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Neuroscience, ISSN 0306-4522, 2008, Volume 157, Issue 2, pp. 414 - 423
Abstract Recent studies suggest that tumor necrosis factor-alpha (TNF) sensitizes primary afferent neurons, and thus facilitates neuropathic pain. Here, we... 
Neurology | DRG | neuropathic pain | ectopic activity | cytokines | TNF receptor | TNF | RHEUMATOID-ARTHRITIS | PRIMARY AFFERENTS | RAT | ALPHA TNF-ALPHA | TACTILE ALLODYNIA | DORSAL-ROOT GANGLION | NEUROSCIENCES | CELL-DEATH | SCIATIC-NERVE | SPINAL NERVE LIGATION | UP-REGULATION | Hyperalgesia - chemically induced | Receptors, Tumor Necrosis Factor - classification | Receptors, Tumor Necrosis Factor, Type II - chemistry | Humans | Male | Ganglia, Spinal - cytology | Ligation - methods | Dose-Response Relationship, Drug | Nerve Fibers - physiology | Receptors, Tumor Necrosis Factor, Type I - chemistry | Time Factors | Reaction Time - drug effects | Receptors, Tumor Necrosis Factor, Type I - pharmacology | Pain Threshold - drug effects | Action Potentials - drug effects | Receptors, Tumor Necrosis Factor - genetics | Physical Stimulation | Receptors, Tumor Necrosis Factor - chemistry | Rats | Nerve Fibers - drug effects | Receptors, Tumor Necrosis Factor - administration & dosage | Mutation - genetics | Receptors, Tumor Necrosis Factor, Type I - genetics | Rats, Sprague-Dawley | Receptors, Tumor Necrosis Factor, Type II - genetics | Spinal Cord - surgery | Hyperalgesia - physiopathology | Animals | Receptors, Tumor Necrosis Factor, Type II - pharmacology | Sensory Receptor Cells - drug effects | Analysis of Variance | In Vitro Techniques | Pain Measurement | Neurosciences | Serum albumin | Tumor necrosis factor | Neurons | Tumors | Necrosis | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2014, Volume 124, Issue 4, pp. 1537 - 1551
Development of host protective immunity against Mycobacterium tuberculosis infection is critically dependent on the inflammatory cytokine TNF. TNF signals... 
MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-NECROSIS-FACTOR | BACILLUS-CALMETTE-GUERIN | TRANSMEMBRANE TNF | DEFICIENT MICE | BACTERIAL-INFECTION | HUMAN ALVEOLAR MACROPHAGES | DENDRITIC CELL MATURATION | FACTOR RECEPTOR | INDUCED GRANULOMA-FORMATION | FACTOR-ALPHA | Tumor Necrosis Factor-alpha - metabolism | Tuberculosis, Pulmonary - microbiology | Tumor Necrosis Factor Decoy Receptors - genetics | Receptors, Tumor Necrosis Factor, Type I - immunology | Tumor Necrosis Factor Decoy Receptors - immunology | Dendritic Cells - immunology | Mycobacterium tuberculosis - immunology | Male | Receptors, Tumor Necrosis Factor, Type II - immunology | Host-Pathogen Interactions - immunology | CD4-Positive T-Lymphocytes - immunology | Mycobacterium tuberculosis - pathogenicity | Receptors, Tumor Necrosis Factor, Type I - deficiency | Signal Transduction - immunology | Female | Interleukin-12 Subunit p40 - metabolism | Tumor Necrosis Factor Decoy Receptors - deficiency | Lymphocyte Activation | Mice, Inbred C57BL | Solubility | Tuberculosis, Pulmonary - pathology | Receptors, Tumor Necrosis Factor, Type I - genetics | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Receptors, Tumor Necrosis Factor, Type II - deficiency | Granuloma - pathology | Tuberculosis, Pulmonary - immunology | Animals | Mice | Prevention | Care and treatment | Immune response | Mycobacterium tuberculosis | Tumor necrosis factor | Physiological aspects | Drug targeting | Regulation | Research | Studies | Tuberculosis | Cytokines | Rodents | Mortality | Ligands | Infections
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 05/2007, Volume 292, Issue 5, pp. 1660 - 1671
Although p38 MAPK activation is essential for myogenesis, the upstream signaling mechanism that activates p38 during myogenesis remains undefined. We recently... 
Myosin heavy chain | Myogenin | Myocyte enhancer factor-2C | Mitogen-activated protein kinase | Akt | Tumor necrosis factor-α | p21 | myocyte enhancer factor-2C | myosin heavy chain | PROTEIN-KINASE PATHWAY | PHYSIOLOGY | myogenin | UBIQUITIN LIGASE | INFLAMMATORY MYOPATHIES | AUTOCRINE SECRETION | CELL BIOLOGY | tumor necrosis factor-alpha | mitogen-activated protein kinase | SKELETAL-MUSCLE | TUMOR-NECROSIS-FACTOR | SIGNALING PATHWAY | GENE-EXPRESSION | TERMINAL DIFFERENTIATION | GROWTH-FACTOR-I | Tumor Necrosis Factor-alpha - metabolism | Cobra Cardiotoxin Proteins | Muscle, Skeletal - metabolism | Muscle Fibers, Skeletal - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Dose-Response Relationship, Drug | MAP Kinase Kinase 6 - metabolism | Receptors, Tumor Necrosis Factor, Type I - deficiency | Myoblasts - metabolism | Muscular Diseases - physiopathology | Receptors, Tumor Necrosis Factor, Type II - metabolism | Muscle, Skeletal - drug effects | Cell Differentiation | p38 Mitogen-Activated Protein Kinases - metabolism | Muscular Diseases - chemically induced | Disease Models, Animal | Autocrine Communication | Cell Line | Muscular Diseases - metabolism | Receptors, Tumor Necrosis Factor, Type I - genetics | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Receptors, Tumor Necrosis Factor, Type II - deficiency | Tumor Necrosis Factor-alpha - pharmacology | Regeneration - drug effects | Animals | Muscle Development - drug effects | Muscle, Skeletal - physiopathology | Mice | Enzyme Activation | Muscle, Skeletal - pathology | tumor necrosis factor-α
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 08/2011, Volume 208, Issue 18, pp. 1563 - 1570
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2007, Volume 117, Issue 12, pp. 3833 - 3845
Journal Article
Journal Article
International Immunopharmacology, ISSN 1567-5769, 03/2017, Volume 44, pp. 143 - 152
Journal Article
Gastroenterology, ISSN 0016-5085, 2015, Volume 149, Issue 4, pp. 993 - 1005.e2
Journal Article
FEBS Letters, ISSN 0014-5793, 04/2017, Volume 591, Issue 8, pp. 1187 - 1195
Exploiting regulatory T cells (Tregs) to control aberrant immune reactions is a promising therapeutic approach, but is hampered by their relative paucity. In... 
TNFRSF25 | DR3 | TNFSF15 | Treg | TL1A | ACTIVATION | IFN-GAMMA PRODUCTION | TNF-LIKE LIGAND | BIOCHEMISTRY & MOLECULAR BIOLOGY | ALLERGIC LUNG INFLAMMATION | PERIPHERAL-BLOOD | VERSUS-HOST-DISEASE | CELL BIOLOGY | BIOPHYSICS | EXPRESSION | MARROW TRANSPLANTATION | T-Lymphocytes, Regulatory - metabolism | Receptors, Tumor Necrosis Factor, Type II - chemistry | Humans | Receptors, Tumor Necrosis Factor, Member 25 - metabolism | Receptors, Tumor Necrosis Factor, Member 25 - agonists | Recombinant Fusion Proteins - metabolism | T-Lymphocytes, Regulatory - immunology | Protein Isoforms - metabolism | Protein Processing, Post-Translational - drug effects | Protein Isoforms - chemistry | Receptors, Tumor Necrosis Factor, Type II - metabolism | T-Lymphocytes, Regulatory - cytology | Protein Interaction Domains and Motifs | Tumor Necrosis Factor Ligand Superfamily Member 15 - chemistry | Phosphorylation - drug effects | Receptors, Tumor Necrosis Factor, Member 25 - genetics | Tumor Necrosis Factor Ligand Superfamily Member 15 - genetics | Immunosuppressive Agents - pharmacology | Peptide Fragments - genetics | Peptide Fragments - metabolism | Cells, Cultured | Recombinant Fusion Proteins - chemistry | Tumor Necrosis Factor Ligand Superfamily Member 15 - metabolism | Receptors, Tumor Necrosis Factor, Type II - genetics | Sirolimus - pharmacology | NF-kappa B p52 Subunit - metabolism | Up-Regulation - drug effects | Peptide Fragments - chemistry | T-Lymphocytes, Regulatory - drug effects | MAP Kinase Signaling System - drug effects | Signal Transduction - drug effects | Receptors, Tumor Necrosis Factor, Member 25 - chemistry | Ligands | Cell Proliferation - drug effects | NF-kappa B p52 Subunit - agonists | Protein Isoforms - genetics
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, p. e90117
Tumour necrosis factor (TNF) is a proinflammatory cytokine that is known to regulate inflammation in a number of autoimmune diseases, including multiple... 
TUMOR-NECROSIS-FACTOR | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | DEMYELINATION | MULTIDISCIPLINARY SCIENCES | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | FACTOR RECEPTORS | CENTRAL-NERVOUS-SYSTEM | MICE | KAPPA-B | MONOCLONAL-ANTIBODIES | FACTOR-ALPHA | Gene Expression | Encephalomyelitis, Autoimmune, Experimental - pathology | Mice, Inbred C57BL | Encephalomyelitis, Autoimmune, Experimental - immunology | Receptors, Tumor Necrosis Factor, Type II - antagonists & inhibitors | Multiple Sclerosis - genetics | Molecular Targeted Therapy | Receptors, Tumor Necrosis Factor, Type I - antagonists & inhibitors | Receptors, Tumor Necrosis Factor, Type I - genetics | Multiple Sclerosis - therapy | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Receptors, Tumor Necrosis Factor, Type II - deficiency | Antibodies - pharmacology | Encephalomyelitis, Autoimmune, Experimental - therapy | Receptors, Tumor Necrosis Factor, Type I - deficiency | Animals | Immunotherapy | Multiple Sclerosis - immunology | Multiple Sclerosis - pathology | Female | Mice | Encephalomyelitis, Autoimmune, Experimental - genetics | Disease Models, Animal | Viral antibodies | Multiple sclerosis | Tumor necrosis factor | Analysis | Antibodies | Models | Inflammation | Tumor necrosis factor receptors | Animal models | Spinal cord | Encephalomyelitis | Disease | Medical services | Clinical trials | Oncology | Nervous system | Biology | Receptors | Immunology | Neurodegeneration | Rodents | Tumor necrosis factor-TNF | Inhibition | Paralysis | Medical research | Cytokines | Experimental allergic encephalomyelitis | Pathology | Neurology | Rheumatoid arthritis | TNF inhibitors | Autoimmune diseases | Tumors
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 05/2008, Volume 28, Issue 19, pp. 5072 - 5081
Journal Article
Journal Article