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PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, pp. e44151 - e44151
Background: Cardiovascular disease (CVD) remains one of the major killers in modern society. One strong risk factor of CVD is cigarette smoking that causes... 
ISCHEMIA-REPERFUSION INJURY | APOPTOSIS | OXIDATIVE STRESS | SIGNALING PATHWAYS | ENDOTHELIAL DYSFUNCTION | MULTIDISCIPLINARY SCIENCES | IN-VIVO | HEART-FAILURE | TOBACCO-SMOKE | CARDIOVASCULAR-DISEASES | ASCORBIC-ACID | Smoking - adverse effects | Inflammation - pathology | Myocardial Infarction - blood | Apoptosis - drug effects | Humans | Middle Aged | Male | Ascorbic Acid - therapeutic use | Myocardial Infarction - pathology | Adult | Ascorbic Acid - blood | Benzoquinones - metabolism | Neutrophil Infiltration - drug effects | Guinea Pigs | Rats | Myocardium - pathology | Enzyme Activation - drug effects | Disease Progression | Myocytes, Cardiac - pathology | Myocardium - enzymology | Animals | Myocardial Infarction - drug therapy | Myocytes, Cardiac - drug effects | Ascorbic Acid - pharmacology | Aged | Myocardial Infarction - prevention & control | Matrix Metalloproteinases - metabolism | Oxidative Stress - drug effects | Medical research | Collagen | Analysis | Quinone | Vitamin C | Medicine, Experimental | Smoking | Ascorbic acid | Heart attacks | Cardiovascular disease | Benzoquinone | Smoke | Damage prevention | Risk factors | Chronic exposure | Antioxidants | Signal transduction | Ischemia | Biocompatibility | Thromboembolism | Heart failure | Dietary supplements | Exposure | Cigarettes | Chemical vapor deposition | Thrombosis | Endothelium | Disease prevention | Injury prevention | Diet | Guinea pigs | Myocardium | Cardiovascular diseases | Oxidative stress | Biotechnology | Laboratories | Science | Population studies | Prevention | Quinones | Rodents | Calcium-binding protein | Supplementation | Heart diseases | Air exposure | Cardiomyocytes | Inflammation | Cigarette smoking | Troponin | Lungs | Genetic engineering | Emphysema | Cigarette smoke | Apoptosis | Index Medicus | Animal models
Journal Article
Journal Article
Jornal brasileiro de pneumologia : publicacao oficial da Sociedade Brasileira de Pneumologia e Tisilogia, ISSN 1806-3713, 01/2016, Volume 42, Issue 1, pp. 7 - 8
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 02/2009, Volume 53, Issue 8, pp. 720 - 729
Objectives: The purpose of this study was to investigate whether FX06 would limit infarct size when given as an adjunct to percutaneous coronary intervention.... 
FX06 | percutaneous coronary intervention | reperfusion injury | cardiac magnetic resonance imaging | acute myocardial infarction | ST-segment elevation | MICROVASCULAR OBSTRUCTION | CARDIAC & CARDIOVASCULAR SYSTEMS | PERMEABILITY | QUANTIFICATION | CARDIAC MAGNETIC-RESONANCE | PIG MODEL | SIZE | VE-CADHERIN | RANDOMIZED CONTROLLED-TRIAL | ISCHEMIA-REPERFUSION | PEPTIDE B-BETA(15-42) | Myocardial Infarction - diagnosis | Myocardial Infarction - mortality | Injections, Intravenous | Double-Blind Method | Humans | Middle Aged | Peptide Fragments - administration & dosage | Fibrin Fibrinogen Degradation Products - administration & dosage | Male | Myocardium - pathology | Combined Modality Therapy | Angioplasty, Balloon, Coronary | Necrosis | Magnetic Resonance Imaging | Myocardial Infarction - therapy | Fibrin Fibrinogen Degradation Products - adverse effects | Myocardial Infarction - pathology | Thrombolytic Therapy | Electrocardiography | Female | Peptide Fragments - adverse effects | Myocardial Reperfusion Injury - prevention & control | Studies | Heart failure | Heart attacks | Free radicals | Nuclear magnetic resonance--NMR | Rodents | Cardiovascular disease | Kinases | Acute coronary syndromes | Potassium | Index Medicus | Abridged Index Medicus | Myocardial Reperfusion Injury/prevention & control | Angioplasty | Intravenous | MEDICIN OCH HÄLSOVETENSKAP | Transluminal | effects | Injections | Peptide Fragments/administration & dosage/adverse effects | Myocardial Infarction/diagnosis/mortality/pathology/therapy | Myocardium/pathology | Percutaneous Coronary | Fibrin Fibrinogen Degradation Products/administration & dosage/adverse | MEDICAL AND HEALTH SCIENCES
Journal Article
Journal Article
The Lancet, ISSN 0140-6736, 01/2018, Volume 391, Issue 10115, pp. 59 - 69
Journal Article
Kidney International, ISSN 0085-2538, 12/2012, Volume 82, Issue 12, pp. 1271 - 1283
Autophagy is induced in renal tubular cells during acute kidney injury; however, whether this is protective or injurious remains controversial. We address this... 
acute kidney injury | autophagy | Atg7 | cisplatin | ischemia–reperfusion | Ischemia-reperfusion | APOPTOSIS | ACTIVATION | ACUTE-RENAL-FAILURE | PATHOLOGICAL ROLE | ischemia-reperfusion | CISPLATIN NEPHROTOXICITY | CELL-DEATH | PATHOPHYSIOLOGY | UROLOGY & NEPHROLOGY | MICE | BODY FORMATION | SELF-DIGESTION | Microtubule-Associated Proteins - genetics | Blood Urea Nitrogen | JNK Mitogen-Activated Protein Kinases - metabolism | Acute Kidney Injury - genetics | Autophagy - drug effects | Reperfusion Injury - blood | Chloroquine - pharmacology | Time Factors | Reperfusion Injury - chemically induced | Acute Kidney Injury - chemically induced | Microtubule-Associated Proteins - deficiency | Reperfusion Injury - genetics | Cytoprotection | Disease Models, Animal | Kidney Tubules, Proximal - pathology | Reperfusion Injury - pathology | Signal Transduction | Acute Kidney Injury - pathology | Acute Kidney Injury - blood | Mice, Inbred C57BL | Cells, Cultured | Tumor Suppressor Protein p53 - metabolism | Biomarkers - blood | Sirolimus - pharmacology | Acute Kidney Injury - prevention & control | Cisplatin | Mice, Knockout | Autophagy-Related Protein 7 | Animals | Reperfusion Injury - prevention & control | Kidney Tubules, Proximal - metabolism | Creatinine - blood | Mice | Enzyme Activation | Apoptosis | Kidney Tubules, Proximal - drug effects | Index Medicus
Journal Article
Journal Article
Kidney International, ISSN 0085-2538, 08/2012, Volume 82, Issue 4, pp. 412 - 427
Endothelial progenitor cells are known to reverse acute kidney injury by paracrine mechanisms. We previously found that microvesicles released from these... 
exosome | ischemia–reperfusion | acute kidney injury | ischemia-reperfusion | HORIZONTAL TRANSFER | STEM-CELLS | CONTRIBUTE | REGENERATION | FAILURE | EXPERIMENTAL GLOMERULONEPHRITIS | REPAIR | EPITHELIAL-CELLS | MESSENGER-RNA | UROLOGY & NEPHROLOGY | PLATELET-ACTIVATING-FACTOR | Chemotaxis, Leukocyte | Cell Proliferation | Epithelial Cells - metabolism | Kidney - blood supply | Capillaries - pathology | Kidney - pathology | Rats, Wistar | Endothelial Cells - transplantation | Cell-Derived Microparticles - transplantation | Male | MicroRNAs - metabolism | Acute Kidney Injury - genetics | Stem Cells - metabolism | Cell Hypoxia | Stem Cell Transplantation | Kidney - metabolism | Transfection | RNA Interference | Time Factors | Cell-Derived Microparticles - pathology | Cell-Derived Microparticles - metabolism | Kidney Tubules - pathology | Kidney Tubules - metabolism | Capillaries - metabolism | Reperfusion Injury - genetics | Reperfusion Injury - metabolism | Disease Models, Animal | Ribonuclease III - genetics | Ribonuclease III - metabolism | Reperfusion Injury - pathology | Acute Kidney Injury - pathology | Endothelial Cells - metabolism | Cells, Cultured | Gene Expression Regulation | Rats | Epithelial Cells - pathology | Acute Kidney Injury - prevention & control | Regeneration | Oligonucleotides - metabolism | Animals | Reperfusion Injury - prevention & control | Fibrosis | Stem Cells - pathology | Acute Kidney Injury - metabolism | Endothelial Cells - pathology | Apoptosis | Index Medicus | Cell proliferation | Intravenous administration | Paracrine signalling | mRNA | Leukocytes | Ribonuclease | Endothelial cells | Kidney | Angiogenesis | Ischemia | Stem cells | Hypoxia | miRNA | Capillaries | Injuries
Journal Article
Journal Article
Journal Article