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Oncogene, ISSN 0950-9232, 2019, Volume 38, Issue 17, pp. 3274 - 3287
High-risk human papillomaviruses (HPVs) constitutively activate ataxia telangiectasia mutated (ATM) and ataxia telangiectasia- and Rad3-related (ATR) DNA... 
LIFE-CYCLE | HUMAN-PAPILLOMAVIRUS TYPE-16 | DNA-DAMAGE RESPONSE | STABILITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | CANCER | P53 | CELL BIOLOGY | REPLICATION | ONCOLOGY | IN-VIVO | GENETICS & HEREDITY | TOPBP1 | NIH 3T3 Cells | Humans | Tumor Protein p73 - genetics | Cell Differentiation - genetics | STAT5 Transcription Factor - genetics | DNA Damage - genetics | Female | Nuclear Proteins - genetics | MRE11 Homologue Protein - genetics | Cell Line | Rad51 Recombinase - genetics | Gene Expression Regulation - genetics | Epithelial Cells - pathology | Signal Transduction - genetics | DNA-Binding Proteins - genetics | Uterine Cervical Neoplasms - genetics | Cervical Intraepithelial Neoplasia - virology | Papillomavirus Infections - genetics | Papillomaviridae - pathogenicity | Carrier Proteins - genetics | Animals | Uterine Cervical Neoplasms - virology | Cervical Intraepithelial Neoplasia - genetics | Gene Amplification - genetics | Mice | Transcription, Genetic - genetics | Ataxia Telangiectasia Mutated Proteins - genetics | E2F1 Transcription Factor - genetics | Checkpoint Kinase 1 - genetics | BRCA2 protein | Transcription | Risk groups | p53 Protein | CHK1 protein | DNA damage | AKT protein | Genomes | Stat5 protein | Cervix | DNA repair | Proteins | MRE11 protein | Cell lines | Ataxia | Ataxia telangiectasia mutated protein | Cervical carcinoma | Deoxyribonucleic acid--DNA | Life cycles | Index Medicus
Journal Article
PLoS Genetics, ISSN 1553-7390, 08/2015, Volume 11, Issue 8, pp. e1005482 - e1005482
Journal Article
Nature Cell Biology, ISSN 1465-7392, 03/2012, Volume 14, Issue 3, pp. 318 - 328
Repair of DNA double-strand breaks is critical to genomic stability and the prevention of developmental disorders and cancer. A central pathway for this repair... 
TARGET | SPINDLE-CHECKPOINT | NUCLEAR RIBONUCLEOPROTEIN-G | REPAIR | STABILIZATION | TAO1 | DOUBLE-STRAND BREAKS | MAMMALIAN-CELLS | PROMOTES | EXPRESSION | CELL BIOLOGY | RNA, Small Interfering - genetics | RNA-Binding Proteins - genetics | Histone Chaperones - genetics | Humans | Histone Chaperones - metabolism | Homologous Recombination | Immunoblotting | Green Fluorescent Proteins - genetics | Gene Regulatory Networks | Nuclear Pore Complex Proteins - genetics | RNA Interference | Cell Cycle Proteins - genetics | RNA Precursors - metabolism | Nuclear Proteins - genetics | Rad51 Recombinase - metabolism | Green Fluorescent Proteins - metabolism | Rad51 Recombinase - genetics | Nuclear Pore Complex Proteins - metabolism | Cell Cycle Proteins - metabolism | Heterogeneous-Nuclear Ribonucleoproteins - metabolism | Nuclear Proteins - metabolism | RNA Precursors - genetics | Transcription Factors - genetics | Reverse Transcriptase Polymerase Chain Reaction | Genome, Human - genetics | Heterogeneous-Nuclear Ribonucleoproteins - genetics | Transcription Factors - metabolism | Poly(ADP-ribose) Polymerases - metabolism | BRCA2 Protein - metabolism | Poly(ADP-ribose) Polymerases - genetics | DNA Repair | Cell Line, Tumor | Models, Genetic | DNA Damage | BRCA2 Protein - genetics | Microscopy, Fluorescence | RNA-Binding Proteins - metabolism | Proteins | Physiological aspects | Research | Binding proteins | DNA damage | Ribonucleoproteins | Index Medicus
Journal Article
Molecular Microbiology, ISSN 0950-382X, 10/2006, Volume 62, Issue 2, pp. 382 - 396
Journal Article
PLoS Genetics, ISSN 1553-7390, 10/2009, Volume 5, Issue 10, pp. e1000680 - e1000680
The p53 family activates many of the same genes in response to DNA damage. Because p63 and p73 have structural differences from p53 and play distinct... 
DELTA-NP73 | APOPTOSIS | TAP73 | MICROARRAY | TUMOR-SUPPRESSOR FUNCTIONS | GENETICS & HEREDITY | P53 FAMILY | BRCA2 | BINDING | CANCER | EXPRESSION | Immunohistochemistry | Transcriptional Activation - genetics | Cell Survival - genetics | Phosphoproteins - metabolism | Tumor Suppressor Protein p53 - genetics | Breast Neoplasms - metabolism | DNA-Binding Proteins - metabolism | MRE11 Homologue Protein | Adenocarcinoma - metabolism | Chromatin Immunoprecipitation | Tumor Suppressor Proteins - genetics | DNA Repair Enzymes - metabolism | Trans-Activators - genetics | Adenocarcinoma - genetics | Nuclear Proteins - genetics | Radiation, Ionizing | Rad51 Recombinase - metabolism | Promoter Regions, Genetic | Tumor Suppressor Proteins - metabolism | Cells, Cultured | Gene Expression Regulation | Tumor Suppressor Protein p53 - metabolism | Nuclear Proteins - metabolism | Phosphoproteins - genetics | DNA-Binding Proteins - genetics | Tumor Protein p73 | Animals | BRCA2 Protein - metabolism | Breast Neoplasms - genetics | DNA Repair | Trans-Activators - metabolism | Mice | Cluster Analysis | Cancer cells | Physiological aspects | Genetic aspects | Research | Tumor proteins | DNA repair | Health aspects | Index Medicus | Medical research | Genotype & phenotype | Genes | Rodents | Leukemia | Genomes | Experiments | Binding sites | Cancer | Apoptosis
Journal Article