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Journal Article
Journal Article
Nature Neuroscience, ISSN 1097-6256, 10/2009, Volume 12, Issue 10, pp. 1333 - 1342
After complete spinal cord transections that removed all supraspinal inputs in adult rats, combinations of serotonergic agonists and epidural electrical... 
HINDLIMB-STEPPING ABILITY | RECOVERY | CATS | ADULT RATS | EPIDURAL STIMULATION | CENTRAL PATTERN GENERATOR | INJURY | LOCOMOTION | INTRASPINAL MICROSTIMULATION | CORD STIMULATION | NEUROSCIENCES | Motor Activity - physiology | Neural Pathways - drug effects | Reflex - physiology | Recovery of Function - drug effects | Quipazine - therapeutic use | Motor Activity - drug effects | Neuronal Plasticity - drug effects | Nerve Net - drug effects | Gait - physiology | Nerve Net - physiology | Spinal Cord Injuries - pathology | Hindlimb - physiopathology | Neuronal Plasticity - physiology | Time Factors | Recovery of Function - physiology | Female | Neural Pathways - physiopathology | Spinal Cord Injuries - therapy | Electric Stimulation - methods | Principal Component Analysis | Physical Conditioning, Animal | Disease Models, Animal | Locomotion - drug effects | Oncogene Proteins v-fos - metabolism | Rats | 8-Hydroxy-2-(di-n-propylamino)tetralin - therapeutic use | Biomechanical Phenomena - physiology | Rats, Sprague-Dawley | Serotonin Receptor Agonists - therapeutic use | Gait - drug effects | Animals | Analysis of Variance | Muscle, Skeletal - physiopathology | Brain - pathology | Spinal Cord Injuries - physiopathology | Muscle, Skeletal - pathology | Electromyography - methods | Locomotion - physiology | Physiological aspects | Sensorimotor integration | Neural transmission | Spinal cord | Neural circuitry | Research | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2012, Volume 7, Issue 1, pp. 315 - 326
Secondary damage following primary spinal cord injury extends pathology beyond the site of initial trauma, and effective management is imperative for... 
SURVIVAL | PROTEIN-KINASE-B | TYROSINE-PHOSPHATASE INHIBITION | ADULT RATS | SUPPRESSOR GENE | CONTUSION INJURY | BIOLOGY | PTEN | ISCHEMIC BRAIN-INJURY | UP-REGULATION | CELL-DEATH | Neuroprotective Agents - therapeutic use | Spinal Cord Injuries - drug therapy | Recovery of Function - drug effects | TOR Serine-Threonine Kinases - metabolism | Microtubule-Associated Proteins - metabolism | Cervical Vertebrae - blood supply | Protein Transport - drug effects | Autophagy - drug effects | Motor Neurons - pathology | Spinal Cord Injuries - pathology | Forelimb - physiopathology | Neuroprotective Agents - pharmacology | Spinal Cord Injuries - enzymology | Vanadium Compounds - pharmacology | Female | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Forelimb - drug effects | Motor Neurons - drug effects | Contusions - complications | Phagosomes - drug effects | Cervical Vertebrae - drug effects | Phagosomes - metabolism | Contusions - physiopathology | PTEN Phosphohydrolase - metabolism | Rats | Vanadium Compounds - therapeutic use | Enzyme Activation - drug effects | Rats, Sprague-Dawley | Contusions - drug therapy | Animals | Signal Transduction - drug effects | Models, Biological | Cervical Vertebrae - pathology | Spinal Cord Injuries - physiopathology | Spinal cord injuries | Phosphatases | TOR protein | Neuroprotection | Neurosciences | Spinal cord | Vanadium compounds | Traumatic brain injury | Central nervous system | Vanadium | Recovery of function | Homology | AKT protein | Spinal cord injury | Kinases | Phosphatase | Autophagy | Western blotting | Proteins | Rodents | Cell cycle | Bisperoxovanadium | Tumorigenesis | Research centers | Tensin | Hematology | Neurons | Rapamycin | Pharmacology | Functional analysis | Trauma | Medicine | Signaling | Injury prevention | Brain research | Insects | Immunofluorescence | Potassium | Histochemistry | Phagocytosis | Apoptosis | Index Medicus
Journal Article
Cell, ISSN 0092-8674, 2002, Volume 110, Issue 4, pp. 429 - 441
The adult brain is extremely vulnerable to various insults. The recent discovery of neural progenitors in adult mammals, however, raises the possibility of... 
RAT SPINAL-CORD | GERBIL HIPPOCAMPUS | STEM-CELLS | TRANSIENT FOREBRAIN ISCHEMIA | NEUROTROPHIC FACTOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | FIBROBLAST-GROWTH-FACTOR | CENTRAL-NERVOUS-SYSTEM | ADULT MAMMALIAN BRAIN | SUBVENTRICULAR ZONE | SYNAPTIC PLASTICITY | CELL BIOLOGY | Neural Pathways - drug effects | Recovery of Function - drug effects | Nerve Regeneration - physiology | Neuronal Plasticity - drug effects | Male | Dendrites - ultrastructure | Nerve Degeneration - metabolism | Neuronal Plasticity - physiology | Recovery of Function - physiology | Hippocampus - ultrastructure | Pyramidal Cells - drug effects | Organ Culture Techniques | Dendrites - drug effects | Synapses - drug effects | Neural Pathways - growth & development | Rats | Synapses - ultrastructure | Brain Ischemia - physiopathology | Cell Division - drug effects | Stem Cells - ultrastructure | Cell Division - physiology | Cell Movement - drug effects | Brain Ischemia - drug therapy | Nerve Regeneration - drug effects | Growth Substances - pharmacology | Pyramidal Cells - metabolism | Rats, Wistar | Microtubule-Associated Proteins - metabolism | Nerve Degeneration - physiopathology | Brain Ischemia - metabolism | Hippocampus - drug effects | Stem Cells - metabolism | Excitatory Postsynaptic Potentials - drug effects | Cell Movement - physiology | Excitatory Postsynaptic Potentials - physiology | Synapses - metabolism | Up-Regulation - physiology | Hippocampus - growth & development | Pyramidal Cells - ultrastructure | Dendrites - metabolism | Transcription Factors - genetics | Microscopy, Electron | Transcription Factors - metabolism | Up-Regulation - drug effects | Animals | Growth Substances - therapeutic use | Stem Cells - drug effects | Neural Pathways - ultrastructure | Nerve Degeneration - drug therapy | Neural circuitry | Neurons | Growth | Physiological aspects | Hippocampus (Brain) | Developmental neurology | Research | Index Medicus
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 04/2008, Volume 28, Issue 14, pp. 3814 - 3823
Journal Article
Nature Neuroscience, ISSN 1097-6256, 06/2012, Volume 15, Issue 6, pp. 862 - 870
Mesenchymal stem cells (MSCs) have emerged as a potential therapy for a range of neural insults. In animal models of multiple sclerosis, an autoimmune disease... 
IMMUNE-RESPONSE | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | LIVER-REGENERATION | C-MET | BONE-MARROW | ENDOTHELIAL-CELLS | ENDOGENOUS REPAIR | SPINAL-CORD-INJURY | CENTRAL-NERVOUS-SYSTEM | MET TYROSINE KINASE | NEUROSCIENCES | Proto-Oncogene Proteins c-met - metabolism | Oligodendroglia - metabolism | Spinal Cord - drug effects | Encephalomyelitis, Autoimmune, Experimental - metabolism | Recovery of Function - drug effects | Humans | Culture Media, Conditioned - pharmacology | Neurons - cytology | Stem Cells - cytology | Hepatocyte Growth Factor - pharmacology | Stem Cells - metabolism | Oligodendroglia - drug effects | Spinal Cord - pathology | Female | Neurogenesis - drug effects | Neurons - metabolism | Oligodendroglia - cytology | Neurons - drug effects | Multiple Sclerosis - metabolism | Encephalomyelitis, Autoimmune, Experimental - pathology | Mice, Inbred C57BL | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Rats | Multiple Sclerosis - therapy | Blotting, Western | Hepatocyte Growth Factor - metabolism | Encephalomyelitis, Autoimmune, Experimental - therapy | Animals | Cell Differentiation - drug effects | Multiple Sclerosis - pathology | Stem Cells - drug effects | Mice | Mesenchymal Stem Cell Transplantation | Physiological aspects | Development and progression | Multiple sclerosis | Research | Growth factors | Stem cells | Index Medicus
Journal Article