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Journal Article
Circulation research, ISSN 1524-4571, 2017, Volume 120, Issue 9, pp. 1477 - 1486
The translation from numerous successful animal experiments on cardioprotection beyond that by reperfusion to clinical practice has to date been disappointing.... 
Cardioprotection | Reperfusion | Remote ischemic conditioning | Myocardial ischemia | Postconditioning | Preconditioning | Reperfusion injury | PERMEABILITY TRANSITION PORE | postconditioning | ISCHEMIA/REPERFUSION INJURY | CARDIAC & CARDIOVASCULAR SYSTEMS | remote ischemic conditioning | reperfusion injury | RANDOMIZED CONTROLLED-TRIAL | BYPASS GRAFT-SURGERY | preconditioning | PROTEIN KINASE-C | PERCUTANEOUS CORONARY INTERVENTION | CYCLOSPORINE-A | IN-VIVO | cardioprotection | PERIPHERAL VASCULAR DISEASE | myocardial ischemia | HEMATOLOGY | reperfusion | ELEVATION MYOCARDIAL-INFARCTION | Coronary Circulation | Reproducibility of Results | Ischemic Postconditioning - methods | Species Specificity | Humans | Risk Factors | Myocardium - pathology | Myocardial Reperfusion - methods | Myocardial Reperfusion Injury - physiopathology | Patient Selection | Myocardial Infarction - therapy | Regeneration | Myocardial Reperfusion - adverse effects | Myocardial Reperfusion Injury - pathology | Animals | Time Factors | Myocardial Infarction - pathology | Ischemic Preconditioning, Myocardial - methods | Myocardial Infarction - physiopathology | Translational Medical Research - methods | Myocardial Reperfusion Injury - prevention & control | Cardiology - methods | Disease Models, Animal | Myocardial infarction | Translation | Heart attacks | Heart surgery | Clinical trials | Risk factors | Ischemia | Surgery | Anesthesia | Clinical medicine | Long-term effects | Hypothermia
Journal Article
Cardiovascular Research, ISSN 0008-6363, 09/2012, Volume 95, Issue 4, pp. 487 - 494
Innate mechanisms of inter-organ protection underlie the phenomenon of remote ischaemic preconditioning (RPc) in which episode(s) of ischaemia and reperfusion... 
Brain | Vagus nerve | Ischaemia/reperfusion injury | Preconditioning | Ischaemia | CARDIAC & CARDIOVASCULAR SYSTEMS | K-ATP CHANNELS | LIMB ISCHEMIA | MYOCARDIAL-ISCHEMIA | BRAIN-STEM | reperfusion injury | LENTIVIRAL VECTORS | CARDIOVASCULAR CONTROL | TRANSGENE EXPRESSION | RAT-HEART | AUTONOMIC NERVOUS-SYSTEM | Myocardial Infarction - genetics | Hindlimb | Receptors, G-Protein-Coupled - metabolism | Brain Stem - metabolism | Vagus Nerve - drug effects | Male | Receptors, Neuropeptide - metabolism | Drosophila Proteins - metabolism | Rhodopsin - metabolism | Recombinant Fusion Proteins - metabolism | Autonomic Fibers, Preganglionic - metabolism | Brain Stem - drug effects | Action Potentials | Myocardial Reperfusion Injury - pathology | Time Factors | Muscarinic Antagonists - pharmacology | Myocardial Infarction - pathology | Neuropeptides - pharmacology | Adenoviridae - genetics | Lentivirus - genetics | Myocardial Infarction - physiopathology | Neural Pathways - physiopathology | Myocardial Reperfusion Injury - genetics | Autonomic Fibers, Preganglionic - drug effects | Disease Models, Animal | Heart - innervation | Muscle, Skeletal - blood supply | Transduction, Genetic | Vagus Nerve - metabolism | Constriction | Rats | Receptors, Neuropeptide - genetics | Atropine - pharmacology | Myocardium - pathology | Myocardial Infarction - metabolism | Rats, Sprague-Dawley | Myocardial Reperfusion Injury - physiopathology | Vagus Nerve - physiopathology | Myocardial Reperfusion Injury - metabolism | Animals | Rhodopsin - genetics | Ischemic Preconditioning, Myocardial - methods | Brain Stem - physiopathology | Myocardial Infarction - prevention & control | Receptors, G-Protein-Coupled - genetics | Drosophila Proteins - genetics | Genetic Vectors | Myocardial Reperfusion Injury - prevention & control | Original
Journal Article
Journal Article
Kidney international, ISSN 0085-2538, 2012, Volume 82, Issue 12, pp. 1271 - 1283
Autophagy is induced in renal tubular cells during acute kidney injury; however, whether this is protective or injurious remains controversial. We address this... 
acute kidney injury | autophagy | Atg7 | cisplatin | ischemia–reperfusion | Ischemia-reperfusion | APOPTOSIS | ACTIVATION | ACUTE-RENAL-FAILURE | PATHOLOGICAL ROLE | ischemia-reperfusion | CISPLATIN NEPHROTOXICITY | CELL-DEATH | PATHOPHYSIOLOGY | UROLOGY & NEPHROLOGY | MICE | BODY FORMATION | SELF-DIGESTION | Microtubule-Associated Proteins - genetics | Blood Urea Nitrogen | JNK Mitogen-Activated Protein Kinases - metabolism | Acute Kidney Injury - genetics | Autophagy - drug effects | Reperfusion Injury - blood | Chloroquine - pharmacology | Time Factors | Reperfusion Injury - chemically induced | Acute Kidney Injury - chemically induced | Microtubule-Associated Proteins - deficiency | Reperfusion Injury - genetics | Cytoprotection | Disease Models, Animal | Kidney Tubules, Proximal - pathology | Reperfusion Injury - pathology | Signal Transduction | Acute Kidney Injury - pathology | Acute Kidney Injury - blood | Mice, Inbred C57BL | Cells, Cultured | Tumor Suppressor Protein p53 - metabolism | Biomarkers - blood | Sirolimus - pharmacology | Acute Kidney Injury - prevention & control | Cisplatin | Mice, Knockout | Autophagy-Related Protein 7 | Animals | Reperfusion Injury - prevention & control | Kidney Tubules, Proximal - metabolism | Creatinine - blood | Mice | Enzyme Activation | Apoptosis | Kidney Tubules, Proximal - drug effects
Journal Article
Journal Article