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Nature Medicine, ISSN 1078-8956, 03/2010, Volume 16, Issue 3, pp. 302 - 307
Journal Article
Neuroscience, ISSN 0306-4522, 2007, Volume 148, Issue 1, pp. 22 - 33
Abstract Post-traumatic stress disorder (PTSD) is a stress-related mental disorder caused by traumatic experience, and presents with characteristic symptoms,... 
Neurology | amygdala | HPA axis | learning | fear conditioning | synaptic plasticity | hippocampus | LOUD TONES | BASOLATERAL AMYGDALA | DIFFERENTIAL CONTRIBUTION | PHYSIOLOGICAL-RESPONSES | CONDITIONED FEAR | NEUROSCIENCES | HIPPOCAMPAL PLASTICITY | DORSAL HIPPOCAMPUS | MEDIAL PREFRONTAL CORTEX | CHILDHOOD SEXUAL-ABUSE | LONG-TERM POTENTIATION | Amygdala - physiopathology | Memory Disorders - physiopathology | Receptors, Glucocorticoid - antagonists & inhibitors | Stress Disorders, Post-Traumatic - metabolism | Reflex, Abnormal - physiology | Nerve Degeneration - physiopathology | Memory Disorders - metabolism | Male | Receptors, Glucocorticoid - metabolism | Stress Disorders, Post-Traumatic - physiopathology | Fear - physiology | Hypothalamo-Hypophyseal System - secretion | Stress, Psychological - metabolism | Long-Term Potentiation - physiology | Anxiety Disorders - metabolism | Corticosterone - blood | Corticosterone - secretion | Hypothalamo-Hypophyseal System - physiopathology | Reflex, Startle - physiology | Disease Models, Animal | Mifepristone - analogs & derivatives | Amygdala - metabolism | Rats | Avoidance Learning - physiology | Anxiety Disorders - etiology | Rats, Sprague-Dawley | Hypothalamo-Hypophyseal System - metabolism | Hippocampus - metabolism | Phenotype | Animals | Memory Disorders - etiology | Cell Death - physiology | Mifepristone - pharmacology | Nerve Degeneration - etiology | Hippocampus - physiopathology | Anxiety Disorders - physiopathology | Memory - physiology | Stress, Psychological - physiopathology | Neurosciences | Dexamethasone | Corticosterone | Neurons | Depression, Mental | Hydrocarbons | Sulfates | Surface active agents | Post-traumatic stress disorder | Genetic aspects | Universities and colleges | Protein kinases | Steroids | PTSD | Neuroendocrinology | Conditioned Emotional Responses | Psychopharmacology | Animal Models | Neurophysiology
Journal Article
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2017, Volume 313, Issue 4, pp. H700 - H707
The last two decades have seen the emergence of Cre-Lox recombination as one of the most powerful and versatile technologies for cell-specific genetic... 
Heart rate | Magnetic resonance imaging | Baroreflex-mediated sympathetic vasomotor tone | Cre-lox recombination | Blood pressure | Cardiac vagal baroreflex | Isoflurane anesthesia | Diffusion tensor imaging | blood pressure | heart rate | magnetic resonance imaging | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | BARORECEPTOR REFLEX | NEUROTROPHIC FACTOR | Cre-Lox recombination | baroreflex-mediated sympathetic vasomotor tone | BLOOD-PRESSURE | HEART-RATE SIGNALS | SITE-SPECIFIC RECOMBINATION | isoflurane anesthesia | cardiac vagal baroreflex | diffusion tensor imaging | PERIPHERAL VASCULAR DISEASE | NEURAL CIRCUITS | SPECTRAL-ANALYSIS | SYSTEMIC ARTERIAL-PRESSURE | BRAIN | Brain-Derived Neurotrophic Factor - genetics | Isoflurane - pharmacology | Blood Pressure - genetics | Reflex, Abnormal - physiology | Neural Pathways | Heart Rate - physiology | Blood Pressure - physiology | Baroreflex - genetics | Reflex, Abnormal - drug effects | Solitary Nucleus - physiopathology | Anesthetics, Inhalation - pharmacology | Consciousness | Animals, Genetically Modified | Vasomotor System | Heart Rate - genetics | Mice, Inbred C57BL | Mice, Transgenic | Reflex, Abnormal - genetics | Diffusion Tensor Imaging | Integrases | Vagus Nerve - physiopathology | Baroreflex - drug effects | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - genetics | Magnetic Resonance Imaging | Phenotype | Animals | Baroreflex - physiology | Mice
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 01/2012, Volume 32, Issue 4, pp. 1336 - 1352
Loss-of-function mutations in human glycine receptors cause hyperekplexia, a rare inherited disease associated with an exaggerated startle response. We have... 
Humans | Reflex, Abnormal - physiology | Stiff-Person Syndrome - genetics | Rats | Signal Transduction - genetics | Mutation - physiology | Animals | Ion Channel Gating - genetics | Glycine - pharmacology | Glycine - physiology | HEK293 Cells | Receptors, Glycine - metabolism | Receptors, Glycine - genetics | Reflex, Startle - physiology
Journal Article