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Journal Article
Nature Neuroscience, ISSN 1097-6256, 03/2004, Volume 7, Issue 3, pp. 261 - 268
Successful axon regeneration in the mammalian central nervous system (CNS) is at least partially compromised due to the inhibitors associated with myelin and... 
PROTEIN-KINASE-C | NEURITE GROWTH-INHIBITORS | CYCLIC-AMP | CONE COLLAPSE | SPINAL-CORD-INJURY | GLYCOPROTEIN INTERACTS | CELL-ADHESION | NOGO RECEPTOR | OUTGROWTH | RHO-GTPASES | NEUROSCIENCES | Protein Kinase C - genetics | Spinal Cord - metabolism | Recombinant Fusion Proteins - pharmacology | Recovery of Function - drug effects | Spinal Cord - growth & development | Nerve Regeneration - physiology | Myelin-Associated Glycoprotein - metabolism | Repressor Proteins - pharmacology | Recombinant Fusion Proteins - metabolism | Central Nervous System - injuries | Central Nervous System - growth & development | Chondroitin Sulfate Proteoglycans - pharmacology | Protein Isoforms - metabolism | Protein Kinase C - metabolism | Myelin Proteins - pharmacology | Recovery of Function - physiology | Female | Nogo Proteins | Spinal Cord - cytology | Repressor Proteins - metabolism | Myelin-Associated Glycoprotein - pharmacology | Neural Pathways - growth & development | Cells, Cultured | Chondroitin Sulfate Proteoglycans - metabolism | Enzyme Inhibitors - pharmacology | Rats | Treatment Outcome | Protein Kinase C - antagonists & inhibitors | Rats, Sprague-Dawley | Animals | Growth Cones - metabolism | Central Nervous System - cytology | Growth Cones - ultrastructure | Signal Transduction - drug effects | Nerve Regeneration - drug effects | Neural Pathways - cytology | Neural Pathways - metabolism | Signal Transduction - physiology | Growth Cones - drug effects | Myelin Proteins - metabolism | Protein Isoforms - antagonists & inhibitors | Protein Isoforms - genetics | Physiological aspects | Axons | Research | Protein kinases | Myelin proteins | Chondroitin
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 04/2011, Volume 20, Issue 8, pp. 1560 - 1573
Mental retardation in Down syndrome (DS) appears to be related to severe neurogenesis impairment during critical phases of brain development. Recent lines of... 
TS65DN MOUSE MODEL | TARGET GENES | METHYLATION | SONIC HEDGEHOG | RESTORES NEUROGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | CELL-PROLIFERATION | MICE | FETUSES | INTRACELLULAR DOMAIN | Up-Regulation | Hedgehog Proteins - pharmacology | Lateral Ventricles - pathology | Cell Proliferation | Humans | Zinc Finger Protein Gli2 | Zinc Finger Protein Gli3 | Male | RNA Interference | Smoothened Receptor | Acetylation | Patched Receptors | Hippocampus - embryology | Repressor Proteins - genetics | Thiophenes - pharmacology | Hippocampus - pathology | Polycomb Repressive Complex 1 | Mice | Receptors, G-Protein-Coupled - genetics | Zinc Finger Protein GLI1 | Kruppel-Like Transcription Factors - genetics | Patched-1 Receptor | Receptors, Cell Surface - genetics | Cell Cycle - genetics | Down Syndrome - metabolism | Lateral Ventricles - metabolism | Receptors, G-Protein-Coupled - agonists | Lateral Ventricles - embryology | DNA Methylation | Hedgehog Proteins - genetics | Cyclohexylamines - pharmacology | Amyloid beta-Protein Precursor - metabolism | Neurons - physiology | Veratrum Alkaloids - pharmacology | Female | Receptors, Cell Surface - biosynthesis | Nuclear Proteins - genetics | Protein Structure, Tertiary | Promoter Regions, Genetic | Mice, Inbred C57BL | Neural Stem Cells - physiology | Proto-Oncogene Proteins - genetics | Forkhead Transcription Factors - genetics | Nerve Tissue Proteins - genetics | Down Syndrome - embryology | Hippocampus - metabolism | Animals | Down Syndrome - genetics | Forkhead Box Protein M1 | Cell proliferation | Cerebellum | Brain | subventricular zone | Animal models | Cell division | Data processing | Mental retardation | Neurogenesis | Amyloid precursor protein | Promoters | Membrane proteins | Molecular modelling | Down's syndrome | Hedgehog protein | Neural stem cells | Alzheimer's disease | Mitogens | Hippocampus
Journal Article
Journal Article
Journal of Virology, ISSN 0022-538X, 01/2011, Volume 85, Issue 1, pp. 208 - 216
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 06/2006, Volume 26, Issue 24, pp. 6413 - 6421
Cerebral ischemic injury proceeds with excitotoxicity-induced acute neuronal death in the ischemic core and with delayed damage processes in the penumbra.... 
MCAO | Inflammation | HMGB1 | Ischemia | shRNA | PAMAM-Arg | ACTIVATION | NEUROSCIENCES | ischemia | CELL-DEATH | NEURITE OUTGROWTH | inflammation | CHROMATIN PROTEIN HMGB1 | CEREBRAL-ARTERY OCCLUSION | MOBILITY GROUP BOX-1 | HMG-1 | GLYCATION END-PRODUCTS | Inflammation - pathology | Microglia - metabolism | Gene Expression - drug effects | Embryo, Mammalian | Culture Media, Conditioned - pharmacology | Male | Repressor Proteins - pharmacology | Infarction, Middle Cerebral Artery - metabolism | RNA, Messenger - metabolism | Brain - metabolism | Inflammation - metabolism | Staurosporine - toxicity | Microglia - physiology | Transfection - methods | Time Factors | Infarction, Middle Cerebral Artery - complications | Enzyme Inhibitors - toxicity | Gene Expression - physiology | Reverse Transcriptase Polymerase Chain Reaction - methods | Neurons - metabolism | Neurons - drug effects | Repressor Proteins - metabolism | Blotting, Western - methods | High Mobility Group Proteins - metabolism | HMGB1 Protein | Cells, Cultured | Rats | Infarction, Middle Cerebral Artery - pathology | Inflammation - etiology | Rats, Sprague-Dawley | Animals | Excitatory Amino Acid Agonists - toxicity | Cell Death - physiology | High Mobility Group Proteins - pharmacology | Brain - pathology | Blotting, Northern - methods | Mice | N-Methylaspartate - toxicity
Journal Article
The Plant Cell, ISSN 1040-4651, 7/2001, Volume 13, Issue 7, pp. 1555 - 1565
RGA (for repressor of ga1-3) and SPINDLY (SPY) are likely repressors of gibberellin (GA) signaling in Arabidopsis because the recessive rga and spy mutations... 
Proteins | Phenotypes | Messenger RNA | Gibberellins | Homeostasis | Biosynthesis | Plants | Genetic mutation | Transgenic plants | Plant cells | INSENSITIVE MUTANT | BIOCHEMISTRY & MOLECULAR BIOLOGY | GAI | PLANT SCIENCES | CELL BIOLOGY | SIGNAL-TRANSDUCTION | GREEN-FLUORESCENT PROTEIN | THALIANA | FUNCTIONAL EXPRESSION | BIOSYNTHESIS | GENES | MOLECULAR-CLONING | LOCUS ENCODES | Green Fluorescent Proteins | Arabidopsis - growth & development | Genes, Plant | Mixed Function Oxygenases - metabolism | Repressor Proteins - pharmacology | Recombinant Fusion Proteins - metabolism | Repressor Proteins - physiology | Arabidopsis Proteins | Cell Nucleus - metabolism | Gibberellins - metabolism | Plants, Genetically Modified | Gene Expression Regulation, Plant | Isotope Labeling | Plant Proteins - metabolism | Repressor Proteins - metabolism | RNA, Plant | Signal Transduction | Plant Proteins - physiology | Repressor Proteins - genetics | Transcription Factors - biosynthesis | Transcription Factors - genetics | Arabidopsis - metabolism | Arabidopsis - genetics | Plant Proteins - genetics | Blotting, Northern | Sequence Alignment | Models, Biological | Genes, Regulator | Gibberellins - pharmacology | Recombinant Fusion Proteins - genetics | Luminescent Proteins - genetics | Mixed Function Oxygenases - genetics | Plant Growth Regulators - metabolism | Plant Growth Regulators - pharmacology | Suppression, Genetic | Plant proteins | Genetic aspects | Research | Growth (Plants) | Arabidopsis
Journal Article
Science, ISSN 0036-8075, 7/1997, Volume 277, Issue 5322, pp. 99 - 101
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2015, Volume 10, Issue 1, p. e116389
Clinical studies suggest that responses to HPV16 E6E7L2 fusion protein (TA-CIN) vaccination alone are modest, and GPI-0100 is a well-tolerated, potent... 
PHASE-II TRIAL | MULTIDISCIPLINARY SCIENCES | HUMAN-PAPILLOMAVIRUS VACCINES | VIRUS-LIKE PARTICLE | VULVAL INTRAEPITHELIAL NEOPLASIA | TA-CIN | CERVICAL-CANCER | MUCOSAL VACCINATION | ANTITUMOR IMMUNITY | HEALTHY-VOLUNTEERS | DNA VACCINATION | Recombinant Fusion Proteins - immunology | Adjuvants, Immunologic - administration & dosage | Adjuvants, Immunologic - pharmacology | Papillomavirus Vaccines - genetics | Recombinant Fusion Proteins - pharmacology | Saponins - administration & dosage | Humans | Papillomavirus E7 Proteins - immunology | Papillomavirus E7 Proteins - genetics | Papillomavirus Infections - therapy | Neoplasms - virology | Antibodies, Neutralizing - immunology | Neoplasms - therapy | Vaccination - methods | HEK293 Cells | Capsid Proteins - immunology | Female | Oncogene Proteins, Viral - genetics | Papillomavirus Infections - virology | Antineoplastic Agents - pharmacology | Papillomavirus Vaccines - administration & dosage | Recombinant Fusion Proteins - administration & dosage | Mice, Inbred C57BL | Repressor Proteins - genetics | Treatment Outcome | Combined Modality Therapy | Cisplatin - pharmacology | Papillomavirus Vaccines - pharmacology | Animals | Repressor Proteins - immunology | Cell Line, Tumor | Antibodies, Viral - immunology | Mice, Inbred BALB C | Oncogene Proteins, Viral - immunology | CD8-Positive T-Lymphocytes - immunology | Capsid Proteins - genetics | Saponins - pharmacology | Drug therapy | Health aspects | Papillomavirus infections | Cisplatin | Adjuvant treatment | Cancer | Animal models | CD8 antigen | Vaccination | Lymphocytes T | CD4 antigen | Chemotherapy | Immunogenicity | Lungs | Neutralizing | Remission | Mice | Fusion protein | Tumors
Journal Article