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Journal Article
Journal Article
PLoS Biology, ISSN 1544-9173, 08/2017, Volume 15, Issue 8, p. e2002354
Microparticles (MPs) are cell-cell communication vesicles derived from the cell surface plasma membrane, although they are not known to originate from cardiac... 
HYPERTROPHY | IN-VITRO | T-TUBULE MEMBRANE | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | ABSCISSION | BIOLOGY | TRAFFICKING | CARDIOVASCULAR-DISEASES | AMPHIPHYSIN-2 BIN1 | PLASMA BIN1 | REQUIREMENT | Adaptor Proteins, Signal Transducing - blood | Humans | Endosomal Sorting Complexes Required for Transport - genetics | RNA Interference | Tumor Suppressor Proteins - chemistry | Cell-Derived Microparticles - metabolism | Endosomal Sorting Complexes Required for Transport - chemistry | Tumor Suppressor Proteins - metabolism | Recombinant Proteins - chemistry | Mice, Transgenic | Nuclear Proteins - chemistry | Particle Size | Myocytes, Cardiac - pathology | Endosomal Sorting Complexes Required for Transport - ultrastructure | Peptide Fragments - chemistry | Myocytes, Cardiac - metabolism | HeLa Cells | Adaptor Proteins, Signal Transducing - chemistry | Nerve Tissue Proteins - blood | Endosomal Sorting Complexes Required for Transport - blood | Exons | Cell-Derived Microparticles - chemistry | Heart Failure - blood | Nerve Tissue Proteins - chemistry | Endosomal Sorting Complexes Required for Transport - antagonists & inhibitors | Tumor Suppressor Proteins - genetics | Peptide Fragments - blood | Protein Interaction Domains and Motifs | Nuclear Proteins - genetics | Cell-Derived Microparticles - ultrastructure | Recombinant Proteins - metabolism | Microscopy, Electron, Transmission | Nuclear Proteins - blood | Peptide Fragments - metabolism | Enzyme-Linked Immunosorbent Assay | Myocytes, Cardiac - cytology | Cells, Cultured | Endosomal Sorting Complexes Required for Transport - metabolism | Nuclear Proteins - metabolism | Heart Failure - pathology | Nerve Tissue Proteins - genetics | Protein Transport | Nerve Tissue Proteins - metabolism | Animals | Tumor Suppressor Proteins - blood | Adaptor Proteins, Signal Transducing - genetics | Heterozygote | Adaptor Proteins, Signal Transducing - metabolism | Myocytes, Cardiac - ultrastructure | Heart | Flow cytometry | Plasma | Neurosciences | Intercellular signalling | Deformation | Calcium | Physicians | Trafficking | Biology | Cell interactions | Cell surface | Microparticles | Fission | Blood plasma | Proteins | Calcium signalling | Osmotic shock | Vesicles | Clonal deletion | Actin | BAR protein | Deletion | Physiology | Ion channels | Bathing | Protein transport | Heart diseases | Enzyme-linked immunosorbent assay | Heart failure | Enzymes | Cardiac muscle | Deformation mechanisms | Membrane vesicles | Cardiomyocytes | Electron microscopy | Membrane proteins | Mutants | Medicine | Cytometry | Data collection | Ventricle | Alzheimers disease | Plasmas (physics)
Journal Article
Hypertension, ISSN 0194-911X, 07/2015, Volume 66, Issue 1, pp. 158 - 166
Activation of the mineralocorticoid receptor has been shown to be deleterious in cardiovascular diseases (CVDs). We have recently shown that lipocalin 2... 
collagen type I | lipocalin 2 | fibroblast | mineralocorticoid receptor | aldosterone | MYOCARDIAL-INFARCTION | CARDIOVASCULAR EVENTS | HEART-FAILURE | TRANSCRIPTION | RECEPTOR | BLOOD-PRESSURE | OBESITY | LEFT-VENTRICULAR DYSFUNCTION | PERIPHERAL VASCULAR DISEASE | EPLERENONE | Aldosterone - toxicity | Oncogene Proteins - genetics | Kidney - pathology | Humans | Obesity, Abdominal - physiopathology | Galectin 3 - biosynthesis | Male | Acute-Phase Proteins - genetics | Oncogene Proteins - physiology | Acute-Phase Proteins - physiology | Lipocalins - blood | Lipocalins - genetics | Lipocalins - physiology | Myocardium - metabolism | Galectin 3 - blood | Peptide Fragments - blood | Fibroblasts | Female | Acute-Phase Proteins - deficiency | Myocytes, Smooth Muscle - drug effects | Cytokines - genetics | Proto-Oncogene Proteins - pharmacology | Lipocalins - pharmacology | Aorta - drug effects | Aldosterone - blood | Cells, Cultured | Myocytes, Smooth Muscle - physiology | Rats | Procollagen - blood | Recombinant Proteins - pharmacology | Galectin 3 - genetics | Hypertension - physiopathology | Myocardium - cytology | Aorta - pathology | Obesity, Abdominal - blood | Animals | Nephrectomy - adverse effects | Cardiomyopathy, Hypertrophic - physiopathology | Lipocalin-2 | Fibrosis | Proto-Oncogene Proteins - physiology | Oncogene Proteins - deficiency | Mice | Proto-Oncogene Proteins - blood | Acute-Phase Proteins - pharmacology | Cytokines - biosynthesis | Cardiomyopathy, Hypertrophic - chemically induced | Hypertrophy | Peptide Fragments | Oncogene Proteins | Recombinant Proteins | Aldosterone | Kidney | Life Sciences | Lipocalins | Cardiomyopathy, Hypertrophic | Nephrectomy | Aorta | Proto-Oncogene Proteins | Cardiology and cardiovascular system | Hypertension | Obesity, Abdominal | Procollagen | Cytokines | Acute-Phase Proteins | Human health and pathology | Myocytes, Smooth Muscle | Galectin 3 | Myocardium
Journal Article
Science, ISSN 0036-8075, 2014, Volume 344, Issue 6184, pp. 649 - 652
Journal Article
Journal Article
Nature Chemical Biology, ISSN 1552-4450, 08/2010, Volume 6, Issue 8, pp. 602 - 609
Journal Article
PLoS Pathogens, ISSN 1553-7366, 11/2016, Volume 12, Issue 11, p. e1005917
Journal Article