inhibitors | Myocardial Reperfusion Injury - prevention & control | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Index Medicus
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 11/2003, Volume 278, Issue 46, pp. 45777 - 45784
.... To examine putative mechanisms and cross-talk with insulin, 3T3-L1 adipocytes were cultured for different times with IL-6 and tumor necrosis factor alpha (TNF-alpha... 
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Tumor Necrosis Factor-alpha - metabolism | Up-Regulation | Phosphorylation | Humans | Middle Aged | Repressor Proteins | Phosphoproteins - metabolism | RNA, Messenger - metabolism | Suppressor of Cytokine Signaling Proteins | Dose-Response Relationship, Drug | Adipose Tissue - metabolism | Mitogen-Activated Protein Kinase Kinases - metabolism | Muscle Proteins | MAP Kinase Kinase 4 | Biological Transport | Time Factors | Insulin Receptor Substrate Proteins | JNK Mitogen-Activated Protein Kinases | Adult | Transcription, Genetic | Interleukin-8 - metabolism | Interleukin-6 - metabolism | Monosaccharide Transport Proteins - metabolism | Tyrosine - chemistry | RNA - metabolism | Cytokines - metabolism | Glucose Transporter Type 4 | Insulin Resistance | 3T3-L1 Cells | Reverse Transcriptase Polymerase Chain Reaction | Suppressor of Cytokine Signaling 3 Protein | Blotting, Northern | Tyrosine - metabolism | Animals | Proteins - metabolism | Adipocytes - metabolism | Glucose - metabolism | Mice | Transcription Factors | insulin receptors | pS-307 protein | peroxisome proliferator-activated receptors | IRS-1 protein | GLUT-4 protein | JNK protein | insulin-receptor substrate 2 | insulin resistance | glucose transporter GLUT4 | insulin-receptor substrate 1 protein | Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2012, Volume 143, Issue 5, pp. 1330 - 1340.e1
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 03/2017, Volume 152, Issue 4, pp. 867 - 879
Journal Article
The Journal of cell biology, ISSN 1540-8140, 08/2007, Volume 178, Issue 5, pp. 829 - 841
The tumor necrosis factor type 1 death receptor (TNFR1) contributes to apoptosis... 
Pathology | Neurons | Transgenic animals | Entorhinal cortex | Memory | Antibodies | Amyloids | Alzheimers disease | Hippocampus | Microglia | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Tumor Necrosis Factor-alpha - metabolism | Amyloid Precursor Protein Secretases - genetics | Memory Disorders - physiopathology | Microglia - metabolism | Memory Disorders - genetics | Humans | Memory Disorders - metabolism | NF-kappa B - metabolism | Neurons - cytology | Aspartic Acid Endopeptidases - genetics | Receptors, Tumor Necrosis Factor, Type I - metabolism | Alzheimer Disease - pathology | Amyloid beta-Peptides - genetics | Gene Deletion | Amyloid beta-Peptides - metabolism | Neprilysin - genetics | Neurons - metabolism | Cerebral Amyloid Angiopathy - genetics | Disease Models, Animal | Microglia - cytology | Insulysin - metabolism | Promoter Regions, Genetic | Alzheimer Disease - physiopathology | Cerebral Amyloid Angiopathy - physiopathology | Gene Expression Regulation | Behavior, Animal - physiology | Mice, Transgenic | Neprilysin - metabolism | Receptors, Tumor Necrosis Factor, Type I - genetics | Mice, Knockout | Learning - physiology | Amyloid Precursor Protein Secretases - metabolism | Cerebral Amyloid Angiopathy - pathology | Animals | Aspartic Acid Endopeptidases - metabolism | Alzheimer Disease - metabolism | Insulysin - genetics | Mice | Alzheimer Disease - genetics | Index Medicus
Journal Article