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Biology of Blood and Marrow Transplantation, ISSN 1083-8791, 2015, Volume 21, Issue 12, pp. 2061 - 2068
Abstract Engraftment syndrome (ES) encompasses a continuum of periengraftment complications after autologous hematopoietic stem cell transplantation. ES may... 
Hematology, Oncology and Palliative Medicine | POEMS | Engraftment syndrome | Lymphoma | Autologous graft-versus-host disease | Multiple myeloma | RISK-FACTORS | BONE-MARROW-TRANSPLANTATION | IMMUNOLOGY | RECIPIENTS | VERSUS-HOST-DISEASE | TREATED RATS | TRANSPLANTATION | CYCLOSPORINE-A | POEMS SYNDROME | PHASE-I TRIAL | PEDIATRIC-PATIENTS | HEMATOLOGY | T-CELLS | Capillary Leak Syndrome - pathology | Graft vs Host Disease - therapy | Capillary Leak Syndrome - immunology | Humans | Immunosuppressive Agents - therapeutic use | Fever - therapy | Multiple Myeloma - immunology | POEMS Syndrome - therapy | Transplantation, Autologous | Graft vs Host Disease - immunology | Renal Insufficiency - etiology | Diarrhea - pathology | Exanthema - etiology | Brain Diseases - immunology | Capillary Leak Syndrome - therapy | Multiple Myeloma - therapy | Lymphoma - therapy | Brain Diseases - therapy | Diarrhea - immunology | Hematopoietic Stem Cell Transplantation - adverse effects | Lymphoma - pathology | Renal Insufficiency - immunology | Brain Diseases - etiology | Fever - immunology | Exanthema - therapy | Fever - pathology | Exanthema - immunology | Risk Factors | Capillary Leak Syndrome - etiology | Lymphoma - immunology | POEMS Syndrome - immunology | POEMS Syndrome - pathology | Graft vs Host Disease - pathology | Multiple Myeloma - pathology | Renal Insufficiency - therapy | Brain Diseases - pathology | Diarrhea - therapy | Renal Insufficiency - pathology | Exanthema - pathology | Hepatic Insufficiency - pathology | Fever - etiology | Diarrhea - etiology | Hepatic Insufficiency - immunology | Hematopoietic Stem Cell Transplantation - methods | Hepatic Insufficiency - etiology | Transplantation Conditioning | Hepatic Insufficiency - therapy | Medical colleges | Algorithms | Stem cells | Management techniques | Transplantation | Management | Hematopoietic stem cells | Diarrhea | Skin diseases | Risk factors
Journal Article
Kidney International, ISSN 0085-2538, 08/2012, Volume 82, Issue 4, pp. 454 - 464
Dense deposit disease and glomerulonephritis with isolated C3 deposits are glomerulopathies characterized by deposits of C3 within or along the glomerular... 
complement | glomerulonephritis | clinical immunology | membranoproliferative glomerulonephritis (MPGN) | LONG-TERM | HEMOLYTIC-UREMIC SYNDROME | MESANGIOCAPILLARY GLOMERULONEPHRITIS | GLOMERULONEPHRITIS TYPE-II | MACULAR DEGENERATION | FACTOR-H DEFICIENCY | GLOMERULAR-FILTRATION-RATE | UROLOGY & NEPHROLOGY | MUTATIONS | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | MEMBRANE COFACTOR PROTEIN | Humans | Child, Preschool | Male | Complement System Proteins - metabolism | Young Adult | Time Factors | DNA Mutational Analysis | Renal Insufficiency - immunology | Child | Glomerulonephritis, Membranoproliferative - pathology | Genetic Predisposition to Disease | Risk Assessment | Complement C3 - metabolism | Gene Frequency | Risk Factors | Glomerulonephritis - genetics | Glomerulonephritis - immunology | Glomerulonephritis - mortality | Biomarkers - blood | Kidney Glomerulus - pathology | Disease Progression | Glomerulonephritis, Membranoproliferative - genetics | Glomerulonephritis, Membranoproliferative - immunology | Phenotype | Complement Factor H - metabolism | Glomerulonephritis, Membranoproliferative - mortality | Glomerulonephritis - pathology | Adolescent | Age of Onset | Membrane Cofactor Protein - genetics | Kidney Glomerulus - immunology | Mutation | Complement Factor I - metabolism | Haplotypes | Complement C3 Nephritic Factor - genetics | Glomerulonephritis - therapy | Infant | Case-Control Studies | Adult | Complement System Proteins - genetics | Female | Membrane Cofactor Protein - metabolism | Complement Pathway, Alternative - genetics | France | Renal Insufficiency - genetics | Kaplan-Meier Estimate | Complement Factor I - genetics | Treatment Outcome | Chi-Square Distribution | Glomerulonephritis, Membranoproliferative - therapy | Biopsy | Complement C3 Nephritic Factor - metabolism | Complement Factor H - genetics
Journal Article
Journal Article
Kidney International, ISSN 0085-2538, 10/2012, Volume 82, Issue 7, pp. 790 - 796
Journal Article
Journal of Diabetes and Its Complications, ISSN 1056-8727, 2016, Volume 31, Issue 1, pp. 245 - 252
Abstract Aims To investigate and describe the relationship between diabetic nephropathy and systemic inflammation in patients with type 1 diabetes mellitus... 
Endocrinology & Metabolism | Endothelial dysfunction | Inflammation | Nephropathy | Monocyte | Type 1 diabetes mellitus | MORTALITY | CELLS | SUBPOPULATIONS | IDENTIFICATION | HEPARANASE | ENDOCRINOLOGY & METABOLISM | HIGH GLUCOSE | CARDIOVASCULAR-DISEASE | CHRONIC KIDNEY-DISEASE | EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | Leukocytes, Mononuclear - metabolism | Up-Regulation | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Middle Aged | Human Umbilical Vein Endothelial Cells - immunology | Male | Monocytes - metabolism | Monocytes - immunology | Diabetes Mellitus, Type 1 - complications | Diabetic Nephropathies - blood | Renal Insufficiency, Chronic - complications | Diabetic Nephropathies - immunology | Renal Insufficiency, Chronic - metabolism | Diabetic Angiopathies - pathology | Plasminogen Activator Inhibitor 1 - metabolism | Kidney Failure, Chronic - pathology | Human Umbilical Vein Endothelial Cells - cytology | Leukocytes, Mononuclear - immunology | Inflammation Mediators - metabolism | Monocytes - pathology | Female | Intercellular Adhesion Molecule-1 - blood | Renal Insufficiency - complications | Renal Insufficiency - immunology | Endothelium, Vascular - immunology | Severity of Illness Index | Diabetic Angiopathies - metabolism | Diabetic Nephropathies - pathology | Kidney Failure, Chronic - immunology | Renal Insufficiency, Chronic - immunology | Diabetic Nephropathies - metabolism | Cells, Cultured | Inflammation Mediators - blood | Renal Insufficiency - metabolism | Biomarkers - blood | Diabetic Angiopathies - blood | Disease Progression | Plasminogen Activator Inhibitor 1 - blood | Kidney Failure, Chronic - metabolism | Renal Insufficiency, Chronic - pathology | Leukocytes, Mononuclear - pathology | Intercellular Adhesion Molecule-1 - metabolism | Endothelium, Vascular - metabolism | Renal Insufficiency - pathology | Diabetic Angiopathies - immunology | Kidney Failure, Chronic - complications | Endothelium, Vascular - pathology | Type 1 diabetes | Care and treatment | Diabetic nephropathies | Medical research | Endothelial growth factors | Medicine, Experimental | Endothelium
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2015, Volume 10, Issue 4, pp. e0124775 - e0124775
Cancer chemotherapy drug cisplatin is known for its nephrotoxicity. The aim of this study is to investigate whether Epigallocatechin 3-Gallate (EGCG) can... 
OXIDATIVE STRESS | GREEN TEA CATECHINS | SUPEROXIDE-PRODUCTION | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | RAT-KIDNEY MITOCHONDRIA | REDOX STATE UNBALANCE | CELL-PROLIFERATION | INDUCED NEPHROTOXICITY | CANCER CHEMOPREVENTION | ENERGETIC METABOLISM | Inflammation - chemically induced | Kidney - pathology | Humans | Kidney - immunology | NF-kappa B - immunology | Male | Inflammation - metabolism | Kidney - metabolism | Renal Insufficiency - chemically induced | Antineoplastic Agents - adverse effects | Inflammation - drug therapy | Anti-Inflammatory Agents - therapeutic use | Catechin - therapeutic use | Renal Insufficiency - drug therapy | Renal Insufficiency - immunology | Catechin - pharmacology | Cell Line | Kidney - drug effects | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Inflammation - immunology | Renal Insufficiency - metabolism | Antioxidants - pharmacology | Antioxidants - therapeutic use | Animals | Cisplatin - adverse effects | Catechin - analogs & derivatives | Mice | Oxidative Stress - drug effects | Antioxidants | Enzymes | Chemotherapy | Catechin | Inflammation | Mitochondrial DNA | Superoxide | Tumor proteins | Green tea | Cisplatin | Cancer | Oxidative stress | Cell culture | Renal function | Syngeneic grafts | Interleukin | Superoxide dismutase | Experiments | Caspase-3 | Urology | Electron transport chain | Mitochondria | Cell growth | Rodents | DNA fragmentation | Polyphenols | Degeneration | Peroxidase | Stretching | Deoxyribonucleic acid--DNA | Glutathione | Creatinine | Glutathione peroxidase | Kidneys | Mortality | Epigallocatechin-3-gallate | Caspase | Metabolism | Gene expression | Anti-inflammatory agents | Medicine | Tea | Urea | Side effects | Hospitals | Cell death | Structural damage | Electron transport | Laboratory animals | Apoptosis | Kidney transplantation | Manganese | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Rheumatology (United Kingdom), ISSN 1462-0324, 03/2017, Volume 56, Issue 3, pp. 362 - 370
Journal Article
Kidney International, ISSN 0085-2538, 06/2017, Volume 91, Issue 6, pp. 1386 - 1397
C3 glomerulopathy is a potentially life-threatening disease of the kidney caused by dysregulated alternative pathway complement activation. The specific... 
properdin | C5a receptor | C3 glomerulopathy | complement factor H | ECULIZUMAB | ACTIVATION | ABNORMALITIES | COMPLEMENT ALTERNATIVE PATHWAY | FOLLOW-UP | NEPHRITIC FACTOR | IN-VIVO | UROLOGY & NEPHROLOGY | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | DENSE-DEPOSIT DISEASE | Receptor, Anaphylatoxin C5a - genetics | Kidney - pathology | Glomerulonephritis - prevention & control | Properdin - deficiency | Properdin - genetics | Kidney - immunology | Complement Factor H - deficiency | Renal Insufficiency - prevention & control | Mice, 129 Strain | Glomerulonephritis - metabolism | Proteinuria - prevention & control | Kidney - metabolism | Time Factors | Complement C5 - immunology | Renal Insufficiency - immunology | Disease Models, Animal | Renal Insufficiency - genetics | Genetic Predisposition to Disease | Complement C5 - metabolism | Kidney - drug effects | Proteinuria - immunology | Complement C3 - metabolism | Antibodies, Monoclonal - pharmacology | Mice, Inbred C57BL | Glomerulonephritis - genetics | Proteinuria - metabolism | Glomerulonephritis - immunology | Renal Insufficiency - metabolism | Complement C3 - immunology | Mice, Knockout | Phenotype | Animals | Signal Transduction - drug effects | Complement Inactivating Agents - pharmacology | Complement Pathway, Alternative - drug effects | Receptor, Anaphylatoxin C5a - deficiency | Complement Factor H - genetics | Complement C5 - antagonists & inhibitors | complement | factor H
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