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Journal of Clinical Investigation, ISSN 0021-9738, 04/2012, Volume 122, Issue 4, pp. 1246 - 1261
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 12, p. e14195
Journal Article
Journal Article
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 01/2018, Volume 314, Issue 1, pp. G1 - G13
Previously, we showed that receptor for activated C kinase 1 (Rack1) regulates growth of colon cells in vitro, partly by suppressing Src kinase activity at key... 
Cell proliferation | Receptor for activated C kinase 1 | Intestinal epithelium | Gene knockout | Apoptosis | KINASE-ACTIVITY | RECOMBINATION | STEM-CELLS | PHYSIOLOGY | PROTEIN | cell proliferation | MOUSE | apoptosis | receptor for activated C kinase 1 | gene knockout | CRE | SRC ACTIVITY | intestinal epithelium | PANETH CELLS | SOX9 | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Intestinal Mucosa - radiation effects | Intestinal Mucosa - metabolism | Epithelial Cells - metabolism | Intestine, Small - pathology | Apoptosis - radiation effects | Intestinal Neoplasms - prevention & control | Mice, 129 Strain | Receptors for Activated C Kinase - genetics | Regeneration - radiation effects | Receptors for Activated C Kinase - metabolism | Intestine, Small - radiation effects | Neoplasms, Radiation-Induced - prevention & control | Neoplasms, Radiation-Induced - pathology | Cell Differentiation - radiation effects | Epithelial Cells - radiation effects | Signal Transduction | Intestinal Neoplasms - metabolism | Mice, Inbred C57BL | Epithelial Cells - pathology | Genotype | Neoplasms, Radiation-Induced - metabolism | Receptors for Activated C Kinase - deficiency | Intestinal Neoplasms - pathology | Mice, Knockout | Cell Lineage | Phenotype | Animals | Intestine, Small - metabolism | Cell Proliferation - radiation effects | Intestinal Mucosa - pathology | Intestines | Epithelial cells | Cellular control mechanisms | Genetic aspects | Observations | Cell differentiation
Journal Article
Acta Biomaterialia, ISSN 1742-7061, 04/2017, Volume 52, pp. 171 - 186
Journal Article
Genes and Development, ISSN 0890-9369, 05/2014, Volume 28, Issue 9, pp. 995 - 1004
Journal Article
Clinical Oral Implants Research, ISSN 0905-7161, 09/2017, Volume 28, Issue 9, pp. 1164 - 1172
ObjectiveRapid and stable fixation of dental implants is crucial for successful treatment. Herein, we examined whether the simultaneous treatment of titanium... 
bone regeneration | ultraviolet photofunctionalization | alendronate | osteoblast differentiation | dental implant | OSTEOBLAST | DENTAL IMPLANTS | ENGINEERING, BIOMEDICAL | OSSEOINTEGRATION | DELIVERING BISPHOSPHONATE | TITANIUM | LOCAL-DELIVERY | DENTISTRY, ORAL SURGERY & MEDICINE | FRACTURE REPAIR | SURFACE | INCREASES FIXATION | UV-PHOTOFUNCTIONALIZATION | Cell Differentiation - radiation effects | Osteoblasts - radiation effects | Humans | Osteoblasts - drug effects | Osteogenesis - drug effects | Cells, Cultured | Osteogenesis - radiation effects | Ultraviolet Rays | Cell Differentiation - drug effects | Bone-Implant Interface - radiation effects | Alendronate - pharmacology | Cell Proliferation - drug effects | Dental Implants | Cell Proliferation - radiation effects | Aluminum compounds | Phosphatases | Implant dentures | Analysis | Sunscreens (Cosmetics) | Radiation | Cell proliferation | Osseointegration | Alkaline phosphatase | Surgical implants | Transplants & implants | Calcium | Toxicity | Cytotoxicity | Osteoblasts | Alizarin | Bisphosphonates | Titanium base alloys | Dental restorative materials | Alendronic acid | Biomedical materials | Bone growth | Cell growth | Dental materials | Computed tomography | Mineralization | Tibia | Cell adhesion | Titanium | Biocompatibility | Scanning electron microscopy | Rabbits | U.V. radiation | Implantation | Dental prosthetics | Electron microscopy | Ultraviolet radiation | Osteoblastogenesis | Bone implants | Fixation | In vivo methods and tests | Dental implants | Differentiation | In vitro methods and tests | Osteogenesis
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, p. e73206
DNA damage can lead to the induction of cellular senescence. In particular, we showed that exposure to ionizing radiation (IR) leads to the senescence of bone... 
IRRADIATION | CHILDHOOD-CANCER | ACTIVATION | MULTIDISCIPLINARY SCIENCES | MICE | IONIZING-RADIATION | STROMAL CELLS | CELLULAR SENESCENCE | EXPRESSION | SURVIVORS | MESENCHYMAL STEM-CELLS | Cell Differentiation - radiation effects | Cell Line | Adipogenesis - radiation effects | Osteogenesis - physiology | Mesenchymal Stromal Cells - radiation effects | Osteocytes - metabolism | Osteocytes - cytology | Tumor Suppressor Protein p53 - metabolism | Mesenchymal Stromal Cells - metabolism | Osteogenesis - radiation effects | Tumor Suppressor Protein p53 - genetics | Adipogenesis - physiology | Cell Lineage - radiation effects | Mesenchymal Stromal Cells - cytology | Animals | Cellular Senescence - radiation effects | Mice | Radiation, Ionizing | Physiological aspects | Ionizing radiation | DNA binding proteins | Tumor proteins | Osteoprogenitor cells | Senescence | Transcription factors | p53 Protein | DNA damage | Homeostasis | Adipocytes | Osteoblasts | Accumulation | Genotype & phenotype | Bone growth | Mineralization | Bone marrow | Aging | Population | Biocompatibility | Bone density | Bone loss | Deoxyribonucleic acid--DNA | Adipogenesis | I.R. radiation | Exposure | Gene expression | Nodules | Regeneration | Stromal cells | Cells (biology) | Differentiation | Osteogenesis | Apoptosis | Deoxyribonucleic acid | DNA
Journal Article