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Kidney International, ISSN 0085-2538, 09/2014, Volume 86, Issue 3, pp. 570 - 581
Angiotensin II type 1 receptor (AT1R)–associated protein (ATRAP) promotes AT1R internalization along with suppression of pathological activation of tissue AT1R... 
epithelial sodium transport | angiotensin | blood pressure | hypertension | renin–angiotensin system | cell signaling | renin-angiotensin system | INFUSED MICE | EPITHELIAL NA+ CHANNEL | DEFICIENT MICE | INTERACTING MOLECULE | MESSENGER-RNA | CORTICAL COLLECTING DUCT | REGULATE BLOOD-PRESSURE | CHLORIDE COTRANSPORTER | UROLOGY & NEPHROLOGY | SMOOTH-MUSCLE-CELLS | DEPENDENT HYPERTENSION | Urinalysis | Blood Pressure - genetics | Male | Sodium - metabolism | RNA, Messenger - metabolism | Renal Reabsorption - genetics | Aldosterone - urine | Sodium-Hydrogen Exchangers - metabolism | Receptor, Angiotensin, Type 1 - genetics | Renal Reabsorption - drug effects | Epithelial Sodium Channels - drug effects | Gene Deletion | Hypertension - chemically induced | Blood Pressure - drug effects | Hypertension - genetics | Vasoconstrictor Agents - pharmacology | Angiotensin II - pharmacology | Mice, Inbred C57BL | Aldosterone - blood | Angiotensinogen - metabolism | Mice, Knockout | Animals | Adaptor Proteins, Signal Transducing - deficiency | Receptor, Angiotensin, Type 1 - metabolism | Adaptor Proteins, Signal Transducing - genetics | Kidney Tubules, Proximal - metabolism | Mice | Adaptor Proteins, Signal Transducing - metabolism | Hydrogen-Ion Concentration | Kidney Tubules, Distal - metabolism | Sodium-Hydrogen Exchanger 3 | Basic Research
Journal Article
Kidney International, ISSN 0085-2538, 01/2018, Volume 93, Issue 1, pp. 147 - 158
We examined activin receptor type IIA (ActRIIA) activation in chronic kidney disease (CKD) by signal analysis and inhibition in mice with Alport syndrome using... 
cardiovascular disease | chronic kidney disease | bone | fibrosis | mineral metabolism | vascular calcification | TGF-BETA | CARDIOVASCULAR EVENTS | RENAL FIBROSIS | ARTERIAL CALCIFICATION | BONE MORPHOGENETIC PROTEIN-7 | GROWTH-FACTOR 23 | UROLOGY & NEPHROLOGY | SMOOTH-MUSCLE-CELLS | LINKED ALPORT-SYNDROME | OSTEOCLAST DIFFERENTIATION | ATHEROSCLEROTIC LESION | Kidney - pathology | Recombinant Fusion Proteins - pharmacology | Blood Vessels - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - genetics | Vascular Calcification - metabolism | Kidney - metabolism | Bone Resorption - genetics | Bone and Bones - metabolism | Myocardium - metabolism | Renal Insufficiency, Chronic - genetics | Bone Resorption - metabolism | Disease Models, Animal | Bone Resorption - physiopathology | Glomerular Filtration Rate | Signal Transduction | Smad2 Protein - metabolism | Cardiomegaly - physiopathology | Nephritis, Hereditary - genetics | Renal Insufficiency, Chronic - physiopathology | Mice, Knockout | Renal Insufficiency, Chronic - prevention & control | Cardiomegaly - prevention & control | Nephritis, Hereditary - metabolism | Collagen Type IV - deficiency | Activin Receptors, Type II - genetics | Fibrosis | Sp7 Transcription Factor - metabolism | Collagen Type IV - genetics | Cardiomegaly - metabolism | Activin Receptors, Type II - metabolism | Bone and Bones - pathology | Phosphorylation | Vascular Remodeling | Blood Vessels - pathology | Vascular Calcification - genetics | Actins - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - physiopathology | Activin Receptors, Type II - antagonists & inhibitors | Blood Vessels - physiopathology | Renal Insufficiency, Chronic - metabolism | Ventricular Remodeling | Chronic Kidney Disease-Mineral and Bone Disorder - prevention & control | Muscle Proteins - metabolism | Vascular Calcification - physiopathology | Microfilament Proteins - metabolism | Nephritis, Hereditary - physiopathology | Kidney - physiopathology | Vascular Calcification - prevention & control | Myocardium - pathology | Bone Resorption - prevention & control | Bone and Bones - physiopathology | Animals | Nephritis, Hereditary - drug therapy | Cardiomegaly - genetics | Bone Remodeling | Chronic kidney disease
Journal Article
Journal Article
Pediatric Nephrology, ISSN 0931-041X, 2017, Volume 32, Issue 7, pp. 1123 - 1135
Magnesium is essential to the proper functioning of numerous cellular processes. Magnesium ion (Mg2+) deficits, as reflected in hypomagnesemia, can cause... 
Homeostasis | Thick ascending limb of Henle’s loop | Magnesium | Hereditary | Distal convoluted tubule | Kidney | PCBD1 CAUSE | FAMILIAL HYPOMAGNESEMIA | SENSORINEURAL DEAFNESS | ATAXIA | CALCIUM-SENSING RECEPTOR | HYPOPARATHYROIDISM | UROLOGY & NEPHROLOGY | Thick ascending limb of Henle's loop | PEDIATRICS | MUTATIONS | KENNY-CAFFEY-SYNDROME | BARTTER-SYNDROME | Seizures - blood | Hypokalemia - blood | Kidney Tubules, Distal - physiology | Humans | Renal Tubular Transport, Inborn Errors - genetics | Renal Reabsorption - genetics | Nephrocalcinosis - complications | Renal Tubular Transport, Inborn Errors - blood | Hypercalciuria - blood | Magnesium Deficiency - genetics | Magnesium Deficiency - complications | Renal Reabsorption - drug effects | Hypokalemia - drug therapy | Magnesium - physiology | Magnesium Deficiency - drug therapy | Hypercalciuria - genetics | Membrane Proteins - metabolism | Child | Hypokalemia - etiology | Renal Tubular Transport, Inborn Errors - drug therapy | Membrane Proteins - genetics | Nephrocalcinosis - drug therapy | Arrhythmias, Cardiac - etiology | Mitochondria - metabolism | Loop of Henle - physiology | Renal Tubular Transport, Inborn Errors - complications | Magnesium - therapeutic use | Mineralocorticoid Receptor Antagonists - therapeutic use | Hypercalciuria - complications | Hypercalciuria - drug therapy | Phenotype | Renal Elimination - genetics | Nephrocalcinosis - genetics | Epithelial Sodium Channel Blockers - therapeutic use | Seizures - etiology | Nephrocalcinosis - blood | Arrhythmias, Cardiac - blood | Mutation | Recommended Dietary Allowances | Hypokalemia - genetics | Magnesium - blood | Homeostasis - genetics | Complications and side effects | Care and treatment | Kidney failure | Arrhythmia | Gene mutations | Research | Magnesium deficiency diseases | Review
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2012, Volume 7, Issue 7, p. e40799
Journal Article