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Journal of Clinical Investigation, ISSN 0021-9738, 09/2011, Volume 121, Issue 9, pp. 3467 - 3478
Hirschsprung (HSCR) disease is a complex genetic disorder attributed to a failure of the enteric neural crest cells (ENCCs) to form ganglia in the hindgut.... 
MEDICINE, RESEARCH & EXPERIMENTAL | NERVOUS-SYSTEM | PROGENITORS | STEM-CELLS | INHIBITION | NOTCH ACTIVATION | NEUROGENESIS | PROLIFERATION | DIFFERENTIATION | NEURAL CREST CELLS | GENOME-WIDE ASSOCIATION | Enteric Nervous System - cytology | Receptors, Notch - metabolism | Humans | Hirschsprung Disease - genetics | Hedgehog Proteins - metabolism | Receptors, Notch - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Neuroglia - cytology | Hedgehog Proteins - genetics | Gastrointestinal Tract - physiology | Gene Expression Regulation, Developmental | Membrane Proteins - metabolism | Patched Receptors | Cell Differentiation - physiology | Intracellular Signaling Peptides and Proteins - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Neural Crest - cytology | Membrane Proteins - genetics | Hirschsprung Disease - physiopathology | Cells, Cultured | Neuroglia - physiology | Receptors, Cell Surface - metabolism | Neural Crest - physiology | Animals | Epistasis, Genetic | Neurogenesis - physiology | Signal Transduction - physiology | Gastrointestinal Tract - cytology | Mice | Polymorphism, Single Nucleotide | Enteric Nervous System - physiology | Patched-1 Receptor | Receptors, Cell Surface - genetics | Hirschsprung's disease | Cell proliferation | Care and treatment | Intestine, Small | Growth | Physiological aspects | Genetic aspects | Cellular signal transduction | Diagnosis | Gene expression | Nervous system, Autonomic
Journal Article
Brain, ISSN 0006-8950, 2007, Volume 130, Issue 2, pp. 357 - 367
Several hereditary small vessel diseases (SVDs) of the brain have been reported in recent years. In 1977, Sourander and Walinder described hereditary... 
NOTCH3 | Genetics | CADASIL | Vascular dementia | Multi-infarct dementia | SUBCORTICAL INFARCTS | vascular dementia | ALZHEIMERS-DISEASE | PATHOLOGY | CEREBRORETINAL VASCULOPATHY | VASCULAR RETINOPATHY | NEUROSCIENCES | CLINICAL NEUROLOGY | STROKE | LEUKOENCEPHALOPATHY | genetics | SMALL-VESSEL DISEASE | AUTOSOMAL-DOMINANT ARTERIOPATHY | MUTATIONS | multi-infarct dementia | Intracranial Arteriosclerosis - pathology | Microcirculation - metabolism | Receptors, Notch - metabolism | Humans | Middle Aged | Intracranial Arteriosclerosis - genetics | Male | Receptors, Notch - genetics | DNA Mutational Analysis - methods | CADASIL - genetics | Polymerase Chain Reaction - methods | Brain - blood supply | Magnetic Resonance Imaging | Dementia, Multi-Infarct - genetics | Dementia, Multi-Infarct - metabolism | Pedigree | Chromosome Mapping - methods | Brain - ultrastructure | Skin - ultrastructure | Adult | Female | Receptor, Notch3 | Dementia, Multi-Infarct - pathology | Mutation | Chromosome Mapping/methods | Medical and Health Sciences | Medicin och hälsovetenskap | CADASIL/genetics | MEDICINE | Intracranial Arteriosclerosis/genetics/pathology | DNA Mutational Analysis/methods | Polymerase Chain Reaction/methods | Brain/blood supply/ultrastructure | MEDICIN | Microcirculation/metabolism | Receptors; Notch/genetics/metabolism | Skin/ultrastructure | Dementia; Multi-Infarct/genetics/metabolism/pathology
Journal Article
Zhonghua yi xue yi chuan xue za zhi = Zhonghua yixue yichuanxue zazhi = Chinese journal of medical genetics, ISSN 1003-9406, 04/2015, Volume 32, Issue 2, p. 274
Notch signaling is highly conserved in evolution and regarded as a key factor in cell fate determination. It mediates cell-to-cell interactions that are... 
Animals | Bone Diseases - genetics | Signal Transduction | Receptors, Notch - metabolism | Bone and Bones - metabolism | Humans | Receptors, Notch - genetics | Bone Development | Bone Diseases - metabolism | Osteoblasts - cytology | Osteoblasts - metabolism | Osteogenesis
Journal Article
Zhonghua gan zang bing za zhi = Zhonghua ganzangbing zazhi = Chinese journal of hepatology, ISSN 1007-3418, 09/2012, Volume 20, Issue 9, p. 677
Journal Article
Brain and nerve = Shinkei kenkyu no shinpo, ISSN 1881-6096, 07/2013, Volume 65, Issue 7, p. 811
The pathomechanisms of cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) are still under debate. Granular... 
CADASIL - pathology | Signal Transduction | Receptors, Notch - metabolism | Humans | Risk Factors | CADASIL - therapy | CADASIL - metabolism | Receptors, Notch - genetics | Myocytes, Smooth Muscle - ultrastructure | Mutation - genetics | Myocytes, Smooth Muscle - metabolism | CADASIL - genetics
Journal Article
Zhongguo shi yan xue ye xue za zhi, ISSN 1009-2137, 08/2014, Volume 22, Issue 4, p. 1068
Journal Article
Zhonghua yi xue za zhi, ISSN 0376-2491, 03/2009, Volume 89, Issue 8, p. 514
Journal Article
Nihon shinkei seishin yakurigaku zasshi = Japanese journal of psychopharmacology, ISSN 1340-2544, 11/2008, Volume 28, Issue 5-6, p. 177
Journal Article
Journal Article
Zhongguo shi yan xue ye xue za zhi, ISSN 1009-2137, 08/2010, Volume 18, Issue 4, p. 927
Jagged-1 protein is one of the ligands belonging to Notch signaling pathway. Notch signaling pathway is one of the major signaling pathways mediated by contact... 
NIH 3T3 Cells | Signal Transduction | Membrane Proteins - genetics | Receptors, Notch - metabolism | Humans | Intercellular Signaling Peptides and Proteins - genetics | Animals | Transfection | Plasmids | Serrate-Jagged Proteins | Ligands | Mice | Genetic Vectors | Calcium-Binding Proteins - genetics | Jagged-1 Protein
Journal Article
Nihon rinsho. Japanese journal of clinical medicine, ISSN 0047-1852, 05/2009, Volume 67, Issue 5, p. 879
Journal Article
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