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Hepatology (Baltimore, Md.), ISSN 0270-9139, 12/2012, Volume 56, Issue 6, pp. 2353 - 2362
Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Gastroenterology. Liver. Pancreas. Abdomen | Biological and medical sciences | Medical sciences | Liver. Biliary tract. Portal circulation. Exocrine pancreas | Tumor Necrosis Factor-alpha - metabolism | Fas Ligand Protein - metabolism | Perforin - genetics | Tumor Necrosis Factor-alpha - genetics | Hepatocytes - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Interleukins - metabolism | Hepatitis, Animal - metabolism | Interleukins - genetics | Receptors, Tumor Necrosis Factor, Type II - metabolism | Acute Lung Injury - metabolism | Hepatitis, Animal - chemically induced | Natural Killer T-Cells | Galactosamine | Gene Expression | Interleukin-33 | Hepatitis, Animal - immunology | Antibodies, Monoclonal | Concanavalin A | Receptors, Tumor Necrosis Factor, Type I - genetics | TNF-Related Apoptosis-Inducing Ligand - metabolism | Receptors, Tumor Necrosis Factor, Type II - genetics | Animals | Acute Lung Injury - chemically induced | Mice | Perforin - metabolism | Primary Cell Culture | Fas Ligand Protein - genetics | RNA, Messenger | TNF-Related Apoptosis-Inducing Ligand - genetics | Index Medicus | Hépatology and Gastroenterology | Receptors, Tumor Necrosis Factor, Type I | Tumor Necrosis Factor-alpha | Fas Ligand Protein | Life Sciences | Hepatitis, Animal | Human health and pathology | TNF-Related Apoptosis-Inducing Ligand | Hepatocytes | Interleukins | Receptors, Tumor Necrosis Factor, Type II | Perforin | Acute Lung Injury
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 11/2012, Volume 122, Issue 11, pp. 4094 - 4104
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Neoplasms - metabolism | Tumor Necrosis Factor-alpha - metabolism | Neoplasm Transplantation | Tumor Escape | Receptors, Tumor Necrosis Factor, Type I - immunology | Tumor Necrosis Factor-alpha - genetics | Caspase 8 - metabolism | Graft Rejection - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Receptors, Tumor Necrosis Factor, Type II - immunology | Caspase 8 - genetics | Signal Transduction - immunology | Neoplasms - genetics | T-Lymphocytes - metabolism | Myeloid Cells - immunology | Receptors, Tumor Necrosis Factor, Type II - metabolism | Tumor Necrosis Factor-alpha - immunology | T-Lymphocytes - pathology | Graft Rejection - pathology | CASP8 and FADD-Like Apoptosis Regulating Protein - genetics | Signal Transduction - genetics | Receptors, Tumor Necrosis Factor, Type I - genetics | CASP8 and FADD-Like Apoptosis Regulating Protein - biosynthesis | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Caspase 8 - immunology | Animals | Neoplasms - immunology | Cell Line, Tumor | Myeloid Cells - metabolism | T-Lymphocytes - immunology | Mice | Mice, Inbred BALB C | CASP8 and FADD-Like Apoptosis Regulating Protein - immunology | Graft Rejection - immunology | Myeloid Cells - pathology | Graft Rejection - genetics | Neoplasms - pathology | Tumor necrosis factor | Genetic aspects | Cellular signal transduction | Research | Properties | Gene expression | Apoptosis | Index Medicus | Abridged Index Medicus
Journal Article
Kidney international, ISSN 0085-2538, 10/2015, Volume 88, Issue 4, pp. 722 - 733
Journal Article
Immunological reviews, ISSN 0105-2896, 11/2011, Volume 244, Issue 1, pp. 9 - 28
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 08/2017, Volume 292, Issue 33, pp. 13714 - 13726
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Activin Receptors, Type I - antagonists & inhibitors | Endothelium, Vascular - cytology | Tumor Necrosis Factor-alpha - metabolism | Activin Receptors, Type II - metabolism | Monocytes - cytology | Humans | Activin Receptors, Type II - antagonists & inhibitors | Endothelium, Vascular - drug effects | Monocytes - metabolism | Monocytes - immunology | Tumor Necrosis Factor-alpha - agonists | Bone Morphogenetic Proteins - metabolism | Protein Processing, Post-Translational - drug effects | RNA Interference | E-Selectin - genetics | Aorta | Vascular Cell Adhesion Molecule-1 - genetics | Phosphorylation - drug effects | Endothelium, Vascular - immunology | Pyrazoles - pharmacology | Cells, Cultured | Activin Receptors, Type I - metabolism | E-Selectin - metabolism | Pyrimidines - pharmacology | Cell Adhesion - drug effects | Activin Receptors, Type I - genetics | Vascular Cell Adhesion Molecule-1 - chemistry | Monocytes - drug effects | Up-Regulation - drug effects | Growth Differentiation Factors - metabolism | Intercellular Adhesion Molecule-1 - metabolism | Intercellular Adhesion Molecule-1 - chemistry | Signal Transduction - drug effects | Endothelium, Vascular - metabolism | Intercellular Adhesion Molecule-1 - genetics | Activin Receptors, Type II - genetics | E-Selectin - chemistry | Protein Kinase Inhibitors - pharmacology | Kinetics | Vascular Cell Adhesion Molecule-1 - metabolism | Index Medicus | atherosclerosis | monocyte | SMAD transcription factor | bone morphogenetic protein (BMP) | endothelial cell | Cell Biology
Journal Article
Cardiovascular research, ISSN 0008-6363, 11/2009, Volume 84, Issue 2, pp. 201 - 208
Ischaemia | Infarction | Reperfusion | Postconditioning | Preconditioning | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cardiology. Vascular system | Heart | Biological and medical sciences | Medical sciences | Coronary heart disease | Tumor Necrosis Factor-alpha - metabolism | Phosphorylation | Tumor Necrosis Factor-alpha - genetics | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Glycogen Synthase Kinase 3 beta | Male | Phosphatidylinositol 3-Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Tumor Necrosis Factor-alpha - deficiency | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Myocardial Reperfusion Injury - pathology | Receptors, Tumor Necrosis Factor, Type II - metabolism | Myocardial Infarction - pathology | Myocardium - metabolism | STAT3 Transcription Factor - deficiency | Flavonoids - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Mice, Inbred C57BL | Myocardium - pathology | Tyrphostins - pharmacology | Myocardial Infarction - metabolism | Glycogen Synthase Kinase 3 - metabolism | Receptors, Tumor Necrosis Factor, Type I - genetics | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Receptors, Tumor Necrosis Factor, Type II - deficiency | Myocardial Reperfusion Injury - metabolism | Animals | Androstadienes - pharmacology | Signal Transduction - drug effects | Mice | Myocardial Infarction - prevention & control | Protein Kinase Inhibitors - pharmacology | STAT3 Transcription Factor - antagonists & inhibitors | Myocardial Reperfusion Injury - prevention & control | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Index Medicus
Journal Article