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Hypertension, ISSN 0194-911X, 09/2004, Volume 44, Issue 3, pp. 264 - 270
Angiotensin II (Ang II) upregulates vascular endothelial growth factor (VEGF) and activates vascular inflammation. However, the decisive role of VEGF in Ang... 
Growth substances | Endothelial growth factors | Angiotensin II | Remodeling | Arteriosclerosis | MACROPHAGE INFILTRATION | FACTOR VEGF | remodeling | NITRIC-OXIDE SYNTHESIS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | endothelial growth factors | FACTOR-KAPPA-B | TUMOR ANGIOGENESIS | CHRONIC BLOCKADE | TYPE-1 RECEPTOR | arteriosclerosis | growth substances | angiotensin II | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | TISSUE ANGIOTENSIN | Genetic Therapy | Tetrazoles - pharmacology | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Male | Gene Expression Profiling | Vasculitis - prevention & control | Vascular Endothelial Growth Factor A - genetics | Angiotensin II Type 1 Receptor Blockers - pharmacology | Transforming Growth Factor beta - biosynthesis | Hypoxia-Inducible Factor 1, alpha Subunit | Interleukin-1 - biosynthesis | Vascular Endothelial Growth Factor Receptor-2 - genetics | Imidazoles - therapeutic use | Receptors, Chemokine - genetics | Vasculitis - chemically induced | Coronary Vessels - pathology | Natriuretic Peptide, Brain - genetics | Transforming Growth Factor beta1 | Hypoxia-Inducible Factor 1 | Interleukin-6 - genetics | Extracellular Matrix Proteins - genetics | Imidazoles - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Aorta - pathology | Macrophages - metabolism | Intercellular Adhesion Molecule-1 - genetics | Chemokine CCL2 - biosynthesis | Nonmuscle Myosin Type IIB | Vascular Cell Adhesion Molecule-1 - biosynthesis | Mice | Vascular Endothelial Growth Factor A - physiology | Hypertrophy | Interleukin-1 - genetics | Tunica Media - pathology | Vascular Endothelial Growth Factor A - biosynthesis | Extracellular Matrix Proteins - biosynthesis | Tunica Media - drug effects | Vasculitis - physiopathology | Recombinant Fusion Proteins - physiology | Ventricular Remodeling | Nuclear Proteins - biosynthesis | Cell Division | Intercellular Adhesion Molecule-1 - biosynthesis | Receptors, Chemokine - biosynthesis | Vascular Cell Adhesion Molecule-1 - genetics | Vascular Endothelial Growth Factor Receptor-2 - biosynthesis | Angiotensin II - toxicity | Nuclear Proteins - genetics | Olmesartan Medoxomil | Hypertrophy, Left Ventricular - etiology | Mice, Inbred C57BL | Extracellular Matrix Proteins - physiology | Natriuretic Peptide, Brain - biosynthesis | Chemokine CCL2 - genetics | Renin-Angiotensin System - physiology | Transcription Factors - biosynthesis | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Animals | Transforming Growth Factor beta - genetics | Interleukin-6 - biosynthesis | Myosin Heavy Chains | Tetrazoles - therapeutic use | DNA-Binding Proteins - biosynthesis | Receptors, CCR2
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2014, Volume 9, Issue 9, p. e107556
Background: Exposure to subclinical levels of lipopolysaccharide (LPS) occurs commonly and is seemingly well tolerated. However, recurrent LPS exposure induces... 
RESPONSES | OBESITY | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | CARDIOVASCULAR-DISEASE | ENDOTOXEMIA | PRESERVED EJECTION FRACTION | MICRORNA THERAPEUTICS | CARDIAC FIBROSIS | NADPH OXIDASES | REVEALS | Matrix Metalloproteinase 9 - biosynthesis | Tissue Inhibitor of Metalloproteinase-1 - biosynthesis | Cell Adhesion Molecules - biosynthesis | Membrane Glycoproteins - biosynthesis | Mice, Inbred C57BL | Cells, Cultured | Male | MicroRNAs - biosynthesis | NADPH Oxidase 2 | Lipopolysaccharides | Collagen Type I - biosynthesis | Hypertrophy, Left Ventricular - chemically induced | Animals | Tissue Inhibitor of Metalloproteinase-2 - biosynthesis | Endomyocardial Fibrosis - chemically induced | Mice | MicroRNAs - genetics | NADPH Oxidases - biosynthesis | Matrix Metalloproteinase 2 - biosynthesis | Cardiomyopathies - chemically induced | Oxidases | RNA | Collagen | Fibrosis | Angiotensin | Bone morphogenetic proteins | Transforming growth factors | Mitogens | Heart | Animal models | Genes | Genomics | Cardiovascular disease | Tissue inhibitor of metalloproteinase 2 | Kinases | Tissue inhibitor of metalloproteinase 1 | NAD(P)H oxidase | Connective tissues | Renin | Rodents | Transcription activation | CYBB protein | Fibroblasts | Cardiology | Heart diseases | Growth factors | Heart failure | Appetite | Research & development--R&D | Mortality | Connective tissue growth factor | Exposure | Metabolism | Gene expression | Ribonucleic acid--RNA | Gelatinase B | Gelatinase A | Medicine | MicroRNAs | Ventricle | Laboratory animals | Research & development | Ribonucleic acid | R&D
Journal Article
Journal Article
Hypertension, ISSN 0194-911X, 01/2005, Volume 45, Issue 1, pp. 133 - 137
Renin is regulated by angiotensin subtype 1 (AT1) receptor, but it is unknown whether angiotensin subtype 2 (AT2) receptor contributes to this regulation. We... 
Renin | Receptors, angiotensin | Kidney | Angiotensin | PRESSURE-NATRIURESIS | PROTEIN | AT RECEPTOR | JUXTAGLOMERULAR CELLS | TYPE-1 RECEPTOR | CONSCIOUS RATS | kidney | renin | angiotensin | NITRIC-OXIDE | GENE-EXPRESSION | receptors, angiotensin II | PERIPHERAL VASCULAR DISEASE | SECRETION | Angiotensin II - genetics | Juxtaglomerular Apparatus - drug effects | Tetrazoles - pharmacology | Valsartan | Receptor, Angiotensin, Type 2 - physiology | Depression, Chemical | Imidazoles - administration & dosage | Systole - drug effects | Valine - administration & dosage | Angiotensin II Type 1 Receptor Blockers - pharmacology | RNA, Messenger - biosynthesis | Tetrazoles - administration & dosage | Female | Sodium, Dietary - administration & dosage | Kidney Tubules - metabolism | Angiotensin II Type 1 Receptor Blockers - administration & dosage | Renin - biosynthesis | Pyridines - administration & dosage | Consciousness | Kidney Tubules - drug effects | Natriuresis - drug effects | Kidney Cortex | Valine - analogs & derivatives | Rats | Renin-Angiotensin System - physiology | Imidazoles - pharmacology | Renin - blood | Reverse Transcriptase Polymerase Chain Reaction | Rats, Sprague-Dawley | Injections | Animals | Juxtaglomerular Apparatus - metabolism | Angiotensin II Type 2 Receptor Blockers | Angiotensin II - biosynthesis | Pyridines - pharmacology | Valine - pharmacology
Journal Article
Heart Failure Reviews, ISSN 1382-4147, 1/2005, Volume 10, Issue 1, pp. 7 - 13
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2012, Volume 7, Issue 3, p. e34026
Background: Uremic toxins are considered to have a determinant pathological role in the progression of chronic kidney disease. The aim of this study was to... 
GROWTH-FACTOR-BETA | P-CRESYLSULPHATE | SULFATE | MULTIDISCIPLINARY SCIENCES | EXPRESSION | PROGRESSION | MOTILITY | Cresols - toxicity | Renin - biosynthesis | Fibrosis - chemically induced | Kidney - pathology | Kidney Tubules | Gene Expression Regulation | Losartan - pharmacology | Male | Renin-Angiotensin System - physiology | Transcription Factors - biosynthesis | Sulfuric Acid Esters - toxicity | Uremia - complications | Indican - toxicity | Animals | Kidney Failure, Chronic - pathology | Models, Biological | Receptor, Angiotensin, Type 1 - biosynthesis | Angiotensinogen - biosynthesis | Epithelial-Mesenchymal Transition | Kidney Failure, Chronic - complications | Mice | Snail Family Transcription Factors | Transforming Growth Factor beta1 - biosynthesis | Fibronectins | Medical research | Corticosteroids | Development and progression | Aldosterone | Transforming growth factors | Sulfates | Muscle proteins | Chronic kidney failure | Fibrosis | Stem cells | Angiotensin | Medicine, Experimental | Bone morphogenetic proteins | Steroids | Transcription factors | Nephrology | Motility | Mesenchyme | Syngeneic grafts | Transforming growth factor | Smooth muscle | Smad3 protein | Kinases | Renal tubules | Smad4 protein | E-cadherin | Fibronectin | Ovarian cancer | Proteins | Cresol | p-Cresol | Mouse devices | Renin | Actin | Smad2 protein | Rodents | Angiotensin AT1 receptors | Angiotensinogen | Growth factors | Cytomegalovirus | Muscles | Breast cancer | Esophagus | Polymerase chain reaction | Toxins | Kidney diseases | Sulfate | Snail protein | Thermal cycling | Binding sites
Journal Article
Gynecologic Oncology, ISSN 0090-8258, 2008, Volume 109, Issue 3, pp. 418 - 425
Journal Article
Journal of Steroid Biochemistry and Molecular Biology, ISSN 0960-0760, 2005, Volume 96, Issue 1, pp. 59 - 66
Journal Article
Journal Article