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The Journal of neuroscience, ISSN 0270-6474, 02/2013, Volume 33, Issue 9, pp. 3953 - 3966
Down syndrome (DS) is associated with neurological complications, including cognitive deficits that lead to impairment in intellectual functioning. Increased... 
Protein Binding - genetics | Electric Stimulation | Cell Count | Male | GABA Modulators - therapeutic use | Learning Disorders - etiology | Hyperkinesis - drug therapy | Neurogenesis - genetics | Long-Term Potentiation - drug effects | Protein Binding - drug effects | Exploratory Behavior - drug effects | Reflex, Startle - drug effects | Imidazoles - therapeutic use | Excitatory Postsynaptic Potentials - genetics | Disease Models, Animal | Mice, Transgenic | Hippocampus - pathology | Imidazoles - pharmacology | Psychomotor Performance - drug effects | Hyperkinesis - etiology | Vesicular Inhibitory Amino Acid Transport Proteins - metabolism | Analysis of Variance | Seizures - etiology | Glutamate Decarboxylase - metabolism | Mice | Receptors, GABA-A - metabolism | Benzodiazepines - therapeutic use | Tritium - pharmacokinetics | Sensory Gating - drug effects | Reflex - genetics | Reflex - drug effects | Hippocampus - drug effects | Excitatory Postsynaptic Potentials - drug effects | Ki-67 Antigen | Reaction Time - drug effects | GABA Modulators - pharmacology | Neurons - physiology | Down Syndrome - complications | Membrane Proteins - metabolism | Neurogenesis - drug effects | Neurons - drug effects | Cues | Down Syndrome - pathology | Benzodiazepines - pharmacology | Acoustic Stimulation | Mice, Inbred C57BL | Learning Disorders - drug therapy | Long-Term Potentiation - genetics | Biophysics | Maze Learning - drug effects | Rotarod Performance Test | Animals | Carrier Proteins - metabolism | Down Syndrome - drug therapy | Cell Proliferation - drug effects
Journal Article
Journal of applied physiology (1985), ISSN 1522-1601, 2012, Volume 112, Issue 6, pp. 1008 - 1014
.... The present study was designed to determine the effects of capsaicin and other chemicals in WAT on the sympathetic outflow and blood pressure and the roles of WAT afferents and hypothalamic paraventricular nucleus (PVN... 
Cardiovascular reflex | Sympathetic activity | Blood pressure | Paraventricular nucleus | blood pressure | RENOVASCULAR HYPERTENSIVE-RATS | NERVOUS-SYSTEM | SPORT SCIENCES | PHYSIOLOGY | LEPTIN SENSORS | sympathetic activity | SENSORY INNERVATION | BODY-FAT | OBESITY | cardiovascular reflex | AFFERENT REFLEX | DOMINANT CONTRIBUTOR | paraventricular nucleus | CAPSAICIN | Paraventricular Hypothalamic Nucleus - physiology | Reflex - physiology | Capsaicin - pharmacology | Sympathetic Nervous System - drug effects | Reflex - drug effects | Male | Muscle, Skeletal - innervation | Heart Rate - drug effects | Sympathetic Nervous System - physiology | Adipose Tissue, White - physiology | Muscle, Skeletal - drug effects | Neurons - physiology | Heart Rate - physiology | Blood Pressure - drug effects | Blood Pressure - physiology | Neurons - drug effects | Heart - innervation | Neurons, Afferent - drug effects | Kidney - drug effects | Rats | Muscle, Skeletal - physiology | Adipose Tissue, White - innervation | Neurons, Afferent - physiology | Rats, Sprague-Dawley | Stimulation, Chemical | Veins - drug effects | Paraventricular Hypothalamic Nucleus - drug effects | Animals | Veins - innervation | Heart - drug effects | Skin - innervation | Kidney - innervation | Adipose Tissue, White - drug effects | Skin - drug effects
Journal Article
Cell reports (Cambridge), ISSN 2211-1247, 2012, Volume 1, Issue 6, pp. 676 - 688
Translational control depends on phosphorylation of eIF2α by PKR-like ER kinase (PERK). To examine the role of PERK in cognitive function, we selectively... 
EUKARYOTIC TRANSLATION INITIATION | UNFOLDED PROTEIN RESPONSE | LONG-TERM-MEMORY | COGNITIVE CONTROL DEFICITS | FEAR EXTINCTION | PREPULSE INHIBITION | ENDOPLASMIC-RETICULUM | WOLCOTT-RALLISON-SYNDROME | BIPOLAR DISORDER | SYNAPTIC PLASTICITY | CELL BIOLOGY | Organ Specificity - drug effects | Protein-Serine-Threonine Kinases - deficiency | Reversal Learning - drug effects | eIF-2 Kinase - metabolism | Humans | Prefrontal Cortex - physiopathology | Receptors, N-Methyl-D-Aspartate - metabolism | Brain - enzymology | Motor Activity - drug effects | Schizophrenia - pathology | Prefrontal Cortex - pathology | eIF-2 Kinase - deficiency | Benzamides - administration & dosage | Prefrontal Cortex - drug effects | Piperidines - pharmacology | Schizophrenia - physiopathology | Gene Deletion | Exploratory Behavior - drug effects | Reflex, Startle - drug effects | Eukaryotic Initiation Factor-2 - metabolism | Behavior, Animal - drug effects | Schizophrenia - enzymology | Benzamides - pharmacology | Phosphorylation - drug effects | Prefrontal Cortex - enzymology | Protein-Serine-Threonine Kinases - metabolism | Piperidines - administration & dosage | Brain - physiopathology | Grooming - drug effects | Cognition - drug effects | Mice, Knockout | Brain - drug effects | Rotarod Performance Test | Animals | Activating Transcription Factor 4 - metabolism | Brain - pathology | Protein Biosynthesis - drug effects | Mice | eIF2α | behavioral flexibility | cognitive control | prefrontal cortex | glycine transporter-1 inhibitor | PERK | translational control | schizophrenia | ATF4
Journal Article
Neuropsychopharmacology (New York, N.Y.), ISSN 1740-634X, 2012, Volume 38, Issue 5, pp. 729 - 742
Journal Article
The Journal of neuroscience, ISSN 1529-2401, 2014, Volume 34, Issue 23, pp. 7931 - 7946
Journal Article
Nature neuroscience, ISSN 1546-1726, 2002, Volume 5, Issue 9, pp. 856 - 860
In the urinary bladder, the capsaicin-gated ion channel TRPV1 is expressed both within afferent nerve terminals and within the epithelial cells that line the... 
CELLS | PAIN | CHANNEL | REFLEX | RESINIFERATOXIN | INTRAVESICAL CAPSAICIN | RELEASE | ATP | NEUROSCIENCES | Immunohistochemistry | Reflex - genetics | Spinal Cord - metabolism | Capsaicin - pharmacology | Muscle Contraction - genetics | Receptors, Drug - genetics | Reflex - drug effects | Urination - genetics | Male | Urinary Bladder - innervation | Urothelium - pathology | Muscle, Smooth - drug effects | Adenosine Triphosphate - metabolism | Urination - drug effects | Receptors, Drug - deficiency | Spinal Cord - cytology | Urothelium - ultrastructure | Mechanoreceptors - metabolism | Urothelium - innervation | Neurons, Afferent - drug effects | Physical Stimulation | Muscle, Smooth - innervation | Cells, Cultured | Proto-Oncogene Proteins c-fos - metabolism | Acetic Acid - pharmacology | Microscopy, Electron | Mice, Knockout | Animals | Receptors, Drug - drug effects | Visceral Afferents - metabolism | Mechanoreceptors - drug effects | Muscle Contraction - drug effects | Muscle, Smooth - physiopathology | Urinary Bladder - physiopathology | Signal Transduction - physiology | Urinary Bladder - drug effects | Mice | Spinal Cord - physiopathology | Visceral Afferents - drug effects | Neurons, Afferent - metabolism | Nitric Oxide - metabolism | Bladder | Physiological aspects | Genetic aspects | Ion channels | Research | Neurons
Journal Article
Journal Article
American journal of physiology. Regulatory, integrative and comparative physiology, ISSN 0363-6119, 04/2016, Volume 310, Issue 8, pp. R697 - R706
We tested the hypothesis that an increase in the anti-inflammatory cholinergic pathway, when induced by pyridostigmine (PY), may modulate subtypes of... 
Myocardial infarction | T lymphocytes | Anticholinesterase drugs | Macrophage activation | Cholinergic anti-inflammatory reflex | IN-VITRO | PHYSIOLOGY | STIMULATION | BAROREFLEX SENSITIVITY | PATHWAY | HEART-RATE-VARIABILITY | AUTONOMIC FUNCTION | REGULATORY T-CELLS | NICOTINIC ACETYLCHOLINE-RECEPTOR | INFLAMMATORY RESPONSE | FAILURE | T-Lymphocytes, Regulatory - metabolism | Myocardium - immunology | Spleen - immunology | Rats, Inbred WKY | Male | Spleen - drug effects | Myocardial Infarction - immunology | T-Lymphocytes, Regulatory - immunology | Heart Rate - drug effects | Chemotaxis, Leukocyte - drug effects | Time Factors | Forkhead Transcription Factors - metabolism | Myocardial Infarction - pathology | Myocardium - metabolism | CD8-Positive T-Lymphocytes - metabolism | Myocardial Infarction - physiopathology | Blood Pressure - drug effects | Macrophages - immunology | Disease Models, Animal | Biomarkers - metabolism | Pyridostigmine Bromide - pharmacology | Anti-Inflammatory Agents - pharmacology | Cholinergic Neurons - metabolism | Ventricular Function, Left - drug effects | Myocardium - pathology | Cholinergic Neurons - drug effects | Myocardial Infarction - metabolism | Macrophages - metabolism | Phenotype | T-Lymphocytes, Regulatory - drug effects | Animals | Myocardial Infarction - drug therapy | Cholinesterase Inhibitors - pharmacology | Interleukin-2 Receptor alpha Subunit - metabolism | Spleen - metabolism | CD8-Positive T-Lymphocytes - drug effects | Macrophages - drug effects | CD8-Positive T-Lymphocytes - immunology | Macrophage Activation - drug effects | Cholinergic Neurons - pathology | Anti-inflammatory drugs | Heart attack | Euthanasia | Physiological aspects | Dosage and administration | Lymphocytes | Cholinesterase inhibitors | Health aspects | cholinergic anti-inflammatory reflex | anticholinesterase drugs | Translational Physiology | macrophage activation | myocardial infarction
Journal Article
Neuropsychopharmacology (New York, N.Y.), ISSN 1740-634X, 2011, Volume 37, Issue 2, pp. 456 - 466
...). Here, we examined whether cannabinoid receptor activation could prevent the effects of traumatic stress on the development of behavioral and neuroendocrine measures in a rat model of PTSD, the single-prolonged stress (SPS) model... 
single prolonged stress | hypothalamic-pituitary-adrenal (HPA) axis | basolateral amygdala | post-traumatic stress-disorder | inhibitory avoidance extinction | CB1 receptors | SYSTEM | LOUD TONES | PSYCHIATRY | PHYSIOLOGICAL-RESPONSES | NEUROSCIENCES | RESTRAINT STRESS | CONSOLIDATION | MEMORY | RECEPTOR ACTIVATION | PHARMACOLOGY & PHARMACY | EXTINCTION | MODULATION | AMYGDALA | Sensory Gating - drug effects | Amygdala - drug effects | Cannabinoid Receptor Antagonists | Humans | Extinction, Psychological - drug effects | Male | Anxiety - drug therapy | Anxiety - prevention & control | Pituitary-Adrenal Function Tests - methods | Piperidines - pharmacology | Morpholines - antagonists & inhibitors | Stress Disorders, Post-Traumatic - drug therapy | Reflex, Startle - drug effects | Benzoxazines - antagonists & inhibitors | Stress, Physiological - drug effects | Morpholines - therapeutic use | Disease Models, Animal | Pyrazoles - pharmacology | Hypothalamo-Hypophyseal System - drug effects | Dexamethasone | Benzoxazines - administration & dosage | Microinjections | Pituitary-Adrenal System - drug effects | Cannabinoids - administration & dosage | Morpholines - administration & dosage | Injections, Intraperitoneal | Morpholines - pharmacology | Rats | Rats, Sprague-Dawley | Benzoxazines - therapeutic use | Cannabinoids - pharmacology | Naphthalenes - pharmacology | Cannabinoid Receptor Agonists | Cannabinoids - therapeutic use | Animals | Naphthalenes - administration & dosage | Benzoxazines - pharmacology | Avoidance Learning - drug effects | Stress Disorders, Post-Traumatic - prevention & control | Naphthalenes - therapeutic use | Naphthalenes - antagonists & inhibitors | PTSD | Prevention | Neuroendocrinology | Drug Therapy | Hallucinogenic Drugs | Conditioned Emotional Responses | Animal Models | Avoidance | Stressors | learning & memory | animal models | amygdala | post-traumatic stress disorder | mood | stress disorders | CB1 receptorsKeywords:CB1 receptors | HPA axis | anxiety | extinction | single-prolonged stress | cannabinoids | Original
Journal Article
Gastroenterology, ISSN 0016-5085, 2013, Volume 144, Issue 1, pp. 145 - 154
Journal Article
Cardiovascular diabetology, ISSN 1475-2840, 2016, Volume 15, Issue 1, p. 157
.... This work was undertaken to investigate the non-glycemic effect of empagliflozin on metabolic syndrome rats with prediabetes. Methods: SHR/NDmcr-cp(+/+) rats (SHRcp... 
Cardiac protection | Oxidative stress | Adipose tissue | Inflammation | Natriuresis | Metabolic syndrome | Prediabetes | CARDIAC & CARDIOVASCULAR SYSTEMS | COTRANSPORTER 2 INHIBITORS | CARDIOVASCULAR OUTCOMES | BLOOD-PRESSURE | DIABETES-MELLITUS | GLUCOSE | ENDOCRINOLOGY & METABOLISM | DOUBLE-BLIND | SGLT2 INHIBITION | TYPE-2 | BARORECEPTOR REFLEX SENSITIVITY | Prediabetic State - metabolism | Intra-Abdominal Fat - physiopathology | Biomarkers - urine | Cardiomegaly - pathology | Male | Metabolic Syndrome - metabolism | Water-Electrolyte Balance - drug effects | Prediabetic State - drug therapy | Metabolic Syndrome - drug therapy | Adipocytes, White - drug effects | Time Factors | Metabolic Syndrome - physiopathology | Weight Gain - drug effects | Intra-Abdominal Fat - drug effects | Prediabetic State - pathology | Disease Models, Animal | Rats, Inbred SHR | Glucosides - pharmacology | Adiposity - drug effects | Sodium-Glucose Transporter 2 - metabolism | Cardiomegaly - physiopathology | Biomarkers - blood | Intra-Abdominal Fat - metabolism | Sodium-Glucose Transporter 2 - antagonists & inhibitors | Hypoglycemic Agents - pharmacology | Diuresis - drug effects | Adipocytes, White - pathology | Animals | Cardiomegaly - prevention & control | Prediabetic State - physiopathology | Benzhydryl Compounds - pharmacology | Hemodynamics - drug effects | Intra-Abdominal Fat - pathology | Metabolic Syndrome - pathology | Oxidative Stress - drug effects | Adipocytes, White - metabolism | Cardiomegaly - metabolism | Hypertrophy
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 2015, Volume 309, Issue 9, pp. H1479 - H1489
...) were continuously determined. With no hemodynamic effect at rest, fentanyl blockade during exercise attenuated both cardiac output and QL similar to 17... 
Dynamic exercise | Exercise pressor reflex | Afferent feedback | CARDIAC-OUTPUT | dynamic exercise | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | CAROTID BAROREFLEX CONTROL | exercise pressor reflex | afferent feedback | AFFERENTS CONTRIBUTE | METABOLIC-RESPONSE | SKELETAL-MUSCLE | ADVANCING AGE | ARTERIAL-BLOOD PRESSURE | FIBER-TYPE | PERIPHERAL VASCULAR DISEASE | Cardiac Output - physiology | Reflex - physiology | Humans | Analgesics, Opioid - pharmacology | Cardiovascular System - innervation | Reflex - drug effects | Male | Fentanyl - pharmacology | Young Adult | Regional Blood Flow - drug effects | Arterial Pressure - drug effects | Quadriceps Muscle - drug effects | Adult | Cardiovascular Physiological Phenomena - drug effects | Vasodilation - physiology | Autonomic Nervous System - drug effects | Cardiac Output - drug effects | Autonomic Nervous System - physiology | Cardiovascular System - drug effects | Arterial Pressure - physiology | Muscle, Skeletal - physiology | Femoral Artery - drug effects | Aging - physiology | Leg - blood supply | Muscle Contraction - physiology | Quadriceps Muscle - physiology | Aged | Femoral Artery - physiology | Vasodilation - drug effects | Regional Blood Flow - physiology | Complications and side effects | Care and treatment | Exercise | Aging | Influence | Research | Cardiovascular diseases | Health aspects | Call for Papers
Journal Article