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The New England journal of medicine, ISSN 1533-4406, 2013, Volume 369, Issue 23, pp. 2183 - 2196
Journal Article
Journal Article
Nature genetics, ISSN 1546-1718, 2012, Volume 44, Issue 8, pp. 910 - 915
Journal Article
Journal of the American Society of Nephrology, ISSN 1533-3450, 2013, Volume 24, Issue 9, pp. 1484 - 1491
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 2015, Volume 308, Issue 4, pp. L314 - L324
.... This population, with age, develops chronic organ injury, such as chronic kidney disease and pulmonary hypertension... 
Sickle cell disease | Nitric oxide | Pulmonary hypertension | Cell free hemoglobin | LACTATE-DEHYDROGENASE | nitric oxide | PHYSIOLOGY | pulmonary hypertension | NITRIC-OXIDE BIOAVAILABILITY | EXERCISE CAPACITY | KNOCKOUT MICE | sickle cell disease | cell free hemoglobin | ACUTE CHEST SYNDROME | RESPIRATORY SYSTEM | ARTERIAL-HYPERTENSION | FETAL-HEMOGLOBIN | TRANSGENIC MOUSE MODEL | PLATELET ACTIVATION | FREE HEMOGLOBIN | Humans | Heart Failure - physiopathology | Immunity, Innate - genetics | Oxidative Stress - immunology | Hypertension, Pulmonary - physiopathology | Acute Chest Syndrome - physiopathology | Hypertension, Pulmonary - therapy | Hemolysis - immunology | Renal Insufficiency, Chronic - therapy | Vascular Resistance - genetics | Heart Failure - immunology | Renal Insufficiency, Chronic - genetics | Reactive Oxygen Species - blood | Hemoglobin, Sickle - immunology | Toll-Like Receptor 4 - genetics | Vascular Diseases - genetics | Heart Failure - genetics | Toll-Like Receptor 4 - immunology | Renal Insufficiency, Chronic - physiopathology | Hemoglobin, Sickle - metabolism | Heme - immunology | Toll-Like Receptor 4 - blood | Nitric Oxide - genetics | Mutation | Vascular Diseases - blood | Practice Guidelines as Topic | Vascular Resistance - immunology | Heme - metabolism | Acute Chest Syndrome - therapy | Age Factors | Hypertension, Pulmonary - blood | Heart Failure - blood | Erythrocytes, Abnormal | Heart Failure - therapy | Heme - genetics | Reactive Oxygen Species - immunology | Acute Chest Syndrome - genetics | Acute Chest Syndrome - immunology | Hemolysis - genetics | Nitric Oxide - blood | Nitric Oxide - immunology | Vascular Diseases - immunology | Renal Insufficiency, Chronic - immunology | Vascular Diseases - therapy | Oxidative Stress - genetics | Hypertension, Pulmonary - genetics | Hypertension, Pulmonary - immunology | Hemoglobin, Sickle - genetics | Vascular Diseases - physiopathology | Renal Insufficiency, Chronic - blood | Acute Chest Syndrome - blood | Genetic aspects | Sickle cell anemia | Vasculitis | Diagnosis | Risk factors | Physiology in Medicine
Journal Article
The Lancet (British edition), ISSN 0140-6736, 2017, Volume 389, Issue 10075, pp. 1238 - 1252
Summary The definition and classification of chronic kidney disease (CKD) have evolved over time, but current international guidelines define this condition as decreased kidney function shown by glomerular filtration rate (GFR... 
Internal Medicine | GLOMERULAR-FILTRATION-RATE | LONGITUDINAL FOLLOW-UP | ESTIMATED GFR | MEDICINE, GENERAL & INTERNAL | HEMODIALYSIS-PATIENTS | RISK | QUALITY-OF-LIFE | IRON SUPPLEMENTATION | CANCER | CKD | BLOOD-PRESSURE | Diagnosis, Differential | Genetic Predisposition to Disease | Glomerular Filtration Rate | Prognosis | Kidney - pathology | Renal Insufficiency, Chronic - etiology | Humans | Image-Guided Biopsy - methods | Risk Factors | Disease Progression | Renal Insufficiency, Chronic - epidemiology | Renal Insufficiency, Chronic - therapy | Renal Insufficiency, Chronic - diagnosis | Proteinuria - etiology | Hypertension | Phosphates | Medical research | Epigenetic inheritance | Medical examination | Anemia | Mortality | Calcifediol | Blood | Vitamin D | Chronic kidney failure | Analysis | Medicine, Experimental | Alfacalcidol | Australia | Health aspects | Contrast media | Low income groups | Nephrology | Transplants & implants | Erythropoietin | Income | Calcium metabolism | Cardiovascular disease | Iron | Sclerosis | Incidence | Atrophy | Hepatitis | Human immunodeficiency virus--HIV | Blood pressure | Mathematical models | Appetite | High income | Nutrient deficiency | Cell survival | Blood cells | Diabetes mellitus | Health risks | Markers | Metabolism | Minority & ethnic groups | Quality of life | Glomerular filtration rate | Calcium phosphates | Ethnicity | Pay for performance | Biopsy | Socioeconomic factors | Coronary vessels | Fibrosis | Lethargy | Death | Kidney diseases | Diabetes | Acute coronary syndromes | Proteinuria | Cancer | Calcium | Hemodialysis | Bone diseases | Red blood cells | Population | Signs and symptoms | Kidneys | Complications | Cost analysis | Cardiovascular diseases
Journal Article
Kidney International, ISSN 0085-2538, 06/2018, Volume 93, Issue 6, pp. 1417 - 1431
... mechanisms by which diabetic kidney disease develops remain unknown. Familial aggregation of diabetic nephropathy indicates that genetic factors may be involved... 
diabetic nephropathy | gene expression | Cell Cycle - genetics | Prognosis | Kidney - pathology | Epigenesis, Genetic | Humans | Middle Aged | Apoptosis - genetics | Male | Diabetic Nephropathies - diagnosis | Kidney Failure, Chronic - ethnology | Case-Control Studies | Albuminuria - physiopathology | DNA Methylation | Diabetic Nephropathies - physiopathology | Renal Insufficiency, Chronic - genetics | Adult | Female | Renal Insufficiency, Chronic - diagnosis | Cytosine | Glomerular Filtration Rate - genetics | Energy Metabolism - genetics | Kidney - physiopathology | Kidney Failure, Chronic - physiopathology | Genetic Predisposition to Disease | Kidney Failure, Chronic - diagnosis | Risk Factors | Albuminuria - ethnology | Diabetic Nephropathies - genetics | Diabetic Nephropathies - ethnology | Renal Insufficiency, Chronic - physiopathology | Disease Progression | Albuminuria - genetics | Kidney Failure, Chronic - genetics | Phenotype | Renal Insufficiency, Chronic - ethnology | Fibrosis | Indians, North American - genetics | CpG Islands | Aged | Renal function | Pathogenesis | Regulatory sequences | Hyperglycemia | Renal insufficiency | Rodents | DNA methylation | Deoxyribonucleic acid--DNA | CpG islands | Creatinine | Kidneys | Epidermal growth factor receptors | DNA probes | Diabetes mellitus | Metabolism | Gene expression | Glomerular filtration rate | Studies | Molecular modelling | Nephropathy | Epigenetics | Diabetes | Kidney diseases | Apoptosis | Diabetic nephropathy
Journal Article
Nature medicine, ISSN 1546-170X, 2018, Volume 24, Issue 11, pp. 1721 - 1731
Chronic kidney disease (CKD), a condition in which the kidneys are unable to clear waste products, affects 700 million people globally... 
FIBROSIS | MEDICINE, RESEARCH & EXPERIMENTAL | ALLELES | BIOCHEMISTRY & MOLECULAR BIOLOGY | RESISTANT NEPHROTIC SYNDROME | RISK | LOCI | VARIANT | CELL BIOLOGY | EPITHELIAL-CELLS | PROXIMAL TUBULE | EXPRESSION | GENOME-WIDE ASSOCIATION | Genetic Predisposition to Disease | Genome-Wide Association Study | Kidney Tubules, Proximal - pathology | Kidney - pathology | Gene Expression Regulation - genetics | Humans | Cell Compartmentation - genetics | Signal Transduction - genetics | Kidney Glomerulus - pathology | Renal Insufficiency, Chronic - physiopathology | Kidney - metabolism | Animals | Transforming Growth Factor beta - genetics | Tumor Suppressor Proteins - genetics | Adaptor Proteins, Signal Transducing - genetics | Kidney Tubules, Proximal - metabolism | Renal Insufficiency, Chronic - genetics | Kidney Glomerulus - metabolism | Polymorphism, Single Nucleotide - genetics | Mice | Disease Models, Animal | Quantitative Trait Loci - genetics | Quantitative trait loci | Kidneys | Chronic kidney failure | Genetic variation | Physiological aspects | Development and progression | Genetic aspects | Health aspects | Nucleotide sequence | Genes | Genomes | Genetic diversity | Ribonucleic acid--RNA | Gene expression | Renal tubules | Gene sequencing | Proteins | Pathways | Genetic analysis | Kidney diseases | Gene mapping | compartment-specific | renal disease | eQTL
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2013, Volume 8, Issue 12, p. e84450
...) and its genetic background has not been completely clarified. The aim of this investigation was to analyze single nucleotide polymorphisms (SNPs... 
RHEUMATOID-ARTHRITIS | POPULATION | VARIANTS | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | IRF5 | SLE | INTERFERON REGULATORY FACTOR-5 | RISK HAPLOTYPE | T-CELLS | ERYTHEMATOSUS | Severity of Illness Index | Genetic Predisposition to Disease | Genome-Wide Association Study | Lupus Nephritis - genetics | Renal Insufficiency, Chronic - etiology | Humans | Male | Case-Control Studies | Renal Insufficiency, Chronic - genetics | Adult | Female | STAT4 Transcription Factor - genetics | Polymorphism, Single Nucleotide | Lupus Nephritis - complications | Lupus | Nephritis | Kidney failure | Systemic lupus erythematosus | Analysis | Genetic aspects | Single nucleotide polymorphisms | Chromosomes | Laboratories | Genes | Science | Genomes | Arthritis | Single-nucleotide polymorphism | Lupus nephritis | Proteins | Ethics | Renal insufficiency | Histopathology | Consent | Gangrene | Population | Tumor necrosis factor-TNF | Proliferative kidney disease | IRAK protein | Kidneys | Blood & organ donations | Rheumatology | Stat4 protein | Genetic diversity | Whites | Patients | Morbidity | Medicine | Studies | Chronic conditions | Hospitals | Biopsy | Lymphomas | Histocompatibility antigen HLA | Mutation | Kidney diseases | Autoimmune diseases | Polymorphism | Kidney transplantation | Rheumatology and Autoimmunity | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Reumatologi och inflammation
Journal Article