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Circulation, ISSN 0009-7322, 05/2010, Volume 121, Issue 18, pp. 2012 - 2022
Background-Whether alterations in mitochondrial morphology affect the susceptibility of the heart to ischemia/reperfusion injury is unknown. We hypothesized... 
Myocardial infarction | Cardiomyocytes | Hypoxia | Reperfusion | Ischemia | hypoxia | PERMEABILITY TRANSITION PORE | APOPTOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | FUSION | myocardial infarction | REPERFUSION INJURY | DEATH | ischemia | cardiomyocytes | HL-1 CELLS | FLUCTUATIONS | MITOFUSIN-2 | PERIPHERAL VASCULAR DISEASE | DYSFUNCTION | HEMATOLOGY | reperfusion | Age Factors | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Male | Green Fluorescent Proteins - genetics | GTP Phosphohydrolases - antagonists & inhibitors | Mitochondria - ultrastructure | Mitochondrial Membranes - drug effects | Myocardial Reperfusion Injury - pathology | Transfection | Myocardial Reperfusion Injury - drug therapy | Quinazolinones - pharmacology | Cell Line | Myocytes, Cardiac - cytology | Mitochondrial Membranes - physiology | Mice, Inbred C57BL | Dynamins | Cardiotonic Agents - pharmacology | Microtubule-Associated Proteins - antagonists & inhibitors | Mitochondria - drug effects | Microscopy, Electron | Myocardial Reperfusion Injury - physiopathology | Microscopy, Confocal | Animals | GTP Phosphohydrolases - metabolism | Myocytes, Cardiac - drug effects | Cell Death - physiology | GTP Phosphohydrolases - genetics | Myocytes, Cardiac - physiology | Mitochondrial Membranes - ultrastructure | Mice | Membrane Fusion - physiology | Mitochondria - physiology | Index Medicus | Abridged Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, pp. e89450 - e89450
Aim: Glycyrrhizin (GL) has been reported to protect against ischemia and reperfusion (I/R)-induced injury by inhibiting the cytokine activity of high mobility... 
ISCHEMIA-REPERFUSION INJURY | STROKE | CELLS | LIVER-INJURY | CHROMATIN PROTEIN HMGB1 | POSTISCHEMIC BRAIN | MEDIATOR | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MOBILITY GROUP BOX-1 | HMG-1 | Neuroprotective Agents - therapeutic use | Cerebral Infarction - drug therapy | Inflammation - pathology | Apoptosis - drug effects | Reperfusion Injury - drug therapy | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Cerebral Infarction - complications | Reperfusion Injury - blood | Inflammation - complications | Brain Ischemia - blood | Glycyrrhizic Acid - pharmacology | Neuroprotective Agents - pharmacology | Infarction, Middle Cerebral Artery - complications | Inflammation - drug therapy | Cerebral Infarction - blood | HMGB1 Protein - metabolism | Infarction, Middle Cerebral Artery - drug therapy | Reperfusion Injury - complications | p38 Mitogen-Activated Protein Kinases - metabolism | HMGB1 Protein - antagonists & inhibitors | Cerebral Infarction - pathology | Brain Ischemia - complications | Reperfusion Injury - pathology | Antioxidants - pharmacology | Infarction, Middle Cerebral Artery - pathology | Rats, Sprague-Dawley | Infarction, Middle Cerebral Artery - blood | Glycyrrhizic Acid - therapeutic use | Animals | MAP Kinase Signaling System - drug effects | Brain Ischemia - drug therapy | Brain Ischemia - pathology | Oxidative Stress - drug effects | Oxidative stress | Brain | Chromosomal proteins | Cytokines | Ischemia | Inflammation | Enzyme-linked immunosorbent assay | Apoptosis | Occlusion | Cytochrome | Cerebral cortex | Kinases | Caspase-3 | HMGB1 protein | Western blotting | Interleukin 6 | Reperfusion | Cerebral blood flow | Rodents | Inhibition | Pretreatment | Stresses | Glycyrrhizin | Cortex | Rats | Caspase | JNK protein | Tumor necrosis factor-α | Nitric-oxide synthase | Stress | Neurological diseases | Polymerase chain reaction | Cytochrome c | Serum levels | Signaling | Injury prevention | Molecular modelling | Nitric oxide | Immunofluorescence | Index Medicus
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 2012, Volume 122, Issue 2, pp. 693 - 710
Journal Article
Nature, ISSN 0028-0836, 03/2016, Volume 531, Issue 7595, pp. 528 - 532
Journal Article
Scientific Reports, ISSN 2045-2322, 01/2017, Volume 7, Issue 1, pp. 41337 - 41337
Enhancing mitochondrial biogenesis and reducing mitochondrial oxidative stress have emerged as crucial therapeutic strategies to ameliorate diabetic myocardial... 
ISCHEMIA-REPERFUSION INJURY | ACTIVATED PROTEIN-KINASE | BIOGENESIS | METABOLISM | PROTECTION | ANTIOXIDANT | MULTIDISCIPLINARY SCIENCES | CADMIUM-INDUCED HEPATOTOXICITY | DYSFUNCTION | STRESS | SIRT3 | AMP-Activated Protein Kinases - metabolism | Diabetes Mellitus, Experimental - drug therapy | Sirtuin 3 - metabolism | Apoptosis - drug effects | Myocardial Reperfusion Injury - complications | Streptozocin | Male | Diabetes Mellitus, Type 1 - complications | Cardiotonic Agents - therapeutic use | Myocardial Reperfusion Injury - pathology | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - metabolism | Glucose - toxicity | Diabetes Mellitus, Experimental - complications | Myocardial Reperfusion Injury - drug therapy | Superoxide Dismutase - metabolism | Glucose Tolerance Test | Signal Transduction | Cytochromes c - metabolism | Diabetes Mellitus, Type 1 - pathology | Myocardium - pathology | Mitochondria - metabolism | Cardiotonic Agents - pharmacology | Mitochondria - drug effects | Rats, Sprague-Dawley | Diabetes Mellitus, Type 1 - drug therapy | Transcription Factors - metabolism | Animals | Melatonin - therapeutic use | Diabetes Mellitus, Experimental - pathology | Cytosol - metabolism | Mice | Oxidative Stress - drug effects | Melatonin - pharmacology | RNA, Small Interfering - metabolism | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 03/2016, Volume 133, Issue 10, pp. 954 - 966
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