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Journal of Ethnopharmacology, ISSN 0378-8741, 2007, Volume 111, Issue 2, pp. 219 - 226
The present study was designed to determine in vivo efficacy of Ganoderma lucidum polysaccharides ( Gl-PS) for enhancing the activity of immunological effector... 
Ganoderma lucidum (Leyss. ex Fr.) Karst. (Ling Zhi) | Cytotoxicity | Cyclophosphamide | Immunomodulation | Polysaccharide | immunomodulation | NKT CELLS | NATURAL-KILLER | CHEMISTRY, MEDICINAL | polysaccharide | MACROPHAGES | IMMUNE FUNCTIONS | Ganoderma lucidum (Leyss. ex Fr.) karst. (Ling Zhi) | CANCER | ANTITUMOR | LYMPHOCYTE-PROLIFERATION | PLANT SCIENCES | RECOVERY | INTEGRATIVE & COMPLEMENTARY MEDICINE | cyclophosphamide | PHARMACOLOGY & PHARMACY | cytotoxicity | LEVAMISOLE | Dose-Response Relationship, Immunologic | Mitogens - pharmacology | Concanavalin A - pharmacology | Cell Count | Male | Spleen - drug effects | T-Lymphocytes, Cytotoxic - drug effects | Time Factors | Polysaccharides - isolation & purification | Killer Cells, Natural - immunology | Bone Marrow Cells - drug effects | Polysaccharides - administration & dosage | Leukocyte Count | Reishi - chemistry | Cytotoxicity, Immunologic - drug effects | Macrophages, Peritoneal - drug effects | T-Lymphocytes, Cytotoxic - immunology | Phagocytosis - drug effects | Mice, Inbred C57BL | Cells, Cultured | Injections, Intraperitoneal | Polysaccharides - adverse effects | B-Lymphocytes - physiology | Spleen - cytology | Immunosuppressive Agents - toxicity | Polysaccharides - pharmacology | T-Lymphocytes, Cytotoxic - physiology | Erythrocytes - drug effects | B-Lymphocytes - drug effects | Immunosuppression | Killer Cells, Lymphokine-Activated - immunology | Animals | B-Lymphocytes - immunology | Leukocytes - drug effects | Cell Proliferation - drug effects | Cyclophosphamide - toxicity | Killer Cells, Natural - drug effects | Mice | Erythrocyte Count | Killer Cells, Lymphokine-Activated - cytology
Journal Article
Journal Article
Journal of Controlled Release, ISSN 0168-3659, 06/2015, Volume 207, pp. 7 - 17
Treatment of ischemia through therapeutic angiogenesis faces significant challenges. Growth factor (GF)-based therapies can be more effective when concerns... 
Myocardial infarction | Coacervate | Therapeutic angiogenesis | Fibrin gel | Growth factors | Controlled release | VEGF | VESSEL MATURATION | ALGINATE HYDROGELS | CHEMISTRY, MULTIDISCIPLINARY | CARDIAC REPAIR | NITRIC-OXIDE | PHARMACOLOGY & PHARMACY | HEPARAN-SULFATE | CELL RECRUITMENT | PDGF-BB | Neovascularization, Physiologic - drug effects | Proto-Oncogene Proteins c-sis - chemistry | Humans | Male | Proto-Oncogene Proteins c-sis - administration & dosage | Angiogenesis Inducing Agents - administration & dosage | Recovery of Function | Drug Carriers | Vascular Endothelial Growth Factor A - chemistry | Fibrin - chemistry | Myocardial Infarction - pathology | Myocardial Infarction - physiopathology | Myocytes, Smooth Muscle - drug effects | Vascular Endothelial Growth Factor A - administration & dosage | Disease Models, Animal | Human Umbilical Vein Endothelial Cells - drug effects | Gels | Ventricular Function, Left - drug effects | Cells, Cultured | Solubility | Myocardium - pathology | Chemotaxis - drug effects | Rats, Sprague-Dawley | Chemistry, Pharmaceutical | Papio | Animals | Myocardial Infarction - drug therapy | Fibrosis | Cell Proliferation - drug effects | Kinetics | Angiogenesis Inducing Agents - chemistry | Delayed-Action Preparations | Drug Combinations | Heparin - chemistry | Heart | Fibrin | Platelet-derived growth factor | Analysis | Physiological aspects | Vascular endothelial growth factor | Heart attack
Journal Article
Cell (Cambridge), ISSN 0092-8674, 2002, Volume 110, Issue 4, pp. 429 - 441
The adult brain is extremely vulnerable to various insults. The recent discovery of neural progenitors in adult mammals, however, raises the possibility of... 
RAT SPINAL-CORD | GERBIL HIPPOCAMPUS | STEM-CELLS | TRANSIENT FOREBRAIN ISCHEMIA | NEUROTROPHIC FACTOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | FIBROBLAST-GROWTH-FACTOR | CENTRAL-NERVOUS-SYSTEM | ADULT MAMMALIAN BRAIN | SUBVENTRICULAR ZONE | SYNAPTIC PLASTICITY | CELL BIOLOGY | Neural Pathways - drug effects | Recovery of Function - drug effects | Nerve Regeneration - physiology | Neuronal Plasticity - drug effects | Male | Dendrites - ultrastructure | Nerve Degeneration - metabolism | Neuronal Plasticity - physiology | Recovery of Function - physiology | Hippocampus - ultrastructure | Pyramidal Cells - drug effects | Organ Culture Techniques | Dendrites - drug effects | Synapses - drug effects | Neural Pathways - growth & development | Rats | Synapses - ultrastructure | Brain Ischemia - physiopathology | Cell Division - drug effects | Stem Cells - ultrastructure | Cell Division - physiology | Cell Movement - drug effects | Brain Ischemia - drug therapy | Nerve Regeneration - drug effects | Growth Substances - pharmacology | Pyramidal Cells - metabolism | Rats, Wistar | Microtubule-Associated Proteins - metabolism | Nerve Degeneration - physiopathology | Brain Ischemia - metabolism | Hippocampus - drug effects | Stem Cells - metabolism | Excitatory Postsynaptic Potentials - drug effects | Cell Movement - physiology | Excitatory Postsynaptic Potentials - physiology | Synapses - metabolism | Up-Regulation - physiology | Hippocampus - growth & development | Pyramidal Cells - ultrastructure | Dendrites - metabolism | Transcription Factors - genetics | Microscopy, Electron | Transcription Factors - metabolism | Up-Regulation - drug effects | Animals | Growth Substances - therapeutic use | Stem Cells - drug effects | Neural Pathways - ultrastructure | Nerve Degeneration - drug therapy | Neural circuitry | Neurons | Growth | Physiological aspects | Hippocampus (Brain) | Developmental neurology | Research
Journal Article
Cardiovascular research, ISSN 0008-6363, 07/2010, Volume 87, Issue 1, pp. 30 - 39
.... The effects of EPO on VEGF expression were studied in cultured cardiac cells and EPO-treated hearts... 
Heart failure | PRESSURE-OVERLOAD | Erythropoietin | ANGIOGENESIS | VEGF EXPRESSION | ISCHEMIA | UMCG Approved | PROGENITOR-CELL | Cardiac function | DOUBLE-BLIND | Neovascularization | DYSFUNCTION | ANEMIA | Vascular endothelial growth factor | DARBEPOETIN-ALPHA | CARDIAC & CARDIOVASCULAR SYSTEMS | Erythropoietin - pharmacology | Neovascularization, Physiologic - drug effects | Transcription, Genetic - drug effects | Up-Regulation | Humans | Rats, Inbred F344 | Heart Failure - physiopathology | Male | Vascular Endothelial Growth Factor A - metabolism | Recombinant Proteins | RNA, Messenger - metabolism | Stem Cells - metabolism | Vascular Endothelial Growth Factor A - genetics | Recovery of Function | Stem Cell Transplantation | Erythropoietin - analogs & derivatives | Time Factors | Epoetin Alfa | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Animals, Newborn | Endothelial Cells - metabolism | Ventricular Function, Left - drug effects | Cells, Cultured | Rats | Heart Failure - metabolism | Cardiotonic Agents - pharmacology | Rats, Sprague-Dawley | Heart Failure - drug therapy | Cell Movement - drug effects | Animals | Myocytes, Cardiac - drug effects | Paracrine Communication - drug effects | Myocytes, Cardiac - metabolism | Stem Cells - drug effects | Cell Proliferation - drug effects | Darbepoetin alfa | Ventricular Remodeling - drug effects | Endothelial Cells - drug effects
Journal Article
Neuroscience, ISSN 0306-4522, 2014, Volume 284, pp. 202 - 216
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 1, pp. e30012 - 326
.... Bisperoxovanadium compounds have proven neuroprotective effects in several central nervous system injury/disease models, however, no mechanism has been linked to such neuroprotection... 
SURVIVAL | PROTEIN-KINASE-B | TYROSINE-PHOSPHATASE INHIBITION | MULTIDISCIPLINARY SCIENCES | CONTUSION INJURY | PTEN | AKT | UP-REGULATION | EXPRESSION | BRAIN | CELL-DEATH | Neuroprotective Agents - therapeutic use | Spinal Cord Injuries - drug therapy | Recovery of Function - drug effects | TOR Serine-Threonine Kinases - metabolism | Microtubule-Associated Proteins - metabolism | Cervical Vertebrae - blood supply | Protein Transport - drug effects | Autophagy - drug effects | Motor Neurons - pathology | Spinal Cord Injuries - pathology | Forelimb - physiopathology | Neuroprotective Agents - pharmacology | Spinal Cord Injuries - enzymology | Vanadium Compounds - pharmacology | Female | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Forelimb - drug effects | Motor Neurons - drug effects | Contusions - complications | Phagosomes - drug effects | Cervical Vertebrae - drug effects | Phagosomes - metabolism | Contusions - physiopathology | PTEN Phosphohydrolase - metabolism | Rats | Vanadium Compounds - therapeutic use | Enzyme Activation - drug effects | Rats, Sprague-Dawley | Contusions - drug therapy | Animals | Signal Transduction - drug effects | Models, Biological | Cervical Vertebrae - pathology | Spinal Cord Injuries - physiopathology | Spinal cord injuries | Phosphatases | TOR protein | Neuroprotection | Neurosciences | Spinal cord | Vanadium compounds | Traumatic brain injury | Central nervous system | Vanadium | Recovery of function | Homology | AKT protein | Spinal cord injury | Kinases | Phosphatase | Autophagy | Western blotting | Proteins | Rodents | Cell cycle | Bisperoxovanadium | Tumorigenesis | Tensin | Hematology | Neurons | Rapamycin | Pharmacology | Functional analysis | Trauma | Medicine | Signaling | Injury prevention | Brain research | Insects | Immunofluorescence | Potassium | Histochemistry | Phagocytosis | Apoptosis
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 3, p. e17328
Repairing trauma to the central nervous system by replacement of glial support cells is an increasingly attractive therapeutic strategy. We have focused on the... 
GLIAL-RESTRICTED PRECURSORS | GAP-JUNCTIONS | GLUTAMATE TRANSPORTERS | OLIGODENDROCYTE PROGENITORS | MULTIDISCIPLINARY SCIENCES | EMBRYONIC STEM-CELLS | LOCOMOTOR RECOVERY | AXONAL REGENERATION | REACTIVE ASTROCYTES | CENTRAL-NERVOUS-SYSTEM | ADULT-RAT-BRAIN | Neurons - pathology | Recovery of Function - drug effects | Humans | Motor Activity - drug effects | Stem Cells - cytology | Glial Fibrillary Acidic Protein - metabolism | Neuroglia - drug effects | Neuroglia - cytology | Cytoprotection - drug effects | Recovery of Function - physiology | Female | Astrocytes - transplantation | Neurons - drug effects | Spinal Cord Injuries - therapy | Bone Morphogenetic Proteins - pharmacology | Ciliary Neurotrophic Factor - pharmacology | Astrocytes - cytology | Astrocytes - drug effects | Cell Survival - drug effects | Rats | Rats, Sprague-Dawley | Cell Shape - drug effects | Cell Movement - drug effects | Animals | Cell Differentiation - drug effects | Stem Cells - drug effects | Spinal Cord Injuries - physiopathology | Graft Survival - drug effects | Ciliary neurotrophic factor | Bone morphogenetic proteins | Spinal cord injuries | Spinal cord | Transcription factors | Transplants & implants | Central nervous system | Recovery of function | Homeostasis | Nervous system | Transplantation | Neurosurgery | Spinal cord injury | Recovery | Proteins | Pain | Rodents | Injuries | Cell survival | Neurons | Astrocytes | Fetuses | Maintenance | Laminates | Gene expression | Survival | Trauma | Feet | Medicine | Regeneration | Adenoviruses | Morphology | Stem cells | Differentiation | Apoptosis | Osteoprogenitor cells | Axonogenesis | Therapeutic applications | Conservation | Embryos | Glial stem cells | Neuronal-glial interactions | Foot | Neural stem cells
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2014, Volume 111, Issue 48, pp. 17272 - 17277
.... In vivo, VEGF-B is required for normal nerve regeneration: mice lacking VEGF-B showed impaired nerve repair with concomitant impaired trophic function... 
Sensation | Debridement | Cornea | Neurites | Neurons | Nerves | Peripheral nerves | Mice | Epithelium | Nerve regeneration | Nerve injury | Neuronal growth | VEGF-B | neuronal growth | cornea | nerve injury | ANGIOGENESIS | PERMEABILITY | ENDOTHELIAL-GROWTH-FACTOR | MULTIDISCIPLINARY SCIENCES | ISCHEMIA | DEFICIENT MICE | KNOCKOUT MICE | RECOVERY | DISPLAY | PATHWAY | DISEASE | Neurites - physiology | Receptors, Notch - metabolism | Nerve Regeneration - physiology | Cell Survival - genetics | Phosphatidylinositol 3-Kinases - metabolism | Peripheral Nerves - drug effects | Cornea - drug effects | Peripheral Nerve Injuries - physiopathology | Neurons - physiology | Neurons - metabolism | Neurons - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Neurites - drug effects | Nerve Regeneration - genetics | Cell Survival - drug effects | Trigeminal Ganglion - cytology | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Peripheral Nerves - pathology | Blotting, Western | Mice, Knockout | Peripheral Nerves - physiopathology | Vascular Endothelial Growth Factor B - pharmacology | Animals | Signal Transduction - drug effects | Vascular Endothelial Growth Factor B - metabolism | Nerve Regeneration - drug effects | Cornea - innervation | Vascular Endothelial Growth Factor B - genetics | Cell Proliferation - drug effects | Cornea - physiopathology | Peripheral Nerve Injuries - genetics | Microscopy, Fluorescence | Sensory receptors | Vascular endothelial growth factor | Health aspects | Biological Sciences
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 9/2016, Volume 53, Issue 7, pp. 4461 - 4483
.... We screened libraries of known drugs for small molecule agonists of L1 and evaluated the effect of hit compounds in cell-based assays in vitro and in mice after femoral nerve and spinal cord injuries in vivo... 
Femoral nerve injury | Neurology | Neurosciences | Biomedicine | Neurobiology | Neuronal survival | Small molecule | Spinal cord injury | Cell adhesion molecule L1 | Neurite outgrowth | Cell Biology | IMMUNOGLOBULIN SUPERFAMILY | MICE DEFICIENT | SPINAL-CORD | NERVE INJURY | NEUROSCIENCES | POLYSIALIC ACID | RECOVERY | TRAUMATIC BRAIN-INJURY | ISCHEMIA-REPERFUSION | NF-KAPPA-B | Microglia - metabolism | Small Molecule Libraries - pharmacology | Astrocytes - pathology | Male | Duloxetine Hydrochloride - pharmacology | Femoral Nerve - injuries | Extracellular Signal-Regulated MAP Kinases - metabolism | Stilbenes - pharmacology | Motor Neurons - pathology | Myelin Sheath - metabolism | Spinal Cord Injuries - pathology | Proteolysis - drug effects | Microglia - pathology | Female | Neurons - metabolism | Neural Cell Adhesion Molecule L1 - metabolism | Phosphorylation - drug effects | Femoral Nerve - drug effects | Motor Neurons - drug effects | Neural Cell Adhesion Molecule L1 - agonists | Astrocytes - drug effects | Cell Survival - drug effects | Glucosides - pharmacology | Microglia - drug effects | Mice, Inbred C57BL | Cells, Cultured | Neuronal Outgrowth - drug effects | Peptides - pharmacology | Cell Movement - drug effects | Animals | Nerve Regeneration - drug effects | Cell Proliferation - drug effects | Spinal Cord Injuries - physiopathology | Astrocytes - metabolism | Neurons | Analysis | Molecules | Spinal cord injuries | Cell adhesion & migration
Journal Article