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Nature, ISSN 0028-0836, 06/2012, Volume 486, Issue 7403, pp. 420 - 428
Highly pathogenic avian H5N1 influenza A viruses occasionally infect humans, but currently do not transmit efficiently among humans. The viral haemagglutinin... 
SINGLE AMINO-ACID | MEMBRANE-FUSION | IN-VITRO | AVIAN H5N1 | HEMAGGLUTININ | A VIRUSES | MULTIDISCIPLINARY SCIENCES | SIALIC-ACID | RECEPTOR-BINDING SPECIFICITY | MOLECULAR-BASIS | SWINE-ORIGIN H1N1 | Pandemics | Reassortant Viruses - pathogenicity | Influenza A Virus, H5N1 Subtype - physiology | Receptors, Virus - metabolism | Humans | Influenza A Virus, H1N1 Subtype - physiology | Reassortant Viruses - genetics | Reassortant Viruses - physiology | Influenza A Virus, H5N1 Subtype - genetics | Body Fluids - virology | Molecular Epidemiology - methods | Ferrets - virology | Hemagglutinin Glycoproteins, Influenza Virus - genetics | Zoonoses - virology | Respiratory System - virology | Adaptation, Physiological - genetics | HEK293 Cells | Female | Influenza, Human - virology | Protein Stability | Influenza A Virus, H1N1 Subtype - genetics | Population Surveillance - methods | Cell Line | Security Measures | Birds - virology | Influenza A Virus, H1N1 Subtype - pathogenicity | Influenza in Birds - transmission | Hot Temperature | Influenza, Human - transmission | Hemagglutinin Glycoproteins, Influenza Virus - metabolism | Reassortant Viruses - isolation & purification | Respiratory System - anatomy & histology | Zoonoses - transmission | Animals | Receptors, Virus - chemistry | Orthomyxoviridae Infections - transmission | Dogs | Influenza in Birds - virology | Bioterrorism - prevention & control | Influenza A Virus, H5N1 Subtype - pathogenicity | HeLa Cells | Influenza, Human - prevention & control | Orthomyxoviridae Infections - virology | Evolution, Molecular | Prevention | Epidemics | Disease transmission | United States | Japan | Avian influenza viruses | Physiological aspects | Genetic aspects | Research | Host-virus relationships | Ferrets | Cell culture | Task forces | Compliance | Laboratories | RNA polymerase | Academic libraries | Experiments | Avian flu | Proteins | Studies | Swine flu | Plasmids | Risk assessment | Occupational health | Mutation
Journal Article
Cell Reports, ISSN 2211-1247, 09/2016, Volume 16, Issue 10, pp. 2576 - 2592
The mechanisms underlying Zika virus (ZIKV)-related microcephaly and other neurodevelopment defects remain poorly understood. Here, we describe the derivation... 
NEURAL PROGENITORS | LONG-TERM | HUMAN BRAIN | ADAPTER | CENTRAL-NERVOUS-SYSTEM | INFECTION | MICE | BINDING KINASE 1 | ORGANOIDS | INNATE IMMUNITY | CELL BIOLOGY | Neocortex - pathology | Neurons - pathology | Transcription, Genetic - drug effects | Brain - embryology | Neuroglia - ultrastructure | Neuroglia - pathology | Humans | Brain - virology | Centrosome - drug effects | Gene Expression Profiling | Microcephaly - virology | Neural Stem Cells - ultrastructure | Zika Virus Infection - virology | Neural Stem Cells - immunology | Neuroepithelial Cells - immunology | Neuroprotective Agents - pharmacology | Spinal Cord - pathology | Microcephaly - pathology | Neuroepithelial Cells - virology | Nucleosides - pharmacology | Fetus - virology | Cell Death - drug effects | Phosphorylation - drug effects | Neurons - drug effects | Protein-Serine-Threonine Kinases - metabolism | Zika Virus - pathogenicity | Proto-Oncogene Proteins - metabolism | Zika Virus - ultrastructure | Neurons - virology | Virus Replication - drug effects | Neuroepithelial Cells - ultrastructure | Immunity, Innate - drug effects | Zika Virus Infection - pathology | Neural Stem Cells - virology | Zika Virus - physiology | Zika Virus - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Receptor Protein-Tyrosine Kinases - metabolism | Neural Stem Cells - enzymology | Centrosome - metabolism | Mitosis - drug effects | Brain - pathology | Protein Kinase Inhibitors - pharmacology | Neuroglia - virology | Neuroepithelial Cells - drug effects | Neurons/pathology | Zika Virus/pathogenicity | Mitochondria/metabolism | Virus Replication/drug effects | Microcephaly/pathology | Neurons/drug effects | Protein-Serine-Threonine Kinases/metabolism | Neural Stem Cells/immunology | Neuroglia/ultrastructure | Neuroepithelial Cells/drug effects | Neuroepithelial Cells/virology | Life Sciences | Brain/pathology | Zika Virus/drug effects | Brain/embryology | Mitochondria/drug effects | Fetus/virology | Neocortex/pathology | Neuroglia/pathology | Cell Death/drug effects | Mitosis/drug effects | Transcription, Genetic/drug effects | Nucleosides/pharmacology | Neural Stem Cells/enzymology | Neural Stem Cells/ultrastructure | Neuroepithelial Cells/ultrastructure | Receptor Protein-Tyrosine Kinases/metabolism | Microcephaly/virology | Proto-Oncogene Proteins/metabolism | Neuroprotective Agents/pharmacology | Zika Virus/ultrastructure | Neuroepithelial Cells/immunology | Brain/virology | Immunity, Innate/drug effects | Spinal Cord/pathology | Zika Virus/physiology | Neuroglia/virology | Microbiology and Parasitology | Zika Virus Infection/pathology | Neurons/virology | Zika Virus Infection/virology | Neural Stem Cells/virology | Centrosome/drug effects | Protein Kinase Inhibitors/pharmacology | Centrosome/metabolism | Phosphorylation/drug effects
Journal Article
Journal Article
American Journal of Tropical Medicine and Hygiene, ISSN 0002-9637, 06/2016, Volume 94, Issue 6, pp. 1362 - 1369
Journal Article
Hepatology, ISSN 0270-9139, 01/2008, Volume 47, Issue 1, pp. 17 - 24
Hepatitis C virus (HCV) infection of Huh‐7.5 hepatoma cells results in focal areas of infection where transmission is potentiated by cell‐cell contact. To... 
IN-VITRO | ENTRY | ASSAY | RNA REPLICATION | HUMAN CYTOMEGALOVIRUS | TISSUE | INFECTION | SPREAD | GASTROENTEROLOGY & HEPATOLOGY | CULTURE | EXPRESSION | Antibodies, Viral - physiology | Hepacivirus - immunology | Membrane Proteins - physiology | Humans | Antigens, CD - physiology | Hepacivirus - physiology | Tetraspanin 28 | HeLa Cells | Claudin-1 | Receptors, Virus - physiology
Journal Article
Journal Article