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EMBO Journal, ISSN 0261-4189, 04/2009, Volume 28, Issue 8, pp. 1055 - 1066
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2014, Volume 289, Issue 3, pp. 1866 - 1875
Journal Article
Oncogene, ISSN 0950-9232, 03/2012, Volume 31, Issue 9, pp. 1143 - 1154
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2011, Volume 6, Issue 10, p. e27081
Inflammation is one of main mechanisms of autoimmune disorders and a common feature of most diseases. Appropriate suppression of inflammation is a key... 
Inflammation - chemically induced | Sirtuin 1 - metabolism | RNA, Small Interfering - genetics | Cell Proliferation | TOR Serine-Threonine Kinases - metabolism | Anti-Inflammatory Agents | NF-kappa B - metabolism | Interleukin-1beta - genetics | Stilbenes - pharmacology | Sirtuin 1 - genetics | Transcription Factor RelA - genetics | Inflammation - metabolism | Matrix Metalloproteinase 9 - metabolism | TOR Serine-Threonine Kinases - genetics | Inflammation - drug therapy | Interleukin-1beta - metabolism | Matrix Metalloproteinase 9 - genetics | Interleukin-6 - metabolism | Real-Time Polymerase Chain Reaction | Fibroblasts - metabolism | Cell Line | Inflammation Mediators | Interleukin-6 - genetics | RNA, Messenger - genetics | Tumor Necrosis Factor-alpha - toxicity | Sirtuin 1 - antagonists & inhibitors | Blotting, Western | Animals | NF-kappa B - genetics | Nitric Oxide Synthase Type II - genetics | Transcription Factor RelA - metabolism | Fibroblasts - drug effects | Fibroblasts - cytology | Mice | Antineoplastic Agents, Phytogenic - pharmacology | Apoptosis | Nitric Oxide Synthase Type II - metabolism | RHEUMATOID-ARTHRITIS | MEDIATED INFLAMMATION | APOPTOSIS | OBSTRUCTIVE PULMONARY-DISEASE | IN-VITRO | PROTEIN TRANSLATION | MULTIDISCIPLINARY SCIENCES | TRANS-RESVERATROL | SMOOTH-MUSCLE-CELLS | RELA ACETYLATION | NF-KAPPA-B | TOR protein | Phosphorylation | Matrix metalloproteinases | Interleukin | Arthritis | Kinases | Interleukin 6 | Proteins | Immunology | Ischemia | Rodents | Resveratrol | Fibroblasts | Acetylation | NF-κB protein | Immunoglobulins | Cytokines | RNA-mediated interference | Medical treatment | Neutrophils | Rheumatology | Inflammation | Rapamycin | Tumor necrosis factor-α | Metabolism | Gene expression | SIRT1 protein | Nitric-oxide synthase | Gelatinase B | Diseases | Hospitals | Ribonucleic acids | Nitric oxide | Sepsis | Ligands | RelA protein | Autoimmune diseases
Journal Article
Oncogene, ISSN 0950-9232, 11/2012, Volume 31, Issue 45, pp. 4798 - 4802
Journal Article
Cancer Letters, ISSN 0304-3835, 2016, Volume 383, Issue 2, pp. 261 - 271
Abstract Inactivation of p53 occurs frequently in various cancers. RITA is a promising anticancer small molecule that dissociates p53-MDM2 interaction,... 
Hematology, Oncology and Palliative Medicine | RITA | NF-κB RelA/p65 | ABCC6 | Chemoresistance | CANCER-CELLS | TRANSCRIPTION | DRUG-RESISTANCE | P53 FUNCTION | NEUROBLASTOMA-CELLS | NECK-CANCER | ONCOLOGY | SIGNALING PATHWAY | IN-VIVO | MULTIDRUG-RESISTANCE | NF-kappa B RelA/p65 | SMALL-MOLECULE RITA | Colonic Neoplasms - genetics | Phosphorylation | Apoptosis - drug effects | Drug Resistance, Multiple | Colonic Neoplasms - drug therapy | Furans - pharmacology | Humans | Serine | Drug Resistance, Neoplasm | Transcription Factor RelA - genetics | Colonic Neoplasms - metabolism | Breast Neoplasms - metabolism | Dose-Response Relationship, Drug | Transfection | MCF-7 Cells | RNA Interference | Time Factors | HEK293 Cells | Female | Multidrug Resistance-Associated Proteins - genetics | Antineoplastic Agents - pharmacology | HCT116 Cells | Breast Neoplasms - drug therapy | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - genetics | Signal Transduction - drug effects | Transcription Factor RelA - metabolism | Tumor Burden - drug effects | Breast Neoplasms - pathology | Mice, Nude | Colonic Neoplasms - pathology | Mutation | Multidrug Resistance-Associated Proteins - metabolism | p53 Protein | Xenotransplantation | Kinases | Drug resistance | Inactivation | Cancer therapies | Blockage | Anticancer properties | Proteins | Cell cycle | Xenografts | Conflicts of interest | Deoxyribonucleic acid--DNA | NF-κB protein | MDM2 protein | Deactivation | Gene expression | Sensitivity | Chemotherapy | Antitumor activity | RelA protein | Transporter | Cancer | Apoptosis
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2012, Volume 109, Issue 31, pp. 12662 - 12667
Chronic sleep deprivation perturbs the circadian clock and increases susceptibility to diseases such as diabetes, obesity, and cancer. Increased inflammation... 
Phosphorylation | Messenger RNA | Cytokines | Fibroblasts | Chronic diseases | Gene expression regulation | Inflammation | Diabetes | Macrophages | Immune system | Biological clock | TRANSCRIPTIONAL ACTIVITY | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | ALPHA | INDIVIDUAL FIBROBLASTS | IN-VITRO | OBESITY | IMMUNE-SYSTEM | biological clock | INFLAMMATION | immune system | GENE-EXPRESSION | NF-KAPPA-B | Inflammation - pathology | Humans | NF-kappa B - immunology | Cryptochromes - genetics | NF-kappa B - metabolism | Transcription Factor RelA - genetics | Inflammation - metabolism | Phosphorylation - genetics | Adenylyl Cyclases - immunology | Transcription Factor RelA - immunology | Cyclic AMP - genetics | Phosphorylation - immunology | Cyclic AMP - metabolism | Cytokines - genetics | Cryptochromes - metabolism | Cytokines - immunology | Cell Line | Gene Expression Regulation | Inflammation - immunology | Adenylyl Cyclases - metabolism | Circadian Rhythm | Mice, Knockout | Animals | NF-kappa B - genetics | Transcription Factor RelA - metabolism | Cryptochromes - immunology | Cyclic AMP - immunology | Inflammation - genetics | Mice | Adenylyl Cyclases - genetics | Cytokines - biosynthesis | Circadian rhythms | Proteins | Physiological aspects | Genetic aspects | Research | Gene expression | Health aspects | Biological Sciences
Journal Article
Nature Immunology, ISSN 1529-2908, 01/2011, Volume 12, Issue 1, pp. 29 - 36
Journal Article