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Molecular and Cellular Biology, ISSN 0270-7306, 04/2008, Volume 28, Issue 7, pp. 2426 - 2436
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
INACTIVATION | APOPTOSIS | PROTEIN | SIGNALING PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | YAP | SIZE-CONTROL | DIFFERENTIATION | DROSOPHILA | TUMOR-SUPPRESSOR PATHWAY | CELL BIOLOGY | 14-3-3 Proteins - physiology | Phosphorylation | Transferases (Other Substituted Phosphate Groups) - genetics | Humans | Receptor Protein-Tyrosine Kinases - physiology | Recombinant Fusion Proteins - physiology | Transferases (Other Substituted Phosphate Groups) - physiology | Mesoderm - cytology | Drosophila Proteins - physiology | Cell Transdifferentiation - physiology | Membrane Proteins - physiology | Cell Division | c-Mer Tyrosine Kinase | Cell Cycle Proteins - genetics | Conserved Sequence | Transcription, Genetic | Epithelial Cells - cytology | Proteins - physiology | Nerve Tissue Proteins - physiology | Transcription Factors - physiology | Membrane Proteins - genetics | Protein-Serine-Threonine Kinases - physiology | Protein-Serine-Threonine Kinases - genetics | Proto-Oncogene Proteins - genetics | Transcription Factors - genetics | Nerve Tissue Proteins - genetics | Protein Processing, Post-Translational - physiology | Amino Acid Motifs | Proteins - genetics | Receptor Protein-Tyrosine Kinases - genetics | Cell Transformation, Neoplastic | Proto-Oncogene Proteins - physiology | Cell Line, Tumor | Nuclear Proteins - physiology | Drosophila Proteins - genetics | Cell Cycle Proteins - physiology | Cell Movement | Index Medicus
Journal Article
Immunity, ISSN 1074-7613, 06/2012, Volume 36, Issue 6, pp. 933 - 946
The mitochondrial protein MAVS (also known as IPS-1, VISA, and CARDIF) interacts with RIG-I-like receptors (RLRs) to induce type I interferon (IFN-I). NLRX1 is... 
ELONGATION-FACTOR TU | HEPATITIS-C VIRUS | RNA | RIG-I | LEUCINE-RICH REPEAT | NUCLEOTIDE-BINDING | ANTIVIRAL IMMUNE-RESPONSES | IMMUNOLOGY | NF-KAPPA-B | ADAPTER PROTEIN | INNATE IMMUNITY | Humans | Recombinant Fusion Proteins - physiology | Molecular Sequence Data | Autophagy - physiology | Mitochondrial Proteins - genetics | Macrophages, Peritoneal - cytology | Interferon Type I - biosynthesis | DEAD-box RNA Helicases - physiology | HEK293 Cells | Microtubule-Associated Proteins - deficiency | Mitochondrial Proteins - deficiency | Cytokines - genetics | Fibroblasts - metabolism | Autophagy-Related Protein 12 | Carrier Proteins - physiology | Amino Acid Sequence | DEAD Box Protein 58 | Proteins - physiology | Peptide Elongation Factor Tu - physiology | Specific Pathogen-Free Organisms | Gene Expression Regulation - immunology | Macrophages, Peritoneal - immunology | Mitochondrial Proteins - physiology | Peptide Elongation Factor Tu - chemistry | Recombinant Fusion Proteins - chemistry | Microtubule-Associated Proteins - physiology | Multiprotein Complexes - physiology | Protein Interaction Mapping | Sequence Homology, Amino Acid | Adaptor Proteins, Signal Transducing - physiology | Sequence Alignment | Animals | Autophagy-Related Protein 5 | Mitochondrial Proteins - chemistry | Interferon Type I - genetics | Mice | Vesiculovirus - physiology | Cytokines - biosynthesis | Virus diseases | Proteins | Magneto-electric machines | Physiological aspects | Interferon | Biological response modifiers | Health aspects | Machinery | Mass spectrometry | Cells | Infections | Plasmids | Autophagy | Rodents | Viral infections | Index Medicus | Infection
Journal Article
Journal Article
Cancer Cell, ISSN 1535-6108, 11/2012, Volume 22, Issue 5, pp. 683 - 697
Journal Article
Nature Medicine, ISSN 1078-8956, 08/2012, Volume 18, Issue 8, pp. 1239 - 1247
The inactivation of the p53 tumor suppressor pathway, which often occurs through mutations in TP53 (encoding tumor protein 53) is a common step in human... 
MEDICINE, RESEARCH & EXPERIMENTAL | N-RAS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUMOR-SUPPRESSOR ACTIVITY | STAPLED P53 | BRAF | MALIGNANT-MELANOMA | P53 PATHWAY | CELL-DEATH | CELL BIOLOGY | BREAST-CANCER | METASTATIC MELANOMA | IN-VIVO | Up-Regulation | Proto-Oncogene Proteins c-mdm2 - genetics | Tumor Suppressor Protein p53 - antagonists & inhibitors | Apoptosis - drug effects | Humans | Neoplasm Proteins - physiology | Gene Expression Regulation, Neoplastic | Recombinant Fusion Proteins - physiology | Skin Neoplasms - chemistry | Male | Melanocytes - metabolism | Neoplasm Proteins - antagonists & inhibitors | Cell Line, Tumor - transplantation | Proto-Oncogene Proteins - biosynthesis | Tumor Suppressor Protein p53 - physiology | Cell-Penetrating Peptides - pharmacology | Nuclear Proteins - biosynthesis | Female | Antineoplastic Agents - pharmacology | Neoplasm Proteins - genetics | Nuclear Proteins - genetics | Cell Line, Tumor - metabolism | Melanoma - chemistry | Proto-Oncogene Proteins c-mdm2 - biosynthesis | Proto-Oncogene Proteins - antagonists & inhibitors | Tumor Stem Cell Assay | Membrane Proteins - genetics | Neoplasm Proteins - biosynthesis | Melanoma, Experimental - etiology | Mice, Inbred C57BL | Mice, Transgenic | Proto-Oncogene Proteins - genetics | Melanoma - pathology | Melanoma - secondary | Proto-Oncogene Proteins B-raf - antagonists & inhibitors | Xenograft Model Antitumor Assays | Animals | Melanoma, Experimental - genetics | GTP Phosphohydrolases - genetics | Keratinocytes - metabolism | Mice, Nude | Nuclear Proteins - antagonists & inhibitors | Proto-Oncogene Proteins - physiology | Signal Transduction - physiology | Mice | Nuclear Proteins - physiology | Drug Resistance, Neoplasm - physiology | Drug Resistance, Neoplasm - drug effects | Care and treatment | Gene mutations | Melanoma | Diagnosis | Research | Gene expression | Identification and classification | Skin cancer | Proteins | Cell growth | Mutation | Cell cycle | Index Medicus
Journal Article
Journal Article
Molecular Biology of the Cell, ISSN 1059-1524, 08/2012, Volume 23, Issue 16, pp. 3041 - 3056
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2011, Volume 121, Issue 6, pp. 2278 - 2289
Angiopoietin-1/Tek signaling is a critical regulator of blood vessel development, with conventional knockout of angiopoietin-1 or Tek in mice being... 
MEDICINE, RESEARCH & EXPERIMENTAL | STEM-CELLS | COMP-ANGIOPOIETIN-1 | ANGIOGENESIS | VEGF | BLOOD-VESSELS | DIABETIC-NEPHROPATHY | MICE | EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | TIE2 RECEPTOR | Kidney Glomerulus - blood supply | Receptor, TIE-2 | Humans | Receptor Protein-Tyrosine Kinases - physiology | Recombinant Fusion Proteins - physiology | Neovascularization, Pathologic - pathology | Blood Vessels - embryology | Diabetic Nephropathies - physiopathology | Gene Expression Regulation, Developmental | Angiopoietin-1 - physiology | Neovascularization, Pathologic - physiopathology | Neovascularization, Pathologic - embryology | Receptor, TIE-1 - physiology | Diabetes Mellitus, Experimental - physiopathology | Recombinant Fusion Proteins - biosynthesis | Pericytes - metabolism | Blood Vessels - injuries | Kidney Glomerulus - pathology | Blood Vessels - cytology | Fetal Heart - pathology | Mice, Knockout | Fetal Heart - growth & development | Angiopoietin-1 - genetics | Myocytes, Cardiac - pathology | Neovascularization, Physiologic - physiology | Liver - blood supply | Animals | Myocytes, Cardiac - physiology | Recombinant Fusion Proteins - genetics | Neovascularization, Pathologic - genetics | Angiopoietin-1 - deficiency | Mice | Wound Healing - physiology | Index Medicus | Abridged Index Medicus | Medical and Health Sciences | Medicin och hälsovetenskap
Journal Article