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Physiological Reviews, ISSN 0031-9333, 07/2006, Volume 86, Issue 3, pp. 747 - 803
Journal Article
Circulation Research, ISSN 0009-7330, 03/2015, Volume 116, Issue 6, pp. 991 - 1006
Excess weight gain, especially when associated with increased visceral adiposity, is a major cause of hypertension, accounting for 65% to 75% of the risk for... 
blood pressure | kidney | chronic renal insufficiency | melanocortins | leptin | mineralocorticoids | CARDIAC & CARDIOVASCULAR SYSTEMS | SYMPATHETIC-NERVOUS-SYSTEM | NATRIURETIC PEPTIDES | CARDIOVASCULAR FUNCTIONS | BLOOD-PRESSURE | MELANOCORTIN SYSTEM | BODY-MASS-INDEX | RENIN-ANGIOTENSIN SYSTEM | ARTERIAL-PRESSURE | SELECTIVE LEPTIN RESISTANCE | PERIPHERAL VASCULAR DISEASE | CHRONIC KIDNEY-DISEASE | HEMATOLOGY | Natriuresis - physiology | Antihypertensive Agents - pharmacology | Intra-Abdominal Fat - physiopathology | Prevalence | Dyslipidemias - complications | Humans | Sodium - metabolism | Sympathetic Nervous System - physiopathology | Pro-Opiomelanocortin - physiology | Hypertension - etiology | Leptin - physiology | Insulin Resistance - physiology | Metabolic Syndrome - physiopathology | Models, Animal | Parasympathetic Nervous System - physiopathology | Hypertension - epidemiology | Kidney - physiopathology | Models, Cardiovascular | Obesity - complications | Aldosterone - physiology | Obesity - physiopathology | Renin-Angiotensin System - physiology | Antihypertensive Agents - therapeutic use | Organ Specificity | Pressure | Renal Insufficiency, Chronic - physiopathology | Hypertension - physiopathology | Metabolic Syndrome - complications | Animals | Heart Conduction System - physiopathology | Receptors, Leptin - physiology | Obesity - epidemiology | Renin-Angiotensin System - drug effects | Hemodynamics | Dyslipidemias - physiopathology | chronic kidney disease | sympathetic activity | Blood pressure | renin-angiotensin system
Journal Article
Circulation Research, ISSN 0009-7330, 04/2013, Volume 112, Issue 8, pp. 1104 - 1111
Journal Article
Circulation Research, ISSN 0009-7330, 03/2015, Volume 116, Issue 6, pp. 960 - 975
Blockers of the renin–angiotensin–aldosterone system (RAAS), that is, renin inhibitors, angiotensin (Ang)-converting enzyme (ACE) inhibitors, Ang II type 1... 
remodeling | gender | aldosterone | AT2 receptor | hyperkalemia | resistant hypertension | receptor | renin | primary aldosteronism | reactive oxygen species | angiotensin | angiotensin-(1-7) | prorenin | angiotensin-(1–7) | CARDIAC & CARDIOVASCULAR SYSTEMS | BLOOD-PRESSURE RESPONSES | PLASMA-RENIN | CONVERTING ENZYME-INHIBITORS | CORONARY VASCULAR BED | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | II TYPE-2 RECEPTOR | HEMATOLOGY | MYOCARDIAL-INFARCTION | AT 2 receptor | CELL MINERALOCORTICOID RECEPTORS | SEX-DIFFERENCES | CONGESTIVE-HEART-FAILURE | Antihypertensive Agents - pharmacology | Drugs, Investigational - pharmacology | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Humans | Drugs, Investigational - therapeutic use | Endothelium, Vascular - drug effects | Hypertension - drug therapy | Male | Ion Channels - physiology | Sex Chromosomes | Molecular Targeted Therapy | Gonadal Steroid Hormones - physiology | Vasoconstriction - physiology | Drug Interactions | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Hyperaldosteronism - physiopathology | Female | Hypertension - genetics | Drug Evaluation, Preclinical | Drug Resistance | Precision Medicine | Aldosterone - physiology | Endothelium, Vascular - physiopathology | Hyperaldosteronism - genetics | Mice, Transgenic | Renin-Angiotensin System - genetics | Renin-Angiotensin System - physiology | Clinical Trials as Topic | Sex Characteristics | Antihypertensive Agents - therapeutic use | Vasoconstriction - drug effects | Hyperaldosteronism - drug therapy | Hypertension - physiopathology | Mice, Knockout | Mineralocorticoid Receptor Antagonists - therapeutic use | Animals | Renin-Angiotensin System - drug effects | Mice | Therapeutic Equivalency
Journal Article